ライフサイエンスコーパス: arterial pressure

1 d a norepinephrine infusion to maintain mean arterial pressure.

2 brain hypoperfusion during acute increase in arterial pressure.

3 ressed partly by increases in heart rate and arterial pressure.

4 n II, and resulted in better preservation of arterial pressure.

5 role of norepinephrine in the regulation of arterial pressure.

6 systolic or diastolic blood pressure or mean arterial pressure.

7 us monitoring of cardiac output and systemic arterial pressure.

8 this exposure to a 5 mm Hg reduction in mean arterial pressure.

9 The primary outcome was 24-hour mean arterial pressure.

10 red norepinephrine to maintain adequate mean arterial pressure.

11 Our primary outcome was 24-hour mean arterial pressure.

12 capillary wedge pressure and mean pulmonary arterial pressure.

13 thout eliciting a supplementary reduction in arterial pressure.

14 ertension but without MetS for the same mean arterial pressure.

15 tients in whom norepinephrine increased mean arterial pressure.

16 lective, as it was associated with decreased arterial pressure.

17 adjusted for age, body mass index, and mean arterial pressure.

18 voke a pressor reflex known to increase mean arterial pressure.

19 Mean pulmonary arterial pressure.

20 vides prognostic utility beyond that of mean arterial pressure.

21 c BP, diastolic BP, pulse pressure, and mean arterial pressure.

22 rdiovascular outcomes, independently of mean arterial pressure.

23 d this correlated with post-brain death mean arterial pressures.

24 ificantly associated with diastolic and mean arterial pressures.

25 st circumference (-1.1 to -1.9 cm), and mean arterial pressure (0.0 to -1.1 mm Hg) at 6 months and Fr

26 CI -0.01, 0.03]; p = 0.36; n = 32,961); mean arterial pressure (-0.06 mm Hg [95% CI -0.19, 0.07]); p

27 8.7, -5.1]; p(group) < 0.0001), similar mean arterial pressure (-1.1 mm Hg [95% confidence limit, -2.

28 Systemic arterial pressure (108.2 +/- 1.4 mm Hg) was unaffected b

29 ge II evaluation, including higher pulmonary arterial pressures (15.4+/-3.0 versus 14.5+/-3.5 mm Hg;

30 quate intravascular volume and adequate mean arterial pressure (1C); avoiding use of intravenous hydr

31 0+/-4 versus 6+/-3 mm Hg; P=0.02), pulmonary arterial pressure (22+/-8 versus 11+/-4 mm Hg; P=0.0001)

32 nts of crystalloid to maintain adequate mean arterial pressure (2C) and the avoidance of hetastarch f

33 Mice underwent 90 minutes of shock (mean arterial pressure 30 mm Hg) and resuscitation via femora

34 % +/- 1% of baseline), hypotension (systemic arterial pressure 31 +/- 1 mm Hg), and acidosis (pH 7.02

35 e (23+/-2 vs. 10+/-3 mmHg, p=0.006) and mean arterial pressure (37+/-1 vs. 24+/-2 mmHg, p=0.006) but

36 n study, 91 patients with PH (mean pulmonary arterial pressure 46+/-15 mm Hg) underwent clinical and

37 , and Mg generated significantly higher mean arterial pressure (48 mm Hg [95% CI, 44-52] vs 33 mm Hg

38 7 mm Hg; 95% CI, -25 to -8; p < 0.001), mean arterial pressure (-7 mm Hg; 95% CI, -12 to -1; p = 0.02

39 dynamic parameters except a decrease in mean arterial pressure (-7 mm Hg; p = 0.041) and in systemic

40 ic variables were relatively preserved (mean arterial pressure 70 [65-77] mm Hg, cardiac index 3.3 [2

41 patients with RAI presented with lower mean arterial pressure (76 +/- 12 versus 83 +/- 14 mmHg, P =

42 significantly (p = 0.01) lower mean systemic arterial pressure (80.1 +/- 15.1 mm Hg) versus control g

43 +/- 8 vs 17 +/- 7; P < .0001) and lower mean arterial pressure (81 +/- 16 vs 85 +/- 15 mm Hg; P < .01

