ライフサイエンスコーパス: aortic arch

1 selective preservation of embryonic vessels (aortic arches).

2 atherosclerosis at both the aortic root and aortic arch.

3 layer of the wall of the ascending aorta and aortic arch.

4 N accumulation in atheroprone regions of the aortic arch.

5 es in NHPs displaying the typical two-artery aortic arch.

6 nfirm the number of arterial branches in the aortic arch.

7 oam cells, in atherosclerotic lesions in the aortic arch.

8 y clipping the arteries originating from the aortic arch.

9 ring repairs of the ascending and transverse aortic arch.

10 994 repairs of the ascending and transverse aortic arch.

11 1107 repairs of the ascending and transverse aortic arch.

12 ring repairs of the ascending and transverse aortic arch.

13 ous system, skin, craniofacial skeleton, and aortic arch.

14 m, resulting in randomized laterality of the aortic arch.

15 terpart underlie left-sided formation of the aortic arch.

16 patterning of the cardiac outflow tract and aortic arch.

17 y in the greater and lesser curvature of the aortic arch.

18 gments 2 cm in length starting 1 cm from the aortic arch.

19 ted attention as a source of thrombus is the aortic arch.

20 rtery is a rare congenital anomaly involving aortic arch.

21 e cardiac outflow tract and the formation of aortic arches.

22 , interrupted aortic arches, and right-sided aortic arches.

23 mation and initial sprouting are normal, but aortic arches 5 and 6 fail to form a lumenized connectio

24 at each of the three levels were as follows: aortic arch, 53.9% with dynamic expiration versus 35.7%

25 It is not known whether progression of aortic arch (AA) atheroma is associated with vascular ev

26 investigate this process, we focused on the aortic arch (AA) blood vessels, which are known to remod

27 AP2B was detected in vivo in mouse and swine aortic arch (AA) endothelia exposed to chronic disturbed

28 1 null mice show defects such as interrupted aortic arch, aberrant subclavian artery and Tetralogy of

29 nal Kv7 channels in the baroreceptors of the aortic arch adjust the sensitivity of the mechanosensiti

30 The shape of the aortic arch also differs in the 2 strains.

31 Aortic arch anatomy was assessed with magnetic resonance

32 of the trachea were measured 1 cm above the aortic arch and 1 cm above the carina.

33 osclerotic plaques in carotid artery, heart, aortic arch and aorta in acute and chronic atheroscleros

34 e had smaller atherosclerotic plaques in the aortic arch and aortic roots, but showed little differen

35 the athero-susceptible regions of the inner aortic arch and aorto-renal branches than elsewhere.

36 of endothelium located in athero-susceptible aortic arch and athero-protected descending thoracic aor

37 The overlapping QTLs for curvature of the aortic arch and atherosclerosis support that the ontogen

38 The aortic arch and atria were examined by using confocal mi

39 Aortic arch and atrioventricular valve reinterventions w

40 lly suspected atherosclerotic disease of the aortic arch and branch vessels underwent breath-hold con

41 ocardiofacial syndrome phenotypes, including aortic arch and cardiac outflow tract abnormalities.

42 netic element of Gbx2, which is required for aortic arch and cardiac outflow tract development, and i

43 rwent nonenhanced computed tomography of the aortic arch and carotid arteries.

44 The genetic basis for aortic arch and carotid artery calcification overlaps wi

45 , reflexogenic areas of the carotid sinuses, aortic arch and coronary arteries and the pulmonary arte

46 in a new era in therapy for diseases of the aortic arch and descending thoracic aorta.

47 define age-related geometric changes of the aortic arch and determine their relationship to central

48 study tested the applicability of published aortic arch and ductal Z scores (measured just before th

49 face immunohistochemical examination of rat aortic arch and experimentally stenosed abdominal aorta

50 ation contributed to 75% of all strokes; for aortic arch and extracranial carotid artery calcificatio

51 ng cells to finely control patterning of the aortic arch and great arteries specifically during the p

52 on that leads to the formation of the mature aortic arch and great vessels.

53 MO4, which showed higher expression in mouse aortic arch and in human coronary endothelium in an asym

54 ed to specific focal localization within the aortic arch and its branches, as detected by fluorescenc

55 scular wall assembly, were restricted to the aortic arch and its branches, compromising the brachioce

56 ignal was detected in vivo in plaques in the aortic arch and its branches.

