ライフサイエンスコーパス: apical cell

1 detectable within the two descendants of the apical cell.

2 l, a stalk cell, and a cytoplasmically dense apical cell.

3 ctly positions the division plane in the bud apical cell.

4 pment and each module develops from a single apical cell.

5 iction imposed on the daughters of the first apical cells.

6 milar between yeast and serum-induced hyphal apical cells.

7 e activity and expression with the number of apical cells.

8 such is crucial for the bursting pattern in apical cells.

9 e occurred at P9-P12 in basal and P12-P15 in apical cells.

10 rity, a pattern that breaks at branching and apical cells.

11 spicuous inverted, pyramidal cell called the apical cell (AC), which is unidentified in angiosperms.

12 We propose that apextrin is involved in apical cell adhesion and that its high level of expressi

13 rophinin is a membrane protein that mediates apical cell adhesion between trophoblastic cells and lum

14 gral structural and functional components of apical cell adhesions (tight junctions).

15 The ATML1 gene was first expressed in the apical cell after the first asymmetric division of the z

16 ter recovery of I(Ca) from inactivation than apical cells allowing them to support a higher AP freque

17 ote divides asymmetrically into an embryonic apical cell and a basal cell with mostly extra-embryonic

18 suggested by Vernon and Meinke, in which the apical cell and its decendents normally suppress the emb

19 We show that Boi is expressed in apical cells and exerts its suppressive effect on FSC pr

20 RABA4d are normally localized at the tip of apical cells and their localization is correlated with r

21 e were able to demonstrate directly that the apical cells are derived from a stratification of the em

22 but the mechanisms of their release from the apical cells are unknown.

23 visions of the zygote, the decendents of the apical cell arrest.

24 Because citrate is released from root apical cells as the deprotonated anion, we used the patc

25 ry sky" pattern in which there was a lack of apical cell binding (hence, dark sky) but increased bind

26 [Ca(2+)](cyto) were seen in the dendrite and apical cell body, while relaxations of the carbachol-ind

27 erved that PMCA2 is rapidly delivered to the apical cell border from where it diffuses to the entire

28 reocilia membrane and is internalized at the apical cell border maintaining an estimated half-life of

29 )hHsp27 associated with basolateral, but not apical, cell borders in injured cells.

30 is integral to the tight junction (TJ), the apical cell-cell adhesion and a key regulator of the epi

31 f either N-cadherin or N-CAM otic cells lose apical cell-cell contact and their epithelial shape.

32 Apical cell-cell contact zones and actomyosin only later

33 cortical tension, but by dynamic linking of apical cell-cell contact zones to an already contractile

34 Sdt heterodimer is not only recruited to the apical cell-cell contacts by binding to Crb but depends

35 The length of the most apical cell-cell junctions was reduced, and basolateral

36 Tight junctions (TJs) are the most apical cell-cell junctions, and claudins, the recently i

37 t of apical-basal polarity, the formation of apical cell-cell junctions, and polarized secretion.

38 crease in Ci(155) levels, nuclear migration, apical cell constriction and an acceleration of the furr

39 nous and ROCK and that it elicited a wave of apical cell constriction that culminated in the formatio

40 dependent morphogenetic events that includes apical cell constriction, localized alignment of groups

41 ct to promote stable myosin accumulation and apical cell constriction, loss-of-function phenotypes fo

42 a conserved mechanism for this kinase during apical cell constriction.

43 We found that shoot apical cells contain high levels of IAA and that IAA dec

44 In response to rings and patches of apical cell contractility, model epithelia smoothly defo

45 ateral cell cortex, and is excluded from the apical cell cortex of dividing cells.

46 ynamics and elevated cortical tension in the apical cell cortex of endoderm precursor cells.

47 Activation of the apical cell death caspase Dronc is necessary and suffici

48 dBruce does not block the activity of the apical cell death caspase Dronc or the proapoptotic Bcl-

49 autonomous induction of proliferation, of an apical cell death caspase.

50 GBE, by tracking cells and quantifying their apical cell deformation over time.

51 Hh is expressed in apical cells, distant from the FSC niche, and diffuses t

52 is no connection between phyllotaxis and the apical cell division pattern indicating a position-depen

53 otaxis (137.5 degrees angle) and an unlinked apical cell division pattern.

54 -based system which links phyllotaxis to the apical cell division pattern.

55 a cultures of P. patens, growing by filament apical cell division, the proportion of apical (dividing

56 fication initiated exclusively from vertical apical cell divisions, both in 3D culture and in vivo.

