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1 Vo2 without altering endothelial function or arterial stiffness.
2 or carbohydrates on endothelial function and arterial stiffness.
3 ressure, forearm blood flow, or estimates of arterial stiffness.
4 flavones are inversely associated with lower arterial stiffness.
5 dial viability ratio were measured to assess arterial stiffness.
6 ve velocity-a clinically relevant measure of arterial stiffness.
7 om fish may improve endothelial function and arterial stiffness.
8 k-favorably affects endothelial function and arterial stiffness.
9 rtension, in part through kidney disease and arterial stiffness.
10 dial artery applanation tonometry to measure arterial stiffness.
11 ides information on both atherosclerosis and arterial stiffness.
12 asonography, and radial tonometry to measure arterial stiffness.
13 complex interaction between ventricular and arterial stiffness.
14 lations of several biomarkers to measures of arterial stiffness.
15 icular hypertrophy and evidence of increased arterial stiffness.
16 and radial applanation tonometry to measure arterial stiffness.
17 tact parathyroid hormone predicted increased arterial stiffness.
18 its hallmark abnormality: increased central arterial stiffness.
19 sfunction are also associated with increased arterial stiffness.
20 predict changes in central pulse pressure or arterial stiffness.
21 d noradrenaline increase aortic and systemic arterial stiffness.
22 lse pressure provides a surrogate measure of arterial stiffness.
23 ows assessment of central pulse pressure and arterial stiffness.
24 amber and myocardial function, and increased arterial stiffness.
25 ts of long-term magnesium supplementation on arterial stiffness.
26 may be assessed by determining the degree of arterial stiffness.
27 n pregnancy has lasting effects on offspring arterial stiffness.
28 ve better performance over AI r in assessing arterial stiffness.
29 althy postmenopausal women is beneficial for arterial stiffness.
30 etin-2 as a link between kidney fibrosis and arterial stiffness.
31 l approach to ameliorate atherosclerosis and arterial stiffness.
32 erial blood pressure, a surrogate marker for arterial stiffness.
33 genes regulate intrinsic SM tone and thereby arterial stiffness.
34 ivity in other conditions is associated with arterial stiffness, a marker of cardiovascular risk.
35 t of asymmetric dimethyl arginine in causing arterial stiffness, a phenomenon which has been linked w
37 ting serum lipids, microvascular reactivity, arterial stiffness, ambulatory blood pressure, and marke
38 inol ameliorates endothelial dysfunction and arterial stiffness among patients without chronic kidney
41 this relationship is unclear but may involve arterial stiffness, an independent risk marker for CVD m
47 t this novel approach significantly improves arterial stiffness and central hemodynamics, which might
48 significant treatment effects were noted for arterial stiffness and central mean or 24-h ambulatory b
50 D insufficiency is associated with increased arterial stiffness and endothelial dysfunction in the co
51 ensitivity, adipose tissue inflammation, and arterial stiffness and exerts a beneficial shift in gut
57 pulse wave velocity (PWV) reflects increased arterial stiffness and is an established cardiovascular
61 ogenesis and maintenance of hypertension and arterial stiffness and more novel cardiovascular risk fa
62 of calibrated tonometry and pulsed Doppler, arterial stiffness and pulsatile hemodynamics were asses
63 uggest distinct polygenic susceptibility for arterial stiffness and salt-sensitive hypertension in Da
64 patients and looked for associations between arterial stiffness and SCD-related vascular complication
65 based studies have evaluated the genetics of arterial stiffness and separate mean and pulsatile compo
66 data for exercise training's improvement of arterial stiffness and system inflammation and reduction
67 These data, which include direct measures of arterial stiffness and thickness, suggest that higher in
71 se pressure (bPP), aoPP, and all measures of arterial stiffness and wave reflections were higher in H
72 sitive patients with AAV are associated with arterial stiffness and whether treatment with valaciclov
73 ed preload, and decreases in blood pressure, arterial stiffness, and afterload as well, thereby impro
76 ed by a forward pressure wave and related to arterial stiffness, and augmentation pressure (AP), thou
82 icate that AASV is associated with increased arterial stiffness, and that stiffness correlates with t
83 nifedipine, and placebo on proteinuria, BP, arterial stiffness, and various cardiovascular biomarker
84 ative stress, high rates of hypertension and arterial stiffness; and those without constitutive expre
86 (AIx), which provides a measure of systemic arterial stiffness, aortic stiffness and central arteria
88 Elevation in plasma ET-1 and increases in arterial stiffness are potential important mechanisms un
90 t gaps were female; P < 0.05), and increased arterial stiffness (arterial stiffness index, 8.5 4.6 in
91 her studies examining the potential value of arterial stiffness as a screening tool to guide initiati
92 s index and, in a subpopulation of patients, arterial stiffness as measured by pulse wave velocity.
