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1 ry effects cannot be explained by changes in appetitive behavior.
2 via MOR, within the CeA promote this form of appetitive behavior.
3  cortex (IL) in the environmental control of appetitive behavior.
4 s capable of supporting distinct theories of appetitive behavior.
5 volved in stimulant-related learning driving appetitive behavior.
6 of hypothalamic nuclei in the stimulation of appetitive behavior.
7 hat have helped elucidate dopamine's role in appetitive behavior.
8 egion that is important in the regulation of appetitive behavior.
9 ated with emotion regulation, attention, and appetitive behavior.
10 in parasympathetic outflow, and decreases in appetitive behavior.
11 ning drug levels into increased drug-related appetitive behavior.
12 CeA neurons define a pathway for suppressing appetitive behaviors.
13 een proposed to be selectively active during appetitive behaviors.
14 or a common neural mechanism associated with appetitive behaviors.
15 or determining their contribution to learned appetitive behaviors.
16 viors and dissect the BLA-to-CeA circuit for appetitive behaviors.
17 output nucleus of the amygdala implicated in appetitive behaviors.
18  signaling and examined several aversive and appetitive behaviors.
19 mprise a network likely involved in approach/appetitive behaviors.
20 ts in the amygdala that promote and suppress appetitive behaviors analogous to the direct and indirec
21 c strength are hypothesized to contribute to appetitive behavior and addiction.
22 ocrine and nonendocrine functions, including appetitive behavior and carbohydrate substrate utilizati
23 ion in nucleus accumbens contributes to both appetitive behavior and fearful behavior that is generat
24 ral hypothalamus (LHA) integrates reward and appetitive behavior and is composed of many overlapping
25                            The NAcc mediates appetitive behavior and is critically modulated by dopam
26 el hypothalamic-AcSh circuit that influences appetitive behavior and mediates the antidepressant acti
27 inct populations of CeA neurons that mediate appetitive behaviors and dissect the BLA-to-CeA circuit
28 ent with roles in the performance of learned appetitive behaviors and in positive reinforcement, resp
29 examine gustatory physiology, tastant-evoked appetitive behavior, and food ingestion to understand cl
30 g trait anxiety, contextual fear memory, and appetitive behavior, and is known to be sensitive to str
31 ed whole body energy demands into subsequent appetitive behavior are incompletely understood.
32 ion, but dissociates from stable conditioned appetitive behavior as this signal returns to preconditi
33 egulating the balance of limbic control over appetitive behavior at the point of learning.
34 Performance levels and lipping intensity (an appetitive behavior) both showed that the monkeys' motiv
35 e and ingestive behavior, opaline can elicit appetitive behavior but can also inhibit ingestion and e
36 may be a reinforcer that elicits conditioned appetitive behavior, but its reinforcing properties stro
37   The results suggest that the inhibition of appetitive behavior by energy state signals may depend,
38  hippocampus is involved with the control of appetitive behavior by interoceptive "hunger" and "satie
39 sed behavior and reveals mechanisms by which appetitive behavior can go awry.
40  variety of physiologic functions, including appetitive behaviors, cognitive functions and metabolism
41 nd was followed by a variety of approach and appetitive behaviors, consistent with electrophysiologic
42 evented DNQX microinjections from generating appetitive behavior (eating) in rostral shell, and equal
43 ion can be quite variable between studies of appetitive behavior, even within the same species.
44 cells may underlie its inhibitory effects on appetitive behavior for rewarding stimuli.
45 f the neural circuit that regulates a sexual appetitive behavior in C. elegans.
46 h natural reward, are less responsive during appetitive behavior in familiar conditions, and are inse
47 supports a multitude of functions related to appetitive behavior in humans and animals, and it has be
48  regulation of competing limbic control over appetitive behavior in mice, we hereby examined the effe
49 amate disruptions typically generate intense appetitive behaviors in rats, but the disruption increme
50  the causal role for CeA circuits underlying appetitive behaviors is poorly understood.
51  on a range of conditioned and unconditioned appetitive behaviors known to depend on mesolimbic DA ac
52                                              Appetitive behaviors of rats were monitored in a runway
53 es associated with hedonic taste evaluation, appetitive behavior, oromotor coordination, and inhibito
54                                              Appetitive behaviors require complex decision making tha
55 rimary role for these neurons in controlling appetitive behaviors such as foraging that promote the d
56  Although compelling within the framework of appetitive behavior, the view that illicit drugs hijack
57  dopamine (DA) is phasically released during appetitive behaviors, though there is substantive disagr
58 ly more trials than nondepleted rats; hence, appetitive behavior was mildly potentiated by depletion,
59          Silencing neuronal activity reduces appetitive behaviors, whereas inducible activation resul
60 ) CeA neurons define a pathway for promoting appetitive behaviors, while R-spondin 2(+) BLA pyramidal

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