コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 thylation might play an important role in AF arrhythmogenesis.
2 ty of ion channel subunit genes that promote arrhythmogenesis.
3 acute stress may have served to promote this arrhythmogenesis.
4 polarizations, known cellular mechanisms for arrhythmogenesis.
5 arization alternans (RA) are associated with arrhythmogenesis.
6 coupling, which is an important mechanism of arrhythmogenesis.
7 (LQTS), a condition associated with enhanced arrhythmogenesis.
8 factor in triggering cardiac enlargement and arrhythmogenesis.
9 function, peripheral vascular resistance and arrhythmogenesis.
10 tribution of the autonomic nervous system to arrhythmogenesis.
11 nfluence electrophysiological properties and arrhythmogenesis.
12 cal link between contractile dysfunction and arrhythmogenesis.
13 physiology and the fundamental mechanisms of arrhythmogenesis.
14 nation of fibrillation without shock-induced arrhythmogenesis.
15 sms of dyslipidaemias contribute directly to arrhythmogenesis.
16 CO pretreatment was confounded by refractory arrhythmogenesis.
17 sion observed and will have implications for arrhythmogenesis.
18 divergent effects on atrial and ventricular arrhythmogenesis.
19 findings may have important implications in arrhythmogenesis.
20 ogy, which could favor a matrix conducive to arrhythmogenesis.
21 tion potential recovery may provide clues to arrhythmogenesis.
22 ensin II-induced gap junction remodeling and arrhythmogenesis.
23 substrate for sex- and age-dependent cardiac arrhythmogenesis.
24 to predict long QT syndrome prolongation and arrhythmogenesis.
25 c nervous system modulation by RDN on atrial arrhythmogenesis.
26 in repolarization, a substrate that promotes arrhythmogenesis.
27 tion system (CCS) development, and increased arrhythmogenesis.
28 system is an important determinant of atrial arrhythmogenesis.
29 the molecular mechanisms associated with AF arrhythmogenesis.
30 one-dependent predisposition to postischemia arrhythmogenesis.
31 s remodeling on atrial electrophysiology and arrhythmogenesis.
32 modulation of cardiac electrophysiology and arrhythmogenesis.
33 onsequent wavebreak, have been implicated in arrhythmogenesis.
34 of ROS and whether ROS played a role in the arrhythmogenesis.
35 nimals, acute blockade of O-GlcNAc inhibited arrhythmogenesis.
36 cts may play a critical role in EAD-mediated arrhythmogenesis.
37 s to study electrophysiologic remodeling and arrhythmogenesis.
38 n of ion channels as a mechanism for cardiac arrhythmogenesis.
39 ral architecture and is a key contributor to arrhythmogenesis.
40 istent INa and EADs) promotes reflection and arrhythmogenesis.
41 nregulated in heart failure and functions in arrhythmogenesis.
42 er to elucidate the role of I(K1) in cardiac arrhythmogenesis.
43 atherosclerosis, thrombosis, vasomotion, and arrhythmogenesis.
44 ance, and enhanced susceptibility to induced arrhythmogenesis.
45 ssion of Cx40 have been implicated in atrial arrhythmogenesis.
46 sympathetic response that may play a role in arrhythmogenesis.
47 CG) as an additional important biomarker for arrhythmogenesis.
48 of RyR2 and how this contributes to cardiac arrhythmogenesis.
49 r may reduce SR Ca content and contribute to arrhythmogenesis.
50 s the cellular basis for QT prolongation and arrhythmogenesis after reversal of the direction of acti
51 optimized platform in a tachycardic model of arrhythmogenesis, an aspect of cardiac electrophysiology
53 itochondrial metabolism in the mechanisms of arrhythmogenesis and contractile dysfunction in cardiac
55 n prior to challenge protected mdx mice from arrhythmogenesis and death, while mdx:utr mice displayed
56 l cell arrays, the tissue's vulnerability to arrhythmogenesis and dynamic behaviour of re-entrant exc
57 To determine the role of IP3R2 in atrial arrhythmogenesis and ECC, we generated IP3R2-deficient m
58 of RyR2s by ROS contributes to CG-dependent arrhythmogenesis and examine the relevant sources of ROS
60 ases of cardiac sympathetic outflow, cardiac arrhythmogenesis and impairment in cardiac function in r
61 ave been postulated to contribute to cardiac arrhythmogenesis and injury during ischemia/reperfusion.
62 shown by WT hearts, these findings attribute arrhythmogenesis and its modification by flecainide and
66 channels at S2814 plays an important role in arrhythmogenesis and sudden cardiac death in mice with h
67 Myocardial ischemia, an important factor for arrhythmogenesis and sudden death, may affect the induci
69 rovide important insights into mechanisms of arrhythmogenesis and suggest that conditional lineage ab
71 eralization contributes significantly to DMD arrhythmogenesis and that selective inhibition may provi
73 istic link between SQT3 mutations and atrial arrhythmogenesis, and potential ion channel targets for
74 ects of anger and other negative emotions on arrhythmogenesis, are important areas of future investig
75 s in myocardial structure, can contribute to arrhythmogenesis around the region of myocardial injury.
