ライフサイエンスコーパス: aorta

1 ing thoracic aorta) and zone III (infrarenal aorta).

2 input function was image-derived (abdominal aorta).

3 ein, peritoneal cavity, and distal abdominal aorta).

4 versible dilation of the lower region of the aorta.

5 using on cyclic expansion of adjacent dorsal aorta.

6 crophages highly expressing CD36, emerged in aorta.

7 were compared with the nontransplanted donor aorta.

8 entiation of the embryonic heart, thorax and aorta.

9 rarenal abdominal aorta, and lower abdominal aorta.

10 ation of blood at a distant site, the dorsal aorta.

11 systolic wall shear stress in the ascending aorta.

12 stic image quality for CT angiography of the aorta.

13 enteric circulation compared to the thoracic aorta.

14 sition of the tracking ROI in the descending aorta.

15 ce of hematopoietic stem cells (HSCs) in the aorta.

16 computed tomographic (CT) angiography of the aorta.

17 nd the sternum and anterior to the ascending aorta.

18 mpaired SMC differentiation in the ascending aorta.

19 transcription factor NF-kappaB levels in the aorta.

20 esonance imaging of the carotid arteries and aorta.

21 on that predominantly affects the infrarenal aorta.

22 on microscopic images of normal rat thoracic aorta.

23 +)CCR5(+) Treg subset (Th1/Tregs) within the aorta.

24 capillaries of many vascular beds and in the aorta.

25 re gradients in the ascending and descending aorta.

26 the Nrf2-regulated enzymes in the heart and aorta.

27 es including liver, adipose, kidney, and the aorta.

28 eccentricity were assessed in the ascending aorta.

29 erating an atheroprotective phenotype in the aorta.

30 are required to drive HSC development in the aorta.

31 d), Foxp3 (1.4 folds) and TGF-beta (1.62) in aorta.

32 ed catheter into either the vena cava or the aorta.

33 he CD4(+) T-cell pool in the atherosclerotic aorta.

34 ia-induced atherosclerosis in the descending aorta.

35 dilatation of the aortic root and ascending aorta.

36 and decreased ADAMTS1 protein levels in the aorta.

37 cle phase as assessed in wounds, tumors, and aorta.

38 correlated with less atherosclerosis in the aorta.

39 ithin the ascending, thoracic, and abdominal aorta.

40 cle cells that make up the wall of the adult aorta.

41 DNA microarrays were performed on abdominal aortas.

42 f porcine coronary arteries and rat thoracic aortas.

43 ntly blocks T-cell homing to atherosclerotic aortas.

44 othelial release of ET-1 from P1 but not P21 aortas.

45 aneurysmal degeneration of TGFBR1 deficient aortas.

46 tors on the structural homeostasis of mature aortas.

47 ndothelium-dependent vasorelaxation in mouse aortas.

48 e propensity on geometry of patient-specific aortas.

49 nd immunofluorescence staining of hearts and aortas.

50 ulation of CD11d(-/-) macrophages within the aortas.

51 it to characterizing the stiffness of mouse aortas.

52 e in both the aortic root (1.2-fold) and the aorta (1.6-fold), despite reduced plasma cholesterol lev

53 mm/m(2); P=0.008) and the proximal ascending aorta (13.8+/-1.9 versus 14.1+/-1.9 mm/m(2); P=0.001).

54 art defects, including 76 coarctation of the aorta, 159 transposition of the great arteries, and 81 t

55 dilatation of the aortic root and ascending aorta (16.2% versus 7.3%, P<0.001) than women.

56 nulomatous arteritis, and in 5 of 18 control aortas (28%) obtained at autopsy.

57 creased pulse wave velocity in the ascending aorta (3.4 versus 2.3 m/s for PAH and controls, respecti

58 graft or stenosis of the anastomosis to the aorta (4 events; 0.006 events per patient-year).

59 um-denuded thoracic aorta (TA) and abdominal aorta (AA) segments, 1-oleoyl-LPA and the LPA1-3 agonist

60 ir is optimal for treating type A (ascending aorta) AAS, whereas thoracic endovascular aortic repair

61 y be optimal for treating type B (descending aorta) AAS.