44 CPAP reduced baseline mean arterial pressure (94 +/- 2 vs. 89 +/- 2 mm Hg, P = 0.00

45 : 0.4, 1.7; p = 0.001), 0.8-mmHg higher mean arterial pressure (95% CI: 0.2, 1.4; p = 0.01), and no s

46 In contrast, an elevated mean arterial pressure along with increased central and perip

47 Antagonism of P2X3 receptors also reduces arterial pressure and basal sympathetic activity and nor

48 Fifteen days later, arterial pressure and carotid thickness were lower in SR

49 In sepsis, the renal nerves help support arterial pressure and determine the initial diuretic res

50 y-induced hypertension, ouabain reduced mean arterial pressure and enhanced placental HSP27 phosphory

51 s, we demonstrate that ELA and 3 both reduce arterial pressure and exert positive inotropic effects o

52 Approximately 200 had portal venous and arterial pressure and flow measurements before and after

53 urons also resulted in a significant fall in arterial pressure and heart rate that was similar in mag

54 ac work was estimated as the product of mean arterial pressure and heart rate.

55 d without LVDD, had significantly lower mean arterial pressure and higher Model for End-Stage Liver D

56 The dynamic information in continuous mean arterial pressure and intracranial pressure monitoring a

57 summary statistics of minute-by-minute mean arterial pressure and intracranial pressure.

58 Organ Dysfunction-2 score now includes mean arterial pressure and lactatemia in the cardiovascular d

59 An inverse relationship between mean arterial pressure and mortality was identified (p = 0.00

60 MRI, in combination with measurement of peak arterial pressure and MRI-derived timing of valvular eve

61 Low mean arterial pressure and need for high doses of vasopressor

62 s calculated as laser-Doppler flowmetry/mean arterial pressure and normalized to maximum.

63 Mean arterial pressure and PP were continuously recorded and

64 terial baroreflex gain, and provoked smaller arterial pressure and R-R interval fluctuations, which w

65 ncreased risk for incident CVD, whereas mean arterial pressure and relative wave reflection (correlat

66 o-obliteration leading to elevated pulmonary arterial pressure and resistance, right ventricular dysf

67 inhibited the effects of vasopressin on mean arterial pressure and significantly reduced the effects

68 tion, the steady-state relationships between arterial pressure and sodium excretion, a correlation th

69 Immediately after implantation, systemic arterial pressure and stroke volume increased and pulmon

70 ined that alarming nocturnal oscillations in arterial pressure and sympathetic nerve activity (SNA) w

71 the 0- to 6-hour time-weighted average mean arterial pressure and used multivariable logistic regres

72 minute; P<0.01) despite a reduction in mean arterial pressure and was inversely related to pulse pre

73 ed inversely with systolic blood pressure or arterial pressure and with parathyroid hormone.

74 etween increasing time-weighted average mean arterial pressures and good neurologic outcome, defined

75 d diastolic blood pressure, 22 SNPs for mean arterial pressure, and 10 SNPs for pulse pressure) with

76 sease, including associations with pulmonary arterial pressure, and adverse neurological sequelae, th

77 on, even after adjustment for sex, age, mean arterial pressure, and BMI.

78 solated aortic rings, cardiac function, mean arterial pressure, and both the renal vascular perfusion

79 ese results were adjusted for age, sex, mean arterial pressure, and cardiovascular risk factors.

80 D with forward pressure wave amplitude, mean arterial pressure, and global reflection coefficient der

81 tonically suppresses splanchnic SNA (SSNA), arterial pressure, and heart rate via projections to the

82 lla (RVLM) lower sympathetic nerve activity, arterial pressure, and heart rate, or when administered

83 percentage of total blood volume (TBV), mean arterial pressure, and heart rate, which were recorded a

84 iomarkers, such as WBC, oxygen content, mean arterial pressure, and heart rate, yielded estimation ac

85 ldosterone improved 5-day survival, invasive arterial pressure, and in vivo and ex vivo arterial resp

86 e, uses shock index as a substitute for mean arterial pressure, and incorporates serum lactate as a b

87 ependently decreased PP, did not modify mean arterial pressure, and increased SVR.

