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1 action with key signaling molecules, such as apoptosis signal-regulating kinase 1.
2 and trace amounts of MEKK3 but not MEKK1 or apoptosis-signal regulating kinase-1.
3 scade is provided by the MAPK kinase kinase, apoptosis-signal-regulating kinase 1.
5 estration of Daxx in the nucleus and keeping apoptosis signal-regulating kinase 1 activation in check
6 ocked nitrative Trx inactivation, attenuated apoptosis signal-regulating kinase-1 activation, and red
7 nase-1) complex formation was abolished, and apoptosis signal-regulating kinase-1 activity was increa
9 acy of selonsertib, a selective inhibitor of apoptosis signal-regulating kinase 1, alone or in combin
10 Oxidized thioredoxin is known to activate apoptosis signal-regulating kinase 1, an upstream activa
11 "inhibitory complex," leading to release of apoptosis signal-regulating kinase 1 and activation of t
12 se-independent pathway through activation of apoptosis signal-regulating kinase 1 and c-Jun NH(2)-ter
14 ogen-activated protein kinase kinase 3/6 and apoptosis signal-regulating kinase 1 and that p38 signal
15 inding to and activating its effector kinase apoptosis signal-regulating kinase 1, and therefore, fro
16 ssociation of TRX with the apoptosis inducer apoptosis signal-regulating kinase-1, and enhanced nucle
17 oxidant defense, which in turn activates the apoptosis signal-regulating kinase-1 apoptotic pathway.
18 uces a transient increase in the activity of apoptosis signal-regulating kinase 1 (ASK-1), a MAPKKK t
19 tress decreased the Cdc25A-bound fraction of apoptosis signal-regulating kinase-1 (ASK-1) and sensiti
20 udies both provided suggestive evidence that apoptosis signal regulating kinase 1 (ASK1) may influenc
21 ciation with this heteromeric complex, while apoptosis signal regulating kinase-1 (ASK1) dissociated
22 its AGP-induced apoptosis signal, regulating apoptosis signal-regulating kinase 1 (ASK1) activity and
23 ed through phosphorylation targets including apoptosis signal-regulating kinase 1 (ASK1) and cyclic A
24 necrosis factor (TNF)-induced activation of apoptosis signal-regulating kinase 1 (ASK1) and germinal
25 rial stress leads to activation of FoxO, via Apoptosis Signal-regulating Kinase 1 (ASK1) and Jun-N-te
26 UV irradiation-induced apoptosis through apoptosis signal-regulating kinase 1 (ASK1) and MAKK4 (S
29 might be mediated by FIP200 interaction with apoptosis signal-regulating kinase 1 (ASK1) and TNF rece
32 ion mutants showed the C-terminal portion of apoptosis signal-regulating kinase 1 (ASK1) bound GRX, a
37 atment with antioxidant N-acetyl-l-cysteine, apoptosis signal-regulating kinase 1 (ASK1) inhibitor th
51 Daxx from the nucleus to the cytoplasm, and apoptosis signal-regulating kinase 1 (ASK1) oligomerizat
53 zed by rapid and sustained activation of the apoptosis signal-regulating kinase 1 (ASK1) signaling pa
55 JNK inhibition correlated with inhibition of apoptosis signal-regulating kinase 1 (ASK1), a JNK and p
56 physically interacts with the N-terminus of apoptosis signal-regulating kinase 1 (ASK1), a kinase be
59 novel functional interaction between Akt and apoptosis signal-regulating kinase 1 (ASK1), a mitogen-a
61 that HIV-1 Nef associates with and inhibits apoptosis signal-regulating kinase 1 (ASK1), a serine/th
62 tivation of the JNK pathway through SEK1 and apoptosis signal-regulating kinase 1 (ASK1), an upstream
64 nine kinases, p21-activated kinase 1 (PAK1), apoptosis signal-regulating kinase 1 (ASK1), and polo-li
65 on induces rapid and sustained activation of apoptosis signal-regulating kinase 1 (ASK1), c-Jun N-ter
66 ed through the Fas receptor and involves the apoptosis signal-regulating kinase 1 (ASK1), the p38 MAP
67 henyl phosphate as well as phosphocasein and apoptosis signal-regulating kinase 1 (ASK1), thought to
68 between inhibitor of kappaB kinase (IKK) and apoptosis signal-regulating kinase 1 (ASK1), unveiling a
70 hydrogen peroxide experiments show that the apoptosis signal-regulating kinase 1 (ASK1)-p38 kinase p
79 tubular cells, and colocalizes with inactive apoptosis signal-regulating kinase-1 (ASK1) phosphorylat
80 ed with nor inhibited the kinase activity of apoptosis signal-regulating kinase-1 (ASK1), an upstream
82 dinucleotide phosphate oxidase activity and apoptosis signal-regulating kinase 1 /c-Jun N-terminal k
83 bstrate (IRS1) through the activation of the apoptosis signal-regulating kinase 1/c-Jun N-terminal Ki
84 ciated with attenuation of the ASK1-JNK/p38 (apoptosis signal-regulating kinase 1/c-Jun NH(2)-termina
85 activity was markedly inhibited, Trx-1/ASK1 (apoptosis signal-regulating kinase-1) complex formation
86 T cell hybrid transfected with a kinase-dead apoptosis-signal-regulating kinase 1 fails to activate p
87 totic, stress-activated protein kinase ASK1 (apoptosis signal-regulating kinase 1) in vitro and in vi
88 rotein/Trx binding and disruption of the Trx/apoptosis signal-regulating kinase 1 "inhibitory complex
89 nce, possibly via the AMPK/mTOR/p70(S6K) and apoptosis signal-regulating kinase 1/JNK/IRS1 pathways.
91 athrin, 3) recruitment and activation of the apoptosis signal-regulating kinase-1/MAPK kinase-3/p38 M
92 ented by pharmacological inhibition of ASK1 (apoptosis signal-regulating kinase 1) or of c-Jun N-term
93 and inhibit Daxx-dependent activation of the apoptosis signal-regulating kinase 1 stress-inducible ki
94 in kinase/ERK kinase kinase (MEKK) 1, MEKK2, apoptosis-signal regulating kinase-1, TGF-beta activated
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