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1 a, hemolytic anemias, and malaria, with both arterial and venous thrombosis.
2 III are associated with an increased risk of arterial and venous thrombosis.
3 antibodies (APLAs) are at increased risk for arterial and venous thrombosis.
4 ne of therapy for conditions associated with arterial and venous thrombosis.
5  of inherited platelet risk factors for both arterial and venous thrombosis.
6 mia) with risk of cardiovascular disease and arterial and venous thrombosis.
7 hromboses, 3 arterial thromboses, 2 combined arterial and venous thrombosis, 2 thromboses secondary t
8 Whether this mutation increases the risks of arterial and venous thrombosis among healthy individuals
9  Whether this polymorphism increases risk of arterial and venous thrombosis among middle-aged men is
10 dy syndrome (APS) often experience recurrent arterial and venous thrombosis and pregnancy losses.
11 arding the extent of shared pathways between arterial and venous thrombosis and whether treatments of
12 odulin (TM) gene occur in patients with both arterial and venous thrombosis, but the effects of these
13 uPA-T provided effective prophylaxis against arterial and venous thrombosis for up to 24 hours.
14 ions of gammaA/gamma' fibrinogen levels with arterial and venous thrombosis have been reported, indic
15                    Because the high rates of arterial and venous thrombosis in lupus cannot be explai
16 e used to treat serious and life-threatening arterial and venous thrombosis in neonates and children.
17 holipid antibodies (aPL) are associated with arterial and venous thrombosis in systemic lupus erythem
18 ntithrombotic and thereby protective against arterial and venous thrombosis, including through the ac
19 eficiency reduced thrombus formation in both arterial and venous thrombosis models, without an appare
20 iphospholipid antibodies are associated with arterial and venous thrombosis, recurrent pregnancy loss
21 ic inflammatory disorders that predispose to arterial and venous thrombosis through similar prothromb

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