ライフサイエンスコーパス: arsenite

1 ic metalloids of arsenic (i.e., arsenate and arsenite).

2 ditions of severe stress induced with sodium arsenite.

3 s may explain the limited phloem mobility of arsenite.

4 Plants chemically reduce arsenate to arsenite.

5 n SGs following exposure to the stress agent arsenite.

6 M arsenite with a linear range up to 100 muM arsenite.

7 were both unstable and partially reduced to arsenite.

8 ation of the arsB mutant restored the MIC of arsenite.

9 resulted in a 16-fold increase in the MIC of arsenite.

10 rease, then decrease of monothioarsenate and arsenite.

11 s showed that arxA was strongly induced with arsenite.

12 to isolate cells with defective response to arsenite.

13 20 microm arsenate [As(V)] or 4 or 20 microm arsenite.

14 n iron plaque, were treated with arsenate or arsenite.

15 nder anaerobic conditions in the presence of arsenite.

16 ts are sensitive to higher concentrations of arsenite.

17 for the proatherogenic effects of inorganic arsenite.

18 sts fail to properly form SGs in response to arsenite.

19 ic accumulation and increased sensitivity to arsenite.

20 lectively neddylated at Lys85 in response to arsenite.

21 nic speciation due to its selectivity toward arsenite.

22 reducing the central arsenic atom (As(V)) to arsenite.

23 er pentavalent organoarsenicals or inorganic arsenite.

24 ollution of groundwater, which is largely by arsenite.

25 ed by exposing p53-knockdown HBECs to sodium arsenite (2.5 muM) for 16 weeks.

26 rew faster in the presence of high levels of arsenite (3 mM).

27 vations indicating that GADD34 is induced by arsenite, a thiol-directed oxidative stressor, in the ab

28 Exposure to arsenite also diminished the recruitment of BRCA1 and RA

29 Treatments with arsenite also led to a dose-dependent decrease in the le

30 Arsenite also recruited a TDP-43 kinase, casein kinase-1

31 Arsenite also stabilized GADD34 protein, slowing its deg

32 Sodium arsenite, an agent that induces oxidative stress, promot

33 e we have studied four stress agents: sodium arsenite, an oxidative agent; Gramicidin, eliciting K(+)

34 sed sensitivities of 0.91 +/- 0.07 mV/mM for arsenite and 0.98 +/- 0.02 mV/mM for arsenate.

35 ted limits of detection (LODs) of 13 muM for arsenite and 132 muM for arsenate.

36 ately we identified 248 mutants sensitive to arsenite and 5 mutants resistant to arsenite exposure.

37 ansgenic lines showed increased tolerance to arsenite and arsenate and a greater capacity for arsenat

38 ion, desorption, and readsorption of aqueous arsenite and arsenate by CuO-NP.

39 We report the first example of arsenite and arsenate removal from water by incorporatio

40 The kinetics and efficiencies of arsenite and arsenate removal from water were evaluated

41 rs emitting bioluminescence as a response to arsenite and arsenate was applied during a field campaig

42 worms from F0 and F1 generations accumulated arsenite and arsenate when F0 L4 larvae were exposed to

43 -through fractionation of inorganic arsenic (arsenite and arsenate) in environmental solids in combin

44 arsenate, and compared it to the sorption of arsenite and arsenate, in suspensions containing 2-line

45 ed in 8- and 4-fold reduction in the MICs of arsenite and arsenate, respectively, and complementation

46 two genes, contributes to the resistance to arsenite and arsenate.

47 cell, yielding a self-powered biosensor for arsenite and arsenate.

48 significantly different decay rates between arsenite and control treatment.

49 ls were further increased by the addition of arsenite and GSH to the medium, indicating that GGCT2;1

50 stress and found that the oxidative stressor arsenite and heat shock-activated stress responses evide

51 f the selective intracellular recognition of arsenite and its pumping out from the cell.

