ライフサイエンスコーパス: aortic dilatation

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1 terial compliance, and, notably, significant aortic dilatation.

2 r cell apoptosis, all of which contribute to aortic dilatation.

3 receptor leads to a significant reduction in aortic dilatation.

4 rd elucidating 1 gene defect responsible for aortic dilatation.

5 ng techniques, with the finding of ascending aortic dilatation (4.7 to 9 cm) in all 9 and significant

6 The role of atherosclerosis in thoracic aortic dilatation and aneurysm formation is poorly defin

7 on of the aortic wall leading to progressive aortic dilatation and eventual rupture.

8 Aortic dilatation and other structural cardiac abnormali

9 d the effects of losartan and doxycycline on aortic dilatation and survival in Fbn1(mgR/mgR) mice.

10 problems, especially mitral valve prolapse, aortic dilatation, and aortic dissection, has resulted i

11 These data demonstrate aortic dilatation, aortic medial degeneration, and alter

12 No patient in either group had aortic dilatation at any level of >20% or more than mild

13 sic aortic abnormalities in BAVs account for aortic dilatation beyond that caused by valvular hemodyn

14 Progressive aortic dilatation, defined as an increase in diameter >0

15 The extent of aortic dilatation (DeltaAD) was determined with AAAs def

16 o determine the natural history of ascending aortic dilatation following AVR.

17 imed to assess the effects of fenofibrate on aortic dilatation in a mouse model of AAA.

18 of aortic regurgitation are associated with aortic dilatation in BAV patients, intrinsic pathology a

19 s to summarize the recent data pertaining to aortic dilatation in congenital heart disease (CHD) and

20 It has been shown that losartan reduces aortic dilatation in patients with Marfan syndrome.

21 g that atherosclerosis plays a minor role in aortic dilatation in the population.

22 ltiple factors predispose this population to aortic dilatation, including underlying genetics, cellul

23 Aortic dilatation is a common finding in CHD.

24 Although aortic dilatation is common in CHD, data about adverse l

25 4 versus 3.9+/-0.6 cm, p=NS) and progressive aortic dilatation occurred in only 10 patients (9.3%).

26 bioavailability is associated with ascending aortic dilatation, outflow tract malrotation, overriding

27 eurysms (P<0.05), while A2AR agonism limited aortic dilatation (P<0.05).

28 It is unknown whether aortic dilatation precedes acute aortic dissection in TS

29 No patients with baseline aortic dilatation (range, 3.5 to 5.3 cm) dilated beyond

30 the aorta of 75 subjects: BAV patients with aortic dilatation stratified by leaflet fusion pattern (

31 ic plaques are weakly associated with distal aortic dilatation, suggesting that atherosclerosis plays

32 mended threshold of aortic root or ascending aortic dilatation that would justify surgical interventi

33 Aortic dilatation was accompanied by changes in the elas

34 Aortic dilatation was present in 29% of the patients, an

35 treatment and posttreatment with rTMD123 on aortic dilatation were measured using the CaCl2-induced

36 to establish a reproducible porcine model of aortic dilatation, which recapitulates the structural an

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