57 C57Bl/6 mice were subjected to 5 minutes of aortic arch and left subclavian occlusion with subsequen

58 ilin receptors npn1 and npn2 is required for aortic arch and outflow tract formation.

59 signaling within isl1 expression domains for aortic arch and outflow tract formation.

60 The main physical findings include aortic arch and outflow tract heart defects, thymus glan

61 ardiovascular defects, including interrupted aortic arch and persistent truncus arteriosus.

62 w tract (OFT) into the functionally separate aortic arch and pulmonary trunk is dependent upon the dy

63 chimeras had equal atherosclerosis burden in aortic arch and root.

64 All aortic arch and RVOT cross-sectional measurements were s

65 e, Apoe (-/-) Tlr7 (-/-) mice showed reduced aortic arch and sinus lesion areas.

66 ith larger calcification volumes in both the aortic arch and the carotid arteries but attenuated afte

67 redisposition with both calcification in the aortic arch and the carotid arteries, providing novel in

68 aneurysms involving both the ascending aorta/aortic arch and the descending thoracic or thoracoabdomi

69 s of atherosclerotic lesion size in both the aortic arch and the root.

70 ry effects of LA were more pronounced in the aortic arch and the thoracic aorta than in the aortic si

71 ngitudinal and transversal remodeling of the aortic arch and their relationship with LV mass and remo

72 mouse models by approximately 20% and in the aortic arch and thoracic aorta of apoE-/- and apoE/low-d

73 tail in endothelium of the atherosusceptible aortic arch and was found to be partially activated.

74 e had ~2-fold greater atherosclerosis in the aortic arch and ~2-8-fold greater atherosclerosis in the

75 ed RNA from freshly isolated EC of DF (inner aortic arch) and UF (descending thoracic aorta) regions

76 ave been shown to interact in patterning the aortic arch, and both genes are required in formation an

77 al septum, retrograde flow in the transverse aortic arch, and compression of the right heart.

78 osis, enhanced macrophage recruitment to the aortic arch, and more abundant mRNA for monocyte chemota

79 t, exercise performance, MRI analysis of the aortic arch, and need for repeat interventions were not

80 luding interrupted aortic arch type B, right aortic arch, and retroesophageal right subclavian artery

81 rsistent truncus arteriosus with interrupted aortic arch, and the Gja1W45X connexin43 mutation caused

82 ted previously in the carotid labyrinth, the aortic arch, and the pulmocutaneous artery of frogs.

83 ger atherosclerotic lesions in aortic roots, aortic arches, and abdominal aortas.

84 hypomorphic pulmonary arteries, interrupted aortic arches, and right-sided aortic arches.

85 More recently, repair of aortic arch aneurysms has been accomplished using both '

86 scle differentiation resulted in interrupted aortic arch, aneurysms and failure to assemble extracell

87 nsions, distensibility, pulse wave velocity, aortic arch angle, left ventricular (LV) mass, LV systol

88 Aortic arch anomalies also were readily detected with an

89 t serve as a genetic modifier of CHDs and/or aortic arch anomalies in individuals with 22q11DS.

90 second of two parts, describes the repair of aortic arch anomalies, left-to-right shunts, valvular di

91 Nissl staining showed that in NHPs with the aortic arch anomaly clipping only two of three arterial

92 extra arterial aortic branch recognizes this aortic arch anomaly in Rhesus macaques that warrants a c

93 Gender, era of birth, aortic arch anomaly, and systemic outflow obstruction we

94 der to reveal the pathological effect of the aortic arch anomaly, we compared the hippocampal cell lo

95 However, aortic arch apoptosis ((99m)Tc-rhAnnexin V-128) increase

96 Anatomic patterns in the aortic arch appear to be predictive of early device fail

97 Atherosclerotic plaques in the aortic arch are a risk factor for ischemic stroke.

98 n, and dissection of the ascending aorta and aortic arch are often associated conditions that complic

99 lformations of the septum, outflow tract and aortic arch are the most common congenital cardiovascula

100 1.5 MRTF-B-/- mutants exhibit deformation of aortic arch arteries 3, 4, and 6 and severe attenuation

101 ich is crucial for normal development of the aortic arch arteries and cranial vasculature during embr

102 ifferentiation into smooth muscle within the aortic arch arteries and impaired cardiac outflow tract

103 neural crest results in misalignment of the aortic arch arteries and outflow tract, contributing to

104 and differentiation to smooth muscle around aortic arch arteries is deficient.

