ライフサイエンスコーパス: arterial lesion

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1 anti-inflammatory M2 phenotype in spleen and arterial lesions.

2 co-localizes with macrophages within murine arterial lesions.

3 that have localized at sites of experimental arterial lesions.

4 raft dysfunction relies on the assessment of arterial lesions.

5 only after histopathologic evaluation of the arterial lesions.

6 onal properties of resident B cells in human arterial lesions.

7 Kawasaki disease (kDa) may develop coronary arterial lesions and subsequent coronary events.

8 onstrated reduced KIS gene expression within arterial lesions and these lesions were significantly sm

9 intima is a key event in the development of arterial lesions, apoptosis of VSMCs also plays an impor

10 baseline NIHSS score, lesion volume on DWI, arterial lesion by magnetic resonance angiography, and c

11 hat MMP-9 is critical for the development of arterial lesions by regulating both SMC migration and pr

12 this was paralleled by a marked reduction in arterial lesion C3 deposition despite similar levels of

13 chromosome 6 gene, which we call Artles (for arterial lesions), did not affect endothelial cell respo

14 Thus, specific arterial lesions (endarteritis, fibrinoid necrosis, acti

15 The arterial lesions following femoral artery injury in LZ/A

16 Podocan-deficient mice showed increased arterial lesion formation compared with wild-type litter

17 xacerbates injury- or hyperlipidemia-induced arterial lesion formation in mice, possibly by excessive

18 Relative to apoE knockout (KO) mice, arterial lesion formation was significantly decreased in

19 ired endothelial regeneration contributes to arterial lesion formation.

20 trating macrophages, which closely resembles arterial lesions from NF1 patients.

21 Compared with WT mice, arterial lesions grew significantly larger in Tg(p22vsmc

22 iting endothelial regeneration and promoting arterial lesion growth in conditions of endothelial inju

23 In addition, SMC migration and arterial lesion growth were significantly impaired in MM

24 Arterial lesions in cardiovascular diseases are characte

25 and inflammatory burden of acute and chronic arterial lesions in mice.

26 We have shown that occlusive arterial lesions in patients with heterozygous ACTA2 mis

27 Genes differentially expressed in arterial lesions included 3 products encoded by the mito

28 Reinjury of rat arterial lesions induces an increase in lesion size that

29 percentage of the volume of human restenotic arterial lesions is occupied by extracellular matrix (EC

30 ion into lipid-laden macrophages at sites of arterial lesions, leading to the development of atherosc

31 ix metalloproteinase 9 (MMP-9) is present in arterial lesions of GCA and may be involved in its patho

32 lasty has become particularly attractive for arterial lesions of Takayasu arteritis.

33 that contrast gradient attenuation along an arterial lesion, or transluminal attenuation gradient (T

34 sative relation between oxidative stress and arterial lesion progression remains unclear.

35 The 2-dimensional representation of the arterial lesion provided by angiography is limited in di

36 NE) in the plasma and protein-HNE adducts in arterial lesions than AR(+/+)/apoE(-/-) mice.

37 Chronic rejection in xenografts involves arterial lesions that bear some histological similaritie

38 The type and location of coronary arterial lesions were determined by coronary angiography

39 Relevant arterial lesions were frequently detected in the middle

40 All arterial lesions were of atherosclerotic etiology.

41 Their coronary arterial lesions, which were strikingly similar to human

42 ection is characterized by the appearance of arterial lesions with concentric intimal thickening.

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