52 ta indicate that signaling events induced by arsenite and oxidative stress may regulate LRRK2 functio

53 was impaired by trivalent arsenicals such as arsenite and phenylarsine oxide.

54 Arsenite and phosphite were confirmed to be the best cat

55 es in the presence of the reduced precursors arsenite and sulfide.

56 that PARP-1 is a direct molecular target of arsenite and that arsenite interacts selectively with zi

57 The kinetic extraction profiles of arsenite and total inorganic arsenic are obtained for ea

58 Arsenite and total inorganic arsenic in each subfraction

59 in situ applicability for treatment of both arsenites and arsenates, and contrary to all known compe

60 s (hydrogen peroxide), a heavy metal stress (arsenite) and an amino acid analogue (azetidine-2-carbox

61 onse to hypertonic stress, oxidative stress (arsenite), and treatment with recombinant angiogenin.

62 exposure of cells to the environmental toxin arsenite, and in a proteasome mutant, loss of Cuz1 enhan

63 53 whose steady-state levels were altered by arsenite, and of these, only 4 exhibited significantly d

64 ceroporin-9 (AQP9) is permeated by glycerol, arsenite, and other small, neutral solutes.

65 i linked to growth traits under heat stress, arsenite, and paraquat, the majority of which were best

66 ure to noncytotoxic concentrations of sodium arsenite, and we confirmed some of these changes using r

67 ANG inhibits protein synthesis and promotes arsenite- and pateamine A-induced assembly of stress gra

68 electron donor for reduction of arsenate to arsenite; and d) As has a high affinity for sulfhydryl g

69 rsenic (TUA): TUA1 was defined as the sum of arsenite, arsenate, monomethylarsonic acid, and dimethyl

70 efficiencies of inorganic oxoanions such as arsenite, arsenate, phosphite, phosphate, and borate is

71 luoride, persulphate, acetate, thiosulphate, arsenite, arsenate, sulphite, and iodide.

72 Our results also suggest arsenite as a general inhibitor for RING finger E3 ubiqu

73 'Photoarsenotrophy', the use of arsenite as an electron donor for anoxygenic photosynthe

74 On the other hand, arsenite As(III) is significantly adsorbed on goethite,

75 The reversible inhibition of laccase by arsenite (As(3+)) and arsenate (As(5+)) is reported for

76 of arsenate (As(V) ) generated by microbial arsenite (As(III) ) oxidation is poorly understood.

77 U-As(IMM), of urinary inorganic As species, arsenite (As(III)) and arsenate (As(V)), and their metab

78 mine drainage (AMD) and are most harmful as arsenite (As(III)) and hexavalent (Cr(VI)).

79 e (S(-II)) with NOM and its consequences for arsenite (As(III)) binding.

80 Resistance to arsenite (As(III)) by cells is generally accomplished by

81 lence and compound speciation, and inorganic arsenite (As(III)) compounds are the most toxic to human

82 robes biotransform both arsenate (As(V)) and arsenite (As(III)) into more toxic methylated metabolite

83 Oxidation of arsenite (As(III)) is a critical yet often weak link in

84 The redox chemistry of chromate (Cr(VI)) and arsenite (As(III)) on the iron oxyhydroxide, ferrihydrit

85 onmental degradation to more toxic inorganic arsenite (As(III)) that contaminates crops and drinking

86 Many microbes methylate inorganic arsenite (As(III)) to more toxic and carcinogenic methyl

87 Binding of arsenite (As(III)) to sulfhydryl groups (Sorg(-II)) play

88 ArsA, the catalytic subunit of the ArsAB arsenite (As(III)) translocating ATPase, is one of the f

89 accompanied by the simultaneous oxidation of arsenite (As(III)) was achieved using an electrochemical

90 us suspension of goethite in the presence of arsenite (As(III)) was investigated with X-ray absorptio

91 lucidated the molecular-scale interaction of arsenite (As(III)) with Fe(III)-NOM complexes under redu

