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1 opathies (i.e., hypertrophic cardiomyopathy, arrhythmogenic right ventricular cardiomyopathy).
2 d to hypertrophic cardiomyopathy and none to arrhythmogenic right ventricular cardiomyopathy.
3 EPI) substrate modification in patients with arrhythmogenic right ventricular cardiomyopathy.
4 electric and histological features known for arrhythmogenic right ventricular cardiomyopathy.
5 S mutations were also found in patients with arrhythmogenic right ventricular cardiomyopathy.
6 attern can also be the first presentation of arrhythmogenic right ventricular cardiomyopathy.
7 n defects of desmin mutants, associated with arrhythmogenic right ventricular cardiomyopathy.
8 ypic heterogeneity seen within families with arrhythmogenic right ventricular cardiomyopathy.
9 overview of the molecular pathophysiology of arrhythmogenic right ventricular cardiomyopathy.
10 nderlie pathogenesis and arrhythmogenesis of arrhythmogenic right ventricular cardiomyopathy.
11 s and in human myocardium from patients with arrhythmogenic right ventricular cardiomyopathy.
12 ar tachycardia, 3; short QT syndrome, 1; and arrhythmogenic right ventricular cardiomyopathy, 23).
13 ic polymorphic ventricular tachycardia (4%), arrhythmogenic right ventricular cardiomyopathy (4%), an
15 in devastating skin blistering diseases and arrhythmogenic right ventricular cardiomyopathy, a heart
16 The results are discussed in the context of arrhythmogenic right ventricular cardiomyopathy, an inhe
17 pholamban R14del mutation causes dilated and arrhythmogenic right ventricular cardiomyopathies and is
18 fQRS has also been defined as a marker of arrhythmogenic right ventricular cardiomyopathy and Brug
19 e shocks in primary prevention patients with arrhythmogenic right ventricular cardiomyopathy and hype
20 The higher appropriate discharge rates in arrhythmogenic right ventricular cardiomyopathy and hype
22 cause life-threatening conditions including arrhythmogenic right ventricular cardiomyopathy, and des
23 physiology of inherited arrhythmias (such as arrhythmogenic right ventricular cardiomyopathy) are dis
24 rtrophy (LVH) and/or fibrosis (n = 59, 16%); arrhythmogenic right ventricular cardiomyopathy (ARVC) (
33 ould expose a latent electrical substrate of arrhythmogenic right ventricular cardiomyopathy (ARVC) i
45 agnetic resonance (CMR) in the evaluation of arrhythmogenic right ventricular cardiomyopathy (ARVC) i
49 Exercise has been proposed as a trigger for arrhythmogenic right ventricular cardiomyopathy (ARVC) p
50 akoglobin (JUP) gene have been identified in arrhythmogenic right ventricular cardiomyopathy (ARVC) p
51 rugs in a rigorously characterized cohort of arrhythmogenic right ventricular cardiomyopathy (ARVC) p
52 possibility of cardiomyopathy, specifically arrhythmogenic right ventricular cardiomyopathy (ARVC).
53 k Force Criteria (rTFC) for the diagnosis of arrhythmogenic right ventricular cardiomyopathy (ARVC).
54 on (TWI), may create diagnostic overlap with arrhythmogenic right ventricular cardiomyopathy (ARVC).
55 osomal proteins are the most common cause of arrhythmogenic right ventricular cardiomyopathy (ARVC).
56 his study was to define the genetic basis of arrhythmogenic right ventricular cardiomyopathy (ARVC).
57 malities precede overt structural disease in arrhythmogenic right ventricular cardiomyopathy (ARVC).
58 onischemic dilated cardiomyopathy [NICM], 15 arrhythmogenic right ventricular cardiomyopathy [ARVC])
59 rdioverter defibrillator (ICD) in males with arrhythmogenic right ventricular cardiomyopathy caused b
60 Arrhythmogenic right ventricular cardiomyopathy, charact
61 ns were described in patients with inherited arrhythmogenic right ventricular cardiomyopathy/dysplasi
62 Heart failure (HF) prevalence in arrhythmogenic right ventricular cardiomyopathy/dysplasi
63 cardiomyopathy, dilated cardiomyopathy, and arrhythmogenic right ventricular cardiomyopathy/dysplasi
64 ers for risk stratification in patients with arrhythmogenic right ventricular cardiomyopathy/dysplasi
65 EVM in a consecutive series of patients with arrhythmogenic right ventricular cardiomyopathy/dysplasi
66 valence of "background genetic noise" in the arrhythmogenic right ventricular cardiomyopathy/dysplasi
67 posed criteria for the clinical diagnosis of arrhythmogenic right ventricular cardiomyopathy/dysplasi
68 ial ventricular tachycardia (VT) ablation in arrhythmogenic right ventricular cardiomyopathy/dysplasi
70 Thus, the hypothesis of an exercise-induced arrhythmogenic right ventricular cardiomyopathy has to b
71 ular magnetic resonance in the evaluation of arrhythmogenic right ventricular cardiomyopathy have cha
72 ocardial infarction, dilated cardiomyopathy, arrhythmogenic right ventricular cardiomyopathy, hypertr
73 ablation of ventricular tachycardia (VT) in arrhythmogenic right ventricular cardiomyopathy improves
74 9 (53%), valvular heart disease in 34 (15%), arrhythmogenic right ventricular cardiomyopathy in 37 (1
77 Possible relevance to the pathogenesis of arrhythmogenic right ventricular cardiomyopathy is discu
78 and adjuvant EPI substrate ablation of VT in arrhythmogenic right ventricular cardiomyopathy is good.
79 xed cardiomyopathies occur infrequently; and arrhythmogenic right ventricular cardiomyopathy is rare.
81 se each (3%) of hypertrophic cardiomyopathy, arrhythmogenic right ventricular cardiomyopathy, long QT
82 With recognition of disease-causing genes in arrhythmogenic right ventricular cardiomyopathy, mutatio
84 hy (n=1), ischemic cardiomyopathy (n=1), and arrhythmogenic right ventricular cardiomyopathy (n=1).
85 had a diagnosis of cardiomyopathy, including arrhythmogenic right ventricular cardiomyopathy (n=3) an
86 We conclude adipocytes in arrhythmogenic right ventricular cardiomyopathy originat
87 at a Dsg2 mutant, V977fsX1006, identified in arrhythmogenic right ventricular cardiomyopathy patients
88 Arrhythmogenic right ventricular cardiomyopathy probands
89 cutive patients with Task Force criteria for arrhythmogenic right ventricular cardiomyopathy referred
90 was higher in nonischemic cardiomyopathy and arrhythmogenic right ventricular cardiomyopathy than in
92 that a mutation in alphaT-catenin linked to arrhythmogenic right ventricular cardiomyopathy, V94D, p
93 ole of genotyping in familial assessment for arrhythmogenic right ventricular cardiomyopathy was inve
94 for a possible primary electric syndrome or arrhythmogenic right ventricular cardiomyopathy were ana
95 hies such as hypertrophic cardiomyopathy and arrhythmogenic right ventricular cardiomyopathy, which a
96 harboring pathogenic mutations suffered from arrhythmogenic right ventricular cardiomyopathy with or
97 pathy, 15 nonischemic cardiomyopathy, and 14 arrhythmogenic right ventricular cardiomyopathy) with a
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