ライフサイエンスコーパス: apneic

1 ariable indicating the presence of increased apneic activity (IAA).

2 If apneic activity does cause acute stress in HF, it should

3 expanded and allow for prolonged periods of apneic airway surgery.

4 entilatory stimuli on the hypocapnia-induced apneic and hypopneic thresholds in sleeping dogs.

5 Lung wet/dry weight was 5.40 +/- 0.93 in apneic animals and 5.00 +/- 0.67 in controls.

6 e experiment fell from 89.6 to 82.8 mm Hg in apneic animals and from 92.2 to 85.5 mm Hg in controls.

7 In patients with OSAS, the EMGgg for apneic breaths during REM (37 +/- 9%) was lower than dur

8 s significantly larger compared with the pre-apneic control.

9 the post-apneic period compared with the pre-apneic control.

10 digging while feeding near the surface, and apneic dives.

11 roscopy showed gross alveolar fluid in three apneic dogs, and electron microscopy showed interstitial

12 showed interstitial fluid in two additional apneic dogs.

13 tively, to 1.7 to 3.2 Torr at the end of the apneic episode.

14 y characterized by hypoxemia due to frequent apneic episodes and fragmentation of sleep due to the br

15 of activation during spontaneous obstructive apneic episodes in non-rapid-eye-movement (NREM) sleep:

16 Apneic episodes induced in animals ventilated with 15%,

17 When the apneic episodes were eliminated by application of nasal

18 due to the brief arousals that terminate the apneic episodes.

19 There is leukocytosis with lymphocytosis and apneic episodes.

20 Repetitive apneic events disrupt the normal physiologic interaction

21 d possibly a higher arousal threshold during apneic events.

22 ear to be linked to the repetitive nocturnal apneic events.

23 The mean amplitude and slope of the post-apneic fEPSP was significantly larger compared with the

24 ves (1 mg lorezapam), and one patient became apneic for a brief interval after receiving additional n

25 o relationship between BNP and the number of apneic/hypopneic episodes or the number of arousals.

26 ssociated with the frequency and severity of apneic/hypopneic episodes, intermittent arousals, and hy

27 ssociated with a significant increase in the apneic index in these infants (28.6 +/- 6.4/hour [S] vs.

28 ices showed good agreement with steady-state apneic left ventricular stroke volume values and moderat

29 There was no difference between apneic oxygenation and usual care in incidence of oxygen

30 Apneic oxygenation does not seem to increase lowest arte

31 These findings do not support routine use of apneic oxygenation during endotracheal intubation of cri

32 west arterial oxygen saturation was 92% with apneic oxygenation versus 90% with usual care (95% confi

33 of supplemental oxygen during laryngoscopy (apneic oxygenation) may prevent hypoxemia.

34 high-flow nasal cannula during laryngoscopy (apneic oxygenation) or no supplemental oxygen during lar

35 that hf-TTI stabilized breathing patterns in apneic patients, and was safe and efficacious for prolon

36 p fragmentation are difficult to separate in apneic patients.

37 cm H2O (mean +/- SD) for three breaths in 19 apneic patients.

38 tilation induced a longer hypocapnia-induced apneic period (51.5 +/- 9.9 versus 11.2 +/- 5.5 seconds,

39 red-pulse facilitation ratio during the post-apneic period compared with the pre-apneic control.

40 diate effect of tracheal stimulation was an "apneic" period at FRC, during which the PCA, a laryngeal

41 atory regulation, including vagally mediated apneic reflexes.

42 way and the respiratory neurons that mediate apneic reflexes.

43 sults showed that PNE markedly prolonged the apneic response and exacerbated the bradycardic response

44 and 12.1% indicated a passive (i.e., infant apneic) response by the infant, despite excluding period

45 ergence of sensory inputs capable of driving apneic responses and that it may represent a common link

46 However, in our earlier study of apneic responses produced by glutamate stimulation in th

47 racterize the sites in the pons that produce apneic responses.

48 We found that apneic sites were consistently associated with the inter

49 were no significant differences with gender, apneic spells, jaundice, or phototherapy.

50 ry sensitivity to CO2 between eupnea and the apneic threshold are changeable in the face of variation

51 3-5 mm Hg > eupnea (and 7-10 mm Hg > normal apneic threshold) throughout CMV trials at raised freque

52 e during sleep may lower the value below the apneic threshold, thereby resulting in central apnea.

53 sed a significant (p < 0.05) increase in the apneic threshold, which we attribute to an inhibitory ef