93 also documented the prognostic importance of arterial stiffness (AS) in various populations as an ind
94 24 wk in overweight and obese adults reduces arterial stiffness, as estimated by a decrease in PWVc-f
95 associations with myocardial infarction and arterial stiffness, as well as coronary artery calcifica
99 d whether this might reflect consequences of arterial stiffness, atherosclerosis, or both is unknown.
100 r system--platelet and endothelial function, arterial stiffness, atherosclerosis, oxidative stress, i
101 history of AF, liver/kidney transplantation, arterial stiffness, atherosclerotic plaques in the aorta
102 rom the lower body, resulting from increased arterial stiffness, augments central aortic pressure and
104 carotid intima-media thickness (a measure of arterial stiffness), brachial artery reactivity (both fl
107 maging and echocardiography, and we assessed arterial stiffness by carotid-femoral pulse wave velocit
109 ort a role for the endothelium in regulating arterial stiffness by release of vasoactive mediators.
110 ort a role for the endothelium in regulating arterial stiffness by release of vasoactive mediators.
111 function (primary outcome), blood pressure, arterial stiffness, cardiac autonomic function, platelet
112 cause of age-associated increases in central arterial stiffness, cardiovagal BRS was expressed with b
114 sma lipids, glucose, C-reactive protein, and arterial stiffness [carotid-femoral pulse wave velocity
115 ual flavonoid intakes and direct measures of arterial stiffness, central blood pressure, and atherosc
116 ally improve central hemodynamics and reduce arterial stiffness compared with conventional beta-block
117 ive protein levels, homocysteine levels, and arterial stiffness compared with those without LV hypert
118 scular endothelial dysfunction and increased arterial stiffness contribute to increased cardiovascula
123 ntation index (AIx) is a measure of systemic arterial stiffness derived from the ascending aortic pre
125 y (2-8 repeated measures, median of 5), and arterial stiffness estimates of 3 large arteries (ultras
128 king blood pressure and different aspects of arterial stiffness from childhood onward, with an aim of
129 , and magnetic resonance imaging, we related arterial stiffness, GFR, urinary albumin excretion, and
133 hip between elevated blood pressure (BP) and arterial stiffness in a biracial (black-white) cohort of
134 sease activity, endothelial dysfunction, and arterial stiffness in a cohort of rheumatoid arthritis (
136 id synthesis ameliorates atherosclerosis and arterial stiffness in apolipoprotein E(-/-) mice and rab
137 rs (ARBs) improves left ventricular mass and arterial stiffness in early-stage chronic kidney disease
140 to carotid atherosclerosis and to increased arterial stiffness in hypertensive patients, independent
141 (Predictive Values of Blood Pressure and Arterial Stiffness in Institutionalized Very Aged Popula
144 medullin, and homocysteine concentrations to arterial stiffness in participants in the Framingham Hea
145 te the relationship between inflammation and arterial stiffness in patients with antineutrophil cytop
147 for quantitative trait loci (QTLs) affecting arterial stiffness in six-week old F2 (Dahl S x R)-inter
151 NAFLD is only associated with increased arterial stiffness in the presence of the "high risk" me
154 t nitric oxide (NO) acting locally regulates arterial stiffness in vivo, and the aim of this experime
156 dex and augmentation pressure, correlates of arterial stiffness, in a cohort of elderly men in the Bo
161 was to ascertain if NAFLD is associated with arterial stiffness, independent of cardiometabolic facto
163 P < 0.05), and increased arterial stiffness (arterial stiffness index, 8.5 4.6 in patients with gaps
166 deposition changes over time and generalized arterial stiffness indicated a relationship between the
168 y improved endothelium-dependent dilatation, arterial stiffness, intima media thickness and blood pre
180 with age in nondemented individuals and that arterial stiffness is strongly associated with the progr
181 wave velocity (PWV), a noninvasive index of arterial stiffness, is a predictor of the longitudinal c
182 ne whether pulse pressure (PP), a measure of arterial stiffness, is an independent predictor of the i
183 A high ABI, a marker of lower extremity arterial stiffness, is associated with CVD events and mo