92 Here we report that arsenite (As[III]) can induce both replication-dependent

93 methylation of As is catalyzed by the enzyme arsenite (As[III]) S-adenosylmethionine methyltransferas

94 I)] to less toxic and carcinogenic inorganic arsenite [As(III)] by C-As bond cleavage.

95 Arsenite [As(III)] can be oxidized to arsenate [As(V)] b

96 strate that Yap8 directly binds to trivalent arsenite [As(III)] in vitro and in vivo and that approxi

97 ability to rice plants, whereas oxidation of arsenite [As(III)] results in As immobilization.

98 Cyanidioschyzon sp. isolate 5508 oxidized arsenite [As(III)] to arsenate [As(V)], reduced As(V) to

99 Some arsenite [As(III)]-oxidizing bacteria exhibit positive c

100 onmental degradation to more toxic inorganic arsenite [As(III)].

101 Arsenite, as well as other agents, triggered relocalizat

102 and the valence state of inorganic arsenic (arsenite, As(III) vs. arsenate As(V)) can be modulated b

103 itions and mechanism(s) for the reduction of arsenite, As(III), by pyrite are incompletely understood

104 adsorption mechanism of arsenate, As(V), and arsenite, As(III), on MNPs by macroscopic adsorption exp

105 tion near-edge spectroscopy showed arsenate, arsenite, As-(GS)3, and As-PCs with varying ratios in va

106 at in many microorganisms genes essential to arsenite (AsIII) oxidation are located immediately adjac

107 Microbial arsenite (AsIII) oxidation forms a critical piece of the

108 t of barley (Hordeum vulgare) seedlings with arsenite (AsIII) rapidly induced physiological and trans

109 -MS)-for the detection of arsenic(III) ions (arsenite, AsO2(-)) in aqueous solution.

110 the root, facilitating efflux of arsenic as arsenite back into the soil to limit both its accumulati

111 o peptides with two cysteines, demonstrating arsenite binding selectivity.

112 Here, we report that arsenite binds to both CCHC DNA-binding zinc fingers of

113 etects bacterial biomethylation of inorganic arsenite both in vivo and in vitro with detection limits

114 within the PARP-1 zinc finger revealed that arsenite bound to peptides containing three or four cyst

115 specifically suppressed ER stress induced by arsenite but not tunicamycin.

116 wed that monothioarsenate is less toxic than arsenite, but more toxic than arsenate at concentrations

117 disassembled canonical SGs induced by sodium arsenite, but not those induced by hydrogen peroxide, le

118 of As(V)/AsSum (total combined arsenate and arsenite concentrations) (0.59-0.78), coupled with high

119 Furthermore, trivalent arsenite coordinated with all three cysteine residues as

120 our study unveiled, for the first time, that arsenite could alter epigenetic signaling by targeting t

121 Herein, we found that arsenite could bind directly to the RING finger domains

122 Herein, we found that arsenite could bind directly to the zinc fingers of Tet

123 xidation coupled to reduction of arsenate to arsenite could simply enhance abiotic desulfidation of t

124 t a variety of weak acids (silicate, borate, arsenite, cyanide, carbonate, and sulfide) cannot only b

125 oncluded that ArsR2 is most likely the major arsenite-dependent regulator of arr and ars operons in S

126 f beta-galactosidase is under control of the arsenite-derepressable arsR-promoter.

127 roducible response generating matrix for the arsenite detection at an ultratrace concentration in aqu

128 development of a whole-cell based sensor for arsenite detection coupling biological engineering and e

129 This finding was not unique to PARP-1; arsenite did not bind to a peptide representing the CCHH

130 Arsenic trioxide and sodium arsenite did not directly modify or inhibit the activity

131 on of the As-hyperaccumulator Pteris vittata arsenite efflux (PvACR3), on As tolerance, accumulation,

132 enite efflux, arsenate reduction followed by arsenite efflux and arsenite methylation.

