ライフサイエンスコーパス: apoptosis

1 n the early stages of programmed cell death (apoptosis).

2 and sensitivity to mitochondrial removal and apoptosis.

3 roliferation, cytoskeletal organization, and apoptosis.

4 sed palmitic acid-induced ROS generation and apoptosis.

5 high-glucose triggered oxidative stress and apoptosis.

6 to promote BOK accumulation and induction of apoptosis.

7 n, segmental sclerosis, podocyte injury, and apoptosis.

8 mately resulting in caspase-3 activation and apoptosis.

9 elated with increased DEN-induced hepatocyte apoptosis.

10 d sustain energy homeostasis and resulted in apoptosis.

11 its translocation to the MOM in the onset of apoptosis.

12 or-related apoptosis-inducing ligand-induced apoptosis.

13 lanoma cells induces G1/S arrest followed by apoptosis.

14 L-2 but unchanged rates of proliferation and apoptosis.

15 Delia radicum, by disrupting the control of apoptosis.

16 by regulating cell growth, proliferation and apoptosis.

17 way, whereas maladaptive UPR outputs trigger apoptosis.

18 r paracrine factors in induction of neuronal apoptosis.

19 orters can disrupt both autophagy and induce apoptosis.

20 ility and by promoting cell-cycle arrest and apoptosis.

21 dylserine translocation that occurs in early apoptosis.

22 invasiveness and increased susceptibility to apoptosis.

23 ANC-1 cells sensitized them to TRAIL-induced apoptosis.

24 ession of Notch1 diminished TNFalpha-induced apoptosis.

25 d induces persistent Ca(2+) mobilization and apoptosis.

26 ic nuphar analogous are able to induce rapid apoptosis.

27 ed upon activation, thus triggering death by apoptosis.

28 ted AKT levels and an increase in myocardial apoptosis.

29 ining why SRp55 depletion triggers beta-cell apoptosis.

30 n-PIM inhibitor, SEL24-B489, induced RS cell apoptosis.

31 anging from cellular growth to migration and apoptosis.

32 inhibition and suppression of stress-induced apoptosis.

33 CCR5 CRPA and both CD4 counts and CD4 T cell apoptosis.

34 s with cell cycle defects, senescence and/or apoptosis.

35 cation of Bax to mitochondria and preventing apoptosis.

36 d them resistant to serum starvation-induced apoptosis.

37 ic manner by massive activation of intrinsic apoptosis.

38 DP5 and PUMA and consequent human beta-cell apoptosis.

39 iferation and cell survival, ERbeta promotes apoptosis.

40 ect correlated with oxidative stress and SMC apoptosis.

41 e metabolites, mitochondrial dysfunction and apoptosis.

42 d found that E2 counteracts TNFalpha-induced apoptosis.

43 epithelial cell mitochondrial DNA damage and apoptosis.

44 ssion of mRNAs encoding proteins involved in apoptosis.

45 ls that accumulate DNA damage from executing apoptosis.

46 plete internalization that leads to cellular apoptosis.

47 vesicle prevented canal fusion by inhibiting apoptosis.

48 ogenitor cells to replicative senescence and apoptosis.

49 dent cells are refractive to PKCdelta-driven apoptosis.

50 irectly involved in smooth muscle cell (SMC) apoptosis.

51 related with increased intestinal epithelial apoptosis.

52 ses p53 nuclear exclusion and cell-intrinsic apoptosis.

53 A) are important mediators and regulators of apoptosis.

54 e cell such as respiration, energy level and apoptosis.

55 protection of beta cells from stress-induced apoptosis.

56 remodelling, transcriptional regulation and apoptosis.

57 ereby primes tumor cells to caspase-mediated apoptosis.

58 iferation with low levels of LMP1 and little apoptosis.

59 d cardiac function and reduced cardiomyocyte apoptosis.

60 acellular superoxide, which then signals RGC apoptosis.

61 nic capacity through cellular senescence and apoptosis.

62 peroxidase function plays a key role during apoptosis.

63 potentiated H2O2-induced DNA damage and SMC apoptosis.

64 nct from the Golgi fragmentation observed in apoptosis.

65 ated EOMA proliferation but did not mitigate apoptosis.

66 t of tunicamycin-induced renal ER stress and apoptosis.

67 -C) was associated with ribosomal stress and apoptosis.

68 n and mediate key cellular processes such as apoptosis.

