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1 CCR2 as a viable therapeutic target for NF1 arterial stenosis.
2 sed for studies of neointima hyperplasia and arterial stenosis.
3 for TAG functional assessment of a coronary arterial stenosis.
4 no benefit in suppressing the development of arterial stenosis.
5 ve a predisposition for premature and severe arterial stenosis.
6 ial atherosclerosis increases with degree of arterial stenosis.
7 terial definition, venous contamination, and arterial stenosis.
8 nd progression of neointimal hyperplasia and arterial stenosis.
9 e to warfarin for patients with intracranial arterial stenosis.
10 n assessing the clinical importance of renal arterial stenosis.
11 assessed for location and extent of carotid arterial stenosis.
12 + cells did not correlate with the degree of arterial stenosis.
13 t contributes to the development of critical arterial stenosis.
14 k patients with atherosclerotic intracranial arterial stenosis.
15 timal proliferation in a murine model of NF1 arterial stenosis.
19 diarrhea, sphincter of Oddi dysfunction and arterial stenosis; all responded to directed treatments.
20 hologies associated with thrombosis, such as arterial stenosis and myeloproliferative neoplasms (MPNs
21 mild, transient graft damage due to gradual arterial stenosis and the development of arterial collat
22 for months to years; LMP causes progressive arterial stenosis and thrombosis and is composed of uniq
23 iovascular abnormalities, including pulmonic arterial stenosis and ventricular septal defects accompa
25 l MR DSA improves the delineation of carotid arterial stenosis by virtually eliminating saturation ef
27 ozygous Nf1 (Nf1(+/-)) mice develop a marked arterial stenosis characterized by proliferating smooth
28 f renal transplant arteries and detection of arterial stenosis comparable with those at contrast-enha
29 grade and hemodynamic significance of renal arterial stenosis, diagnostic quality, and presence of a
30 VEC MR imaging at baseline, single coronary arterial stenosis, dipyridamole stress, and reactive hyp
32 specificity (92%) for the detection of renal arterial stenosis, including all four distal stenoses en
37 hed definitions; FCA was defined as cerebral arterial stenosis not attributed to specific diagnoses s
40 multiphoton microscopy revealed that in vivo arterial stenosis of a damaged carotid artery markedly i
41 a non-lacunar brain infarct without proximal arterial stenosis or cardioembolic sources, with a clear
42 flow reserve and (a) assessments of coronary arterial stenosis severity by quantitative coronary angi
43 renal arterial stenosis, kidneys with renal arterial stenosis showed 50% (0.14/0.28) EF reduction (P
44 ry groups, an approximately 50% reduction in arterial stenosis was observed with targeted NP treatmen
46 0.44 to 3.57+/-0.65 and 3.45+/-0.58 mm2, and arterial stenosis was reduced from 58+/-11% to 37+/-8% a
48 icity of DSC CT for detecting 50% or greater arterial stenosis were calculated by using a bivariate s
50 ading to graft loss, whereas two episodes of arterial stenosis were successfully treated with percuta
51 sal status is a risk factor for intracranial arterial stenosis when compared with premenopausal statu
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