ライフサイエンスコーパス: apnea-hypopnea index

1 en the groups and correlated negatively with apnea-hypopnea index.

2 Nocturnal polysomnography to evaluate apnea-hypopnea index.

3 andard overnight sleep study to determine an apnea-hypopnea index.

4 ifferent gradations of OSA severity based on apnea-hypopnea index.

5 es in leg fluid volume and either DeltaNC or apnea-hypopnea index.

6 obstructive sleep apnea was assessed by the apnea-hypopnea index.

7 s were matched for age, body mass index, and apnea/hypopnea index.

8 provement in nocturnal oxygen saturation and apnea/hypopnea index.

9 Seventy-one subjects (ages, 55-76 yr; apnea-hypopnea index, 0.2-96.6 events/h) were evaluated

10 (29 male) with Cheyne-Stokes breathing (mean apnea-hypopnea index 19.8 [SD 2.6] and stable symptomati

11 ft tissue structures in 48 control subjects (apnea-hypopnea index, 2.0 +/- 1.6 events/hour) and 48 pa

12 We recruited 30 obese control subjects (apnea-hypopnea index, 4.7 +/- 3.1 events per hour) and 7

13 ge, 54 [10] yr; median [interquartile range] apnea-hypopnea index, 41 [35-53]; mean [SD] Epworth slee

14 Seventeen subjects (apnea-hypopnea index, 42.6 +/- 6.2 [SEM]) were studied d

15 our) and 72 obese patients with sleep apnea (apnea-hypopnea index, 43.5 +/- 28.0 events per hour).

16 ents/hour) and 48 patients with sleep apnea (apnea-hypopnea index, 43.8 +/- 25.4 events/hour).

17 OSA prevalence was 65% (median apnea-hypopnea index, 7.2; range, 0-93), 40% of which we

18 Nasal occlusion increased the apnea hypopnea index (AHI) (occlusion mean = 6.6 +/- 8.0

19 SDB was quantified with the apnea hypopnea index (AHI) and oxygen desaturation measu

20 association of head and facial form with the apnea hypopnea index (AHI) in 364 white individuals and

21 leep fragmentation, all participants with an apnea-hypopnea index (AHI) > or = 1 were analyzed separa

22 r the OSA-associated quantitative phenotypes apnea-hypopnea index (AHI) and body mass index (BMI).

23 of portable monitors, or association between apnea-hypopnea index (AHI) and health outcomes among com

24 A polysomnographically derived apnea-hypopnea index (AHI) and hypoxemia index (percent

25 Apnea-hypopnea index (AHI) and percent nighttime with ox

26 d significant dose-relationships between REM apnea-hypopnea index (AHI) and prevalent hypertension.

27 SDB was categorized using the apnea-hypopnea index (AHI) and the hypoxemia index (perc

28 risk for all-cause mortality, with FEV1 and apnea-hypopnea index (AHI) as the primary exposure indic

29 sleep-disordered breathing, depending on the apnea-hypopnea index (AHI) cutoff, ranged from 40 to 60%

30 moderate to severe SDB, defined as having an Apnea-Hypopnea Index (AHI) greater than 15 as assessed b

31 In fully adjusted models, patients with apnea-hypopnea index (AHI) greater than 30 had a 30% hig

32 week, on a nondialysis day, subjects with an apnea-hypopnea index (AHI) greater than or equal to 20 h

33 revalence of periodontal disease between the apnea-hypopnea index (AHI) groups, with a negligible Spe

34 Women with an apnea-hypopnea index (AHI) less than 10 comprised the co

35 Women with an apnea-hypopnea index (AHI) less than 10 were the control

36 Patients with an apnea-hypopnea index (AHI) less than 15 were the control

37 patients with resistant hypertension and an apnea-hypopnea index (AHI) of 15 or higher.

