ライフサイエンスコーパス: archenteron

1 y inside the ectoderm, and generation of the archenteron.

2 ension movements during the formation of the archenteron.

3 both ectodermal lineages and portions of the archenteron.

4 efore this territory invaginates to form the archenteron.

5 y of animal blastomeres to contribute to the archenteron.

6 s also regulated to that new position in the archenteron.

7 derm and fusing at the dorsal midline of the archenteron.

8 promote expression in the vegetal plate and archenteron.

9 o locate expression in the vegetal plate and archenteron.

10 ere also common, including reduced or absent archenterons.

11 t primordium is similar to elongation of the archenteron and also of the entire embryonic axis (both

12 e, at the gastrula stage in the whole of the archenteron and in postgastrular stages only in the midg

13 The archenteron and its derivatives were measured three dime

14 the Endo16 gene, expressed in vegetal plate, archenteron and midgut.

15 endodermal target genes are expressed in the archenteron and might be terminal differentiation enzyme

16 (4) that interactions between the tip of the archenteron and the presumptive oral ectoderm are not re

17 region and the posterior of the invaginating archenteron, and finally to the midgut and hindgut of th

18 e, expressed in the embryonic vegetal plate, archenteron, and then midgut.

19 eposited in the basal lamina surrounding the archenteron as well as in other areas of the blastocoel

20 As the archenteron begins to elongate, marking the onset of the

21 ge embryos SpHmx is expressed throughout the archenteron, but particularly strongly in delaminating s

22 e layered epithelium, and whether or not the archenteron cavity actually gives rise to the gut lumen.

23 Instead the archenteron cavity almost closes at tailbud stages befor

24 eavage pattern in determining ectodermal and archenteron cell fates.

25 Both archenteron cell number and wall volume continued to inc

26 ondary invagination involves the addition of archenteron cells and an increase in volume of the arche

27 during secondary invagination, the number of archenteron cells increased by at least 38% (over 50% wh

28 e rearrangement and reshaping of the primary archenteron cells.

29 RhoA induces precocious invagination of the archenteron, complete with the actin rearrangements and

30 eneration gives rise to larvae containing no archenteron derivatives at all, endoderm only, or both e

31 alone each regenerate the full complement of archenteron derivatives; thus, they are uninformative as

32 In addition, epithelial cells in the archenteron display a significant decrease in adherens j

33 mall micromere descendents at the tip of the archenteron during gastrulation and are then enriched in

34 econdary mesenchyme cells and the elongating archenteron during gastrulation; Cadherin (G form) has a

35 cellular matrix of cells of the invaginating archenteron during sea urchin gastrulation.

36 In addition, much of archenteron elongation was found to be independent of Xd

37 cell division being a responsible force for archenteron elongation.

38 g sites of mesoderm specification within the archenteron endomesoderm.

39 The volume of the archenteron epithelial wall plus the volume of 17 new SM

40 teron cells and an increase in volume of the archenteron epithelium, we conclude that secondary invag

41 convergent extension, blastopore closure and archenteron formation in a single embryo.

42 ction is included in the kinetic analysis of archenteron formation.

43 s an extensive rearrangement of cells of the archenteron giving rise to secondary mesenchyme at the a

44 enotype in C5a knockdown embryos, and causes archenteron hyper-invagination in control embryos.

45 ort-range communication between cells of the archenteron in order to reorganize the tissues and posit

46 Although invagination of the archenteron in sea urchins and dorsal closure in Drosoph

47 bilaterally symmetric, and flank the ectopic archenteron, in some cases resulting in mirror-image, sy

48 , but they accompany the invagination of the archenteron initially, in much the same way vertebrate m

49 dy reports that RhoA is necessary to trigger archenteron invagination in the sea urchin embryo.

50 r SMCs but also causes decreased and delayed archenteron invagination.

51 movements and to cell rearrangements during archenteron invagination.

52 egetal plate and primary invagination of the archenteron, involves only the Endo16-expressing cells o

53 Furthermore, they suggest that the archenteron is competent to form mesoderm or endoderm, a

54 During gastrulation, the archenteron is formed using cell shape changes, cell rea

55 These results show that the prospective archenteron is produced by a larger population of cleava

56 be into a long thin tube; secondly, that the archenteron lining does not become the definitive gut lu

57 somitic morphology while still a part of the archenteron lining.

58 While the archenteron lumen doubled in length during secondary inv

59 ng Notch signal as one upstream component of archenteron morphogenesis.

60 re, as an initial step towards examining how archenteron precursors are specified, a clonal analysis

61 eric embryo approach, we show that implanted archenteron precursors differentiate autonomously to pro

62 s demonstrate that mesoderm induction in the archenteron requires contact with ectoderm, and loss-of-

63 Recombination of ectoderm and archenteron rescues development.

64 gesting that this region of the fully formed archenteron retains an unexpected pluripotency.

65 al effect of disrupting morphogenesis of the archenteron, revealing a previously unsuspected function

66 Long after the archenteron reveals territorial specification through ex

67 nsion of deep ectoderm just underlain by the archenteron roof is twice that of not-yet-underlain deep

68 nic mesenchyme, the CyIIa gene, expressed in archenteron, skeletogenic and secondary mesenchyme, and

69 After removal of any or all parts of the archenteron, the remaining veg 1 and /or veg 2 tissue re

70 y retain apical projections extending to the archenteron throughout gastrulation.

71 n giving rise to secondary mesenchyme at the archenteron tip followed by the foregut, midgut and hind

72 rived from the host, the ectopic presumptive archenteron tissue can act to 'organize' ectopic axial s

73 ition, the ectopically implanted presumptive archenteron tissue induces ectopic skeletal patterning s

74 Both veg1 and veg2 descendants form archenteron tissues, revealing that the ectoderm and end

75 Oral veg1 clones extended along the archenteron up to the foregut region, while aboral veg1

76 t decline until after full elongation of the archenteron was completed.

77 mbryo but the recognizable morphology of the archenteron was re-established.

78 when SMCs that emigrated from the tip of the archenteron were included).

79 ve capacity of this structure, pieces of the archenteron were removed or transplanted at different st

80 After removal of the archenteron (which includes presumptive coelomic mesoder