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1 cells optogenetically with halorhodopsin or archaerhodopsin.
2 that the same is true for bacterioruberin in archaerhodopsin.
3 uorescence of a microbial rhodopsin protein, Archaerhodopsin 3 (Arch) from Halorubrum sodomense, expr
8 activation of a light-activated proton pump, Archaerhodopsin-3 (Arch), proton transients induced ASIC
9 cy tuning of the stimulated neurons, whereas archaerhodopsin-3 (Arch)-mediated inactivation biased de
10 decreased spiking of excitatory neurons, as archaerhodopsin-3 (Arch)-mediated optical silencing of c
12 transduced to express either ChR2(E123A) or archaerhodopsin-3 from the Halorubrum sodomense strain T
14 ciated virus expressing the inhibitory opsin archaerhodopsin, and fiber-optic cannulae were implanted
16 The light-activated inhibitory proton pump Archaerhodopsin (Arch) was expressed under control of th
19 pens when both are activated together, using Archaerhodopsin as an optical voltage clamp to provide t
24 annelrhodopsin, CheRiff, and a near infrared Archaerhodopsin-derived voltage indicator, QuasAr2, via
26 n contrast, hydrolysis of the Schiff base in archaerhodopsin does not abolish the CD bands of bacteri
27 reely behaving mice, whereas inhibition with archaerhodopsin for 30 min suppressed LH pulsatility.
28 based on green fluorescent proteins (FPs) or archaerhodopsin has emerged as a powerful approach for d
30 ed mice co-expressing Channelrhodopsin-2 and Archaerhodopsin in pyramidal cells in the hippocampal CA
33 with channelrhodopsin-2, or inhibition with archaerhodopsin, simulated an instantaneous increase or
34 et CRF neurons with the optogenetic silencer archaerhodopsin tp009 (CRF-ArchT) to examine the role of
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