105 5-11.0 had well-developed pharyngeal arches, aortic arch arteries, and no signs of cardiac failure.

106 sel malformations, hypoplastic pulmonary and aortic arch arteries, cardiac malformations, micrognathi

107 ands, craniofacial skeleton, cranial nerves, aortic arch arteries, cardiac outflow tract and cephalic

108 Fbln1 nulls also display anomalies of aortic arch arteries, hypoplasia of the thymus and thyro

109 iver of smooth muscle differentiation in the aortic arch arteries.

110 to the misalignment of the outflow tract and aortic arch arteries.

111 apoptosis and the embryos had clearly patent aortic arch arteries.

112 ral crest-derived mesenchyme surrounding the aortic arch arteries.

113 ections of the outflow tract with the caudal aortic arch arteries.

114 ssal nerves, and defective remodeling of the aortic arch arteries.

115 ly abnormal semilunar valves and concomitant aortic arch artery abnormalities.

116 second heart field tissues results in murine aortic arch artery and cardiac anomalies.

117 ized by the NC mediated morphogenesis of the aortic arch artery and differentiation of NC cells into

118 smooth muscle differentiation and results in aortic arch artery defects.

119 smooth muscle differentiation and resultant aortic arch artery defects.

120 wave of smooth muscle differentiation during aortic arch artery development.

121 ion assays and is sufficient to recapitulate aortic arch artery expression of Jagged1 in transgenic m

122 nd endocardial precursors does not result in aortic arch artery patterning defects or embryonic death

123 t ventricle, semilunar valve hyperplasia and aortic arch artery patterning defects.

124 Affected embryos are deficient in aortic arch artery smooth muscle during midgestation, de

125 However, aortic arch atheroma is a common post-mortem finding, an

126 t and subsequent embolization of debris from aortic arch atheroma or from the calcified valve itself

127 Here we review the evidence for aortic-arch atheroma as an important independent risk fa

128 l atrial fibrillation, patent foramen ovale, aortic arch atherosclerosis, atrial cardiopathy, and sub

129 Aortic arch atherosclerotic lesions of ApoE(-/-) mice we

130 imum FDG activity divided by the mean of the aortic arch background).

131 ections with intimal flap extension into the aortic arch between the innominate and left subclavian a

132 rom 2-dimensional regions of interest in the aortic arch blood pool and in the right lobe of the live

133 ncy of IL-17A/IL-17RA preferentially reduced aortic arch, but not thoracoabdominal aortic T cell, neu

134 Deficiency of IL-17A or IL-17RA reduced aortic arch, but not thoracoabdominal aortic TNFalpha an

135 Most prominent associations were found for aortic arch calcification and cardiovascular mortality (

136 In particular, aortic arch calcification volume yields unique informati

137 measured manually at 3 predetermined levels (aortic arch, carina, and bronchus intermedius) to confir

138 tter was performed only at the levels of the aortic arch, carina, and bronchus intermedius).

139 The mean diameter at the aortic arch, carina, and one vertebral body above the ga

140 rfusion circuit and pressures distending the aortic arch, carotid sinus and coronary artery barorecep

141 ponse to stimulation of baroreceptors in the aortic arch, carotid sinuses and coronary arteries, stim

142 non-occlusive atherosclerotic plaques in the aortic arch, cervical, or cerebral arteries.

143 permeability in the lesser curvature of the aortic arch compared with the greater curvature.

144 Imaging of the aortic arch (computed tomography angiography, DSA) revea

145 The present simulations suggest that aortic arch curvature is an important risk factor for em

146 ), mostly of the conotruncal type, and/or an aortic arch defect.

147 ce develop double outlet right ventricle and aortic arch defects, and the defects are caused by mutat

148 ice deficient in Tbx1 exhibit pharyngeal and aortic arch defects, the developmental program and mecha

149 toward the understanding of conotruncal and aortic arch defects.

150 erase chain reaction analysis of lesion-rich aortic arches demonstrated a marked reduction in mRNA fo

151 development of the cardiac outflow tract and aortic arch derivatives.