62 The proximal aorta acts as a coupling device between heart and brain

63 METHODS AND LD were present in EC lining the aorta after the peak in plasma triglycerides initiated b

64 take was evaluated in murine atherosclerotic aortas after stimulation with M-CSF or GM-CSF by using q

65 led to a 30% increase in Treg percentages in aorta and aorta-draining lymph nodes (LNs) of these mice

66 eloped more atherosclerotic lesions in whole aorta and aortic root area, with markedly increased SRA

67 following: type 1, dilation of the ascending aorta and aortic root; type 2, isolated dilation of the

68 scular contrast enhancement in the abdominal aorta and brachiocephalic artery was quantified by measu

69 In zebrafish, HSCs emerge from the dorsal aorta and colonize the caudal hematopoietic tissue (CHT)

70 Enlarged ascending thoracic aorta and descending thoracic aorta were not significant

71 nd progenitor cells accumulate in the dorsal aorta and fail to colonize the fetal liver.

72 ethylation within the dilated human thoracic aorta and in SMCs cultured from these tissues, which inv

73 teritis, a chronic autoimmune disease of the aorta and its large branches, is complicated by aneurysm

74 (GCA) causes autoimmune inflammation of the aorta and its large branches, resulting in aortic arch s

75 rix described here can be delivered near the aorta and locally limit AAA expansion.

76 clinical CVD, enlarged infrarenal abdominal aorta and lower abdominal aorta, on noncontrast multidet

77 It is shown that elastic deformation of the aorta and of the endoprosthesis induced by static forces

78 number of elastic lamellae in the ascending aorta and progressive aortic root dilation as assessed b

79 d blood samples collected from the abdominal aorta and renal vein in 17 participants undergoing simul

80 in atherosclerotic plaques of the abdominal aorta and right carotid artery as compared with normal c

81 in atherosclerotic plaques of the abdominal aorta and right carotid artery as compared with normal c

82 from the vascular endothelium of the dorsal aorta and subsequently switch niche to the fetal liver t

83 ulating T-cell homing to the atherosclerotic aorta and the functionality of the CCR5Teff cells.

84 y disease that develops around the abdominal aorta and the iliac arteries, and spreads into the adjac

85 uited to assess the function of the proximal aorta and the left ventricle (eg, aortic arch pulse wave

86 the left renal vein (LRV) lodged between the aorta and the superior mesenteric artery.

87 from the vascular endothelium of the dorsal aorta and then the fetal liver but what regulates this s

88 of blood pressure loci showed enrichment in aorta and tibial artery.

89 n patients with a dilated proximal ascending aorta and trileaflet aortic valve, we aimed to assess (1

90 he best support for cannulation of the swine aorta and vena cava.

91 showed the presence of apatite in both donor aortas and allografts.

92 as assessed by Oil Red O staining of en face aortas and aortic root cross-sections, and changed plaqu

93 Transfection of TNF-alpha-treated mouse aortas and cultured ECs with miRs was more efficient wit

94 albumin and microvessel density in diseased aortas and especially in ruptured plaque.

95 to a marked reduction in lipid deposition in aortas and isolated macrophages.

96 Aortas and tail arteries were isolated from young (3-4 m

97 oss of calcium was less in heavily calcified aortas and was associated with an increase in the Ca/P r

98 teric arteries, and the descending abdominal aorta) and catheters (jugular vein, peritoneal cavity, a

99 pubis to aortic zone I (descending thoracic aorta) and zone III (infrarenal aorta).

100 rmation, sprouting of neovessels from murine aorta, and angiogenesis in Matrigel plugs.

101 n cross-linking sites in skin, bone, tendon, aorta, and cornea.

102 y in brain, spleen, lung, lymph node, liver, aorta, and kidney.

103 y in brain, spleen, lung, lymph node, liver, aorta, and kidney.

104 ing thoracic aorta, the infrarenal abdominal aorta, and lower abdominal aorta.

105 trol group in various tissues, including the aorta, and this expression correlated strongly with (99m

106 e organs: liver, kidneys, spleen, descending aorta, and upper large intestine.

107 matory lipid (lipoxin A4), ex vivo in murine aortas, and in vivo via tail vein injection in a 24-h mu

108 ; type 2, isolated dilation of the ascending aorta; and type 3, isolated dilation of the sinus of Val

109 tic regression identified the left ventricle/aorta angle as an indicator of indexed aortic diameter >

110 ether the pulmonary artery (PA)-to-ascending aorta (Ao) ratio is associated with outcome in unselecte

111 nd the graft was transplanted by end-to-side aorta-aorta and porto-cava anastomoses and end-to-end co

112 In most cases, significant injuries to the aorta, aortic valve annulus, and left ventricle require

113 tegies for major complications involving the aorta, aortic valve annulus, and left ventricle.

114 ng aneurysms and dissections of the thoracic aorta, are a major cause of morbidity and mortality.

