ライフサイエンスコーパス: arterial

1 Intra-arterial administration of lactic acid (to simulate exer

2 reatment options, such as systemic and intra-arterial administration of therapeutics ( 3 - 5 ).

3 te, therefore, that chronological resistance arterial aging is a prominent factor leading to weakened

4 esized that subjects with CKD have increased arterial and cellular inflammation, reflected by (18)F-f

5 agulation is not necessary for grade 1 and 2 arterial and grade 1 venous thrombosis.

6 esults in a greater improvement of coronary, arterial, and myocardial function than TNF-alpha inhibit

7 cipates pivotally in many diseases including arterial aneurysms.

8 e of less than 1% of all selective bronchial arterial angiograms.

9 ty during mental stress, as well as impaired arterial baroreflex sensitivity (BRS).

10 Arterial blood CO2 tension when increased by 25 mm Hg ca

11 by the level of impaired O2 extraction from arterial blood during peak exercise.

12 The arterial blood flow that underlies the stigmata rarely i

13 In IUGR baboons there was increased carotid arterial blood flow velocity during late systole and dia

14 echanically ventilated patients may not have arterial blood gas measurements available at relevant ti

15 Toward the end of each phase, we measured arterial blood gases, inspiratory effort, and work of br

16 rinsulinemic (4x basal) hyperglycemic clamp (arterial blood glucose 146 +/- 2 mg/dL) with portal GLC

17 lin is a ubiquitous peptide that can elevate arterial blood pressure (ABP) yet understanding of the m

18 Mean velocity index based on arterial blood pressure did not reach statistical signif

19 EY POINTS: Dysfunctions in CNS regulation of arterial blood pressure lead to an increase in sympathet

20 st compressions for >/=1 minute and invasive arterial blood pressure monitoring before and during CPR

21 vity, adrenal sympathetic nerve activity and arterial blood pressure whereas equi-osmotic mannitol/so

22 by peak exercise cardiac power output (mean arterial blood pressure x cardiac output) and functional

23 uctuations of cerebral perfusion pressure or arterial blood pressure.

24 Arterial blood samples were drawn for arterial input fun

25 Full tracer kinetic models require arterial blood sampling and dynamic image acquisition.

26 culation, a dynamic (18)F-FHNP PET scan with arterial blood sampling was acquired from rats treated w

27 underwent 180-min PET with (18)F-AV-1451 and arterial blood sampling.

28 min after bolus injection of (18)F-T807 with arterial blood sampling.

29 Sufficient blood flow to tissues relies on arterial blood vessels, but the mechanisms regulating th

30 nd B-type NP, regulate fluid homeostasis and arterial BP through renal actions involving increased GF

31 development and is maintained postnatally at arterial branch sites.

32 alized arterial calcification of infancy and arterial calcification due to CD73 deficiency.

33 development in individuals with generalized arterial calcification of infancy (GACI) due to loss-of-

34 rs, with phenotypic overlap with generalized arterial calcification of infancy and arterial calcifica

35 After arterial cannulation, the specimens were perfused using

36 d pressure (BP) was directly measured by the arterial catheter and found 13.8% higher in HS vs Cont r

37 aroscopic liver lobe resection combined with arterial catheter blood withdrawal to achieve a sustaine

38 thetized and ABP was monitored via a femoral arterial catheter.

39 CLI was induced by external iliac arterial cauterization in 10-12-month-old mice.

40 The ability to noninvasively assess arterial CD206+ macrophages may lead to improved underst

41 nts treated predominantly with primary intra-arterial chemotherapy.

42 o develop bone tumors when inoculated in the arterial circulation with human prostate cancer (PCa) ce

43 Although small changes in arterial CO2 are particularly potent to change CBF (1 mm

44 ly potent to change CBF (1 mmHg variation in arterial CO2 changes CBF by 3%-4%), the coupling mechani

45 Prospectively targeted arterial CO2 has the capability to evolve as an alternat

46 tilation was reduced (P < 0.001) at constant arterial CO2 tension and pH (P = 0.27 and P = 0.23, resp

47 ow ceases, the reservoir pressure related to arterial compliance and the excess pressure caused by wa

48 Pulmonary arterial compliance did not independently predict outcom

49 Recent studies have suggested that pulmonary arterial compliance may also predict prognosis in PAH.

50 iables during follow-up, including pulmonary arterial compliance, after initial management in PAH.

