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1 ured affective dimensions (e.g., valence and arousal).
2 nuclei, poised to activate CeM for autonomic arousal.
3 depending on attentional demand and state of arousal.
4 mediated mechanisms that regulate behavioral arousal.
5 vation of autonomic structures and emotional arousal.
6 caudate nucleus) covaried with the level of arousal.
7 ested a role for noradrenergic modulation of arousal.
8 between-network cohesion peaked at moderate arousal.
9 le for noradrenergic mechanisms in mediating arousal.
10 executive control network peaks at moderate arousal.
11 eontological) and showed increased emotional arousal.
12 more" effects in perception and memory under arousal.
13 ral neurons (l-LNvs) regulating light-driven arousal.
14 in PDF-expressing neurons or l-LNvs promoted arousal.
15 ite through which the orexin neurons promote arousal.
16 ally active during periods of wakefulness or arousal.
17 stress, thus contributing to accumulation of arousal.
18 uggesting critical involvement in behavioral arousal.
19 2] and can change size and shape upon sexual arousal.
20 identify genes that affect larval zebrafish arousal.
21 cative of cortical activation and behavioral arousal.
22 brain regions involved in the regulation of arousal.
23 dopaminergic neurons differentially modulate arousal.
24 their effects are enhanced by noradrenergic arousal.
25 PDF-expressing neurons promotes light-driven arousal.
26 ear to be clearly associated with changes in arousal.
27 expectations about internal states, such as arousal.
28 he neuronal mechanisms controlling sleep and arousal.
29 ating in conjunction with systems regulating arousal.
30 ions, including circadian timing, sleep, and arousal.
31 to humans in comparable states of emotional arousal.
32 he neuronal mechanisms controlling sleep and arousal.
33 urprised facial expressions, unconfounded by arousal.
34 gions that integrate with the LC to regulate arousal.
35 ion of salient stimuli prompts vigilance and arousal.
36 on aggression, independent of any effect on arousal.
37 evels disinhibits CeM and triggers autonomic arousal.
38 thin the salience network dynamically tracks arousal.
39 negative) at a consistent level of emotional arousal.
40 iated with different categories of emotional arousal.
41 n to the LC, also covaried with the level of arousal.
42 ed significant increases in mean sympathetic arousal accompanied by significant increases in mean abs
43 ght be tuned to either (1) general emotional arousal (activation vs deactivation) or (2) specific emo
44 istent with the notion of increased cortical arousal after anodal stimulation and decreased cortical
46 vity and muscle tone to promote and maintain arousal along with learned adaptive behavioral responses
47 al phenotypes (extreme diurnal preference in arousal and activity) and sleep/mood disorders, includin
49 e effects through investigation of autonomic arousal and alterations of activation and functional con
50 ported by several mechanisms, such as social arousal and attention, and facilitates social monitoring
52 ated by a number of factors, from changes in arousal and attentional state to learning and task engag
54 rontal-striatal circuitry involved in limbic arousal and executive control in 36 individuals-18 cocai
55 esthesia-induced unconsciousness and altered arousal and further establish principled neurophysiologi
56 lcholine (ACh) levels, such as attention and arousal and in pathological conditions such as Alzheimer
57 nistered following a delay led to heightened arousal and increased generalization of SCRs and explici
58 methylphenidate are effective for increasing arousal and inducing reanimation, or active emergence fr
61 t occipital lesions associated with impaired arousal and left insular lesions associated with decreas
66 wever, nonphotic factors, such as behavioral arousal and metabolic cues, can also phase shift the mas
69 sensorimotor, and mixed stressors on driver arousal and performance with respect to (wrt) baseline.
70 xin (also known as hypocretin) neurons drive arousal and promote the maintenance of normal wakefulnes
71 ic stress disorder (PTSD) are alterations in arousal and reactivity which could be related to a malad
72 then show that treatments that both produce arousal and reset the phase of circadian clock activate
73 rane produced behavioral and EEG evidence of arousal and restored the righting reflex in 6/6 mice.
75 Homer1a serves as a molecular integrator of arousal and sleep need via the wake- and sleep-promoting
80 significant calming effect on physiological arousal and subjective experience during a socially stre
81 would have a calming effect on physiological arousal and subjective reports of state anxiety during t
82 t VTA dopaminergic neurons are necessary for arousal and that their inhibition suppresses wakefulness
83 known to drive brain states of attention and arousal and to be deficient in pathologies such as Alzhe
85 tion could facilitate transmission of global arousal and/or cognitive signals to the dLGN with retino
86 by the less-sleeping hemisphere caused more arousals and faster behavioral responses than those dete
87 evel of EEG-vigilance (an indicator of brain arousal) and b) a more stable EEG-vigilance regulation t
88 cal pain that may arise from excessive PANIC arousal, and 3) facilitation of social joy through the s
89 postural avoidance, increases physiological arousal, and enhances visual perception of affective sti
90 ght and increased sympathetic nervous system arousal, and lisdexamfetamine reduced weight and appetit
91 neurons play an essential role in promoting arousal, and loss of the orexin neurons results in narco
92 eus, a brain center implicated in attention, arousal, and panic that projects throughout the brain.