115 nterval [CI], 0.62-0.85), coarctation of the aorta (aRR, 0.77; 95% CI, 0.61-0.96), ventricular septal

116 lve patients with dilated proximal ascending aorta, ascending aortic area/height ratio was independen

117 g enzyme was upregulated in TGFBR1 deficient aortas at the early stage of aneurysmal degeneration.

118 m male murine hearts subjected to transverse aorta banding surgery.

119 rentiating ccRCC from other RCC subtypes are aorta-based corrected AV and aorta-based corrected relat

120 ther RCC subtypes, a cut-off AV of 86-89 HU, aorta-based corrected AV of 89-95 HU and renal parenchym

121 ncement ratio, a cutoff of 2.59-2.74 for the aorta-based corrected relative contrast enhancement rati

122 CC subtypes are aorta-based corrected AV and aorta-based corrected relative contrast enhancement valu

123 second generation DPP1 inhibitors free from aorta binding liabilities found for earlier compound ser

124 he patient population with dilated ascending aorta, both peak TKE and total TKEsys were significantly

125 ven without equal efficiency in all tissues (aorta, brain and kidney), resulted in rapid lethality ma

126 rgeons attempted analogous operations on the aorta, but even the renowned Sir Astley Cooper and Willi

127 thelium in the floor of the embryonic dorsal aorta by an endothelial-to-hematopoietic transition (EHT

128 cutaneous transcaval access to the abdominal aorta by electrifying a caval guidewire and advancing it

129 sults demonstrated that curvature pattern of aorta can play a determinant role in AF-induced stroke p

130 However, CIH-induced aorta changes were absent in CD36 knockout mice, Our res

131 Prenatal diagnosis of coarctation of the aorta (CoA) is still challenging and affected by high ra

132 dothelium in the ventral floor of the dorsal aorta concurrent with arteriovenous specification and in

133 n of aggrecan and a dilation of the thoracic aorta, confirming that aggrecanase activity regulates ag

134 evel in the DCM (40-fold) than in transverse aorta constriction (4-fold).

135 d to chronic pressure-overload by transverse aorta constriction (TAC).

136 ine failing heart of both DCM and transverse aorta constriction mice that was accompanied by a decrea

137 ustly in DCM, and partially after transverse aorta constriction, by stabilizing myocardial NAD(+) lev

138 d to chronic pressure-overload by transverse aorta constriction.

139 cardiac hypertrophy triggered by transverse aorta constriction.

140 entified in patients with coarctation of the aorta contained at least 1 gene with FOXC1-binding sites

141 VZV DNA was found in most aortas containing VZV antigen.

142 2+) concentration ([Ca(2+)]i) and stimulated aorta contraction.

143 which the lumen and the outer surface of the aorta could be clearly seen.

144 We examined the development of the dorsal aorta (DA) and the cardinal vein (CV) in Ncx1 (-/-) muta

145 oderm covered the ventral side of the dorsal aorta (DA), but not the posterior cardinal vein.

146 specialized endothelial cells of the dorsal aorta (DA).

147 ), located in the ventral wall of the dorsal aorta (DA).

148 ts, which are immunogenic, were increased in aortas, DCs, and macrophages of tg(sm/p22phox) mice.

149 ly defined locations: the ascending thoracic aorta, descending thoracic aorta, the infrarenal abdomin

150 BAV undergoing surgery for aortic stenosis (aorta diameter </=45 mm: BAVnon-dil or >45 mm: BAVdil).

151 ed as pulmonary artery diameter to ascending aorta diameter [PA:A] ratio >1), a marker of pulmonary v

152 t ventricular and pulmonary artery/ascending aorta diameter ratios were higher (P<0.001, P=0.02, and

153 althy fetuses (P</=0.001), but the ascending aorta diameter, expressed as z score or millimeters, was

154 nction, isolated dilatation of the ascending aorta distal to the sinotubular junction, or diffuse dil

155 as evident in ApoE(-/-)Irf5(-/-) mice in the aorta, draining lymph nodes, and bone marrow cell cultur

156 0% increase in Treg percentages in aorta and aorta-draining lymph nodes (LNs) of these mice compared

157 nal role of mural cells investing the dorsal aorta during early development using the zebrafish.