57 also induced stratification through vertical apical cell divisions.

58 markers to follow the appearance and loss of apical cells during development.

59 on in a non-cell-autonomous manner to induce apical cell enlargement on both sides of their expressio

60 histocompatibility complex (MHC); one is the apical cell expressing both MHC classes I and II, and th

61 s, crb2b and crb3a, promote the formation of apical cell features: photoreceptor inner segments and c

62 PIN-mediated auxin transport regulates apical cell function, leaf initiation, leaf shape, and s

63 hypA or hypB function lead to a cessation of apical cell growth but activated isotropic growth and mi

64 The GUS-negative daughters of apical cells had a strong tendency to contribute primari

65 Apical cells have active mitotic cycles, whereas subapic

66 In normal eyes, the antibody bound to apical cells in a mosaic pattern, with cells exhibiting

67 reeping body (the thallus), which grows from apical cells in an invaginated "notch." The genetic mech

68 At days 9 and 13, single apical cells in conjunctival epithelium stained with AB/

69 ly development but is then restricted to the apical cells in developing aggregates, which are thought

70 We propose that Hh diffuses from apical cells, including cap cells, and regulates the pro

71 Loss of crumbs function disrupts the apical cell junction belt and crumbs overexpression expa

72 trated at the lateral membranes close to the apical cell junction complexes.

73 e targeting of exogenous myc-tagged PMP22 to apical cell junctions in polarized epithelia and to anti

74 Yap and the Hippo pathway kinases Lats1/2 at apical cell junctions to induce Yap phosphorylation and

75 nopus neural plate, being enriched at medial apical cell junctions.

76 birth, ASGP mRNA was diffusely spread in the apical cell layer of both conjunctival and corneal epith

77 oteins that are expressed exclusively in the apical cell layer of the entire tadpole epidermis, which

78 MUC16 mRNA and protein localized to the apical cell layers of the cornea and to the suprabasal r

79 The GalNAc-T4 isoenzyme was found in the apical cell layers, whereas GalNAc-T2 was found in the s

80 ver a form of cell subdivision that abscises apical cell membrane and mediates neuron detachment from

81 e antibody (OE-1) that both localized to the apical cell membrane and significantly inhibited PMN tra

82 irus can infect via receptors located at the apical cell membrane but that the glycocalyx impedes int

83 he purpose of this study was to identify the apical cell membrane component and viral protein that me

84 the formation of inverted cysts, wherein the apical cell membrane faces the cyst exterior, and the ba

85 rming the requirement for SA residues on the apical cell membrane for efficient infectivity of SA-dep

86 studies, we detected endogenous sgk1 at the apical cell membrane of aldosterone-stimulated mpkCCD(c1

87 tein that is asymmetrically localized to the apical cell membrane of dividing cortical progenitor cel

88 ins 2 and 3 can selectively permeabilize the apical cell membrane of epithelial cells in culture to e

89 We track single toxin receptors on the apical cell membrane of MDCK cells with Eu-doped oxide n

90 ore, association of endogenous sgk1 with the apical cell membrane of mpkCCD(c14) cells could be modul

91 ordinated actomyosin dynamics contributes to apical cell membrane organization.

92 cell adhesion molecules, thus regulating the apical cell membrane remodeling and cytoskeletal dynamic

93 racellular and intracellular surfaces of the apical cell membrane that activate apical Cl(-) conducta

94 at Cby facilitates basal body docking to the apical cell membrane through proper formation of ciliary

95 o examine the transmission of force from the apical cell membrane to the basal cell membrane.

96 PCP protein complexes fail to traffic to the apical cell membrane, although other aspects of apical-b

97 roplicae, a well-developed glycocalyx on the apical cell membrane, and a normal appearance of goblet

98 asal bodies mature but fail to dock with the apical cell membrane, are misorientated and almost compl

99 ropose that the association of sgk1 with the apical cell membrane, where it interacts with ENaC, is a

100 pigmented epithelium (PE), primarily in the apical cell membrane, with minimal extension to the prox

101 diffuse and localized to the basolateral and apical cell membrane.

102 ndogenous sgk1 is functionally active at the apical cell membrane.

103 availability of anionic phospholipids on the apical cell membrane.

104 cAMP-regulated chloride channel localized at apical cell membranes and exists in macromolecular compl

105 However, the expression of CFTR in apical cell membranes or its function as a Cl(-) channel

106 hways: (i) by direct cell-to-cell contact of apical cell membranes with EBV-infected lymphocytes; (ii

107 idney fluid output, expansion of caudal duct apical cell membranes, and occlusion of the caudal prone

108 It was revealed that the apical cells (MHC-positive) were recognized directly by

109 al surface to the rest of the cell or in the apical cell migration and tubulogenesis machinery.