38 ricular ejection fraction of 45% or less, an apnea-hypopnea index (AHI) of 15 or more events (occurre

39 A total of 14.7% of subjects with an apnea-hypopnea index (AHI) of 15 or more had a diagnosis

40 th recently diagnosed (<6 months) OSA and an apnea-hypopnea index (AHI) of 20 events/hour or more.

41 The prevalence varies based on the apnea-hypopnea index (AHI) threshold used for the evalua

42 Mean apnea-hypopnea index (AHI) was 52.6 +/- 28.2 (SD) events

43 The apnea-hypopnea index (AHI) was derived from standardized

44 nce of OSA, physiological data including the apnea-hypopnea index (AHI), and nocturnal oxygen saturat

45 r, OAs have variable success at reducing the apnea-hypopnea index (AHI), and predicting responders is

46 ated with hsCRP and MRP 8/14 levels and with apnea-hypopnea index (AHI), BMI z score, and apolipoprot

47 Our main exposures were the obstructive apnea-hypopnea index (AHI), central apnea index (CAI >/=

48 ed into four severity groups on the basis of apnea-hypopnea index (AHI), followed by comparisons of c

49 In combination with apnea-hypopnea index (AHI), identification of "respirato

50 y was associated with a 55% reduction in the apnea-hypopnea index (AHI), which decreased from a prefl

51 p-disordered breathing was quantified by the apnea-hypopnea index (AHI)-the average number of apneas

52 for the diagnosis of OSAS and calculation of Apnea-Hypopnea Index (AHI).

53 rence, hours of use, mask leak, and residual apnea-hypopnea index (AHI).

54 OSA severity was defined by the apnea-hypopnea index (AHI): severe >30, moderate >15-30,

55 Apnea-hypopnea index (AHI, the average number of apneas

56 Outcome measures included apnea-hypopnea index (AHI; average number of apneas plus

57 The primary outcome measures were the apnea-hypopnea index (AHI; the number of apnea or hypopn

58 OSAS was defined by an apnea/hypopnea index (AHI) greater than or equal to 1 ev

59 a resolution of symptoms and a reduction in Apnea/Hypopnea Index (AHI) to < 5/h, and partial respons

60 p was divided into 3 sub-groups based on the apnea/hypopnea index (AHI): mild, moderate, or severe OS

61 ne 30, 1997, and had to have normal results (apnea hypopnea index [AHI] <5).

62 al of 126 patients with moderate-severe OSA (apnea hypopnea index [AHI], 25.6 [SD 12.3]) were randoml

63 of hypersomnolence in 741 patients with SDB (apnea-hypopnea index [AHI] >/= 10 events/h).

64 surgical responses (>/=50% reduction in the apnea-hypopnea index [AHI] and <10 events/hour) in patie

65 participants were identified as free of OSA (apnea-hypopnea index [AHI] of <5 events/h and not treate

66 terval 1.00 to 1.21] per 10-unit increase in apnea-hypopnea index [AHI]) but not in older men or in w

67 in 68 patients (55 males, 13 females; median apnea-hypopnea index [AHI], 35) not receiving hypotensiv

68 We examined 55 sleep apnea probands (apnea-hypopnea index [AHI]: 43.2 +/- 26.3 events/h), 55

69 A total of 25 adults with positional OSAS (apnea-hypopnea index [AHI]supine:AHInon-supine >/= 2) we

70 both sleep laboratory and clinical criteria (apnea/hypopnea index [AHI] > or = 10 and the presence of

71 The 24-h prevalence of predominant CAs (apnea/hypopnea index [AHI] >/=5 events/h, with CA of >50

72 ecutive new outpatients diagnosed with SAHS (apnea/hypopnea index [AHI] >or= 5/hour, and >or= 2 sympt

73 ore episodes of apnea and hypopnea per hour (apnea-hypopnea index, AHI).

74 y of apneas and hypopneas per hour of sleep (apnea-hypopnea index, AHI).