51 troke, transient ischemic attack, peripheral arterial complication, and cardiac arrhythmia), as well.

52 Consequently, arterial conduits other than the left internal thoracic

53 edge, a new multiscale mathematical model of arterial contractility accounting for structural and fun

54 Changes in volumes, strain, and ventricular-arterial coupling were consistently associated with conc

55 Tracer uptake was quantified on arterial cryosections using autoradiography and compared

56 me chronic and non-resolving and can lead to arterial damage and thrombosis-induced organ infarction.

57 nce interval [CI]: 1.82 to 2.29), peripheral arterial disease (HR: 1.95; 95% CI: 1.72 to 2.21), unher

58 a to lower limbs in patients with peripheral arterial disease (PAD).

59 al arterial revascularization for peripheral arterial disease and to assess whether readmission risk

60 (smoking, diabetes mellitus, and peripheral arterial disease).

61 rior stroke/transient ischemic attack, 6.5%; arterial disease, 15.9%; all CHADS-VASc risk factors wer

62 ADA HbA1c clinical categories for peripheral arterial disease, and 0.683 for ADA fasting glucose conc

63 d CHD, stroke, heart failure, and peripheral arterial disease.

64 nd surgical revascularization for peripheral arterial disease.

65 hemic heart disease implying a role in human arterial disease.

66 ography, which was followed by transcatheter arterial embolization (TAE) of the BAA and of the pathol

67 can be considered as an alternative to trans-arterial embolization when the pseudoaneurysm cannot be

68 The cell-specific deletion of CXCR4 in arterial endothelial cells (n=12-15) or SMCs (n=13-24) m

69 Vulnerability of pulmonary arterial endothelial cells (PAEC) to apoptosis was incre

70 Here, we report the derivation of arterial endothelial cells from human pluripotent stem c

71 Arterial endothelial cells were robustly generated from

72 r alpha) in circulating monocytes, pulmonary arterial endothelial cells, and also activated B cells.

73 e Ca(2+) -activated K(+) channels present in arterial endothelium to generate endothelium-derived hyp

74 pecific complications (thromboembolic venous/arterial events, amputations, recurrent/persistent throm

75 oxygenation for respiratory failure, 2) veno-arterial extracorporeal membrane oxygenation for cardiog

76 Veno-arterial extracorporeal membrane oxygenation offers the

77 wall shear stress (WSS) typical of venous or arterial flow inhibit taxis.

78 py can be achieved both through preferential arterial flow to the tumor and through selective cathete

79 Measures of maternal uterine arterial flow, including resistance index and mean veloc

80 erable for larger tumors and those with high arterial flow.

81 Significantly lower arterial fraction and higher vascular transit time, frac

82 metastatic lesions, and significantly higher arterial fraction and lower vascular transit time were o

83 from volunteer livers, significantly higher arterial fraction, fractional volume of the interstitial

84 ion, restores endothelial growth control and arterial gene expression.

85 The use of 3 arterial grafts was associated with statistically signif

86 grafting with the use of 2-arterial versus 3-arterial grafts was performed.

87 consider lowering their threshold for using arterial grafts, and the radial artery may be the prefer

88 lay a part in the complex pathway leading to arterial growth.

89 stem cell pool and plays important roles in arterial homeostasis and disease.

90 iously used and newly constructed metrics of arterial hyperoxia and systematically assess their assoc

91 f existing and newly constructed metrics for arterial hyperoxia were examined, and the associations w

92 ng evidence has shown the potential risks of arterial hyperoxia, but the lack of a clinical definitio

93 In familial pulmonary arterial hypertension (FPAH), the autosomal dominant dis

94 sociated with ASST positivity (P = .009) and arterial hypertension (P = .005), but not with other pos

95 in the rat monocrotaline model of pulmonary arterial hypertension (PAH) are described.

96 y been associated with survival in pulmonary arterial hypertension (PAH) at the time of diagnosis.

97 ascular risk, however, its role in pulmonary arterial hypertension (PAH) has not been determined.