93 ognitive performance shifts as a function of arousal, and provide new insights into vulnerability for
95 between heart rate, a physiological index of arousal, and within-network cohesion in the salience net
98 s efficacy is increased when tonic levels of arousal are maintained in an optimal range, in manners t
101 iations between alterations of female sexual arousal as well as vaginal lubrication and the site of c
104 fluenced by internal states (e.g., shifts in arousal, attention or cognitive ability) or external sti
109 energic effects) model basically explains an arousal-based amplification of emotional stimuli, wherea
111 ev exposed and unexposed mice, avoidance and arousal behaviors were examined 7-15 days after exposure
114 ulness, and their silencing not only impairs arousal but is sufficient to rapidly and selectively ind
115 wakefulness is sufficient to not only impair arousal but to rapidly and selectively induce slow-wave
116 hrine (NE) control brain-wide states such as arousal, but whether they control complex social behavio
117 variety of behaviors that rely on heightened arousal, but whether they directly and causally control
118 ne is an anesthetic that alters the level of arousal by selectively targeting alpha2 adrenergic recep
119 itation, but a manifestation of the infant's arousal by the modeler's exhibition of the same behavior
120 ctivity accompanying shifts in vigilance and arousal can interfere with the study of other intrinsic
127 However, in vivo insights into noradrenergic arousal circuitry have been constrained by the fundament
129 mus, and basal ganglia comprise a functional arousal circuitry.SIGNIFICANCE STATEMENT Electrophysiolo
132 operates in parallel with frontal inputs to arousal circuits to regulate task-dependent modulation o
138 e tracked pupil responses (a proxy of phasic arousal) during sensory-motor decisions in humans, acros
141 can be counteracted by raising their sexual arousal, either by engaging the flies with prolonged cou
142 o several hours include cycles in behavioral arousal, episodic glucocorticoid release, and gene expre
143 EST1 = .48 and EST2 = .72; P < .001), sexual arousal (EST2 = .50; P = .008), and vaginal lubrication
144 minergic (DRN(DA)) activity upon exposure to arousal-evoking salient cues, irrespective of their hedo
146 of the decision, an increase in pupil-linked arousal, fixational eye movements, and fluctuations in b
148 ed that pupil responses reflected endogenous arousal fluctuations opposed to differences in stimulus
149 physiology of sleepwalking, i.e. the partial arousal from slow-wave sleep, is today well-documented,
150 eport nor other secondary processes, such as arousal, from perceptual decision processing per se.
151 es and circuits involved in sleep and sexual arousal have been extensively studied in Drosophila.
152 onsive during the peak period of sympathetic arousal, heart rate increase, and cardiorespiratory sens
154 by periodic rewarming (REW) during interbout arousal (IBA), proapoptotic conditions that are lethal t
156 Studies of subjective and genital sexual arousal in monosexual (i.e. heterosexual and homosexual)
157 ited higher levels of separation anxiety and arousal in response to social separation, but infants ca
158 s and subjective ratings of pleasantness and arousal in response to the pictures was tested for cued
160 d during the task showed increased emotional arousal in the hippocampal-damaged patients and they sta
166 he psychological processes thought to elicit arousal - in particular, the processes involved in the a
167 e to the chemogenetic manipulation enhancing arousal, increasing asymptotic levels of fear learning o
169 Ap in PDF-expressing neurons did not promote arousal, indicating that a reduced Ap function in PDF-ex
170 Taken together, these findings suggest that arousal induced by sounds can facilitate attention in a
171 acilitation of attention is studied: whether arousal induced by task-irrelevant auditory stimuli coul
172 in contrast to nocturnal rodents, behavioral arousal induced either by sleep deprivation or caffeine
175 ent an impressive interdisciplinary model of arousal-induced norepinephrine release and its role in s
179 orts the concept that dysregulation of brain arousal is a possible predictor of treatment response in
180 ur data indicate that impaired female sexual arousal is associated with MS lesions in the occipital r
184 al and memory processing, and its links with arousal, is discussed with respect to the GANE (glutamat
185 disgust-cues induced unexpected, unconscious arousal just before participants discriminated motion si
186 lso been advocated in modulation of state of arousal leading to transition from wakefulness to sleep
187 (electroencephalogram [EEG]) and behavioral arousal: lesions of the PB complex produce a monotonous
194 siological states corresponding to different arousal levels from deep sleep to focused attention.