158 ta suggest that cells ensheathing the dorsal aorta emerge from a sub-population of cells in the adjac

159 levels, reduced the plaque area of the total aorta en face and the cross-sectional aortic sinus, decr

160 scle cells in the primitive embryonic dorsal aorta establishes the long-lived lineages of smooth musc

161 abbit models were essentially similar in the aorta, even though they exhibited different types of hyp

162 rotic plaque size at the aortic root and the aorta for high-fat diet animals as compared with Ldlr(-/

163 In liver, kidney, and aorta from animals with cirrhosis, treatment with MS-PPO

164 hat are critical in protecting the abdominal aorta from injury.

165 ed by immunofluorescence microscopy thoracic aortas from 16 simian immunodeficiency virus (SIV)- or s

166 Aortic lipid mediator profiling of aortas from Apoe(-/-) mice fed a high-fat diet for 4 wee

167 Abdominal aortas from female XY mice selectively expressed Y chrom

168 METHODS AND Ascending aortas from patients with dilated aortopathy were immuno

169 To address this, calcified aortas from uremic mice were transplanted orthotopically

170 Moreover, aortas from XY females had augmented matrix metalloprote

171 lesion divided by iodine attenuation in the aorta) from dual-energy CT and arterial perfusion (AP),

172 y synthesis of IkappaBalpha mRNA in thoracic aortas (gestational day 20, postnatal week 7 and 16).

173 re is needed for dissection of the ascending aorta, given the high mortality (26%-58%) and proximity

174 hematopoietic stem cells (HSCs) arise in the aorta-gonad-mesonephros (AGM) and mature as they transit

175 atopoietic stem cells (HSCs) emerge from the aorta-gonad-mesonephros (AGM) region, but the molecular

176 ly in the ventral domain of the aorta in the aorta-gonad-mesonephros (AGM) region.

177 ells emerge, similar to hematopoiesis in the aorta-gonad-mesonephros (AGM).

178 re-HSC pool undergoes dramatic growth in the aorta-gonad-mesonephros region and by E11.5 reaches the

179 tenance in an identical manner to vertebrate aorta-gonadal-mesonephros (AGM) HSCs.

180 ients (50+/-13 years; 87% men) with proximal aorta >/=4 cm, who also had a gated contrast-enhanced th

181 NFL athletes were twice as likely to have an aorta >40 mm after adjusting for the same parameters.

182 he area surrounding the infrarenal abdominal aorta halfway between the right renal artery and aortic

183 in hemogenic endothelium (HE) of the dorsal aorta have been relatively well studied, the molecular r

184 and concomitant replacement of the ascending aorta (hazard ratio: 7.7; p = 0.0003).

185 e interval=1.06 to 2.32) and lower abdominal aorta (hazard ratio=1.53; 95% confidence interval=1.00 t

186 risk factors, enlarged infrarenal abdominal aorta (hazard ratio=1.57; 95% confidence interval=1.06 t

187 pite increased VEGF-C in the atherosclerotic aortas, how adventitial lymphatics regress.

188 the great arteries (n=7), coarctation of the aorta/hypoplastic aortic arch (n=5), tetralogy of Fallot

189 del and the Patlak method using a descending aorta image-derived input function, and mean tumor Ki va

190 Besides, in vitro rat aorta images were collected successfully at dynamic foci

191 s for CT angiography of the thoracoabdominal aorta in 129 consecutive patients (hereafter, cohort A).

192 originated in the distal arch or descending aorta in 71%; 52% of affected patients, including 68% wi

193 es in carotid artery, heart, aortic arch and aorta in acute and chronic atherosclerosis induced in ap

194 itment and activation of immune cells to the aorta in atherosclerosis is regulated by adhesion molecu

195 ntiated Treg cells relocated to the inflamed aorta in atherosclerosis-prone low-density lipoprotein r

196 t loss of arterial connections to the dorsal aorta in Notch pathway-deficient zebrafish.

197 We observe stiffening of the aorta in regulator of G protein signaling 5-deficient mi

198 and increased SMC apoptosis in the ascending aorta in response to increased biomechanical forces, thu

199 preferentially in the ventral domain of the aorta in the aorta-gonad-mesonephros (AGM) region.

200 Aortas in Adamts-4-/- mice showed reduced elastic fibre

201 in vitro, ex vivo, and in AngII-infused mice aortas in vivo.

202 ) recapitulated the pathology seen in Marfan aortas, including defects in fibrillin-1 accumulation, e

203 ogressive disease course (P=0.017), thoracic aorta involvement (P=0.009), and retinopathy (P=0.002) w

204 The ascending thoracic aorta is designed to withstand biomechanical forces from

205 Stiffening of the proximal aorta is strongly associated with age and hypertension.

206 The aorta is the largest artery in the body, yet processes u

207 hemodynamic load on a structurally defective aorta is the primary driver of thoracic aortic disease,

208 , and that TGF-beta overactivity in diseased aortas is a secondary, unproductive response to restore

209 largement of the root and ascending thoracic aorta, leading to ascending aortic dissections.