110 However, neither marker accumulates in apical cells of Deltabrk1 filaments.

111 HCLE cultures, MUC16 protein was detected in apical cells of islands of stratified cells.

112 The apical cells of the archegonium, the canal cells, and th

113 Conversely, transgene expression in the apical cells of the colon positive for endogenous u-PAR

114 istry revealed that HCC was localized in the apical cells of the epithelium in the normal conjunctiva

115 Membrane-associated mucins present in the apical cells of the ocular surface epithelium (MUC1, -4,

116 Apical cells of the stratified cultures were the cells t

117 Apical cells of the stratified islands produced MUC16 an

118 ostatic ducts and somewhat less uniformly in apical cells of transition and central zone glands.

119 ds in the subcuticular cavity just above the apical cells of trichomes or emit them into the headspac

120 mponent of the crumbs pathway that regulates apical cell polarity and also may play a role in photore

121 triggered downregulation of PARD6B, loss of apical cell polarity, disorganization of F-actin, and ac

122 reased microvilli density and maintenance of apical cell polarity.

123 n-myosin contraction, and results in loss of apical cell polarity.

124 e presence of a membrane mucin, MUC1, at the apical cell pole, beta-catenin at the apical-lateral mem

125 Further divisions of the apical cell produce a peltate trichome with one basal ce

126 Estimates of the density of the apical cell range from 1.004 to 1.085.

127 ay inherit the radial glial fibre, while the apical cell sequesters the majority of the Numb protein.

128 sarcomeres concomitantly impacts changes in apical cell shape and tissue geometry.

129 We investigated the apical cell-shape changes that characterize amnioserosa

130 n apical release of progeny virus, increased apical cell sloughing, apoptosis, and occasional syncyti

131 Individual cells in the two-apical-cell stage embryo responding to heat shock produc

132 by inducing genetic chimerism during the two-apical-cell stage of embryogenesis to determine if the o

133 e report that virus attachment to DAF on the apical cell surface activates Abl kinase, triggering Rac

134 but the mechanism by which changes near the apical cell surface affect changes along the entire apic

135 TPase subunits, showed colocalization at the apical cell surface and coassociation by immunoprecipita

136 of the ZA but is distributed over the entire apical cell surface and concentrated in the immediate vi

137 Virus binds to VLA-2 on the apical cell surface and moves rapidly to early endosomes

138 emonstrated that hSVCT1 was expressed at the apical cell surface and video rate measurements revealed

139 isms that restrict these determinants to the apical cell surface are unknown.

140 Expression was about sixfold greater on the apical cell surface as assessed biochemically by selecti

141 inding protein filamin A, potentially at the apical cell surface associated with the basal body.

142 centrated in actin-rich protrusions from the apical cell surface colocalized with the RNA-binding ret

143 It is proposed that the marginal zone of the apical cell surface contains a crumbs- and stardust-depe

144 in tubular fluid and anions anchored on the apical cell surface could determine whether a crystal bi

145 Nucleolin was enriched on the apical cell surface domain of A549 cells, and HPIV-3 int

146 piregulin (EREG) from the basolateral to the apical cell surface drives transformation.

147 e protein and chitin matrix packaging at the apical cell surface during development.

148 The apical cell surface is locally perturbed by atomic force

149 bble-like cysts that selectively incorporate apical cell surface markers.

150 p-ERK was localized at the apical cell surface of bronchiolar and alveolar type II

151 -1 is a transmembrane mucin expressed at the apical cell surface of mouse uterine epithelial cells (U

152 ass are preferentially exocytosed toward the apical cell surface of polarized cells, include antigens

153 ution of functional hSVCT1 expression at the apical cell surface of polarized epithelia and define an

154 In vivo, T-cadherin was detected on the apical cell surface of the chick intestinal epithelium.

155 receptor tyrosine phosphatase present on the apical cell surface of the glomerular podocyte.

156 t with defective recycling of megalin to the apical cell surface of the proximal tubules and thus dec

157 t of the mechanism of mucin removal from the apical cell surface of UEC.

158 stalline arrays of uroplakin proteins to the apical cell surface of urothelial umbrella cells.

159 g with ezrin and alpha(3)beta(1) integrin in apical cell surface protrusions.

160 of the F-actin cytoskeleton similarly affect apical cell surface remodeling and lumen formation.