152 ntified at the level of the ascending aorta, aortic arch, descending thoracic aorta, and coronary art

153 bserved at the level of the ascending aorta, aortic arch, descending thoracic aorta, and the coronary

154 ular smooth muscle cells (SMCs) around those aortic arches destined for survival and reorganization,

155 Early aortic arch development occurs simultaneously with rapid

156 We show that Shh is necessary for aortic arch development, similar to Tbx1, and is also re

157 NCCs resulted in defects in craniofacial and aortic arch development, the latter with variable penetr

158 nsight into the factors that guide branchial aortic arch development, we examined the process by whic

159 apoE-null mice, the lesser curvature of the aortic arch develops lesions before the greater curvatur

160 ere isolated from the inner curvature of the aortic arch (DF; athero-susceptible) and a nearby UF reg

161 relation between wall shear stress (WSS) and aortic arch diameter in the developing embryo, and obser

162 Mean aortic arch diameter increased from 39.8 (30.5; 42.6) mm

163 es such as cardiac thrombi, cardiac tumours, aortic arch disease and other rare cardiac anomalies.

164 Hybrid approaches to aortic arch disorder continue to evolve as techniques an

165 chest but is constrained to incorporate the aortic arch down through the heart.

166 Swine aortic arch endothelia exhibited elevated ROS, NOX4, HIF

167 its formation correlates with blood flow and aortic arch enlargement.

168 otic calcification in the coronary arteries, aortic arch, extracranial, and intracranial internal car

169 In wild type embryos, each branchial aortic arch first appears as an island of angioblasts in

170 rosclerosis support that the ontogeny of the aortic arch formation is a potential risk factor for ath

171 sclerosis regression in which plaque-bearing aortic arches from apolipoprotein E-deficient (apoE(-/-)

172 r CCR7-dependent regression, we transplanted aortic arches from atherosclerotic Apoe-/- mice, or from

173 rosclerosis, P2X7 expression was analyzed in aortic arches from low density lipoprotein receptor(-/-)

174 h, diameters, height, width, and curvature), aortic arch function (local aortic distensibility and ar

175 sing magnetic resonance imaging to determine aortic arch geometry (length, diameters, height, width,

176 us work, we examined hemodynamic loading and aortic arch growth in the chick embryo at Hamburger-Hami

177 the following anatomic locations: diaphragm, aortic arch, heart, thoracic spine, and lung apices.

178 tellation of congenital malformations of the aortic arch, heart, thymus, and parathyroid glands descr

179 The presence of coarctation shelf and aortic arch hypoplasia were more common in fetuses with

180 s, including interrupted aortic arch type B, aortic arch hypoplasia, double-outlet right ventricle, a

181 sposition of the great arteries, interrupted aortic arch, hypoplastic left heart syndrome, and aortic

182 Interrupted aortic arch (IAA) is a rare congenital malformation of t

183 ent truncus arteriosus (PTA) and interrupted aortic arch (IAA), which are associated with the faulty

184 ent truncus arteriosus (PTA) and interrupted aortic arch (IAA-B).

185 as high across all territories imaged except aortic arch (ICC values from 0.90 to 0.97, arch 0.71).

186 achieved superior hemodynamic and integrated aortic arch imaging outcomes compared with BA patients.

187 s and smooth muscle apoptosis throughout the aortic arch in affected patients.

188 lesser curvature and branching point of the aortic arch in mice as well as human pulmonary artery br

189 report for the first time an anomaly of the aortic arch in some Rhesus macaques that appears as a ke

190 Aortic arch inflammation ((18)F-FDG uptake) did not diff

191 Aortic arch inflammation ((18)F-FDG) and apoptosis ((99m

192 The IL-17A/IL-17RA axis increases aortic arch inflammation during atherogenesis through th

193 -1-dependent fashion and that, while loss of aortic arch integrity contributes to the early lethality

194 However, the treatment of TB-AAD with aortic arch involvement (AAI) remains an unresolved issu

195 Patients with TB-AAD and aortic arch involvement do not differ with regards to mo

196 Age-related unfolding of the aortic arch is related to increased proximal aortic stif

197 ed that the extent of atherosclerosis in the aortic arch is significantly correlated in males, but no

198 5 and alphav integrins developed interrupted aortic arches, large brachiocephalic/carotid artery aneu

199 LR -/- mice, compound 13 showed reduction of aortic arch lesion progression and no plasma or hepatic

200 analysis identified 2 significant peaks for aortic arch lesion size on chromosome 1 (105 Mb, LOD=5.0

201 er, overall area and cellular composition of aortic arch lesions did not differ significantly among g

202 is model results in significant reduction in aortic arch lesions.