184 ity-dwelling older adults, aPWV, a marker of arterial stiffness, is associated with higher CV mortali
185 reflections and PP amplification, but not of arterial stiffness, is observed when hypertension is stu
187 ion was evaluated by 24-hour blood pressure, arterial stiffness, low (LF)- and high (HF)-frequency he
188 pathophysiological processes that determine arterial stiffness may help guide therapeutic approaches
191 s of cardiovascular disease, suggesting that arterial stiffness, measured via carotid-femoral pulse w
192 ardiovascular evaluation, including imaging, arterial stiffness measurements, and biochemical studies
193 to examine the associations between baseline arterial stiffness measures (exposure variables) and inc
197 hange BP (office BP and 24-h ambulatory BP), arterial stiffness, nitric oxide, endothelin 1, or blood
199 but no significant difference in the odds of arterial stiffness (OR, 1.07; 95% CI, 0.63-1.84; P = .80
202 vestigate the effects of oral astaxanthin on arterial stiffness, oxidative stress, and inflammation i
203 xanthin (12 mg/d for 12 mo) had no effect on arterial stiffness, oxidative stress, or inflammation in
204 s indicated that only female sex (P < 0.02), arterial stiffness (P < 0.002), and atherosclerotic plaq
205 d pressure variability (P = .01), peripheral arterial stiffness (P = .02), carotid intima-media thick
206 Furthermore, patients demonstrated higher arterial stiffness (P = 0.005), LF and HF heart rate var
208 tients with coronary heart disease (CHD) and arterial stiffness progression in young healthy subjects
209 nificantly and independently associated with arterial stiffness progression, incident subclinical ath
211 ustment for clinical factors known to modify arterial stiffness, proximal thoracic aortic stiffness w
213 se pressure (cPP), their relation to central arterial stiffness (pulse wave velocity [PWV]) and arter
214 na, heart failure, or stroke) in relation to arterial stiffness (pulse wave velocity [PWV]), wave ref
215 (laser Doppler imaging with iontophoresis), arterial stiffness (pulse wave velocity and analysis), b
216 monitor), renin and aldosterone levels, and arterial stiffness (pulse wave velocity and augmentation
218 icular mass (cardiac magnetic resonance) and arterial stiffness (pulse wave velocity/analysis, aortic
219 an 8.2 +/- 3.2% vs. 8.1 +/- 3.3%), and lower arterial stiffness (pulse wave velocity: mean 6.99 +/- 1
220 (laser Doppler imaging with iontophoresis), arterial stiffness [pulse wave velocity, pulse wave anal
222 A demonstrated comparable relationships with arterial stiffness (r(2) = 0.616 for homocysteine and r(
223 increased flow-mediated dilatation, reduced arterial stiffness, reduced mean arterial and diastolic
225 function, with increases in small and large arterial stiffness representing a characteristic change
229 ular disease, factors capable of influencing arterial stiffness, such as exercise and the use of reni
230 than those with CRI (P < 0.001) and greater arterial stiffness than both CRI patients and control su
232 evalence of left ventricular hypertrophy and arterial stiffness that confer an adverse prognosis.
233 ce imaging assessment of cardiac morphology, arterial stiffness (total aortic compliance and arterial
235 osclerosis is, in part, related to increased arterial stiffness, vascular dysfunction, elevated oxida
236 greater insight into hemodynamics of altered arterial stiffness versus impaired peripheral resistance
239 To look at effects on subclinical disease, arterial stiffness was evaluated at baseline and after 5
243 n of arterial pulse pressure (a surrogate of arterial stiffness) was linked to deterioration of the w
244 duction in pulse-wave velocity, a measure of arterial stiffness, was associated with a decrease in AD
245 Pulse pressure/stroke volume, a measure of arterial stiffness, was higher in participants with DM (
251 T or BP, clinically relevant improvements in arterial stiffness were observed; equol producers were p
253 gether with augmentation index, a measure of arterial stiffness, were determined before and at interv
256 igate the association of local and segmental arterial stiffness with incident cardiovascular events a
257 derives performance improvement in assessing arterial stiffness, with a stronger correlation with cfP
258 elected nucleotide excision repair genes and arterial stiffness within the AortaGen Consortium and fo
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