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1 ne of the pair must be knocked out to confer arrhythmicity.
2  reliably predicted the spontaneous onset of arrhythmicity.
3 ter at higher levels of ZTL, to the point of arrhythmicity.
4 elevated levels of TIM and approximately 50% arrhythmicity.
5  correlated with altered periods or apparent arrhythmicity.
6 r ablation of just six DH44+ PI cells causes arrhythmicity.
7 ted knockdown of CG17282 produced behavioral arrhythmicity and long periods and high levels of hypoph
8 nt background restored LL-induced behavioral arrhythmicity and wild-type neuronal activity patterns,
9 alizes to nuclei produce dominant behavioral arrhythmicity, and (2) constitutively nuclear PER repres
10 adian periods at lower expression levels and arrhythmicity at higher levels.
11 hrony among these regions does not result in arrhythmicity because core body temperature and explorat
12 (LL) can induce behavioral and physiological arrhythmicity by desynchronizing clock cells in the SCN.
13                            After the initial arrhythmicity, cell populations are then desynchronized.
14                                Although this arrhythmicity could be caused by a loss of light entrain
15  Mop3), the single knockout of which confers arrhythmicity, despite the presence of its paralog, Bmal
16         elf3 mutations cause light-dependent arrhythmicity, elongated hypocotyls, and early flowering
17 PDF-expressing LNv causes initial behavioral arrhythmicity followed by gradual long-term emergence of
18 e age-related transition from rhythmicity to arrhythmicity for each parameter occurs unpredictably, w
19 ient, or calcium signaling lead to circadian arrhythmicity in adult behaving Drosophila.
20 rsistent oscillation in constant conditions, arrhythmicity in constant light, resetting by brief ligh
21  role of the engraftment patterns of MSCs on arrhythmicity in controllable in vitro models is investi
22 n in the clarity of the rhythm, and apparent arrhythmicity in some tubes.
23 nd causes long-period behavioral rhythms and arrhythmicity, indicating that cycling vri is required f
24 F in the circadian control circuit overcomes arrhythmicity induced by pdf(01) null mutation, most lik
25 reatretain-->ment counteracted the circadian arrhythmicity induced by the DPS protocol: only 6% of re
26                                          The arrhythmicity is transient and is followed by the resump
27                                         This arrhythmicity might result from a requirement for PKA ac
28                       Despite the behavioral arrhythmicity, molecular oscillations are still detectab
29 ture changes, we found that population-level arrhythmicity occurs when certain perturbations cause st
30                     Lesions of the SCN cause arrhythmicity of locomotor activity, and transplants of
31                           Adult hMSCs affect arrhythmicity of neonatal rat cardiomyocyte cultures by
32 ish the FRQ self-association and lead to the arrhythmicity of the overt rhythm.
33 d by constant light, resulting in behavioral arrhythmicity or 'splitting' of rhythms of activity and
34  circadian period, and overexpression causes arrhythmicity, suggesting a more comprehensive role for
35  neurons alters tim expression and increases arrhythmicity under standard constant darkness (DD) cond
36 rony within clock nuclei is disrupted during arrhythmicity, whereas neurons in the left and right clo
37 hened significantly, ultimately resulting in arrhythmicity, while blue light-based phase shifts show
38 mozygous viable adults, as well as molecular arrhythmicity, with constitutively high levels of PER pr
39 city between SCN-lesioned individuals but to arrhythmicity within individuals.

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