203 ate and subclavian arteries arising from the aortic arch, macroscopic visualization of these two arte

204 Aortic arch malformations are common congenital disorder

205 ing craniofacial, cardiac outflow tract, and aortic arch malformations.

206 phologies of vagal afferent terminals in the aortic arch may serve as substrates for the future inves

207 Lipid content in the aortic arch measured by oil red-O staining revealed a 1.

208 eft common carotid artery using an idealized aortic arch model.

209 indow of increased vulnerability to aberrant aortic arch morphogenesis with the potential for profoun

210 rtic arch (n=28) and coarctation/interrupted aortic arch (n=12).

211 AS (n=2), coarctation (n=5), and interrupted aortic arch (n=2).

212 This included right aortic arch (n=28) and coarctation/interrupted aortic ar

213 (n=7), coarctation of the aorta/hypoplastic aortic arch (n=5), tetralogy of Fallot (n=1), hypoplasti

214 rventions on the pulmonary arteries (n=8) or aortic arch (n=5).

215 f the inner, but not the outer, curvature of aortic arch, nor the straight segment of thoracic aorta

216 have undergone balloon aortoplasty (BD) for aortic arch obstruction (COA) after the Norwood procedur

217 Thirty-seven neonates with an aortic arch obstruction presenting for univentricular or

218 Aortic arch obstruction was present in 30 patients (70%)

219 ion of lesions was observed in the aorta and aortic arch of anti-OX40L-treated mice compared with con

220 sham: 0.97+/-0.05 s(-1); P=0.068) and in the aortic arch of ApoE(-/-) mice compared with WT mice (Apo

221 Notably, the extent of lesion size in the aortic arch of Senp2(+/-)/Ldlr(-/-) mice was much larger

222 in reduced glucose uptake in the spleen and aortic arch of these mice.

223 -17A was preferentially expressed within the aortic arch of WD-fed Apoe(-/-) mice.

224 ed with genes progressively regulated in the aortic arches of 2 mouse models of atherosclerosis durin

225 e injections revealed that the transition in aortic arch pattern is not a uniform process and multipl

226 These results explain why aortic arch patterning defects and OFT defects can occur

227 xhibited cardiovascular anomalies, including aortic arch patterning defects, pulmonary artery stenosi

228 fluorescent dye microinjections to identify aortic arch patterns and measured diameters using both i

229 ed that these two stages contained different aortic arch patterns with no inter-embryo variation.

230 Selective Aortic Arch Perfusion (SAAP) combines thoracic aortic ba

231 Murine aortic arch perivascular adipose tissues likewise expres

232 Aortic arch plaques are a risk factor for ischemic strok

233 eurons, immunohistochemistry and an isolated aortic arch preparation were used to demonstrate the pre

234 e proximal aorta and the left ventricle (eg, aortic arch pulse wave velocity and distensibility) as w

235 The authors estimated that a 1% increase in aortic arch PWV (in meters per second) is related to a 0

236 d ascending aortic distensibility, increased aortic arch PWV (p < 0.001), and increased central blood

237 Aortic arch PWV helped predict WMH volume independent of

238 Aortic arch PWV measured with phase-contrast MR imaging

239 l predictive model of subsequent WMH burden, aortic arch PWV provides a distinct contribution along w

240 Aortic arch PWV was measured with phase-contrast magneti

241 Linear regression was conducted with aortic arch PWV, 15 other cardiovascular risk factors, a

242 lume adjusted for sex and ethnicity included aortic arch PWV, age, systolic blood pressure, hypertens

243 ants undergoing biventricular repair without aortic arch reconstruction, 89 (86%) returned for neurod

244 root replacement in 21 (38), with ascending aortic/arch reconstruction in 13 (23) and mitral valve s

245 However, why some aortic arches regress while others are incorporated into

246 sm that impacts the timing of events such as aortic arch regression and generation, leading to the va

247 e factors associated with increased risk for aortic arch reintervention in patients who have undergon

248 s arteriosus, and abnormal maturation of the aortic arch reminiscent of common forms of human congeni

249 important contributions to OFT formation and aortic arch remodeling.

