ライフサイエンスコーパス: aromatic L-amino acid decarboxylase

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1 and VMAT2, and by carbidopa, which inhibits aromatic L-amino acid decarboxylase.

2 t decrement in tyrosine hydroxylase (TH) and aromatic L-amino acid decarboxylase (AAAD) activities.

3 functional recovery mechanisms, with reduced aromatic L-amino acid decarboxylase (AAAD) activity clos

4 Aromatic L-amino acid decarboxylase (AAAD) and tyrosine

5 The aromatic L-amino acid decarboxylase (AAAD) is involved i

6 st cells (PF) were genetically modified with aromatic L-amino acid decarboxylase (AADC) and VMAT-2 ge

7 Aromatic l-amino acid decarboxylase (AADC) deficiency is

8 Aromatic l-amino acid decarboxylase (AADC) deficiency is

9 The rat aromatic l-amino acid decarboxylase (AADC) gene contains

10 Homozygous mutations in the aromatic l-amino acid decarboxylase (AADC) gene result i

11 and non-NMDA antagonists on the activity of aromatic l-amino acid decarboxylase (AADC) in the corpus

12 techniques that cells containing the enzyme aromatic l-amino acid decarboxylase (AADC) occur not onl

13 ted whether the downregulation of the enzyme aromatic l-amino acid decarboxylase (AADC) or the TH cof

14 F110 up-regulates tyrosine hydroxylase (TH), aromatic l-amino acid decarboxylase (AADC), and GTP cycl

15 atic l-amino acids may also be substrates of aromatic l-amino acid decarboxylase (AADC), the level of

16 ericytes that ectopically express the enzyme aromatic L-amino acid decarboxylase (AADC), which synthe

17 The aromatic L-amino-acid decarboxylase (AADC) defect appear

18 Alveolar Type II cells express the enzyme aromatic-L-amino acid decarboxylase (AADC) and, when inc

19 Aromatic L-amino acid decarboxylases (AADCs) are key enz

20 urine analyses indicated reduced activity of aromatic L-amino acid decarboxylase and other PLP-depend

21 1 in immature neurons and the expression of aromatic l-amino acid decarboxylase and tyrosine hydroxy

22 nces upstream of the C.elegans gene encoding aromatic L-amino-acid decarboxylase and genes involved i

23 ween these two epithelial lineages), and (c) aromatic l-amino-acid decarboxylase, but not its dopamin

24 1) devised a novel in-house assay for PGD of aromatic L-amino acid decarboxylase deficiency, based on

25 howed the clinical phenotype to be caused by aromatic L-amino acid decarboxylase deficiency.

26 dopaminergic markers, tyrosine hydroxylase, aromatic L-amino acid decarboxylase, dopamine, 3,4-dihyd

27 ssion tomography (PET) imaging agent for the aromatic L-amino acid decarboxylase enzyme.

28 rmal growth factor-induced proliferation, or aromatic L-amino acid decarboxylase expression.

29 nin transporter, tryptophan hydroxylase, and aromatic L-amino acid decarboxylase genes whose expressi

30 inflammation by inducing vagal activation of aromatic L-amino acid decarboxylase, leading to the prod

31 tudied in PC12 cells following inhibition of aromatic L-amino acid decarboxylase with m-hydroxybenzyl

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