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1 able in risk stratification of patients with arterial occlusive disease.
2 scularization for lower extremity peripheral arterial occlusive disease.
3 9 years; range, 54-80 years) with peripheral arterial occlusive disease.
4 tic arteries of patients suspected of having arterial occlusive disease.
5 and economical assessment of lower extremity arterial occlusive disease.
6 d on the order of testing or the severity of arterial occlusive disease.
7 ively assess the severity of lower extremity arterial occlusive disease.
8 for the treatment of atherosclerotic carotid arterial occlusive disease.
9 tion of diagnosis of chronic lower-extremity arterial occlusive disease.
10 mechanism that protects against ischemia in arterial occlusive disease.
11 5% CI, 1.24-3.33]; P = .002), and peripheral arterial occlusive disease (adjusted HR, 2.15 [95% CI, 1
13 alities may contribute to the development of arterial occlusive disease and associated clinical event
14 patients treated with dasatinib, peripheral arterial occlusive disease and other arterial disorders
15 s in patients suspected of having peripheral arterial occlusive disease, and diagnostic performance w
17 ity due to worsening pre-existing peripheral arterial occlusive disease in a patient who had received
19 ng abdominal aortic aneurysm (33 percent) or arterial occlusive disease of the legs (67 percent).
21 Intermittent claudication due to peripheral arterial occlusive disease (PAOD) is a common cause of p
23 ls in 60 randomized patients with peripheral arterial occlusive disease referred for 64-section multi
26 mocysteine, which are frequently elevated in arterial occlusive disease, we hypothesized that folic a
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