77 -wave alternans, known to be associated with arrhythmogenesis, as well as increasing inducibility of
78 Neural dysregulation is central to atrial arrhythmogenesis associated with endurance exercise trai
79 xcess of PCs promotes triggered activity and arrhythmogenesis at lower levels of stress than VMs.
80 ncoupling induced by acute ischemia enhances arrhythmogenesis, but it may also protect the heart by l
81 ent alternans (CTA) has a recognized role in arrhythmogenesis, but its origin is not yet fully unders
82 vity is known to be important in ventricular arrhythmogenesis, but there is little information on the
84 To date, information on the role of NCX in arrhythmogenesis derived from models with increased NCX
86 These findings suggest that stress-related arrhythmogenesis due to the WTC tragedy was not restrict
92 Papillary muscles have been implicated in arrhythmogenesis; however, their role in post-infarction
93 ved the way for an improved understanding of arrhythmogenesis in a wide spectrum of life-threatening
94 ors (RyR2s) is hypothesized to contribute to arrhythmogenesis in AF, but the molecular mechanisms are
96 wave initiation, potentially accounting for arrhythmogenesis in CPVT linked to mutations in CASQ2.
98 o contribute to the increased propensity for arrhythmogenesis in diseased myocardium, although a caus
103 ing the process of wavefront propagation and arrhythmogenesis in human atria, technical concerns and
106 e we investigated mechanisms of KCNE3-linked arrhythmogenesis in Kcne3(-/-) mice using real-time qPCR
108 plasma membrane-related pathways involved in arrhythmogenesis in long QT syndrome, whereas proarrhyth
110 hether ion channel remodeling contributes to arrhythmogenesis in N-cadherin conditional knock-out (N-
112 s phenomenon may contribute significantly to arrhythmogenesis in patients with Brugada syndrome.
115 al heterogeneities and thus to contribute to arrhythmogenesis in the long QT and Brugada syndromes.
117 peri-infarct zone contributes to ventricular arrhythmogenesis in the postmyocardial infarction settin
118 Vm and the shortened APD95 could facilitate arrhythmogenesis in the presence of underlying ischemia.
119 pathways that may contribute to ventricular arrhythmogenesis in the settings of HF-associated remode
122 id-derived mediators that may play a part in arrhythmogenesis include phospholipids and leucotrienes
123 ingle-cell analysis revealed a substrate for arrhythmogenesis, including a complete absence of transi
124 analysis revealed an anatomic substrate for arrhythmogenesis, including a decrease and mislocalizati
127 ated by the fact that cardiac excitation and arrhythmogenesis involve the three-dimensional ventricul
128 ce of the autonomic nervous system in atrial arrhythmogenesis is also supported by circadian variatio
130 A popular biological model used to study arrhythmogenesis is the cultured cardiac cell monolayer,
132 se higher doses of digoxin may predispose to arrhythmogenesis, lower dose digoxin should be considere
133 that Cl- channels may contribute to cardiac arrhythmogenesis, myocardial hypertrophy and heart failu
135 ic substrates that underlie pathogenesis and arrhythmogenesis of arrhythmogenic right ventricular car
145 e essential for mechanistic understanding of arrhythmogenesis, since cells are subjected to rapid per
146 tress triggers myocardial ischemia, promotes arrhythmogenesis, stimulates platelet function, and incr
147 ation is a critical component of ventricular arrhythmogenesis that can be noninvasively assessed with
148 yanodine receptors (RyR2) has been linked to arrhythmogenesis, the molecular mechanisms triggering re
149 rcoplasmic reticulum (SR) has been linked to arrhythmogenesis, the role played by SR Ca(2+) uptake re
150 mechanism suggests that fibroblasts promote arrhythmogenesis through direct electrical interactions
151 gen species (ROS), which could contribute to arrhythmogenesis through redox modification of cardiac r
152 immunohistochemistry, and the potential for arrhythmogenesis was examined using programmed electrica
153 be the relationship between excitability and arrhythmogenesis, we explored conditions for new wavelet
154 m our understanding of mechanisms underlying arrhythmogenesis, we extended this approach, identifying
155 annelopathy on cardiac electrophysiology and arrhythmogenesis, we generated a murine model of ODDD by
156 e spatial organization of repolarization and arrhythmogenesis were determined in a surrogate model of
157 tions produced multiple mechanisms of atrial arrhythmogenesis, with significant differences between t
158 rtant effect on atrial electrophysiology and arrhythmogenesis, with the overall response depending on
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。