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1 2 (PKP2), is the most common causal gene for arrhythmogenic cardiomyopathy.
2 and drug screens for effective therapies in arrhythmogenic cardiomyopathy.
3 in is the first sarcomeric protein linked to arrhythmogenic cardiomyopathy.
4 ed phenotype of an early onset biventricular arrhythmogenic cardiomyopathy.
5 es has prompted proposal of the broader term arrhythmogenic cardiomyopathy.
6 cause loss of junctional Pg is a hallmark of arrhythmogenic cardiomyopathy.
7 excess cardiac fibroadipocytes, as in human arrhythmogenic cardiomyopathy.
8 ause hypertrophic, dilated, restrictive, and arrhythmogenic cardiomyopathies.
9 and the development of high-risk dilated and arrhythmogenic cardiomyopathies.
10 of titin's spring region is associated with arrhythmogenic cardiomyopathy, a disease characterized b
11 Desmoglein 2 gene (DSG2) mutations cause arrhythmogenic cardiomyopathy (AC) in human and transgen
18 milies, including hypertrophic, dilated, and arrhythmogenic cardiomyopathies and inherited arrhythmia
19 r early cardiac arrhythmias in patients with arrhythmogenic cardiomyopathy and cardiocutaneous syndro
20 is review highlights current knowledge about arrhythmogenic cardiomyopathy and considers clinical, pa
21 ith Naxos disease, which is characterized by arrhythmogenic cardiomyopathy and the cutaneous disorder
23 as sarcomeric, force generation disease; and arrhythmogenic cardiomyopathy as desmosome, cell junctio
24 Cardiomyocyte ILK deletion produces a lethal arrhythmogenic cardiomyopathy associated with important
25 rdium has revealed mechanistic insights into arrhythmogenic cardiomyopathy but cardiac samples are di
26 inally hailed as a putative gold standard in arrhythmogenic cardiomyopathy, cardiovascular magnetic r
27 pping phenotype of dilated and left-dominant arrhythmogenic cardiomyopathies complicated by frequent
29 anish family with inherited left ventricular arrhythmogenic cardiomyopathy/dysplasia and a high incid
30 nts presented data consistent with inherited arrhythmogenic cardiomyopathy/dysplasia phenotype with v
31 auses predominant inherited left ventricular arrhythmogenic cardiomyopathy/dysplasia with a high inci
32 histochemistry was compatible with inherited arrhythmogenic cardiomyopathy/dysplasia, and the functio
34 magnetic resonance assessment include proven arrhythmogenic cardiomyopathy in the family, unexplained
40 anding of the pathogenesis of this aspect of arrhythmogenic cardiomyopathy may shed light onto the ba
41 human PKP2 associate with a life-threatening arrhythmogenic cardiomyopathy, often of right ventricula
43 kedly diminished in buccal mucosa cells from arrhythmogenic cardiomyopathy patients with known desmos
44 on of Jup in cardiomyocytes in mice leads to arrhythmogenic cardiomyopathy similar to Naxos disease i
45 lar junction proteins are the major cause of arrhythmogenic cardiomyopathy, whereas recessive mutatio
46 re responsible for a subset of patients with arrhythmogenic cardiomyopathy who exhibit cardiac arrhyt
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