88 cipient age, body mass index, mean pulmonary arterial pressure, and pretransplant diagnosis, higher E

89 s subjects, right atrial pressure, pulmonary arterial pressure, and pulmonary capillary wedge pressur

90 had larger reductions in diastolic BP, mean arterial pressure, and PWV (-2.24 +/- 1.31 mm Hg, -1.24

91 improvement in exercise capacity, pulmonary arterial pressure, and quality of life.

92 Heart rate, mean arterial pressure, and total peripheral resistance were

93 iovascular parameters, including heart rate, arterial pressures, and body temperature.

94 y (EEG) power; (3) a modest decrease in mean arterial pressure; and (4) a progressive shift of the EE

95 N) is activated and drives SNA in support of arterial pressure (AP).

96 Systolic and pulse arterial pressures, as well as indices of vascular funct

97 ts during vasoactive drug-induced changes in arterial pressure assessed at the internal carotid and v

98 Every 10 mmHg drop from baseline in mean arterial pressure associated with a 3% increase in ische

99 rats displayed significant reduction in mean arterial pressure associated with attenuation of both ce

100 There was no difference in mean arterial pressure at 1, 24, or 48 hours between groups.

101 On average, the 24-hour mean arterial pressure at 12 weeks was lower in the group rec

102 blood removal/retransfusion to maintain mean arterial pressure at 30 mm Hg).

103 e blood volume, subsequent titration of mean arterial pressure at 35 mm Hg), anesthetized and instrum

104 Mean arterial pressure at 6 hours was 72.2 mm Hg in the renin

105 guidelines, which recommend maintaining mean arterial pressure at 85 to 90mm Hg for a week after spin

106 nd point was a response with respect to mean arterial pressure at hour 3 after the start of infusion,

107 OGTT, maternal height at OGTT, maternal mean arterial pressure at OGTT, maternal smoking and drinking

108 on, norepinephrine titrated to maintain mean arterial pressure at preshock values, mechanical ventila

109 points comprised cardiac index and pulmonary arterial pressure at rest and during exercise after 9 we

110 imates and invasive measurement of pulmonary arterial pressure at rest and peak exercise were simulta

111 ood pressure targets were not achieved (mean arterial pressure at study end: NE: 81 mm Hg [76-85 mm H

112 r systolic and diastolic blood pressures and arterial pressure at week 20 before clinical recognition

113 Mean end-inspiratory and end-expiratory arterial pressures at t = 13 minutes (mm Hg) were as fol

114 onized ventilation elicited the highest mean arterial pressure, best oxygenation, and a normal mixed

115 systolic BP (beta=-4.11; P=2.8x10(-4)), mean arterial pressure (beta=-3.50; P=8.9x10(-6)), and reduce

116 tation velocity-time integral, and pulmonary arterial pressure between patients with and without post

117 o significant difference in the 24-hour mean arterial pressure between the control group and the grou

118 lass and higher left ventricular volumes and arterial pressure both at rest and during exercise than

119 Pulmonary ventilation and pulmonary arterial pressure both rise progressively during the fir

120 Advancing age and lower mean arterial pressure, but not the presence of a transthyret

121 L-NAME increased mean arterial pressure by approximately 17 mm Hg in both grou

122 Cocaine increased mean arterial pressure (by 14+/-2 mm Hg [mean+/-SE]), heart r

123 ut no effect on macrocirculatory parameters (arterial pressure, cardiac index, heart rate, and pulse

124 PP, mean arterial pressure, cardiac output, SVR, and ascites volu

125 s: preflight, late mission and landing day.) Arterial pressure changed systematically from preflight

126 uencies and probabilities increased, even as arterial pressure climbed to new levels); or altered pul

127 NOS3 and GUCY1A3 expression and reduced mean arterial pressure, combined them into a genetic score, a

128 than the intact group (2-month fall in mean arterial pressure: control-intact, -10 +/- 1 mm Hg; cont

129 Coronary vascular resistance (CVR; mean arterial pressure/coronary blood velocity) was used as a

130 cardiographic measures of systolic pulmonary arterial pressure correlated reasonably well with invasi

131 ar reflexes while also producing lability of arterial pressure, damage to cardiac myocytes, and in so

132 Heart rate was unchanged, and arterial pressure declined modestly.

133 Mean arterial pressure decreased similarly during endotoxemia

134 degree of hemodynamic instability (mean [SD] arterial pressure decreases of 25 [1] and 41 [11] mm Hg,

135 nd 170 mm Hg of steady-state changes in mean arterial pressure, defined as static CA.

