ライフサイエンスコーパス: arterialization

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1 nspositions, 6 renoportal anastomoses, and 1 arterialization).

2 ory response in a canine model of vein graft arterialization.

3 e inflammatory cells that follows vein graft arterialization.

4 ta provide insights into miRNA regulation of arterialization and highlight the importance of vascular

5 al malignancy are associated with increasing arterialization and loss of portal blood supply; therefo

6 tudies suggest the role of Alk1 signaling in arterialization and remodeling of arteries.

7 Thus, this novel mouse model of venous arterialization displays anatomical and cellular feature

8 Although the anatomical changes of venous arterialization have been well characterized, the molecu

9 and may lead to novel approaches to improve arterialization in human disease.

10 Here, we present a novel model of venous arterialization in mice wherein the external jugular vei

11 suggesting that Notch can no longer promote arterialization in the cardinal vein during this develop

12 ediated vascular defects were accompanied by arterialization, including ectopic venous expression of

13 This treatment effectively abolished arterialization-induced ERK-1/2 activation, decreased me

14 Percutaneous in situ coronary venous arterialization is in its infancy, but may be able to tr

15 The vascular injury caused by arterialization is responsible for vein graft intimal hy

16 Percutaneous in situ venous arterialization may be an effective therapy for diffuse,

17 niques (portacaval hemitransposition, portal arterialization, multivisceral transplantation) are asso

18 Venous arterialization occurs when a vein segment is transposed

19 by inhibiting Notch signaling and subsequent arterialization of endothelial cells, yet nothing is kno

20 supported and survive using the technique of arterialization of the venous network.

21 gy revealed severely dilated lymphatics and "arterialization" of the pulmonary veins in those with th

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