133 (III)) by cells is generally accomplished by arsenite efflux permeases from Acr3 or ArsB unrelated fa

134 pressed the yeast (Saccharomyces cerevisiae) arsenite efflux transporter ACR3 into Arabidopsis to eva

135 plants, both the lsi2 mutant lacking the Si/arsenite efflux transporter Lsi2 and its wild-type culti

136 Known mechanisms include arsenite efflux, arsenate reduction followed by arsenite

137 sistance to arsenate is reduction coupled to arsenite efflux.

138 te DNA damage with a high-fat diet or sodium arsenite exacerbated adipocyte senescence and metabolic

139 )-(oxyhydr)oxides and that both arsenate and arsenite exclusively formed monodentate-binuclear ("brid

140 In arsenite-exposed cells, 186 probe set-identified transcr

141 g capacity of PARP-1 immunoprecipitated from arsenite-exposed cells.

142 , transgenerational reproductive toxicity by arsenite exposure and the underlying mechanisms in C. el

143 Our study demonstrates that maternal arsenite exposure causes transgenerational reproductive

144 e of C. elegans was significantly reduced by arsenite exposure in F0 and that this reduction in brood

145 aggregation and proteolysis after prolonged arsenite exposure, GADD34-bound CK1 catalyzed TDP-43 pho

146 transcription as well as protein levels upon arsenite exposure, indicating that p27 provides a negati

147 NK2/c-Jun- and HSF-1-dependent pathways upon arsenite exposure, which provides additional important m

148 00637 human skin fibroblast cells induced by arsenite exposure.

149 itive to arsenite and 5 mutants resistant to arsenite exposure.

150 d arsenate when F0 L4 larvae were exposed to arsenite for 24 h.

151 king water with 0, 250 ppb, or 25 ppm sodium arsenite for 5 wk and then challenged intratracheally wi

152 Some arsenite formed intermediately, which was subsequently a

153 uent generations (F1-F5) were cultured under arsenite-free conditions.

154 ts lacking HAC1 lose their ability to efflux arsenite from roots, leading to both increased transport

155 Irradiation of the arsenite/goethite under conditions where dissolved oxyge

156 ferent exogenous stressors, including sodium arsenite, heat, and hippuristanol.

157 trated to confer resistance to As(III) in an arsenite hypersensitive strain of Escherichia coli.

158 particulate matter (PM), i.e., the inorganic arsenite iAs(III) and arsenate iAs(V), and the methylate

159 toxic levels of arsenic, including inorganic arsenite (iAs3+, </= 5 muM), inorganic arsenate (</= 20

160 tINT2 in Xenopus laevis oocytes also induced arsenite import.

161 technique can determine the concentration of arsenite in a few min with a detection limit of 0.1 ppb

162 nges in mRNA stability in response to sodium arsenite in human fibroblasts.

163 proved that the intermediate accumulation of arsenite in the drainage channel is microbially catalyze

164 C1 therefore functions to reduce arsenate to arsenite in the outer cell layer of the root, facilitati

165 eover, GO-SH demonstrates selectivity toward arsenite in the presence of arsenate.

166 oring a possibility of the quantification of arsenite in the ultratrace concentration range, the Ru N

167 expression and activity are up-regulated by arsenite, in a manner dependent on activating transcript

168 As concentrations, the percentage of unbound arsenite increased in the vein and mesophyll of young ma

169 Cells exposed to 10muM sodium arsenite increased the stability of HIPK1 and HIPK2 prot

170 Similarly, in dopaminergic MN9D cells, arsenite induced the export of endogenous Nurr1, resulti

171 Consistently, arsenite-induced activation of JNK2/c-Jun and HSF-1 path

172 culture (SILAC) to assess quantitatively the arsenite-induced alteration of global kinome in human ce

173 However, the relevance of Tpl2 in arsenite-induced carcinogenesis and the underlying mecha

174 eatment, suggesting that Tpl2 is critical in arsenite-induced carcinogenesis.

