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1  lumbar vein injuries, 1 renal artery, and 1 aortic injury).
2 model in rats on the basis of CaCl2-mediated aortic injury.
3 management have expanded for solid organ and aortic injury.
4  no evidence of PH near the diaphragm and no aortic injury.
5 horacic aorta to evaluate potential thoracic aortic injury.
6 ng a high-cholesterol diet after endothelial aortic injury.
7 ed in patients suspected of having traumatic aortic injury.
8 years) were evaluated for possible traumatic aortic injury.
9 graphy directly without CT, 10 had traumatic aortic injury.
10 dictive value for the detection of traumatic aortic injury.
11 d mediastinal hematoma or direct findings of aortic injury.
12 th CT scans that were positive for traumatic aortic injury.
13 heir diagnostic algorithm for acute thoracic aortic injury.
14 ssion of iNOS in response to balloon-induced aortic injury.
15 of protease activity in a rat model of focal aortic injury.
16 insensitive but relatively specific sign for aortic injury after blunt trauma.
17 ockout mice were resistant to CaCl2-mediated aortic injury and did not develop elastin degeneration a
18 oups of patients who are suspected of having aortic injury and have indeterminate angiograms may bene
19 with moderate to high suspicion of traumatic aortic injury and in all patients with CT scans that wer
20                                          For aortic injury and mediastinal hemorrhage, respectively,
21 There was no significant difference in major aortic injury and permanent pacemaker implantation rates
22 d PH near the level of the diaphragm without aortic injury, and 72 had no evidence of PH near the dia
23 the thorax (HCTT) for the diagnosis of blunt aortic injury (BAI).
24 iography to indicate suspicion for traumatic aortic injury before angiography resulted in savings of
25  hemorrhage alone is sensitive for traumatic aortic injury, but the finding of aortic injury is more
26       Endovascular repair of blunt traumatic aortic injury can be performed with a low morbidity and
27 ve cases of endograft treatment of traumatic aortic injury from December 2004 to November 2008 were r
28              Likewise, the therapy for acute aortic injuries has changed substantially.
29              The imaging evaluation of acute aortic injuries has undergone radical change over the pa
30              Endovascular repair of thoracic aortic injury has been shown in multiple studies to have
31 s at angiography were positive for traumatic aortic injury in 19 (90%).
32                        CT signs of traumatic aortic injury in 21 patients included contour abnormalit
33 racic CT was performed to evaluate traumatic aortic injury in 677 patients with positive or equivocal
34 s at angiography were negative for traumatic aortic injury in 77 (97%).
35 facilitating the diagnosis of acute thoracic aortic injury in the patient with blunt chest trauma has
36             Endovascular repair of traumatic aortic injury is becoming routine.
37  traumatic aortic injury, but the finding of aortic injury is more specific.
38  Moreover, endovascular imaging of the focal aortic injury model enabled successful measurement of en
39 ; 95% CI: 1.81, 7.64; P = .0001), as well as aortic injuries (n = 4 vs n = 0; P = .0015; OR, unavaila
40  = 197), pulmonary vein stenosis (n = 2), or aortic injury (n = 2) were routinely reviewed in transve
41 advances, timely diagnosis of acute thoracic aortic injury remains a challenge.
42  both PH near the level of the diaphragm and aortic injury; six had aortic injuries without PH, five
43                                              Aortic injury status was determined by reviewing angiogr
44  been used for serial examination of minimal aortic injuries that are treated conservatively.
45 iology and pathophysiology of acute thoracic aortic injury, the current status of the individual imag
46 ositive likelihood ratio for the presence of aortic injury was 10.8, and the negative likelihood rati
47 near the level of the diaphragm as a sign of aortic injury was 70%; specificity, 94%; positive predic
48 age, respectively, specificity for traumatic aortic injury was 99% and 87% and sensitivity was 90% an
49 m there was very high suspicion of traumatic aortic injury were examined with aortography only.
50 scans positive for mediastinal hemorrhage or aortic injury were retrospectively reviewed and interpre
51 us of imaging and therapy of acute traumatic aortic injuries will be reviewed.
52  of the diaphragm and aortic injury; six had aortic injuries without PH, five had PH near the level o

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