75 For each interval, we assessed the median apnea-hypopnea index and the relative risk of sudden dea

76 Body mass index, apnea-hypopnea index, and cognitive abilities were model

77 ea or Cheyne-Stokes respiration, obstructive apnea-hypopnea index, and percentage of sleep time with

78 rs11691765, GPR83, P = 1.90 x 10(-8) for the apnea-hypopnea index, and rs35424364; C6ORF183/CCDC162P,

79 en saturation as measured by pulse oximetry, apnea-hypopnea index, and the fraction of events that we

80 e four-year follow-up study according to the apnea-hypopnea index at base line were estimated after a

81 The effects of severe apnea (apnea-hypopnea index, at least 30 episodes/h), which occ

82 om 12-channel home polysomnography, were the apnea-hypopnea index (average number of apneas/hypopneas

83 s were metaanalyzed for association with the apnea-hypopnea index, average oxygen saturation during s

84 incident CVD experienced larger increases in apnea-hypopnea index between polysomnograms.

85 The relation of incident CVD to change in apnea-hypopnea index between the 2 polysomnograms was te

86 ion was assessed for baseline sleep indices: apnea-hypopnea index, central sleep apnea (central apnea

87 The difference in adjusted mean apnea-hypopnea index change between subjects with and wi

88 rdiac causes during other intervals, and the apnea-hypopnea index correlated directly with the relati

89 Mean number of events measured by the apnea-hypopnea index decreased from 35.7/h to 2.1/h at 6

90 Apnea hypopnea index, defined as number of apneas and hy

91 his hypothesis, we correlated loop gain with apnea-hypopnea index during supine, nonrapid eye movemen

92 ntification of all children with obstructive apnea-hypopnea index greater than 5/hour total sleep tim

93 ss quality of life in 122 patients with SDB (apnea-hypopnea index > or = 5 events/hour), this study f

94 with polysomnography between 1992 and 2004 (apnea-hypopnea index > or =15) who subsequently underwen

95 overweight/obese matched patients with OSA (apnea-hypopnea index >/= 15 events per hour) and 11 norm

96 OSA (apnea-hypopnea index >/= 5 events/h) was assessed using

97 SDB was characterized by apnea-hypopnea index >/=15 events per hour (polysomnogra

98 derate-to-severe sleep-disordered breathing (apnea-hypopnea index >/=15%) was significantly higher in

99 ients with newly revascularized CAD and OSA (apnea-hypopnea index >/=15/h) without daytime sleepiness

100 with paroxysmal AF (43 with >/=moderate OSA [apnea-hypopnea index >/=15] and 43 without OSA [apnea-hy

101 best predicted by age >60 years (HR: 5.53), apnea-hypopnea index >20 (HR: 1.60), mean nocturnal O2sa

102 ed that, for clinically defined sleep apnea (apnea/hypopnea index > or = 10 and daytime symptoms), me

103 women diagnosed with moderate to severe OSA (apnea-hypopnea index, >/=15) in 19 Spanish sleep units.

104 eepiness: severe sleep-disordered breathing (apnea-hypopnea index, >30 episodes/hr), self-report of p

105 M and non-REM sleep was quantified using the apnea-hypopnea index in REM (AHIREM) and non-REM sleep (

106 correlation was found between loop gain and apnea-hypopnea index in the atmospheric group only (r =

107 At baseline, the mean apnea-hypopnea index in the patients with the syndrome w

108 18 overweight/obese subjects without apnea (apnea-hypopnea index < 15 events per hour) with 25 overw

109 ife in normal subjects (n = 15) without SDB (apnea-hypopnea index < 5 events/hour) recruited from the

110 to moderate degrees of sleep apnea (5 < or = apnea-hypopnea index < or = 30).

