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1 of cerebral blood flow despite correction of arterial hypotension.
2 us- pituitary-adrenal (HPA) axis response to arterial hypotension.
3  pulmonary disease (1.8 [1.2-2.7]), systolic arterial hypotension (2.9 [1.7-5.0]), tachypnoea (2.0 [1
4 rtension acting as a predisposing factor and arterial hypotension actually producing the disorders.
5             These associations indicate that arterial hypotension after ROSC may represent a potentia
6  a life-threatening kidney disease featuring arterial hypotension along with electrolyte abnormalitie
7                   Lipopolysaccharide-induced arterial hypotension and arterial hypoxemia were attenua
8 ked human hemoglobin can ameliorate systemic arterial hypotension and improve organ perfusion in a po
9 eletal muscle injury produced early systemic arterial hypotension and vasodilation, and a decrease in
10 tations of NCC cause renal salt wasting with arterial hypotension (Gitelman syndrome).
11 S) mRNA and protein expression, and systemic arterial hypotension in a rat model of septic shock.
12  M40401, modulates serum cytokine levels and arterial hypotension in an Escherichia coli-infected con
13 an important factor contributing to systemic arterial hypotension in septic shock.
14 secondary analyses we found that exposure to arterial hypotension induced E-selectin and thrombin-ant
15 nsive medications for arterial hypertension, arterial hypotension (particularly nocturnal hypotension
16  are significantly correlated with nocturnal arterial hypotension, particularly in hypertensive patie
17                                         With arterial hypotension, postarrest, hypothermic piglets ha
18 objective was to determine whether post-ROSC arterial hypotension predicts outcome among postcardiac
19 ion, but significantly increased the risk of arterial hypotension, pruritus, urinary retention, and m
20 ith etomidate, episodes of apnea, hypoxia or arterial hypotension requiring therapeutic intervention

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