136 on, normalization of serum lactate, and mean arterial pressure did not differ among groups.

137 for first trimester body mass index and mean arterial pressure, differences in intima thickness and I

138 Work rate and mean arterial pressure during exercise were similar in contro

139 regarding its accuracy to estimate pulmonary arterial pressure during exercise.

140 y two and fivefold, respectively, normalized arterial pressure during LVR, and lowered plasma lactate

141 C) activation and reflexively increases mean arterial pressure; endomorphin release is also increased

142 These results indicate that arterial pressure equilibrates within the endothelium an

143 ed with hemodynamic instability (higher mean arterial pressure extrema points frequencies were associ

144 isodes of hypotension were common, with mean arterial pressure falling by a median of 22 mmHg (interq

145 Lipid infusion increased aortic flow and arterial pressure faster and to a greater degree than di

146 Arterial pressure fell and then rose in space, and drift

147 If, despite fluid management, mean arterial pressure fell by more than 10 mm Hg from baseli

148 In response to hemorrhage, mean arterial pressure fell in all groups, with the fall bein

149 CR, or HS/CR+MC-2 (HS = 40% of baseline mean arterial pressure for 60 minutes; CR = return of shed bl

150 litude, and 1 ms pulse width, restoring mean arterial pressure from 0 to 37 mmHg.

151 o extracorporeal membrane oxygenation, lower arterial pressure, fungal pneumonia, and advancing age.

152 5-year-old female uni-x and sham sheep, mean arterial pressure, glomerular filtration rate, and renal

153 uded the following physiologic targets: mean arterial pressure greater than 70 mm Hg, cerebral perfus

154 od neurologic outcome at a threshold of mean arterial pressure greater than 70 mm Hg.

155 This threshold (mean arterial pressure > 70 mm Hg) had the strongest associat

156 he first-choice vasopressor to maintain mean arterial pressure >/= 65 mm Hg (1B); epinephrine when an

157 confirm the diagnosis of PAH (mean pulmonary arterial pressure >/=25 mm Hg and pulmonary capillary we

158 Mean arterial pressure >/=65 mm Hg and central venous pressur

159 management at 33 degrees C with target mean arterial pressure >/=65 mm Hg is associated with increas

160 sistent with POPH (defined as mean pulmonary arterial pressure >25 mm Hg and pulmonary vascular resis

161 d treatment with vasopressors targeting mean arterial pressure (>/=65 mm Hg) and blood transfusion (f

162 th lower baseline diastolic BP (DBP) or mean arterial pressure had more progression of subcortical at

163 ood flow in response to transient changes in arterial pressure has been used to assess dynamic CA.

164 1.09; P=0.027), exercise systolic pulmonary arterial pressure (hazard ratio, 1.03; P<0.001), and res

165 s associated with incident CVD, whereas mean arterial pressure (hazard ratio, 1.10; 95% confidence in

166 Mean arterial pressure, heart rate, and lactate were register

167 objective physical variables (including mean arterial pressure, heart rate, respiratory rate, and oxy

168 tion (HR: 23.2; P = 0.01), and baseline mean arterial pressure (HR: 0.92; P = 0.01) were found to be

169 atory modulation of sympathetic activity and arterial pressure in both normotensive and CIH hypertens

170 Terutroban did not significantly change arterial pressure in CCl4 -cirrhotic rats but decreased

171 tensive and CIH hypertensive rats, but basal arterial pressure in CIH rats remained higher compared t

172 Valsartan significantly reduced arterial pressure in knockout mice but not in controls.

173 fect lactic acid-mediated reflex increase in arterial pressure in patients with PAD.