98 onary phenotype and BPD-associated pulmonary arterial hypertension (PAH) in BPD mouse models, which,

99 to seek evidence for alteration in pulmonary arterial hypertension (PAH) in which apelin signaling is

100 Pulmonary arterial hypertension (PAH) is an obstructive disease of

101 RATIONALE: Pulmonary arterial hypertension (PAH) is an obstructive vasculopat

102 treatment guidelines for pediatric pulmonary arterial hypertension (PAH) is hampered by lack of pedia

103 ns of the pulmonary vasculature in pulmonary arterial hypertension (PAH) is primarily provided by aut

104 whether it would be beneficial for pulmonary arterial hypertension (PAH) remains to be explored.

105 o occlusive vascular remodeling in pulmonary arterial hypertension (PAH) that is hereditary, idiopath

106 in the CAV1 gene in patients with pulmonary arterial hypertension (PAH), but the mechanisms by which

107 recognized as a cause of angina in pulmonary arterial hypertension (PAH).

108 d defining treatment strategies in pulmonary arterial hypertension (PAH).

109 e in various pathologies including pulmonary arterial hypertension and cardiomyopathy.

110 -Carnitine (LC) exerts beneficial effects in arterial hypertension due, in part, to its antioxidant c

111 ients with idiopathic or heritable pulmonary arterial hypertension from London (UK; cohorts 1 and 2),

112 les from 43 incident patients with pulmonary arterial hypertension in cohort 3 (p=0.0133).

113 ients with idiopathic or heritable pulmonary arterial hypertension in cohort 4, with 4.4 years' follo

114 ients with idiopathic or heritable pulmonary arterial hypertension to improve risk stratification.

115 om 2005 to 2014, 228 patients with pulmonary arterial hypertension were prospectively enrolled.

116 proteins identifies patients with pulmonary arterial hypertension with a high risk of mortality, ind

117 application in progressive/severe pulmonary arterial hypertension with inadequate response to conven

118 pulmonary hypertension because of pulmonary arterial hypertension, and in patients with postcapillar

119 le, lower prevalence rates of comorbidities (arterial hypertension, diabetes mellitus, and heart fail

120 able goal using the combination of pulmonary arterial hypertension-targeted therapies and LT in patie

121 35 patients who were treated with pulmonary arterial hypertension-targeted therapies before LT resul

122 Cpc-PH bears similarities to pulmonary arterial hypertension.

123 prognostic marker in patients with pulmonary arterial hypertension.

124 ell-validated prognostic scores in pulmonary arterial hypertension.

125 cause of this presentation, although primary arterial hypoperfusion may also be an etiology.

126 Arterial hypoperfusion secondary to outflow obstruction

127 High work of breathing and exercise-induced arterial hypoxaemia (EIAH) can decrease O2 delivery and

128 eceptor expression during the acquisition of arterial identity in vertebrates.

129 sed bone-marrow activity (r=0.47; p<0.0001), arterial inflammation (r=0.49; p<0.0001), and risk of ca

130 r activity was significantly associated with arterial inflammation (r=0.70; p=0.0083).

131 To test the hypothesis that arterial inflammation is linked to HIV disease activity

132 mygdalar activity, bone-marrow activity, and arterial inflammation were assessed with validated metho

133 etween perceived stress, amygdalar activity, arterial inflammation, and C-reactive protein.

134 In addition to systemic inflammation, local arterial inflammation, driven by monocyte-derived macrop

135 associated with LN inflammation but not with arterial inflammation.

136 anges in vascular conductance (FVC) to intra-arterial infusion of phenylephrine (PE; alpha1 -agonist)

137 To investigate whether perioperative hepatic arterial infusion pump chemotherapy (HAI) was associated

138 and forearm blood flow in response to intra-arterial infusions of endothelial-dependent and -indepen

139 rtant as platelets to thrombosis at sites of arterial injury and that platelets contribute to venous

140 atio (DVR) estimated with (blDVR) or without arterial input function (refDVR), and VT Additional voxe

141 inetic analysis using a metabolite-corrected arterial input function and to compare parameters derive

142 uantitative small-animal PET imaging with an arterial input function can be performed routinely.

143 Arterial blood samples were drawn for arterial input function determination.

144 ed using a bolus-plus-infusion protocol, the arterial input function was measured, and the animals un

145 Arterial input functions were based on individual blood

146 vided the best estimate of tau binding using arterial input functions.

147 terial percent medial thickness (r=0.41) and arterial %IT (r=0.35) but more strongly with venous %IT

148 Arterial load and wave reflections in HFpEF were similar

149 n between post-TAVR blood pressure, systemic arterial load, and mortality.