175 Arsenite-induced dephosphorylation is accompanied by los

176 Here, we identify genes required for arsenite-induced ER stress response in a genome-wide RNA

177 r screen discovered several genes modulating arsenite-induced ER stress, including sodium-dependent n

178 ay, which is at least partially required for arsenite-induced ER stress.

179 el are assessed against experimental data of arsenite-induced genotoxic damage to human hepatocytes;

180 ibitor, flavopiridol, partially restored the arsenite-induced growth inhibition of human skin fibrobl

181 but not JNK1, led to dramatic inhibition of arsenite-induced Hsp27 and Hsp70 expression.

182 The majority of the arsenite-induced L-glutamate influx was via a high-affin

183 Here, we report that arsenite-induced oxidative stress differs from thapsigar

184 dylation promotes SG assembly in response to arsenite-induced oxidative stress.

185 d that the deletion of p50 (p50-/-) impaired arsenite-induced p53 protein expression, which could be

186 We also found that arsenite-induced phosphorylation of extracellular signal

187 dylatable SRSF3 (K85R) mutant do not prevent arsenite-induced polysome disassembly, but fails to supp

188 Furthermore, inhibition of Tpl2 reduced the arsenite-induced promoter activity of NF-kappaB and acti

189 ine norovirus (MNV) infection did not impact arsenite-induced SG assembly or G3BP1 integrity, suggest

190 l-time RT-PCR confirmed a major, significant arsenite-induced stabilization of the mRNA encoding delt

191 m target histone-3 are phosphorylated during arsenite-induced stress.

192 nin, but not related ribonucleases, inhibits arsenite-induced tiRNA production and translational arre

193 RNH1 enhances tiRNA production and promotes arsenite-induced translational arrest.

194 We have identified arsenite- inducible regulatory particle-associated prote

195 -1), encoded by a previously uncharacterized arsenite-inducible gene in budding yeast.

196 inger AN1-type domain 2a gene, also known as arsenite-inducible RNA-associated protein (AIRAP), was r

197 AIRAPL (arsenite-inducible RNA-associated protein-like) is an ev

198 icals, including arsenic trioxide and sodium arsenite, inhibited activation of the NLRP1, NLRP3, and

199 direct molecular target of arsenite and that arsenite interacts selectively with zinc finger motifs c

200 Enrichment cultures converted 63% of arsenite into methylated products, with dimethylarsinic

201 d-type background showed increased efflux of arsenite into the external medium.

202 roscope and useful to estimate the amount of arsenite ions in various water samples.

203 Arsenite is a well-known human carcinogen that especiall

204 a differential pulse voltammetric technique, arsenite is oxidized to arsenate leading to its quantita

205 were pursued with an oxidative agent (sodium arsenite), K-releasing agent (Gramicidin) and a metal io

206 ects of targeting telomeres with sodium meta-arsenite (KML001) (an agent undergoing early clinical tr

207 s, inositol transporters are responsible for arsenite loading into the phloem, the key source of arse

208 Rice (Oryza sativa) takes up arsenite mainly through the silicic acid transport pathw

209 Following arsenite maternal exposure of the F0 generation, subsequ

210 Thus, sodium arsenite may confer its cytotoxic effect partly through

211 ent study revealed, for the first time, that arsenite may exert its carcinogenic effect by targeting

212 te reduction followed by arsenite efflux and arsenite methylation.

213 a previously uncharacterized, human-specific arsenite methyltransferase (AS3MT) isoform (AS3MT(d2d3))

214 e (AS3MT) isoform (AS3MT(d2d3)), which lacks arsenite methyltransferase activity and is more abundant

215 We conclude that arsenite modification of mRNA stability is relatively un

216 ative to wild type, after exposure to sodium arsenite (NaAsO(2)).