69 utophagosomes/amphisomes with lysosomes, and apoptosis.

70 cell death, namely cornification rather than apoptosis.

71 gradation and induction of caspase-dependent apoptosis.

72 (2 W/cm(2)) to achieve maximal induction of apoptosis.

73 oroquine (CQ) enhanced palmitic acid-induced apoptosis accompanied by increased ROS generation, and t

74 t not NSCs (quiescent and activated) undergo apoptosis after 2 Gy IR.

75 iptionally activated a biosensor and induced apoptosis after DNA damage.

76 on of XPO1 cargos (p53, PTEN), and increased apoptosis after treatment.

77 Evasion of apoptosis allows many cancers to resist chemotherapy.

78 omarkers related to mitochondrial stress and apoptosis also were significantly lower in the CD47mAb-t

79 gyrus (DG), drastically increased perinatal apoptosis, altered DG cell composition, and impaired lea

80 osteoblast numbers by suppressing osteoblast apoptosis and activating bone-lining cells.

81 iac overexpression of Qki5 prevented cardiac apoptosis and cardiac atrophy induced by doxorubicin and

82 Using drugs to protect HG MKs from apoptosis and CD42b shedding, we also demonstrate that a

83 ation in human cancer cells by inducing both apoptosis and cell cycle arrest, and that reducing DHX33

84 phospho-histone H3 (PH3) staining to assess apoptosis and cell proliferation, respectively, showed a

85 p53 transcriptional target genes that induce apoptosis and cell-cycle arrest.

86 rvations demonstrate that Ly6C(+) macrophage apoptosis and decreased ingress of circulating monocytes

87 decreased cervical cell number by increasing apoptosis and decreasing cell proliferation through init

88 When we disrupted feedback, apoptosis and divisions were uncoupled, and the organ de

89 rotection to keratinocytes from UV-B-induced apoptosis and DNA damage via ATR.

90 TP-deficient neutrophils exhibited decreased apoptosis and enhanced accumulation at the infection sit

91 volved in the control of alloreactive T cell apoptosis and expansion.

92 required for proliferation, protection from apoptosis and expression of activation/memory genes duri

93 nction, vascularization, and amelioration of apoptosis and hypertrophy.

94 emonstrate enhanced proliferation, decreased apoptosis and increased cytotoxicity in the presence of

95 flammation, steatosis, oxidative stress, and apoptosis and increased mitochondrial biogenesis.

96 al. show that Sirt6 protects podocytes from apoptosis and inflammation by increasing autophagic flux

97 target RNAs includes positive regulators of apoptosis and inflammation, and modulators of signalling

98 Suppressing PLK2 attenuated HDG-induced apoptosis and inflammatory responses both in vitro and i

99 diabetes, where it contributes to beta-cell apoptosis and insufficient insulin secretion.

100 rrest and subsequent intrinsic mitochondrial apoptosis and is shared by all preimmune murine B cell s

101 o neonatal animals cause widespread neuronal apoptosis and later neurocognitive impairment.

102 hs of age preceded by spontaneous hepatocyte apoptosis and liver inflammation within the first month

103 Human T2D kidneys exhibited more RPTC apoptosis and lower expression of hnRNP F, SIRTUIN-1, an

104 Murine cytomegalovirus triggers both apoptosis and necroptosis in infected cells; however, en

105 The extrinsic apoptosis and necroptosis pathways regulate each other a

106 drial ribosome biogenesis, and regulation of apoptosis and nuclear transcription.

107 otein accumulation in the ER, contributes to apoptosis and organ injury.

108 showed markedly better inhibitory effects on apoptosis and oxidative/inflammatory stresses in the RGC

109 contributed to the synergistic induction of apoptosis and proliferation inhibition in AML cell lines

110 -) mice were reduced without affecting their apoptosis and proliferation.

111 l permeability and flow cytometry assays for apoptosis and proliferation.

112 dence that acetylation promotes FOXO3-driven apoptosis and recently a specific JNK (c-Jun N-terminal

113 al stimulation alters the spatial pattern of apoptosis and sensory deprivation leads to exacerbated a

114 ne kinase inhibitor and Akt inhibitor causes apoptosis and synergistic growth inhibition in multiple

115 zing protein tristetraprolin (TTP) regulates apoptosis and the numbers of activated infiltrating muri

116 n of BCL-2 family members enables evasion of apoptosis and tumor resistance to chemotherapy.