111 ea-hypopnea index >/=15] and 43 without OSA [apnea-hypopnea index <5]), right atrial and left atrial

112 of 1 to each criterion that was satisfied: (apnea-hypopnea index, <30 events per hour) + (nadir oxyg

113 derate to severe sleep-disordered breathing (apnea-hypopnea index, measured as events/hour, >/=15) ar

114 es, death, sleep study measures (such as the Apnea-Hypopnea Index), measures of cardiovascular status

115 omnography, and severe SDB was defined as an apnea-hypopnea index of >30 per hour of sleep.

116 Relative to the reference category of an apnea-hypopnea index of 0 events per hour at base line,

117 t confidence interval, 1.13 to 1.78) with an apnea-hypopnea index of 0.1 to 4.9 events per hour at ba

118 d adjusted rate ratio [caffeine/placebo] for apnea-hypopnea index of 0.89 [95% CI = 0.55-1.43]; P = 0

119 Sleep-disordered breathing was defined as an apnea-hypopnea index of 15 or more events per hour of sl

120 he prevalence of sleep-disordered breathing (apnea-hypopnea index of 15 or more) among hormone users

121 Participants with an apnea-hypopnea index of 15 to 50 events per hour were ra

122 t confidence interval, 1.46 to 5.64) with an apnea-hypopnea index of 15.0 or more events per hour.

123 the syndrome was 35, as compared with a mean apnea-hypopnea index of 2 in the comparison group.

124 onsecutive patients were enrolled who had an apnea-hypopnea index of 20 h(-1) or greater and an Epwor

125 ructive sleep apnea syndrome was based on an apnea-hypopnea index of 5 or higher (five or more events

126 t confidence interval, 1.29 to 3.17) with an apnea-hypopnea index of 5.0 to 14.9 events per hour, and

127 e or more events per hour); patients with an apnea-hypopnea index of less than 5 served as the compar

128 An apnea-hypopnea index of more than 10 per hour was signif

129 omes were total sleep time on actigraphy and apnea-hypopnea index on polysomnography.

130 leep-disordered breathing was ascertained by apnea-hypopnea index or clinical diagnosis.

131 al apnea (OR, 6.31; 95% CI, 1.94-20.51), and apnea-hypopnea index (OR, 1.22; 95% CI, 1.08-1.39 [per 5

132 associated with significant improvements in apnea-hypopnea index (P < 0.001); microarousal index (P

133 versely with DeltaNC (r=-0.755, P<0.001) and apnea-hypopnea index (r=-0.765, P<0.001), it did not in

134 ificant positive correlation between SAA and apnea-hypopnea index (r=0.40, P=0.03).

135 saturation (SaO2); right putamen tCho/Cr and apnea hypopnea index; right putamen GABA/Cr and baseline

136 mes were nocturnal oxygen saturation and the apnea/hypopnea index; secondary outcomes were sleep stru

137 us mean diffusivity correlated with a higher apnea hypopnea index (Spearman's r = -0.50, p = 0.008) a

138 idnight to 6 a.m. had a significantly higher apnea-hypopnea index than those with sudden death from c

139 leep-disordered breathing, as defined by the apnea-hypopnea index (the number of episodes of apnea an

140 oke or cardiovascular events with increasing apnea-hypopnea index values.

141 Mean (SEM) apnea-hypopnea index was also significantly higher in pa

142 Initially, the apnea-hypopnea index was determined by polysomnography f

143 The mean (+/-SD) of apnea-hypopnea index was significantly higher in patient

144 bjects (1,839 in fully adjusted models), the apnea-hypopnea index was used to classify OSA as none (0

145 and 2590 m than placebo and autoCPAP: median apnea/hypopnea index was 5.8 events per hour (5.8/h) (IQ

146 y of apneas and hypopneas per hour of sleep (apnea-hypopnea index) was determined by unattended, sing

147 studies focused on traits defined using the apnea-hypopnea index, which contains limited information

148 (Epworth Sleepiness Scale score >10) and an apnea-hypopnea index with 3% desaturation and from 5 to