ライフサイエンスコーパス: aortic valve stenosis

1 existing cardiovascular disease or calcific aortic valve stenosis.

2 tain high-risk surgical patients with severe aortic valve stenosis.

3 ctor for cardiovascular disease and calcific aortic valve stenosis.

4 mic and functional characteristics of severe aortic valve stenosis.

5 functional properties of severe degenerative aortic valve stenosis.

6 or cardiovascular disease (CVD) and calcific aortic valve stenosis.

7 for treatment of severe symptomatic calcific aortic valve stenosis.

8 e best approach to treat neonatal congenital aortic valve stenosis.

9 PPM and regression of SMR following AVR for aortic valve stenosis.

10 alve thickening to severe calcification with aortic valve stenosis.

11 enic signaling, and halts the progression of aortic valve stenosis.

12 entify therapeutic targets for prevention of aortic valve stenosis.

13 rdiographic findings in patients with severe aortic valve stenosis after transcatheter aortic valve r

14 egies aiming to prevent PPM in patients with aortic valve stenosis and concomitant SMR.

15 will effectively reduce the risk of calcific aortic valve stenosis and CVD.

16 Aortic valve stenosis and insufficiency develop over tim

17 Patients >/= 70 years old with severe aortic valve stenosis and no significant coronary artery

18 ies that highlight Lp(a) in CVD and calcific aortic valve stenosis and propose pathways to clinical r

19 d Lp(a) is causally associated with calcific aortic valve stenosis and the need for aortic valve repl

20 Thirty-five patients with aortic-valve stenosis and 10 healthy controls underwent

21 tive cardiomyopathy (HOCM), 10 patients with aortic valve stenosis, and 14 healthy individuals using

22 nvasive diagnostic tool in the assessment of aortic valve stenosis, and how the results compare with

23 are clinical risk factors for development of aortic valve stenosis, and hypercholesterolemia is a put

24 nto the haemodynamic cardiac consequences of aortic valve stenosis (AS) and aortic valve regurgitatio

25 Aortic valve stenosis (AS) can cause angina despite unob

26 vere pulmonary hypertension (PHT) and severe aortic valve stenosis (AS) from 1987 to 1999.

27 Aortic valve stenosis (AS) induces compensatory alterati

28 Calcific aortic valve stenosis (AS) is a life-threatening disease

29 The presence of syncope in patients with aortic valve stenosis (AS) predicts a grave prognosis.

30 In aortic valve stenosis (AS), the occurrence of heart fail

31 nd obesity impacted outcome in patients with aortic valve stenosis (AS).

32 treatment can modify the natural history of aortic valve stenosis (AS).

33 Accurate quantification of aortic valve stenosis (AVS) is needed for relevant manag

34 Congenital aortic valve stenosis (AVS), coarctation of the aorta (C

35 A region was associated with the presence of aortic valve stenosis (AVS), no prospective study has su

36 ospectively associate with increased risk of aortic valve stenosis (AVS).

37 the severity and hemodynamic consequences of aortic valve stenosis (AVS).

38 s assessed by FFR in 54 patients with severe aortic valve stenosis before and after transcatheter aor

39 ctor for cardiovascular disease and calcific aortic valve stenosis, but no approved specific therapy

40 Calcific aortic valve stenosis (CAVS) is a major health problem f

41 to cardiovascular disease (CVD) and calcific aortic valve stenosis (CAVS) is substantial.

42 f hypertrophy was also seen in patients with aortic valve stenosis: ERK(Thr188) phosphorylation was i

43 stenosis (AS) according to the new proposed aortic valve stenosis grading classification.

44 nts, and risk factors described for critical aortic valve stenosis have been shown to be inapplicable

45 en have a better prognosis when experiencing aortic valve stenosis, hypertrophic cardiomyopathy, or h

46 ortic valve sclerosis was present in 26% and aortic valve stenosis in 2% of the entire study cohort;

47 risk of cardiovascular disease and calcific aortic valve stenosis in patients with elevated Lp(a) co

48 g hypertension in patients with asymptomatic aortic valve stenosis is scarce.

49 Although aortic-valve stenosis is clearly associated with adverse

50 Aortic valve stenosis may influence fractional flow rese

51 In contrast to patients with aortic valve stenosis, MEE was not improved in patients

52 or redo TAVR were moderate-severe prosthetic aortic valve stenosis (n=10, 21.7%), moderate-severe cen

53 Patients with severe aortic valve stenosis (n=161) undergoing aortic valve re

54 aortic valve is frequently an antecedent to aortic valve stenosis or insufficiency and is often asso

55 ion of pathophysiological conditions such as aortic valve stenosis or insufficiency, making it possib

56 w-up measurements were performed in HOCM and aortic valve stenosis patients 4 months after surgery.

57 in FFR values was found before and after the aortic valve stenosis removal (0.89+/-0.10 versus 0.89+/

58 Coronary hemodynamics are influenced by aortic valve stenosis removal.

59 ons for diseases such as atherosclerosis and aortic valve stenosis, since it strongly suggests a gene

60 t to randomize all-comers with severe native aortic valve stenosis to either transcatheter aortic val

61 idney transplant recipients (KT) with severe aortic valve stenosis underwent transfemoral TAVI.

62 from the right sinus of Valsalva, congenital aortic valve stenosis (with bicuspid valve) and myocardi

63 Aortic valve stenosis (with or without aortic regurgitat

64 tested the hypothesis that calcification and aortic valve stenosis would develop in genetically hyper