1 g block 1 is described, also in 6 steps from arabitol.

2 butenolide 1 is described in four steps from arabitol.

3 poxypentane building block 4 in 6 steps from arabitol.

4 d, and tested chemical sensors determining d-arabitol.

5 ent bonds between vicinal hydroxyl groups of arabitol and boronic acid substituents of the bithiophen

6 teps are the selective kinetic protection of arabitol and the cyclization of 11 to form the butenolid

7 , as well as elevated amounts of erythritol, arabitol, and ribitol in the plasma of affected individu

8 previously described selective protection of arabitol as its 1,2:4,5-bis-pentylidene acetal 5, we rep

9  With the QCR and EG-FET chemosensors, the d-arabitol concentration was determined under flow-injecti

10 .00 mM, respectively, being lower than the d-arabitol concentrations in urine of patients with invasi

11 n addition, a number of these saccharides (L-arabitol, D-fructose, sucrose, D-glucose, cellotetraose,

12 ls mannitol, sorbitol, erythritol, adonitol, arabitol, galactitol, and xylitol revealed that diols co

13  with respect to common interferences, and l-arabitol in particular, of the devised chemosensors was

14               An elevated concentration of d-arabitol in urine, especially compared to that of l-arab

15 l in urine, especially compared to that of l-arabitol or creatinine, is indicative of a fungal infect

16    Elevated urinary excretion of erythritol, arabitol, ribitol, and pent(ul)ose-5-phosphates was dete

17 ol, ethanol, glycerol, erythritol, rhamnose, arabitol, sorbitol, galactitol, mannitol, arabinose, glu