150 ect systemic, associated with an increase in arterial LPS and a susceptibility to sepsis.

151 atch-clamp experiments using native cerebral arterial myocytes, membrane stretch-induced cation curre

152 tch clamp electrophysiology and rat cerebral arterial myocytes, we monitored STOCs in the presence an

153 lso, US28-positive cells were present within arterial neointima.

154 UWFA 4 of 24 (16.7%) SCD eyes had peripheral arterial occlusion (Goldberg I), and 20 of 24 eyes (83.3

155 These complications are caused by thrombotic arterial occlusion localized at the site of high-risk at

156 RATIONALE: Atherosclerotic-arterial occlusions decrease tissue perfusion causing is

157 Ocular arterial occlusive disorders were divided into central (

158 cardial ischemia differ among various ocular arterial occlusive disorders.

159 Since anemia reduces arterial oxygen content, it further threatens oxygen del

160 We measured arterial oxygen partial pressure continuously with a fas

161 ssigned to "control" (FIO2 0.3, adjusted for arterial oxygen saturation >/= 90%) and "hyperoxia" (FIO

162 the effect of postoperative IS on hypoxemia, arterial oxygen saturation (Sao2) level, and pulmonary c

163 of low flow supplemental oxygen to normalize arterial oxygen saturation.

164 Reduction of maternal arterial oxygen tension by 50% over 30 min resulted in a

165 Higher PEEP can improve arterial oxygenation, reduce tidal lung stress and strai

166 PEEP/FiO2 table that prioritizes support for arterial oxygenation.

167 cal iSS is not associated with macrovascular arterial pathology.

168 Arterial patrolling is a prominent new feature of noncla

169 RTN neurons encode arterial PCO2 /pH via cell-autonomous and paracrine mech

170 ive end-tidal targeting to precisely control arterial Pco2 and PET to measure the outcome variable, M

171 Central respiratory chemoreceptors regulate arterial PCO2 by adjusting lung ventilation.

172 ir ultimate effect is invariably to minimize arterial PCO2 fluctuations.

173 PASP correlated with arterial percent medial thickness (r=0.41) and arterial

174 nal transplants performed using the standard arterial PF technique served as a control group.

175 Impaired ability of arterial phagocytes to uptake apoptotic cells (efferocyt

176 Results Arterial phase breath holds were shorter after gadoxetat

177 In 80% (35 of 44) of subjects, arterial phase breath holds were shorter after gadoxetat

178 bo-controlled trial, whether maximal hepatic arterial phase breath-holding duration is affected by ga

179 Conclusion Maximal hepatic arterial phase breath-holding duration is reduced after

180 Arterial phase breath-holding duration was timed after e

181 Exclusion of rim arterial phase hyperenhancement as a means of satisfying

182 Among masses with arterial phase hyperenhancement, the rim pattern was mor

183 validated two-tissue compartment model with arterial plasma input function with total volume of dist

184 sistent with POPH (defined as mean pulmonary arterial pressure >25 mm Hg and pulmonary vascular resis

185 (systolic blood pressure </=90 mm Hg or mean arterial pressure </=65 mm Hg) presenting to the emergen

186 vels of estradiol-17beta (E2) increases mean arterial pressure (MAP) in young female Sprague-Dawley (

187 Mean arterial pressure (MAP), heart rate (HR), BT, motor acti

188 Every 10 mmHg drop from baseline in mean arterial pressure associated with a 3% increase in ische

189 There was no difference in mean arterial pressure at 1, 24, or 48 hours between groups.

190 Mean arterial pressure at 6 hours was 72.2 mm Hg in the renin

191 imates and invasive measurement of pulmonary arterial pressure at rest and peak exercise were simulta

192 These results indicate that arterial pressure equilibrates within the endothelium an

193 fect lactic acid-mediated reflex increase in arterial pressure in patients with PAD.

194 Mean arterial pressure initially decreased further under bolu

195 c events rose rapidly below an absolute mean arterial pressure of 60 mmHg.

196 Wireless transmission of detected arterial pressure signals to a smart phone demonstrates

197 the lactic acid-mediated reflex increase in arterial pressure that is MOR stimulation-independent an

198 dex was then calculated as the ratio of mean arterial pressure to regional cerebral blood flow.

