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1 and obese Smad3 null animals had significant aortic dilation.
2 b, but not miR-195, led to an attenuation of aortic dilation.
3 e (Mfap2(-/-);Mfap5(-/-)) show age-dependent aortic dilation.
4 nal flow abnormalities, aortic function, and aortic dilation.
5 tance of flow pattern in the pathogenesis of aortic dilation.
6 ributes modestly to both atherosclerosis and aortic dilation.
7 expressed uPA stimulates atherosclerosis and aortic dilation.
8  cardiac disease are at risk for progressive aortic dilation.
9 g antibody diminished angiotensin II-induced aortic dilation.
10 bryos treated at E10.5 exhibited significant aortic dilation.
11 ct manipulation of the M1/M2 balance altered aortic dilation.
12 ation, and neutralization of EDPs attenuated aortic dilation.
13 ith iliac vein produces a fusiform abdominal aortic dilation (AAD).
14 njection of M2-polarized macrophages reduced aortic dilation after aneurysm induction.
15 picardial and endocardial abnormalities, and aortic dilation, among others.
16  some animals disappeared over time, whereas aortic dilation and aortic valve thickening were absent
17 ological inhibition of Nos2 rapidly reversed aortic dilation and medial degeneration in young Adamts1
18 ) is a degenerative disease characterized by aortic dilation and rupture leading to sudden death.
19  syndrome and to determine the prevalence of aortic dilation and valve dysfunction associated with di
20 he chronic phase, most FIDs evolve with slow aortic dilation and without complications.
21 ate and severe) aortic valve dysfunction and aortic dilation and/or dissection.
22 nital aortic valve fusion is associated with aortic dilation, aneurysm, and rupture in girls and wome
23          uPA-accelerated atherosclerosis and aortic dilation are largely, if not completely, independ
24 ith BAV showed abnormal flow patterns but no aortic dilation, both further supporting the importance
25 forms of vasculopathy, including progressive aortic dilation, dissection, and peripheral artery aneur
26 genous PAME but not SAME induced significant aortic dilation (EC(50) = 0.19 nM), indicating that PAME
27 a and IL-1R significantly decreased thoracic aortic dilation (IL-1beta knockout=54.2+/-16.8% and IL-1
28  abnormalities may be a major contributor to aortic dilation in BAV.
29                                              Aortic dilation in db/db Smad3 null mice occurred despit
30                    The mechanisms underlying aortic dilation in this patient cohort are described, an
31                                     Thoracic aortic dilation in Turner syndrome must be evaluated in
32 lect to note a substantial proportion of new aortic dilations in the EMR.
33         This finding, in turn, suggests that aortic dilation is due primarily to the failure by the m
34                                    Ascending aortic dilation is important in bicuspid aortic valve (B
35 , Ab-mediated neutralization of EDPs reduced aortic dilation, matrix metalloproteinase activity, and
36 treatment in WT mice with small TAAs reduced aortic dilation on day 14 (control treatment: 89.1+/-18.
37 c remodeling" (false lumen thrombosis and no aortic dilation or rupture).
38                                         Mean aortic dilation was 96 +/- 13% for vehicle-treated mice,
39                                              Aortic dilation was not observed at 1 year after stent g
40 of 4112 patients were reviewed and 440 (11%) aortic dilations were identified, of which 91 were new f

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