217 studied the influence of heat shock, sodium arsenite (NaAsO2), cycloheximide (CHX) and Lipofectamine

218 osure to cadmium chloride (CdCl2) and sodium arsenite (NaAsO2).

219 Pregnant CD-1 mice were exposed to sodium arsenite [none (control), 10 ppb, or 42.5 ppm] in drinki

220 py indicated that As was mainly arsenate and arsenite, not As-bearing sulfides.

221 ular heme caused by the massive induction by arsenite of heme oxygenase mRNA (HMOX1; 68-fold increase

222 To investigate the impact of arsenite on PARP-1 zinc finger function, we measured the

223 When exposed to 25 muM arsenite or arsenate overnight, most inorganic arsenic w

224 ooded conditions, and rice plants exposed to arsenite or DMA(V) were grown to maturity in nonsterile

225 Here, we show that arsenite or H2O2-induced stresses promote loss of Ser(91

226 n FCV-infected cells that were stressed with arsenite or hydrogen peroxide.

227 senate respiratory reductase (ArrA) and (ii) arsenite oxidase (AoxB).

228 nes faecalis) that was an early isolate with arsenite oxidase activity.

229 A new arsenite oxidase clade, ArxA, represented by the haloalk

230 nature of the molybdenum active site of the arsenite oxidase from the Alphaproteobacterium Rhizobium

231 CIB 8687, the betaproteobacterium from which arsenite oxidase had its structure solved and the first

232 This "new" arsenite oxidase is referred to as ArxA and was identifi

233 Arsenite oxidase is thought to be an ancient enzyme, ori

234 he genetic identification of a "new" type of arsenite oxidase that fills a phylogenetic gap between t

235 ly arsenate respiratory reductase, ArrA, and arsenite oxidase, AioA (formerly referred to as AroA and

236 ere arxA is predicted to encode for the sole arsenite oxidase.

237 about the evolutionary relationships between arsenite oxidases (AoxB) and arsenate respiratory reduct

238 These results suggest that ArxA-type arsenite oxidases appear to be widely distributed in the

239 ces as distinct members of the ArxA clade of arsenite oxidases.

240 ated Mlg_0216 (ArxA) of MLHE-1 in reversible arsenite oxidation and arsenate reduction in vitro.

241 Arsenotrophy, growth coupled to autotrophic arsenite oxidation or arsenate respiratory reduction, oc

242 LHE-1, a chemolithoautotroph that can couple arsenite oxidation to nitrate reduction.

243 The role of arxA in photosynthetic arsenite oxidation was confirmed by disrupting the gene

244 um, resulting in the loss of light-dependent arsenite oxidation.

245 mlg_0216) was required for chemoautotrophic arsenite oxidation.

246 ax borkumensis (Na(V)Ab1p), and one from the arsenite oxidizer Alkalilimnicola ehrlichei (Na(V)Ae1p).

247 acNaV derived from NaVAe1, a BacNaV from the arsenite oxidizer Alkalilimnicola ehrlichei found in Mon

248 L15 is an alphaproteobacterium isolated from arsenite-oxidizing biofilms whose draft genome contains

249 ation studies using E. coli or C. glutamicum arsenite permease mutants clearly show that CgAcr3-1 is

250 Members of the Acr3 family of arsenite permeases confer resistance to trivalent arseni

251 sulfur, suggesting that As may be stored as arsenite-phytochelatin complexes.

252 Arsenite preconcentrates onto the Ru surface just by dip

253 hat CgAcr3-1 is an antiporter that catalyzes arsenite-proton exchange with residues Cys129 and Glu305

254 treatment of cells with puromycin or sodium arsenite, reagents that arrest translation, also resulte

255 we report that the zinc finger protein Ars2 (arsenite-resistance protein 2; also known as Srrt) is ex

256 Here, we show that menin interacts with arsenite-resistant protein 2 (ARS2), a component of the

257 ne or thioredoxin proteins by copper (II) or arsenite, respectively, provided further support for the

258 40 in vitro and in cells, and treatment with arsenite resulted in substantially impaired H2B ubiquiti

259 volatilization can be mediated by the enzyme arsenite S-adenosylmethionine methyltransferase (ArsM) o

260 Thus formed Ru NPs have the arsenite selective surface and conducting core that is i

261 We analyzed the proteins corresponding to arsenite-sensitive mutants and determined that they belo

262 n a proteasome mutant, loss of Cuz1 enhances arsenite sensitivity.