199 Mean arterial pressure was reduced to 30 mm Hg for 90 minutes

200 tonically suppresses splanchnic SNA (SSNA), arterial pressure, and heart rate via projections to the

201 ldosterone improved 5-day survival, invasive arterial pressure, and in vivo and ex vivo arterial resp

202 NOS3 and GUCY1A3 expression and reduced mean arterial pressure, combined them into a genetic score, a

203 pital location, era, systolic pressure, mean arterial pressure, lactate, bundle compliance, amount of

204 udy was to examine the effect of RDN on mean arterial pressure, renal function, and the reflex respon

205 ressed partly by increases in heart rate and arterial pressure.

206 this exposure to a 5 mm Hg reduction in mean arterial pressure.

207 C) activation and reflexively increases mean arterial pressure; endomorphin release is also increased

208 QRS-gated DPD demonstrated higher pulmonary arterial pressures versus isolated postcapillary pulmona

209 iovascular parameters, including heart rate, arterial pressures, and body temperature.

210 tastases include chemoinfusion via a hepatic arterial pump or port, irinotecan-loaded drug-eluting be

211 l role for ER stress in mechanoregulation at arterial regions of atherosusceptible-SS inflamed by low

212 SS) dependent leukocyte TEM and compensatory arterial remodeling obeying Glagov's phenomenon.

213 Arterial remodeling participates pivotally in many disea

214 e arterial pressure, and in vivo and ex vivo arterial response to phenylephrine at 18 hours after ind

215 ate nationwide readmissions after peripheral arterial revascularization for peripheral arterial disea

216 The ART (Arterial Revascularization Trial) was designed to compar

217 VD) (myocardial infarction, unstable angina, arterial revascularization, stroke, or cardiovascular de

218 Intra-arterial route is very promising, but MSCs are missing m

219 Arterial shear (1002.6 s(-1)) induced a sustained increa

220 al secretion and affected thrombus growth at arterial shear rate, indicating a role for Vps34 kinase

221 ition on immobilized collagen in response to arterial shear.

222 to Ca(2+) entry and thrombus formation under arterial shear.

223 In arterial SMCs, CXCR4 sustained normal vascular reactivit

224 lymeric particles were safe to rat pulmonary arterial smooth muscle cell and to the lungs, as evidenc

225 ming BKalpha and auxiliary beta1 subunits in arterial smooth muscle cells (myocytes).

226 In parallel, pulmonary arterial smooth muscle cells (PASMCs) from Cox4i2(-/-) m

227 nd phosphorylated SMAD2/3 in human pulmonary arterial smooth muscle cells on treatment with cocaine a

228 om human pluripotent stem cells that exhibit arterial-specific functions in vitro and in vivo.

229 Notch1 activation, known to promote arterial specification, was decreased in mutant DA endot

230 steps causes hyperproliferation and loss of arterial specification.

231 dy, identical magnetic resonance scans using arterial spin labeling (ASL) were performed to study the

232 aging was performed using velocity-selective arterial spin labeling (VSASL) and 3D image acquisition

233 (AC) glutamate (Glu) and glutamine (Gln) and arterial spin labeling evaluation for rCBF.

234 dyspnea in healthy individuals (n=23) during arterial spin labeling functional magnetic resonance ima

235 [(1)H]) images and perfusion images by using arterial spin labeling were obtained for comparison.

236 ted (1)H images and to perfusion images from arterial spin labeling.

237 al blood flow (rCBF) using pseudo-continuous arterial spin labelling.

238 but no significant difference in the odds of arterial stiffness (OR, 1.07; 95% CI, 0.63-1.84; P = .80

239 ensitivity, adipose tissue inflammation, and arterial stiffness and exerts a beneficial shift in gut

240 gents that target the NO pathway in reducing arterial stiffness in HFpEF.

241 owever, the performance of AI r in assessing arterial stiffness is limited.

242 tress markers (glutathione and cystine), and arterial stiffness were evaluated.

243 opathy, cardiovascular autonomic neuropathy, arterial stiffness, and hypertension.

244 associations with myocardial infarction and arterial stiffness, as well as coronary artery calcifica

245 Arterial stiffness, flow-mediated dilation (FMD), nitrog

246 109 +/- 9 mmHg, P = 0.049) without impacting arterial stiffness, FMD, GMD, or NO.

247 ve better performance over AI r in assessing arterial stiffness.

