ライフサイエンスコーパス: arachnoid

1 drug resistance or demonstrated presence of arachnoid adhesions respectively.

2 breakage of a catheter, pump malfunction and arachnoid adhesions.

3 in the more internal connective tissues (pia-arachnoid and endoneurium-perineurium) was also found in

4 cal parenchyma may be transmitted to the pia-arachnoid and might then serve in the induction of neuro

5 pear as the meninges differentiate into pia, arachnoid, and dura.

6 tral retinal vessels, and in the dura mater, arachnoid, and pia mater of the meningeal sheath surroun

7 eling of cells of the outer meninges (mainly arachnoid), blood vessels (arteries, veins, and capillar

8 nical stimulation of adult human and rat pia-arachnoid cell cultures (loaded with calcium indicator d

9 Gap junction coupling between pia-arachnoid cells and astrocytes was shown by dye transfer

10 Calcium waves in pia-arachnoid cells could invade contiguous astrocytes, and

11 The differentiation of arachnoid cells in the mutant meninges is also abnormal.

12 after a trauma, spontaneous hemorrhage in an arachnoid cyst is a rare and serious complication with a

13 mental abnormalities of the corpus callosum, arachnoid cyst, abnormalities of the septum pellucidum a

14 Arachnoid cysts are congenital or developmental intra-ar

15 Arachnoid cysts are congenital, benign and intra-arachno

16 A great majority of arachnoid cysts are congenital.

17 Arachnoid cysts are mostly asymptomatic and they can dev

18 cerebellar dysplasia, ventriculomegaly, and arachnoid cysts are nearly invariant.

19 Arachnoid cysts form 1% of the non-traumatic lesions whi

20 here is a male dominance at a rate of 3/1 in arachnoid cysts which locate mostly in the middle fossa.

21 has proved useful, such as ventriculomegaly, arachnoid cysts, and abdominal masses, are described.

22 e is an uncommon and serious complication of arachnoid cysts, which can give rise to atypical feature

23 bdural hygroma is a rare complication of the arachnoid cysts.

24 ion defects leading to disruption of the pia-arachnoid, ectopia of fibroblasts in the cortex, and rea

25 enhancement patterns are characterized: dura-arachnoid enhancement and pia-subarachnoid space enhance

26 an ex vivo model of CSF outflow across human arachnoid granulations (AGs) as an approximation of in v

27 lytic lesions on skull radiographs, seen as arachnoid granulations fovea in CT.

28 ood vessels and later within neurons and pia arachnoid (> or =3 hours), particularly within piriform

29 sive mononeuropathies, 2 (CI: 2, 3); and sub-arachnoid haemorrhage, 1 (CI: 0.8, 2).

30 xtent that the injury is associated with sub-arachnoid hemorrhage and hematoma.

31 vessels in the subarachnoid space and in the arachnoid layer of the meninges.

32 over trigeminal sensory neurons and over the arachnoid layers surrounding trigeminal ganglia supports

33 both TNFR subtypes was also present over the arachnoid layers surrounding trigeminal ganglia.

34 widespread infection of cells of the pia and arachnoid mater of the leptomeninges over large areas of

35 ake mechanisms, GABA transporters in the pia-arachnoid may help to regulate the amount of GABA availa

36 r indirectly across the choroid plexuses and arachnoid membrane (blood-CSF barrier) into the CSF and

37 outflow may occur in a similar manner in the arachnoid membrane adjacent to the granulations, in addi

38 suggesting the need for a model using human arachnoid membrane with granulations for the study of co

39 The brain's arachnoid membrane with granulations is an important bio

40 a result of splitting or duplication of the arachnoid membrane.

41 y of CSF but also of metabolites through the arachnoid membrane.

42 can develop anywhere in the brain along the arachnoid membrane.

43 The arachnoid membranes thicken.

44 ell lines were implanted between the pia and arachnoid meninges as well as in the sciatic nerve to mi

45 tor outer segments, and in the fascicles and arachnoid of the optic nerve.

46 The dura-arachnoid pattern consists of curvilinear enhancement ov

47 overmigrated neurons into the developing pia-arachnoid, scattering its mesenchymal cells throughout t

48 igration of neurons and glial cells into the arachnoid space results in the formation of cortical dys

49 ir transport of fluorescent solutes from sub-arachnoid space to brain in mice or rats.

50 , and Scleral Thickness (at the Anterior Sub-arachnoid space) were calculated for each ONH.

51 accumulate in the ventricles, meninges, sub-arachnoid spaces, and injection site.

52 Arachnoid villi occlude and degenerate.

53 The pia-arachnoid waves were blocked by either octanol or apyras

54 3 was almost entirely restricted to the pia-arachnoid, whereas mGATs 1 and 4 were present only in sp