161 th additional N-glycans redistributed to the apical cell surface similar to that of rat FcRn.

162 Efficient transduction of AEC from the apical cell surface supports the feasibility of using VS

163 ctive tract, and gut, requires a specialized apical cell surface that prevents adhesion.

164 s preferentially secreted RANTES through the apical cell surface thereby establishing a chemical grad

165 te proteins and enzymes that assemble at the apical cell surface to provide epithelial integrity and

166 glutinin from the trans-Golgi network to the apical cell surface was severely inhibited in cells over

167 ha- and gamma-ENaC subunits that reached the apical cell surface were considerably longer (t(12) > 24

168 tinal epithelial cells depends on DAF at the apical cell surface, and expression of human DAF on muri

169 its heterodimerization partner ErbB3, to the apical cell surface, effectively segregating the two rec

170 I) formed biofilms in close proximity to the apical cell surface, followed by invasion and destructio

171 ion by binding to and sequestering Hh on the apical cell surface, thereby inhibiting Hh diffusion.

172 l and delivery sites at the periphery of the apical cell surface.

173 anosomes to mature and to associate with the apical cell surface.

174 l caused Polycystin-2 mislocalization to the apical cell surface.

175 ithelia that can withstand freshwater on the apical cell surface.

176 ng that at least a portion is exposed on the apical cell surface.

177 ed for the efficient targeting of GCC to the apical cell surface.

178 onductance regulator chloride channel at the apical cell surface.

179 s that control the expression of ENaC at the apical cell surface.

180 e newly synthesized protein delivered to the apical cell surface.

181 ly synthesized endolyn were delivered to the apical cell surface.

182 ased the fluorescent labeling of ENaC at the apical cell surface.

183 ecting a set of force curves over the entire apical cell surface.

184 ng factor (DAF), a receptor expressed on the apical cell surface.

185 eta2 subunits accumulated selectively in the apical cell surface.

186 e necessary for sorting hemagglutinin to the apical cell surface.

187 mbly zone that controls aECM assembly at the apical cell surface.

188 or that there are inhibitory factors on the apical cell surface.

189 ic from the trans-Golgi network (TGN) to the apical cell surface.

190 of large patches (or plaques) at or near the apical cell surface.

191 ic of secretory proteins from the TGN to the apical cell surface.

192 and V159G substitutions redirect EREG to the apical cell surface.

193 beta1-integrin on microprotrusions from the apical cell surface.

194 TLR7, and TLR9) are mostly expressed on the apical cell surfaces of epithelial cells in the human tr

195 Syntaxin 1A is seen near the apical cell surfaces of human bronchial airway epitheliu

196 folding is characterized by constriction of apical cell surfaces, and the resulting cell shape chang

197 When applied to apical cell surfaces, wild type toxins elicited a brisk

198 It was observed that in most EMTs, the apical cell tail is retracted cleanly from the lumen of

199 we find that Cdc42 is critical for limiting apical cell tension by antagonizing Rho activity at AJs.

200 o contributes, downstream of Rho1 to sustain apical cell tension.

201 egrity independent of its role in sustaining apical cell tension.

202 s in BRK1 being localized only in the tip of apical cells, the exclusive site of cell extension and d

203 In addition, SIK1 fails to partition the apical cell; thus, nuclei are not likely to arrest mitos

204 ), suggesting that the higher sensitivity of apical cells to salt is not related to either enhanced N

205 The apical cells uniformly cover the surface of the tadpole

206 was the most important factor in determining apical cell uptake of lutein, with cookies and muffins e

207 Apical cells vary in length and number of nuclei, wherea

208 f an exogenous PME induces thickening of the apical cell wall and inhibits pollen tube growth.

209 es to the focused secretion of pectin to the apical cell wall and, thus, to the polarized growth of t

210 interference contrast (DIC), (2) changes in apical cell wall fluorescence in cells stained with prop

211 We obtained evidence that apical cell wall synthesis occurs through examining the

212 ch constitute the principal component of the apical cell wall.

213 at both types of media penetrate through the apical cell wall.

214 essation of elongation and thickening of the apical cell wall.

215 respond to osmotic stress by softening their apical cell walls, sustaining extension growth despite r

216 The act7-4 root apical cells were not in straight files and contained ob

217 leaky; the intercellular spaces between the apical cells were penetrated by sulfosuccinimidyl-6-(bio

218 of actin is observed at the tip of wild-type apical cells, whereas in Deltabrk1, smaller, more distin

219 e cells and more numerous apoptotic condylar apical cells, while chondroprogenitors displayed higher