ライフサイエンスコーパス: aphasic

1 n published a 'Schema for the Examination of Aphasic and Amnesic Persons'.

2 tion as well as functional imaging in stroke aphasic and neurologically intact participants.

3 endering the patient quadriparetic, globally aphasic, and minimally responsive.

4 ual pathway, nor any synthesis of normal and aphasic behavior.

5 One aphasic case displayed higher right-hemisphere tangle de

6 inal tangles was significantly higher in the aphasic cases (P = 0.034).

7 ocampus and/or entorhinal cortex) in several aphasic cases and the severe occipito-parietal involveme

8 ged from neuropsychological studies in other aphasic cohorts, and functional imaging studies in healt

9 euroimaging work with progressive non-fluent aphasics, compared directly to non-aphasic patients with

10 Aphasic deficits are usually only interpreted in terms o

11 band and an affected brother had progressive aphasic dementia, leukoencephalopathy, and occipital cal

12 clinical profile did not include non-fluent aphasic features.

13 We found that, while the aphasic groups were impaired relative to normal controls

14 Five of seven aphasics had more leftward tangle asymmetry in all four

15 structurally disconnected regions relate to aphasic impairment and its recovery.

16 emic differences for control subjects versus aphasics in left primary auditory cortex and bilateral s

17 The most common aphasic manifestation was of the logopenic type for Alzh

18 The same region was activated in the aphasic patients as they listened to standard (undistort

19 andard test of spoken language in 16 chronic aphasic patients both before and after a rehabilitation

20 As a group, aphasic patients demonstrated a selective disruption of

21 compare SD and comprehension-impaired stroke aphasic patients directly on the same battery of semanti

22 to characterize a large group of progressive aphasic patients from a single center (n = 38), first cl

23 entification of Alzheimer pathology in these aphasic patients is puzzling since tangles and related n

24 graphy) and word rate in normal subjects and aphasic patients listening to monosyllabic words at rate

25 In addition, the aphasic patients made associative semantic errors in pic

26 Severely aphasic patients tended to perform worse in both domains

27 ng the ability of 30 left hemisphere-damaged aphasic patients to match environmental sounds and lingu

28 Preliminary experiments in aphasic patients using transcranial magnetic stimulation

29 Chronic aphasic patients were impaired at the task and demonstra

30 ol groups: 12 normal subjects and 7 anterior aphasic patients whose infarcts spared the left POp.

31 on-fluent aphasics, compared directly to non-aphasic patients with frontotemporal dementia, has demon

32 We studied seven aphasic patients with left anterior perisylvian infarcti

33 the neuropsychology component of the study, aphasic patients with multimodal semantic deficits follo

34 Subgroup analysis revealed that aphasic patients with preserved positive intertemporal c

35 The aphasic patients' picture naming performance improved co

36 sfunctional in comprehension-impaired stroke aphasic patients).

37 paring and impairment of those structures in aphasic patients, and the structures that normal adults

38 bust predictor of impaired speech fluency in aphasic patients, even when motor speech, lexical proces

39 Here we demonstrate for the first time that aphasic patients, who have largely recovered language fu

40 d a detailed set of procedures for examining aphasic patients.

41 may even be useful for enhancing recovery in aphasic patients.

42 Asymmetric cortical degeneration syndromes (aphasic, perceptual-motor, frontal lobe and bitemporal c

43 separating the principal aspects of chronic aphasic performance and isolating their neural bases.

44 concordance of Alzheimer pathology with the aphasic phenotype.

45 The aphasic presentations include both fluent and non-fluent

46 in normal speakers and damage to the LIFG in aphasic speakers was associated with performance on the

47 he results of lesion-deficit correlations in aphasic speakers who performed the same word production

48 amage to those regions results in non-fluent aphasic speech; when they are undamaged, fluent aphasias

49 ed sensitivity to phonemic boundaries in the aphasics' speech network in both hemispheres.

50 een language comprehension performance after aphasic stroke and the functional connectivity of a key

51 Although many more studies of aphasic stroke are required with larger patient numbers

52 left and right ATLs in patients with chronic aphasic stroke has been illustrated.

53 studies investigating language outcome after aphasic stroke have tended to focus only on the role of

54 y in the recovery of speech production after aphasic stroke may relate in part to differences in pati

55 Functional neuro-imaging studies of aphasic stroke offer the potential for a better understa

56 al cortex connectivity in a group of chronic aphasic stroke patients.

57 as posterior regions described in studies on aphasic stroke patients.

58 n the impaired speech production observed in aphasic stroke patients.

59 a marker of receptive language outcome after aphasic stroke, and illustrate that language system orga

60 ntact neural networks promote recovery after aphasic stroke, either spontaneously or in response to i

61 en that this brain region is often spared in aphasic stroke, we propose that it is a sensible target

62 ral superior temporal cortex connectivity in aphasic stroke.

63 ence comprehension following left hemisphere aphasic stroke.

64 one factor that may determine recovery from aphasic stroke.

65 tributing to the potential for recovery from aphasic stroke.

66 e clinical classification of the progressive aphasic syndromes is also debated.

67 tia within the classification of progressive aphasic syndromes, and for contemporary models of semant

68 entations include both fluent and non-fluent aphasic syndromes.

69 s of tangles and plaques were greater in the aphasic than amnestic cases (P < 0.05), especially in ne

70 speech patterns of each primary progressive aphasic variant adequately, and to reveal associations b

71 Within the aphasics, we also found behavioural correlates with conn

72 9 prospectively enrolled primary progressive aphasics were selected for this study because of peak at

73 port the performance of two Italian-speaking aphasics who show contrasting, selective difficulties in

74 Our results suggest that aphasics with impaired speech comprehension have less ve

75 stages and processes involved in normal and aphasic word production.

76 sonance imaging, to 14 chronic patients with aphasic word retrieval deficits.