263 eover, cells induced to undergo apoptosis by arsenite show increased R-CRT on their cell surface.

264 urface just by dipping the RuNPs/GC into the arsenite solution as it interacts chemically with Ru NPs

265 mpetitive effect of silicate on arsenate and arsenite sorption increased with increasing silicate pre

266 polymerization was slowest, was arsenate and arsenite sorption not affected by the presence of silica

267 icate surface polymerization on arsenate and arsenite sorption was studied by use of hematite pre-equ

268 Highly selective separation of arsenite species [As(III)] was achieved by chelation wit

269 erall changes in Ago2-mRNA interactions upon arsenite stress by cross-linking immunoprecipitation (CL

270 nscript to be translationally induced during arsenite stress conditions.

271 Arsenite stress quickly and reversibly decreased asymmet

272 Arsenite stress stimulates LRRK2 self-association and as

273 tress granule breakdown during recovery from arsenite stress, indicating a possible role for hYVH1 in

274 sporter, LAT1, suppressed mTOR activation by arsenite, supporting a role for these transporters in mo

275 When exposed to arsenite, the endodermis was again a site of accumulatio

276 nd seed arsenic concentrations when fed with arsenite through the leaves.

277 ed arsenic concentrations in plants fed with arsenite through the roots, relative to wild-type plants

278 Results suggest an effective oxidation of arsenite to arsenate on the surface of CuO-NP.

279 by coupling the intracellular recognition of arsenite to the generation of an electrochemical signal.

280 0.44, 0.74, 0.15, 0.17 and 0.67 ng L(-1) for arsenite, total inorganic, mono-, dimethylated As and tr

281 apped onto a protein interactome to identify arsenite-toxicity-modulating networks.

282 a PERK-independent eIF2alpha kinase through arsenite treatment and is independent of activating tran

283 response to hyperosmolarity, heat shock, and arsenite treatment but rapidly dephosphorylated after ox

284 in and accumulates in stress granules during arsenite treatment of human cells.

285 reversible: oxidative stress resulting from arsenite treatment transformed large IBs into a scatteri

286 BP1 co-localize to stress granules following arsenite treatment, but not during mitosis.

287 expression and PGE(2) production induced by arsenite treatment, suggesting that Tpl2 is critical in

288 on also occurs in cells during heat-shock or arsenite treatment, when poly-ubiquitinated proteins acc

289 in steady-state ALAS1 mRNA levels seen after arsenite treatment.

290 appaB and AP-1 are downstream transducers of arsenite-triggered Tpl2.

291 ry reduction pathway in response to elevated arsenite under anaerobic conditions.

292 Thioarsenates form from arsenite under sulfate-reducing conditions, e.g., in ric

293 Arsenite was present only as a minor species (3-5%) in t

294 Here, treatment of human erythrocytes with arsenite was shown to induce the uptake of L-glutamate a

295 anism against toxic arsenic species, such as arsenite, which consists of the selective intracellular

296 d biosensor has a detection limit of ~40 muM arsenite with a linear range up to 100 muM arsenite.

297 ed as an intermediate by abiotic reaction of arsenite with sulfide.

298 hich grows by coupling arsenate reduction to arsenite with the oxidation of sulfide to sulfate.

299 onothioarsenate forms by abiotic reaction of arsenite with zerovalent sulfur.

300 was generally lower compared to arsenate and arsenite, with the exception of the near instantaneous a