ライフサイエンスコーパス: aortic calcification

1 ng (psoas area, psoas density, and abdominal aortic calcification).

2 Ps with biochemical parameter in relation to aortic calcification.

3 with significant differences in coronary or aortic calcification.

4 coronary artery calcification and abdominal aortic calcification.

5 e culture and mouse model, respectively, for aortic calcification.

6 rmation by micro-CT of anatomic location and aortic calcification.

7 n CKD, perhaps explaining this inhibition of aortic calcification.

8 have contributed to their protection against aortic calcification.

9 d reduced flow-mediated dilation and greater aortic calcification.

10 coronary artery calcification, and abdominal aortic calcification.

11 xpression per se is not sufficient to induce aortic calcification.

12 calcification, but higher dosages stimulated aortic calcification.

13 ty-five patients had at least one measurable aortic calcification.

14 Little is known about the heritability of aortic calcification.

15 role in the presence and extent of abdominal aortic calcification.

16 all shear stress in relation to distribution aortic calcifications.

17 ry artery calcification (CAC), and abdominal aortic calcification (AAC) at the first follow-up visit

18 se; little is known, however, about thoracic aortic calcification (AC).

19 enetic difference was found only between the aortic calcification and control group with VEGF SNP -25

20 ew also analyzes the frequent coexistence of aortic calcification and coronary artery disease in term

21 were significantly different in control and aortic calcification and could enhance the aortic calcif

22 tudy was to investigate the relation between aortic calcification and VEGF SNPs (-2578C>A, -1154G>A a

23 both of which also associated with abdominal aortic calcifications and smaller global reflection coef

24 65 days were small body surface area, severe aortic calcification, and falls within the past 6 months

25 Lmna(G609G/+) mice showed excessive aortic calcification, and primary aortic vascular smooth

26 e-dependent increases in serum phosphate and aortic calcification associated with increased risk of m

27 a high-phosphate medium, warfarin increased aortic calcification but only in the absence of pyrophos

28 iol and paricalcitol were protective against aortic calcification, but higher dosages stimulated aort

29 Aortic calcification, collagen accumulation, and wall th

30 ted dilation and greater coronary artery and aortic calcification compared with women without hot fla

31 d aortic calcification and could enhance the aortic calcification development.

32 rent genotypes of VEGF SNPs which may induce aortic calcification development.

33 DR activators calcitriol and paricalcitol on aortic calcification in a mouse model of chronic kidney

34 e established as aggravating factors for the aortic calcification in association with different VEGF

35 nversely, parathyroid hormone (PTH) inhibits aortic calcification in low-density lipoprotein receptor

36 t the first and twelfth month after KTx: the aortic calcification index (ACI), fibroblast growth fact

37 Aortic calcification is developed due to accumulation of

38 ricardial fat, was associated with abdominal aortic calcification (odds ratio 1.32, 95% confidence in

39 dilation (beta=-0.97; SE, 0.44; P=0.03) and aortic calcification (odds ratio, 1.63; 95% confidence i

40 -/-) (LRP6-VKO) siblings exhibited increased aortic calcification on high-fat diet without changes in

41 ith no known CV risk factors but significant aortic calcification on preoperative imaging will have i

42 ation (P=0.02) and 17% decrease in abdominal aortic calcification (P=0.03).

43 eam tomography to assess coronary artery and aortic calcification, reported hot flashes (any/none, pr

44 Aortic calcification scores at CT colonography are signi

45 coronary artery calcification and abdominal aortic calcification scores.

46 Aortic calcification was diagnosed by examining the post

47 Moderate aortic calcification was present on prior chest X-ray.

48 vo study with injury-induced local abdominal aortic calcification, we showed that elastin antibody-co

49 ent levels of uremia, hyperphosphatemia, and aortic calcification were induced by feeding nephrectomi

50 , 2.87; 95% confidence interval: 1.22, 6.72) aortic calcifications were associated with leakage.

51 ed to the progression of coronary artery and aortic calcification, whereas sevelamer attenuated or ar