210 procedures require careful navigation of the aorta, left atrium, and right heart, including detailed

211 The cultures generate a network of aorta-like SOX17(+) vessels from which RUNX1C(+) blood c

212 efficient of variation in different regions (aorta, liver, spleen, kidney, small bowel, lumbar verteb

213 and diabetes but a higher rate of porcelain aorta, lower glomerular filtration rate, and higher mean

214 ontrast material-filled outpouching from the aorta lumen with a communicating orifice of >3 mm.

215 l and pathological situations such as normal aorta, lungs, and wound healing, tumors, and placenta, a

216 such as inflammation and oxidative stress in aorta macrophages.

217 VE-cadherin tension that occur as the dorsal aorta matures and upon genetic and chemical perturbation

218 AV-cKL expression had a 74%-78% reduction in aorta mineral content and a 72%-77% reduction in mineral

219 y over various image-based patient-dependent aorta models.

220 ment for uncomplicated AAS in the descending aorta (n = 61) revealed no dissection-related deaths in

221 Tumor-to-background (aorta, normal breast, and marrow) ratios were also signi

222 her proportion of former NFL athletes had an aorta of >40 mm (29.6% versus 8.6%; P<0.0001).

223 on of the right carotid artery and abdominal aorta of 7 rabbits fed an atherogenic diet.

224 on of the right carotid artery and abdominal aorta of 7 rabbits fed an atherogenic diet.

225 e formation was significantly reduced in the aorta of apoE(-/-)/PDE3B(-/-)and LDL-R(-/-)/PDE3B(-/-)mi

226 he heart and kidney but not in the liver and aorta of Fabry mice.

227 dly observed in aortic root, arch, and total aorta of male mice, whereas the effect was less pronounc

228 escribe blood flow patterns in the ascending aorta of patients with AS and determine their associatio

229 cells that reside in the wall of the dorsal aorta of the embryo at E10.5.

230 iR-155) is significantly up-regulated in the aortas of apoE(-/-) mice, and miR-155 deficiency in apoE

231 anilic acid (3-HAA) levels in the plasma and aortas of Apoe(-/-) mice, but not in IDO(-/-) mice.

232 In the aortas of atherosclerotic animals, we found markedly inc

233 ownregulated, while Sirt1 is upregulated, in aortas of germ-free mice.

234 ed gene expression variations (versus normal aorta) of transforming growth factor-beta1 (TGF-beta1),

235 Similarly, in isolated aorta, oleic acid treatment generates LD in EC ex vivo.

236 frarenal abdominal aorta and lower abdominal aorta, on noncontrast multidetector computed tomography

237 truction, or reconstruction of the ascending aorta or aortic arch) with intraoperative bleeding (bloo

238 ld-type mice by constricting their abdominal aorta or by infusion of angiotensin II.

239 En face staining of the murine aorta or carotid arteries modified with flow-altering cu

240 AV) and differences in contrast density; the aorta or renal parenchyma were evaluated based on correc

241 AngII levels are not increased in Acta2(-/-) aortas or kidneys.

242 time integrals, and the energies of forward (aorta-originating) and backward (microcirculatory-origin

243 tes still had significantly larger ascending aortas (P<0.0001).

244 A greater tubular ascending aorta, presence of mitral regurgitation, reduced left ve

245 to-background ratio (TBR) from the ascending aorta (primary endpoint) (adalimumab: TBR = 0.002, 95% c

246 ction of ROI area in normal control tissues (aorta, psoas) and in mathematical models (P < 0.01).

247 tative Endovascular Balloon Occlusion of the Aorta (REBOA) at the thoracic aorta (Zone 1) can limit s

248 tative endovascular balloon occlusion of the aorta (REBOA) is an innovative procedure in the treatmen

249 Chronic FID was not associated with aorta-related events (hazard ratio: 0.98; 95% confidence

250 Patients with acute FID had a higher risk of aorta-related events than those without FID (hazard rati

251 ensin system (RAS) over-activity in thoracic aortas, resulting in reduced blood pressure in offspring

252 asured in right and left carotids, abdominal aorta, right and left iliofemoral arteries, and coronary

253 Aorta rings and isolated VSMC obtained from wild type or

254 uced by patients' platelets on isolated mice aorta rings.