195 te-specific pharmacological manipulations of arousal levels may be used to study brain arousal circui
196 ide an impressive cross-level account of how arousal levels modulate behavior, and they support it wi
200 ions of the brain that are involved in sleep arousal (locus coeruleus) and preference/aversion (nucle
202 l. offers a neurophysiological basis for the arousal mechanism which is essential for empirical aesth
203 he apparent tone-evoked responses reflect an arousal-mediated resumption of place-specific firing.
210 pil size, which reflects the activity of an 'arousal' network, related to the norepinephrine system.
211 la to alter potassium channel conductance in arousal neurons after light exposure, and in many animal
214 oject to several other established sleep and arousal nodes, including the tuberomammillary nucleus, v
215 project directly and/or indirectly to nearby arousal nuclei of the brainstem and to more distant targ
216 disturbance, and sympathetic nervous system arousal occurred more frequently with lisdexamfetamine t
217 The time scale of the effects of emotional arousal on neutral information processing is crucial for
218 g the pure effects of valence independent of arousal or emotion category is a challenging task, given
220 nocturnal light exposure can have either an arousal- or sleep-promoting effect, and that these respo
221 both sleep deprivation and caffeine-induced arousal potentiate the photic entrainment in a diurnal r
222 ian clock in a manner similar to that of our arousal procedures and that these shifts are also blocke
223 te that cholinergic cells activated by these arousal procedures project to the circadian clock in the
227 d whether a measure of women's physiological arousal (pupil diameter change) was correlated with rati
232 ments in focused and selective attention and arousal regulation, and to identify the specific nature
233 ional preparedness, selective attention, and arousal regulation, whereas associative ability (learnin
234 re suggests that through global projections, arousal-related neuromodulatory changes can rapidly alte
236 s a reversible drug-induced state of altered arousal required for more than 60,000 surgical procedure
240 before optical VTA stimulation inhibited the arousal responses and restoration of righting in 6/6 ChR
241 and physiologic evidence points to impaired arousal responses to hypercarbia and hypoxia, which ulti
243 is shows that the accumulation of endogenous arousal signals informs gaze-shift timing judgements.
244 up mechanisms in conditions of attention and arousal.SIGNIFICANCE STATEMENT The pedunculopontine tegm
246 centers involved in the control of cortical arousal state, including the noradrenergic locus coerule
248 photometry in freely behaving mice and found arousal-state-dependent alterations in VTA dopaminergic
250 s occurred when animals were in high and low arousal states as measured by animal speed and pupillome
252 d motor systems, and its ability to regulate arousal states puts the PPN in a key position to modulat
253 uit properties underlying neuromodulation of arousal states such as sleep and wakefulness remain uncl
254 eted as a continuous processing of competing arousal states, yielding selective amplification and inh
263 grey (PAG) lies at the heart of the defence-arousal system and its integrity is paramount to the exp
264 n is an important component of the ascending arousal system and may be a key site through which the o
265 the 'set point' for mobilizing their limbic arousal system has been elevated-an interpretation consi
266 GNIFICANCE STATEMENT The acetylcholine (ACh) arousal system in the brain is needed for robust attenti
267 lamus and is a critical part of an ascending arousal system that controls the firing mode of thalamic
268 alamus comprises a key node of the ascending arousal system, but the cell types underlying this are n
270 ) is an essential component of the ascending arousal systems and may be a key site through which the
271 othesized that phasic responses of brainstem arousal systems are a significant source of this variabi
272 terface between the Nmu system and brainstem arousal systems that represents a novel wake-promoting p
273 sitional tracking of individual animals, and arousal threshold can be determined by vibrational stimu
274 sibility and muscle function, loop gain, and arousal threshold), and baseline predictors of which pat
278 c inhibition of PBel(CGRP) neurons prevented arousal to CO2, but not to an acoustic tone or shaking.
280 Psychophysiological insomnia (PI) includes arousal to sleep-related stimuli (SS), which can be trea
283 s opposed to either a more general emotional arousal value or a more specific emotion category distin
284 ression exemplars are attributed valence and arousal values that are uniquely and naturally uncorrela
285 ed models, we find that Nmu does not promote arousal via the hypothalamic-pituitary-adrenal axis, but
289 ceptual bias, uncertainty, and physiological arousal we found that arousing disgust cues modulated th
290 and animal locomotive speed as indicators of arousal, we found that 3-5 Hz oscillations were not rest
291 etween HA neuron excitability and behavioral arousal, we investigated both the electrophysiological d
292 unctional constructs of negative valence and arousal were applied to the EPDS dimensions that reflect
294 These findings indicate that at moderate arousal, which has been associated with optimal noradren
295 to affective monitoring, competition raises arousal, which, when sustained, results in negative affe
298 alience network monotonically increases with arousal, while cohesion of this network with the executi
299 hesized that dexmedetomidine-induced altered arousal would manifest with reduced functional connectiv
300 uously involved in motivation and behavioral arousal, yet the contributions of other DA populations t
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