117 anism that involves caspase-dependent T cell apoptosis and upregulation of inhibitory immune checkpoi

118 it decreased neurogenesis, enhanced neuronal apoptosis, and an increased ratio of excitatory to inhib

119 APK3, a gene involved in cell proliferation, apoptosis, and autophagy.

120 bition of NPM1 impairs caspase-2 processing, apoptosis, and caspase-2-dependent inhibition of cell gr

121 unculin-A-induced actin depolymerization and apoptosis, and cell line transfection.

122 ctional effects on viability, proliferation, apoptosis, and cytotoxicity were monitored.

123 t effects on growth inhibition, induction of apoptosis, and delay of acquired resistance.

124 pression with relevance to oxidative stress, apoptosis, and ion transport.

125 AIL-sensitive cells attenuated TRAIL-induced apoptosis, and shRNA-mediated HOTAIR knockdown in TRAIL-

126 ions (OCDL), DNA double-strand breaks (DSB), apoptosis, and the local and systemic immune responses.

127 pathy, nonsyndromic intellectual disability, apoptosis, and the Warburg effect.

128 lbumin challenge (P < .05), higher levels of apoptosis (Annexin V positivity, P < .005), and less lun

129 This "lock-in and apoptosis" approach performed by our novel compound in H

130 ial cell (AEC) mitochondrial dysfunction and apoptosis are important in idiopathic pulmonary fibrosis

131 h forms disjunct from immunologically silent apoptosis are, in theory, more likely to be relevant.

132 osing pathways leading to caspase-8-mediated apoptosis as well as nuclear factor kappaB (NF-kappaB)-d

133 exhibited decreased survival and pronounced apoptosis associated with a decreased GSH/GSSG ratio, au

134 b or glycan ligand binding causes eosinophil apoptosis associated with reactive oxygen species (ROS)

135 transcripts related to stress responses and apoptosis at the wound healing stage, signaling pathways

136 p27T187A KI activated an E2F1-p73-apoptosis axis in DKO prostate tumorigenesis, slowed dis

137 inflammation resolution, neutrophils undergo apoptosis before they are removed by macrophages, but if

138 s, cyt c detection is not only serving as an apoptosis biomarker, but also is of great importance to

139 s that promote cellular survival and prevent apoptosis, both of which are important drivers of tumori

140 not only repressed proliferation and induced apoptosis but also impaired tumorigenicity.

141 response to EZH2 inhibition is required for apoptosis, but not for growth arrest, through a mechanis

142 suppressor FOXO3 is an important mediator of apoptosis, but the mechanisms that control its proapopto

143 ic apoptosis by death receptors or intrinsic apoptosis by chemotherapeutic agents.

144 Increased apoptosis by cleaved caspase-3 (P = 0.03) and decreased

145 cells in response to induction of extrinsic apoptosis by death receptors or intrinsic apoptosis by c

146 dual role in breast cancer cell invasion and apoptosis by demethylating histone and the non-histone p

147 t Nur77, an orphan nuclear receptor, induces apoptosis by targeting mitochondria.

148 ession of lung fibrosis, attenuated cellular apoptosis (caspase-3/7) and lung deposition of collagen

149 ondria in ATP production (bioenergetics) and apoptosis (cell life/death decision) were thought to be

150 ichment Analysis revealed pathways including apoptosis, DNA repair and early estrogen response that w

151 by influencing multiple functions including apoptosis, fibrosis and inflammation.

152 iomyocyte hypertrophy, reduced cardiomyocyte apoptosis, fibrosis, calcium/calmodulin-dependent protei

153 ell characterized cellular proliferation and apoptosis guards (NF-kappaB, Bcl-2 and p53) in these NPs

154 roles for mitochondria in calcium handling, apoptosis, heme turnover, inflammation, and oxygen and n

155 rus-induced type I interferon production and apoptosis; however, the regulation of MAVS-mediated apop

156 Survivin, an inhibitor of apoptosis (IAPs) family member, exhibited a decreased ex

157 RAIL) has been implicated in cellular growth/apoptosis, immune cell function and bone-resorbing osteo

158 minate the structural basis of resistance to apoptosis, immune evasion, and loss of cell junctions se

159 roduced by a mutated form of Kv2.1 mimicking apoptosis in a mammalian expression system, and protecte

160 asL induced potent target antigen-restricted apoptosis in a panel of cancer lines and in primary pati

161 ed their ability to sensitize TRAIL-mediated apoptosis in a panel of renal carcinoma cells.