174 asal blood pressure and acute change in mean arterial pressure in response to angiotensin II (Ang II)

175 erence in the primary outcome of 6-hour mean arterial pressure in septic shock patients receiving vas

176 r 1 (BLT1) receptor with CP-105,696, reduced arterial pressure in the SHR compared to the normotensiv

177 tive arterial elastance despite similar mean arterial pressures in control subjects.

178 served in CIH and HC rats, although the mean arterial pressure increase was lower after CIH.

179 After infusion of angiotensin II, mean arterial pressure increased by 61.6 mmHg in MD-NOS1KO mi

180 After fluid challenge, mean arterial pressure increased from 73 mm Hg (interquartile

181 Mean arterial pressure increased significantly in MD-NOS1KO m

182 intervention attenuated not only the fall in arterial pressure induced by LPS, but also the associate

183 Mean arterial pressure initially decreased further under bolu

184 The Intraoperative Norepinephrine to Control Arterial Pressure (INPRESS) study was a multicenter, ran

185 eye at each of the 8 time points as 2/3(mean arterial pressure-intraocular pressure [IOP]).

186 A significant increase in mean arterial pressure is observed in early adulthood in both

187 aggressively treated in this period if mean arterial pressure, lactate clearance, and diuresis are m

188 pital location, era, systolic pressure, mean arterial pressure, lactate, bundle compliance, amount of

189 Exclusion criteria for both groups were mean arterial pressure less than 60 mm Hg, contraindications

190 when shock was present, animals with a mean arterial pressure less than 65 mm Hg (n = 6) had similar

191 n which animals were exsanguinated to a mean arterial pressure level of 40 mm Hg during 30 minutes, h

192 n from the 30th to the 60th minute to a mean arterial pressure level of 40 mm Hg; or control group.

193 was a composite of severe hypotension (mean arterial pressure < 60 mm Hg) and bradycardia (heart rat

194 (systolic blood pressure </=90 mm Hg or mean arterial pressure </=65 mm Hg) presenting to the emergen

195 ndomized to strict or usual BP control (mean arterial pressure </=92 mmHg or 102-107 mmHg, respective

196 namically compromised patients with systolic arterial pressure <100 mm Hg was higher among those who

197 albumin <2.5 g/dL, heart rate >90 bpm, mean arterial pressure <60 mmHg, white blood cell count >/=15

198 002), as was the proportion of patients with arterial pressure <82 mm Hg (64% of those taking NSBBs v

199 Mean arterial pressure (MAP) and OPP (systolic, diastolic, an

200 systolic BP (SBP), diastolic BP (DBP), mean arterial pressure (MAP) and pulse pressure (PP), we geno

201 reover, inflammation increased maternal mean arterial pressure (MAP) and was associated with renal st

202 diastolic BP (DBP) were measured, with mean arterial pressure (MAP) calculated.

203 vels of estradiol-17beta (E2) increases mean arterial pressure (MAP) in young female Sprague-Dawley (

204 nt of CA during steady-state changes in mean arterial pressure (MAP) induced by intravenous infusion

205 of Doppler BP to systolic BP (SBP) and mean arterial pressure (MAP) is uncertain and Doppler measure

206 males; however, i.c.v. leptin increased mean arterial pressure (MAP) only in males.

207 The nocturnal mean arterial pressure (MAP) was compared with the daytime MA

208 Renal blood flow (RBF), mean arterial pressure (MAP), and heart rate (HR) were contin

209 ndstage Liver Disease (MELD) score, low mean arterial pressure (MAP), and non-SBP infections.

210 systolic BP (SBP), diastolic BP (DBP), mean arterial pressure (MAP), and pulse pressure (PP) average

211 systolic BP (SBP), diastolic BP (DBP), mean arterial pressure (MAP), and pulse pressure (PP), we gen

212 Mean arterial pressure (MAP), heart rate (HR), BT, motor acti

213 ow systolic blood pressure (SBP) and/or mean arterial pressure (MAP).