248 e repairs (pulmonary artery band followed by arterial switch operation).

249 Most (80%) were single-stage arterial switch operations, but 20% were atrial baffling

250 ion of the blood flow as spiral in the whole arterial system and is believed to improve the graft lon

251 zero-heat-flux, esophageal temperature, and arterial temperature.

252 at-flux method to esophageal temperature and arterial temperature.

253 lux method compared with esophageal or iliac arterial temperatures measurements.

254 The more arterial the pattern of spot sign presentation, the grea

255 Increased arterial thickness measured with ultrasound correlates w

256 been detected in atherosclerotic lesions and arterial thrombi in humans and mice.

257 e performed to uncover the effects of NAC on arterial thrombi.

258 of thrombotic stroke induced by either intra-arterial thrombin injection or ferric chloride applicati

259 entify relevant safety end points, including arterial thromboembolic events, MI, stroke or transient

260 The risk of arterial thromboembolism in patients with cancer is inco

261 nts with aspirin for secondary prevention in arterial thrombosis and aspirin with anticoagulants for

262 These data suggest RBCs promote arterial thrombosis by enhancing platelet accumulation a

263 Two eltrombopag recipients (arterial thrombosis n=1; myocardial infarction n=1) and

264 In addition to the aPL thrombophilic effect, arterial thrombosis was related to accelerated atheroscl

265 role of platelets, coagulation, and flow in arterial thrombosis, little attention has been paid to f

266 tential therapeutic target for prevention of arterial thrombosis.

267 d cells, play a major role in hemostasis and arterial thrombosis.

268 inositol 3- kinase (PI3K) and contributes to arterial thrombosis.

269 th cardiovascular disease were identified in arterial tissue.

270 ssociated with mRNA expression of ZCCHC14 in arterial tissues (p = 9.4 x 10(-7) ) and DNA methylation

271 ns was correlated with endothelial function (arterial tonometry).

272 Intra-arterial treatment may be at least as effective in patie

273 ), hypoplastic right heart (n=1), and common arterial trunk (n=1).

274 relative contribution in vivo, we quantified arterial-venous concentration gradients across the human

275 eater extent, exercise independently promote arterial-venous delivery gradients of intravascular nitr

276 of 4 mL/Kg increases transient stop-flow arm arterial-venous equilibrium pressure and reliably detect

277 Hemoglobin concentration and arterial-venous oxygen content difference have large eff

278 syndrome (APS) is characterized by recurrent arterial/venous thrombosis and miscarriages in the persi

279 ary artery bypass grafting with the use of 2-arterial versus 3-arterial grafts was performed.

280 s and potentially negligible toxic effect on arterial vessel walls.

281 In partial UFE, only the small arterial vessels to the fibroids were embolized, leaving

282 level, confirming the non-linearity between arterial volume distensibility and external pressure.

283 ifferent effect of external cuff pressure on arterial volume distensibility between peripheral arteri

284 , 20, 30 and 40 mmHg, the overall changes in arterial volume distensibility referred to those without

285 , 30 and 40 mmHg) on the whole arm to obtain arterial volume distensibility.

286 Arterial wall (68)Ga-pentixafor uptake is significantly

287 determined that DNMT1-specific inhibition in arterial wall ameliorates the disturbed flow-induced ath

288 mography computed tomography (PET/CT) of the arterial wall and a migratory phenotype of monocytes.

289 effective siRNA transfection of the injured arterial wall and provide a clinically effective and tra

290 eover, cell-specific effects of CXCR4 in the arterial wall and underlying mechanisms remain elusive,

291 While LNs and, to a lesser degree, the arterial wall are inflamed in HIV, inflammation in these

292 Arterial wall dendritic cells attract CD4(+) T cells and

293 le impact of oversized stent implantation on arterial wall injury has not been systematically investi

294 ascular diseases such as atherosclerosis and arterial wall injury.

295 that maladaptive autoimmune responses in the arterial wall play critical roles in the process of athe

296 the increased number of plaques or abnormal arterial wall thickness.

297 olesterol in the subendothelial space of the arterial wall to the liver) is terminated by selective h

298 g of CXCR4 expression in the atherosclerotic arterial wall.

299 ar preload and afterload influence pulmonary arterial wave propagation as demonstrated by changes in

300 previously reported mathematical analysis of arterial waveform that extracts hidden oscillations in t