162 we show that 100 microM of ascorbate induced apoptosis in A2058 melanoma cells.

163 , we show that ABBV-075 efficiently triggers apoptosis in acute myeloid leukemia (AML), non-Hodgkin l

164 ors of heat shock protein 90 (HSP90) induced apoptosis in BL cells in vitro at concentrations that di

165 b-AP15 induces apoptosis in cancer cells, but the underlying mechanisms

166 nally, activated cofilin is unable to induce apoptosis in cells genetically lacking p53.

167 increased DNA damage and selectively induced apoptosis in cells overexpressing oncogenes, suggesting

168 elial cell numbers, triggering extrusion and apoptosis in crowded regions and cell division in sparse

169 cell adhesion, AMPK signaling, autophagy and apoptosis in different cell types.

170 understanding of cytokine-induced beta-cell apoptosis in early T1D.

171 Furthermore, LMP1 blocks IRF5-mediated apoptosis in EBV-infected cells.

172 Our data revealed no significant apoptosis in either HCFs or HKCs following CXL.

173 sumption, oxidative stress, and subsequently apoptosis in epithelial cells during ischemia-reperfusio

174 T-627 failed to decrease renal ER stress and apoptosis in ETB def rats.

175 mitochondria and triggers caspase-dependent apoptosis in HeLa cells, which are more sensitive to inh

176 orphyrin IX based SDT (ALA-SDT) could induce apoptosis in human tongue squamous carcinoma SAS cells t

177 ion, leading to increased ROS production and apoptosis in hypoxic cancer cells as well as impaired gr

178 ellular ATP /AMP during chemotherapy-induced apoptosis in Jurkat human leukemia cells.

179 PTEN inhibition enhanced tubular cell apoptosis in kidneys with IRI, which was associated with

180 attenuated mitogenic signaling and triggered apoptosis in KRAS-mutant lung cancer cells and inhibited

181 xpression, which increased cisplatin-induced apoptosis in lung cancer cells.

182 (13b) decreased proliferation and activated apoptosis in MDA-MB-231 breast cancer cells in vitro and

183 ole of physiological ascorbate to potentiate apoptosis in melanoma.

184 blocks cellular proteasome function, induces apoptosis in MM cells and overcomes resistance to protea

185 However, TGF-beta induced apoptosis in normal and benign but not in carcinoma cult

186 erent from the canonical SOCE/Ca(2+)-induced apoptosis in other tumors.

187 lin-3 induced G1 arrest but did not activate apoptosis in p53(-/-) sarcoma cells.

188 al ANKZF1, as shown by an increased level of apoptosis in patients' lymphocytes, a decrease in mitoch

189 whether the reduced necrosis and macrophage apoptosis in plaques of these mice was a manifestation o

190 ein kinase B (AKT) inhibitor AZD5363 induced apoptosis in PTEN-deficient DLBCLs irrespective of their

191 1 and SIRT7 and failed to induce p-FOXO3 and apoptosis in response to LPS.

192 f IRS-2 sensitizes breast carcinoma cells to apoptosis in response to treatment with microtubule-disr

193 encoded by RS1, regulates ERK signaling and apoptosis in retinal cells.

194 t ability to track cells that have undergone apoptosis in situ has revealed a division of labor among

195 S-phase entry in DCX(+) cells and increased apoptosis in Sox2(+) neural stem and progenitor cells, a

196 n of human ABCD1 in oligodendrocytes rescued apoptosis in the abcd1 mutant.

197 lcohol feeding causes lipid accumulation and apoptosis in the liver.

198 ults in induction of ER stress and increased apoptosis in the pancreas, potentially explaining the lo

199 ion of activated caspase-3 (AC3) to quantify apoptosis in the postnatal mouse ventral forebrain and h

200 Further, PU-H71 induced apoptosis in the presence of stromal co-culture or cytop

201 itors reduced p27 expression and potentiated apoptosis in thyroid cancer cells while not affecting su

202 sed keratinocyte proliferation and increased apoptosis in vitro and in skin grafts regenerated on mic

203 the physiological fate of cells that die by apoptosis in vivo is their rapid recognition and engulfm

204 FRD significantly augmented cardiac apoptosis in WT vs. CD-WT mice, which was prevented by c

205 tress, and increasing programmed cell death (apoptosis) in the tissues required for neurulation.

206 em-like cells and rendered them sensitive to apoptosis induced by chemotherapeutic drugs.