214 rst 24 hrs of intracranial pressure and mean arterial pressure monitoring to known baseline risk fact

215 ension (PH) is diagnosed by a mean pulmonary arterial pressure (mPAP) value of at least 25 mm Hg duri

216 llation of liposomal fasudil, mean pulmonary arterial pressure (MPAP) was reduced by 37.6+/-5.7% and

217 ression of Kv1.5 channels), we measured mean arterial pressure, myocardial blood flow, myocardial tis

218 Hypertension, elevated arterial pressure, occurs as the consequence of increase

219 o, 2.71; 95% CI, 1.67-4.39; p < 0.001), mean arterial pressure (odds ratio, 0.979; 95% CI, 0.963-0.99

220 agic shock by blood withdrawn until the mean arterial pressure of 30 mm Hg and maintained at this pre

221 35-40 kg) were anesthetized and bled to mean arterial pressure of 35-40 mm Hg for 90 minutes, followe

222 c events rose rapidly below an absolute mean arterial pressure of 60 mmHg.

223 a vasopressor requirement to maintain a mean arterial pressure of 65 mm Hg or greater and serum lacta

224 = 0.009) compared with animals with an mean arterial pressure of 65-70 mm Hg (n = 4).

225 n daily dose of vasopressor to insure a mean arterial pressure of 65-75 mm Hg.

226 Targeting a mean arterial pressure of 80 to 85 mm Hg, as compared with 65

227 Sepsis Campaign recommends targeting a mean arterial pressure of at least 65 mm Hg during initial re

228 ension was defined as a decrease in the mean arterial pressure of greater than or equal to 15% compar

229 by a progressive elevation in mean pulmonary arterial pressure, often leading to right ventricular fa

230 changes in right atrial pressure, pulmonary arterial pressure, or pulmonary resistance.

231 ant alterations in urinary sodium excretion, arterial pressure, or pulse rate.

232 atinine, bilirubin or albumin, baseline mean arterial pressure, or study design, size or time period.

233 patients with higher baseline BP (DBP, mean arterial pressure, or systolic BP), those with declining

234 in the crystalloid group, had a higher mean arterial pressure (P=0.03) and lower net fluid balance (

235 cording of hemodynamic parameters [pulsatile arterial pressure (PAP) and heart rate (HR)].

236 ty patients with COPD had PH (mean pulmonary arterial pressure [PAPm] >/= 25 mm Hg).

237 with CHD risk tended to be modified by mean arterial pressure (Pinteraction=0.08) and was modified b

238 use, blood pressure medication use, and mean arterial pressure, PP quartile was still associated with

239 In vehicle-treated sheep, mean arterial pressure progressively declined from 25 to 32 h

240 an+/-SD, 8.3+/-2.8 per subject) of pulmonary arterial pressure, pulmonary arterial wedge pressure and

241 ure were not correlated with changes in mean arterial pressure (r = 0.0002; p = 0.85).

242 y correlated with decreases in the diastolic arterial pressure (r = 0.92) and to a lesser extent with

243 scular resistance, and pulmonary-to-systemic arterial pressure ratio resolved the observed survival d

244 r and the pulmonary artery pressure/systemic arterial pressure ratio was lower in doxycycline groups,

245 (p = 0.76) associated with hypotensive mean arterial pressure readings (</=60 mm Hg) were independen

246 of valvular events and a measurement of peak arterial pressure, regional myocardial work could be cal

247 udy was to examine the effect of RDN on mean arterial pressure, renal function, and the reflex respon

248 alpha2-adrenergic agonist clonidine on mean arterial pressure, renal sympathetic nerve activity, and

249 n Earth with electrocardiogram, non-invasive arterial pressure, respiratory carbon dioxide concentrat

250 ctrocardiogram, finger photoplethysmographic arterial pressure, respiratory carbon dioxide levels, ti

251 herapy in the improvement of 6MWD, pulmonary arterial pressure, right atrial pressure, cardiac index

252 Salt sensitivity of arterial pressure (salt-sensitive hypertension) is a ser

253 inephrine required to maintain a target mean arterial pressure; secondary outcomes included hemodynam

254 re and invasive arterial blood pressure mean arterial pressures showed better agreement; acute kidney