255 NG), and superoxide dismutase 3 in ascending aorta samples from 50 tricuspid and 70 patients with BAV

256 e analyzed also by Western blot in ascending aorta samples from other 10 tricuspid aortic valve, 10 B

257 In engrafted aorta, selected peptides containing Arg or Arg-Asn, not

258 xplanted smooth muscle cells from Acta2(-/-) aortas show increased production of reactive oxygen spec

259 t AoD (both p < 0.05), whereas the ascending aorta size was similar.

260 hancer signature at rs9349379 exclusively in aorta, suggesting a regulatory function for this SNP in

261 A4, and LPA6 In endothelium-denuded thoracic aorta (TA) and abdominal aorta (AA) segments, 1-oleoyl-L

262 start of treatment in TBR from the ascending aorta (TBR = -0.006, 95% CI = -0.049 to 0.038; P = 0.796

263 d as target-to-background ratio (TBR) in the aorta (TBRmax: CKD, 3.14+/-0.70 versus control, 2.12+/-0

264 herosclerotic lesions in the aortic root and aorta than did mice that had received control bone marro

265 ersized device, and to have erosion into the aorta, than survivors.

266 out mice were viable but with mildly dilated aortas that had a 43% reduction in NAD(+) in the media.

267 scending thoracic aorta, descending thoracic aorta, the infrarenal abdominal aorta, and lower abdomin

268 Moreover, in human aortas, the aortic content of isoketal adducts correlate

269 Results In normal aortas, the pulse waves propagated at relatively constan

270 aortic aneurysms of the descending thoracic aorta thoracic endovascular aortic repair (TEVAR) is rel

271 tion of mural cell recruitment to the dorsal aorta through disruption of pdgfr signaling leads to a r

272 or-matched healthy and atherosclerotic human aorta tissue (n = 15) and human carotid plaque samples (

273 ys and could be further replicated in blood, aorta tissue and carotid plaque material of atherosclero

274 ct alignment defects ranging from overriding aorta to double-outlet right ventricle and dextro-transp

275 Exposure of WKY rat aortas to IL-17F impaired endothelium-dependent vascular

276 Abdominal allogeneic aorta transplantation from Dark Agouti to Brown Norway r

277 In mouse aorta transplantation GA models, endothelial-specific SE

278 ncreased hemodynamic forces on the ascending aorta, typically due to poorly controlled hypertension,

279 e information on physiological motion of the aorta under various hemodynamic circumstances.

280 tissue homogenates of rat liver, kidney, and aorta, using an enzyme-linked immunosorbent assay.

281 Results Aorta versus muscle CNR (mean +/- standard deviation) wi

282 ne, which in turn results in enhanced dorsal aorta vessel elasticity and failure to restrict aortic d

283 However, not all HPCs in the aorta, vitelline and umbilical arteries, and fetal liver

284 H did not elicit significant improvements in aorta wall macrophage phenotype.

285 SUVmax in the liver and aorta was determined using automatic volumes of interest

286 aortic size (descending thoracic, abdominal aorta) was larger among patients with AoD versus those w

287 aortic valve and dilated proximal ascending aorta, we sought to assess (1) factors associated with i

288 -141, miR-200a, miR-204, and miR-211) in the aorta were analyzed.

289 ncy or a dilated aortic annulus or ascending aorta were at greater risk for reintervention.

290 nding thoracic aorta and descending thoracic aorta were not significantly associated with CVD events.

291 systolic wall shear stress in the ascending aorta were quantified.

292 ohistochemical studies, and whole descending aorta were stained with Oil Red O.

293 Aortas were collected 14 d after elastase perfusion.

294 ticoid receptor expressions studied in mouse aortas were down-regulated at 6 and 18 hours in endotoxe

295 tive oxygen species (ROS) levels in thoracic aortas when challenged with deoxycorticosterone acetate

296 , with 100% penetrance of ascending thoracic aortas, whereas TGFBR2 deletion only caused mild aortic

297 nd breaks in SMCs within the human ascending aorta, which were specifically enriched in SMCs with low

298 n of PAH, we hypothesized that the ascending aorta will present signs of apparent stiffness in childr

299 odies, we identified VZV antigen in 11 of 11 aortas with pathologically verified granulomatous arteri

300 and activity of matrix metallopeptidase 2 in aortas without affecting metabolic parameters.

301 clusion of the Aorta (REBOA) at the thoracic aorta (Zone 1) can limit subdiaphragmatic blood loss and