207 Endothelial cell apoptosis induced by oxidative stress is an early event

208 AML may protect myeloid precursor cells from apoptosis induced by the NE mutants.

209 he potential of this approach by quantifying apoptosis-induced intracellular activity in individual c

210 Our TUNEL experiments using tunicamycin, an apoptosis inducer, and GADD34, an inhibitor of eIF2alpha

211 While tumor necrosis factor-related apoptosis inducing ligand (TRAIL) has been implicated in

212 ng exposure to tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) and by visualizing and

213 TNF-alpha related apoptosis-inducing ligand (TRAIL) selectively kills tumo

214 TNF-related apoptosis-inducing ligand (TRAIL) was initially describe

215 ated arrest of tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis.

216 This hypoxia-specific apoptosis induction by YC-1 involved excessive reactive

217 rface resulting in receptor crosslinking and apoptosis induction.

218 insights, including in sphingolipid-mediated apoptosis, inflammation and energy storage/usage.

219 We show that transient apoptosis inhibition by short-term overexpression of pro

220 t apoptosis precedes CD42b shedding and that apoptosis inhibition enriches the FV(+) HG/CD42b(+) MKs,

221 Apoptosis is an important antiviral host defense mechani

222 Apoptosis is an important component of normal tissue phy

223 fore they are removed by macrophages, but if apoptosis is delayed, neutrophils can cause extensive ti

224 The ability to suppress host macrophage apoptosis is essential for M. tuberculosis (Mtb) to repl

225 expressed in polyclonal Drosophila S2 cells, apoptosis is induced.

226 hicken Ntn1 in the mouse otic vesicle, where apoptosis is less prominent, resulted in canal truncatio

227 is; however, the regulation of MAVS-mediated apoptosis is poorly understood.

228 ell [Breg] counts, and B cell activation and apoptosis) is specifically associated with pathogenic SI

229 gy impairment, cellular organelle stress and apoptosis, leading to an NTD reduction.

230 r sensitizers of RCC cells to TRAIL-mediated apoptosis led to identification of the 17beta-hydroxywit

231 roinflammatory cytokines), cardiac fibrosis, apoptosis, lower CAR (Coxsackievirus and adenovirus rece

232 rylation and a PKR-eIF2alpha pathway in cell apoptosis may be an important part of the mechanism unde

233 ellular processes, including the cell cycle, apoptosis, migration and invasion.

234 yme activities and redox status that lead to apoptosis, necrosis, and autophagy of tumour cells.

235 into one of the canonical categories such as apoptosis, necrosis, paraptosis, and autophagy, suggesti

236 latory events were investigated: cell death (apoptosis), neutrophil influx and cytokine/chemokine exp

237 nly autophagy but also staurosporine-induced apoptosis occurring in a Bax, Drp1-dependent manner.

238 Estrogens protect against apoptosis of endothelial cells (ECs), one of the hallmar

239 Moreover, increased apoptosis of GCs and follicular atresia, reduced ovulati

240 ylation, demonstrated that PC1-5TMC inhibits apoptosis of HEK293T cells in a PKR-eIF2alpha-dependent

241 The TRAIL pathway can mediate apoptosis of hepatic stellate cells to promote the resol

242 leic acid-binding dye causing dose-dependent apoptosis of individual cells without collateral damage.

243 hage reprogramming, reactivation of T cells, apoptosis of Kras mutant neoplastic ductal cells and pan

244 eavage of caspase-3, indicating the onset of apoptosis of LX-2 cells, as was confirmed by the termina

245 uces autophagy and fibrosis with concomitant apoptosis of LX-2 cells, which may explain some potentia

246 estation of the selective effect of TRPC3 on apoptosis of M1 macrophages previously observed in vitro

247 nuclear factor kappaB (NF-kappaB) causes the apoptosis of MDCC-MSB1 cells.

248 virus thus "locked-in" subsequently induces apoptosis of the host cells.

249 nd (TRAIL) was initially described to induce apoptosis of tumor cells and/or virally infected cells,

250 herapy induce DNA damage to drive cells into apoptosis or senescence as outcomes of the DNA damage re

251 to selectively induce programmed cell death (apoptosis) or uncontrolled cell death (necrosis).

252 or ISO-1 significantly blocked photoreceptor apoptosis, outer nuclear layer (ONL) thinning, and retin

253 These cells are defective in apoptosis owing to blocked degradation of Mcl-1, a pro-s

254 with ADHD-associated volumetric reductions: apoptosis, oxidative stress and autophagy.