250 In particular, aortic arch repair, requiring either deep hypothermic ci

251 ement, 5 valve surgery, 2 septal myectomy, 1 aortic arch replacement, 1 myocardial bridge unroofing,

252 oss-sectional measurements were taken of the aortic arch, right ventricular (RV) outflow tract (RVOT)

253 wed decreased atherosclerotic lesions in the aortic arch, root (57%, P<0.001), and the entire aorta (

254 eptor did not develop smaller lesions in the aortic arch, root, and thoracoabdominal aorta compared w

255 ty of abdominal aortic aneurysms, and caused aortic arch ruptures and dissections, indicating that al

256 he conotruncus region, leading to defects in aortic arch septation.

257 TLR7 deficiency also reduced aortic arch SMC loss and lesion intima and media cell ap

258 reduced suprarenal aortic diameter, reduced aortic arch Sudan IV staining, higher serum HDL levels,

259 the current techniques and results of hybrid aortic arch surgery centered around a new classification

260 Aortic arch surgery has a high incidence of brain injury

261 perimental, totally endoscopic approaches to aortic arch surgery have been performed successfully.

262 Open aortic arch surgery is highly invasive and may result in

263 Secondary aortic arch surgery was necessary only in patients with

264 e aorta and its large branches, resulting in aortic arch syndrome, blindness, and stroke.

265 o HLHS had retrograde flow in the transverse aortic arch (TAA), 88% had left-to-right flow across the

266 entiation in vivo, and drive preservation of aortic arches that ought to regress.

267 inal organs and dissection or rupture of the aortic arch, the prognosis of focal single-organ vasculi

268 lipid deposition and intimal thickening from aortic arch throughout the abdominal aorta compared with

269 f neural crest-derived structures, including aortic arch, thymus, and cranial nerves.

270 the FimA- mutant were detected in blood and aortic arch tissue of apoE-/- mice by PCR after challeng

271 tion of the FimA- mutant in the blood and in aortic arch tissue, apoE-/- mice challenged with the Fim

272 In the aortic arch, TMR-D labeled large and small vagal afferen

273 TKE values were integrated over the aortic arch to obtain peak TKE.

274 ion from the bilaterally symmetric embryonic aortic arches to the mature great vessels is a complex m

275 ascular abnormalities, including interrupted aortic arch type B, aortic arch hypoplasia, double-outle

276 aryngeal arch arteries including interrupted aortic arch type B, right aortic arch, and retroesophage

277 h arteries can result in interruption of the aortic arch type B, typically found in DiGeorge syndrome

278 ontralateral internal carotid artery, and an aortic arch type II, with a trend for aortic arch type I

279 ion, lesion length, lesion eccentricity, and aortic arch type III were significantly associated with

280 and an aortic arch type II, with a trend for aortic arch type III.

281 contralateral carotid stenosis, and complex aortic arch type were predictive for bilateral ischemic

282 Age, hypertension, lesion morphology, and aortic arch type were predictive for procedural-related

283 exposed to atheroprotective flow than in the aortic arch under atheroprone flow.

284 lengthening, and decreased curvature of the aortic arch (unfolding) were all significantly associate

285 Aortic arch variations noted during normal rapid vascula

286 psular bleed of unknown cause with associate aortic arch vessel anomaly.

287 ressed during morphogenesis of the heart and aortic arch vessels and at early stages of cartilage dev

288 ) locus cause a pronounced dilatation of the aortic arch vessels as well as aberrant patterning of th

289 This dilatation of the aortic arch vessels does not appear to be caused by incr

290 The side of the aortic arch was not associated with aortic root dimensio

291 (-/-) mice, the (64)Cu-GPVI-Fc uptake in the aortic arch was significantly higher compared with WT mi

292 These geometric changes of the aortic arch were significantly related to decreased asce

293 AA) is a rare congenital malformation of the aortic arch, which might be accompanied with other coexi

294 etected 3 arterial branches arising from the aortic arch, which prompted us to subsequently search fo

295 a length increased most, with age leading to aortic arch widening and decreased curvature.

296 We report a case of the right-sided aortic arch with aplasia of the left brachiocephalic tru

297 Developmental anomalies of the carotid and aortic arch with intracranial bleeding is a rare occurre

298 The right aortic arch with mirror-image of branching arteries with

299 or reconstruction of the ascending aorta or aortic arch) with intraoperative bleeding (blood volume

300 gy have facilitated methods of replacing the aortic arch without deep hypothermic circulatory arrest