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1 and obese Smad3 null animals had significant aortic dilation.
2 b, but not miR-195, led to an attenuation of aortic dilation.
3 e (Mfap2(-/-);Mfap5(-/-)) show age-dependent aortic dilation.
4 nal flow abnormalities, aortic function, and aortic dilation.
5 tance of flow pattern in the pathogenesis of aortic dilation.
6 ributes modestly to both atherosclerosis and aortic dilation.
7 expressed uPA stimulates atherosclerosis and aortic dilation.
8 cardiac disease are at risk for progressive aortic dilation.
9 g antibody diminished angiotensin II-induced aortic dilation.
10 bryos treated at E10.5 exhibited significant aortic dilation.
11 ct manipulation of the M1/M2 balance altered aortic dilation.
12 ation, and neutralization of EDPs attenuated aortic dilation.
16 some animals disappeared over time, whereas aortic dilation and aortic valve thickening were absent
17 ological inhibition of Nos2 rapidly reversed aortic dilation and medial degeneration in young Adamts1
18 ) is a degenerative disease characterized by aortic dilation and rupture leading to sudden death.
19 syndrome and to determine the prevalence of aortic dilation and valve dysfunction associated with di
22 nital aortic valve fusion is associated with aortic dilation, aneurysm, and rupture in girls and wome
24 ith BAV showed abnormal flow patterns but no aortic dilation, both further supporting the importance
25 forms of vasculopathy, including progressive aortic dilation, dissection, and peripheral artery aneur
26 genous PAME but not SAME induced significant aortic dilation (EC(50) = 0.19 nM), indicating that PAME
27 a and IL-1R significantly decreased thoracic aortic dilation (IL-1beta knockout=54.2+/-16.8% and IL-1
35 , Ab-mediated neutralization of EDPs reduced aortic dilation, matrix metalloproteinase activity, and
36 treatment in WT mice with small TAAs reduced aortic dilation on day 14 (control treatment: 89.1+/-18.
40 of 4112 patients were reviewed and 440 (11%) aortic dilations were identified, of which 91 were new f
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