255 nated in the activation of the mitochondrial apoptosis pathway.

256 gical MAPK inhibition restores migration and apoptosis potential in a mouse LCH model, as well as in

257 and CD42b shedding, we also demonstrate that apoptosis precedes CD42b shedding and that apoptosis inh

258 vealed time-dependent changes in endometrial apoptosis preceding neutrophil influx and cytokine/chemo

259 Vascular smooth muscle cell (VSMC) apoptosis precipitates AAA formation, whereas VSMC proli

260 activity was observed without an increase in apoptosis rate.

261 lymphoma 2) protein Bax (Bcl-2 associated X, apoptosis regulator) can commit cells to apoptosis via o

262 turnover and expression of proliferation and apoptosis-related genes in gastric cancer (GC) and adjac

263 rved PCD regulatory mechanisms include plant apoptosis remains enigmatic.

264 Yersinia-induced apoptosis requires the kinase activity of receptor-inter

265 Invasive growth and apoptosis resistance of breast cancer cells are associat

266 ts against lethal insults through conferring apoptosis resistance to hepatocytes.

267 e cell (PASMC) proliferation, migration, and apoptosis resistance.

268 sted a beneficial role for host survival and apoptosis resistance.

269 disease characterized by the accumulation of apoptosis-resistant fibroblasts in the lung.

270 Ls and, in turn, triggers cell-cycle arrest, apoptosis, senescence and autophagy in many cancer cells

271 tumor cell lysis, induction of cytotoxic or apoptosis-sensitizing cytokines and promotion of antitum

272 Activation of apoptosis signal-regulating kinase 1 (ASK1) in hepatocyt

273 pression of GMAP6 protected Huh-7 cells from apoptosis, suggesting that GIMAP6 is an anti-apoptotic p

274 re surrounded by uninfected cells undergoing apoptosis, suggesting that programmed cell death may lim

275 ntly greater inhibition of proliferation and apoptosis than when used alone.

276 vae showed a significantly increased rate of apoptosis that could be ameliorated with cysteamine, the

277 associated with Abeta deposits and neuronal apoptosis, the critical upstream factors contributing to

278 sensitive to the cellular changes caused by apoptosis, the sensitivity of spectral analysis to oncos

279 However, the precise contribution of apoptosis to GC biology and selection is not well define

280 moved by a dual mechanism of cell egress and apoptosis to re-establish the stable microglial network.

281 (ATM), mitosis (PMF1, CENPN and MAD1L1) and apoptosis (TP53).

282 splayed hyperactivation of p53 and increased apoptosis under genotoxic and hematopoietic stress.

283 odes of cell death, such as cells undergoing apoptosis versus necrosis/necroptosis.

284 X, apoptosis regulator) can commit cells to apoptosis via outer mitochondrial membrane permeabilizat

285 as viral mitochondria-localized inhibitor of apoptosis (vMIA) traffics from the ER to mitochondria an

286 Apoptosis was also lower in the rapamycin treated cells.

287 Apoptosis was evaluated by a TUNEL assay.

288 ility of ROL or Bt2cAMP to induce neutrophil apoptosis was impaired in AnxA-knock-out mice.

289 Apoptosis was observed in both infected and uninfected b

290 Macrophage apoptosis was reduced and collagen content was enhanced

291 tizes lung cancer cells to cisplatin-induced apoptosis, we for the first time found that knockdown of

292 mental NEC resulting in increased epithelial apoptosis, we investigated the effects of C. sakazakii o

293 , defective insulin secretion, and increased apoptosis when a combined high-fat and high-glucose diet

294 GAPDH-overexpressing cells to DNA damage and apoptosis, which indicated that Ape1 is indispensable fo

295 rdiac morphology, and elevated cardiomyocyte apoptosis, which were mitigated in the cathepsin K knock

296 -apoptotic p53 program to induce and execute apoptosis, while PLD1 functions in a regulatory multi-br

297 potential in podocytes and induced podocyte apoptosis, while the inhibition of autophagy by chloroqu

298 a new role for EBNA3A in the suppression of apoptosis with implications for EBV lymphomagenesis.

299 uction of tumor suppressor protein, p53, and apoptosis with suppression of urokinase-type plasminogen

300 entative model by integrating the control of apoptosis with the relevant signaling pathways.