255 Wireless transmission of detected arterial pressure signals to a smart phone demonstrates

256 Mean arterial pressure, skin blood flow via laser-Doppler flo

257 atment prevented the further decline in mean arterial pressure, substantially reduced heart rate and

258 c shock to undergo resuscitation with a mean arterial pressure target of either 80 to 85 mm Hg (high-

259 eduction in systolic blood pressure and mean arterial pressure than did either CPAP or weight loss al

260 the lactic acid-mediated reflex increase in arterial pressure that is MOR stimulation-independent an

261 ctrocardiogram, finger photoplethysmographic arterial pressure, tidal carbon dioxide concentrations a

262 ctrocardiogram, finger photoplethysmographic arterial pressure, tidal volume, respiratory carbon diox

263 dex was then calculated as the ratio of mean arterial pressure to regional cerebral blood flow.

264 added to norepinephrine to either raise mean arterial pressure to target or to decrease norepinephrin

265 capillary wedge pressure and mean pulmonary arterial pressure to volume loading with rapid saline in

266 ents with grade A TR signals, mean pulmonary arterial pressure-to-workload ratio at a threshold of 1.

267 roteinuria were repeat transplantation, mean arterial pressure, transplant glomerulopathy, microcircu

268 ontributes significantly to the elevation of arterial pressure under these conditions.

269 The ability to measure subtle changes in arterial pressure using devices mounted on the skin can

270 Arterial pressure, vagal tone and muscle sympathetic out

271 reshold effect at time-weighted average mean arterial pressure value of 70 mm Hg.

272 liance decreased stroke volume and increased arterial pressure variations.

273 QRS-gated DPD demonstrated higher pulmonary arterial pressures versus isolated postcapillary pulmona

274 nce (FVC; Doppler ultrasound, brachial intra-arterial pressure via catheter) to local intra-arterial

275 Basal mean arterial pressure was 15 mm Hg higher and glomerular fil

276 minophen-induced hypotension, the nadir mean arterial pressure was 64 mm Hg (95% CI, 54-74).

277 We found that time-weighted average mean arterial pressure was associated with good neurologic ou

278 Systemic arterial pressure was higher and the pulmonary artery pr

279 Arterial pressure was monitored via an arterial catheter

280 A compensatory increase in systemic arterial pressure was observed in rats treated with lipi

281 LT1) receptors were blocked with CP-105,696, arterial pressure was reduced in the SHR compared to the

282 Mean arterial pressure was reduced to 30 mm Hg for 90 minutes

283 Mean arterial pressure was reduced to 30 mm Hg for 90 minutes

284 l-L-thiocitrulline (1 mumol/kg, IV) and mean arterial pressure was registered.

285 mathematical model for long-term control of arterial pressure was the model of Guyton and Coleman; r

286 The effect of vasopressin on mean arterial pressure was unaltered and that on renal vascul

287 aneous vascular conductance (CVC = flux/mean arterial pressure) was expressed as a change from baseli

288 transmitral gradient, and systolic pulmonary arterial pressure were assessed at different stages of e

289 Changes in mean arterial pressure were closely correlated with decreases

290 Leg blood flow and mean arterial pressure were determined, whereas leg vascular

291 ne systolic excursion and systolic pulmonary arterial pressure were measured at rest and during exerc

292 index, left ventricular dimensions, and mean arterial pressure were measured using bilateral venous o

293 The risk scores for blood pressure and mean arterial pressure were not associated with any of the ch

294 PVR and mean pulmonary arterial pressure were not significant predictors of pos

295 olume, cardiac output and reductions in mean arterial pressure were similar between age groups and co

296 ar otolith system and a decrease in the mean arterial pressure when a person stands up.

297 s from a decrease in cardiac output and mean arterial pressure, whereas treated rats survived until t

298 ficant increases (P < 0.05; n = 7-8) in mean arterial pressure, which were generally accompanied by s

299 ssin was titrated to raise and maintain mean arterial pressure within no less than 10 mm Hg from base

300 one of the other indices including pulmonary arterial pressure (WMD: -0.97 mmHg, 95%CI: -4.39, 2.44,