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1 s with pituitary tumor, medulloblastoma, and arteriovenous malformation.
2 nt feeding artery, which was consistent with arteriovenous malformation.
3 mab to treat the macular oedema in a case of arteriovenous malformation.
4 ted] as assessed by development of pulmonary arteriovenous malformation.
5 pared with those presenting with hemorrhagic arteriovenous malformation.
6 emorrhage after embolization of a cerebellar arteriovenous malformation.
7 in 18 patients with head and neck tumors and arteriovenous malformations.
8 well as the presence of multiple and massive arteriovenous malformations.
9 on congenital heart disease and intracranial arteriovenous malformations.
10 ular assist devices (LVADs) and is caused by arteriovenous malformations.
11 ication of nascent blood vessels and develop arteriovenous malformations.
12 , congestive heart failure due to a cerebral arteriovenous malformation (1), tracheal occlusion incur
14 ovascular accident caused by a right pontine arteriovenous malformation and destruction of the right
15 Mice mutant for Notch1 and Notch3 develop arteriovenous malformations and display hallmarks of the
17 f some other conditions, such as parenchymal arteriovenous malformations and intracerebral hemorrhage
18 y related Fox transcription factors, exhibit arteriovenous malformations and lack of induction of art
21 terized by multisystemic vascular dysplasia, arteriovenous malformations, and focal dilatation of pos
23 estigate the proper management of unruptured arteriovenous malformations, and the key factors in endo
26 el density was comparable for hemangioma and arteriovenous malformation (AVM) but significantly great
27 n about whether to treat an unruptured brain arteriovenous malformation (AVM) depends on a comparison
34 better outcome prediction for patients with arteriovenous malformation (AVM)-related intracerebral h
48 age (ICH) for patients with unruptured brain arteriovenous malformations (AVMs) in the natural course
49 n about the frequency and clinical course of arteriovenous malformations (AVMs) of the brain in adult
61 nterventional treatment for unruptured brain arteriovenous malformations (bAVMs) is uncertain because
63 cally associated with capillary malformation-arteriovenous malformation, but sporadic reports of lymp
64 s achieved satisfactory obliteration of deep arteriovenous malformations, but with increased actuaria
69 RASA1 mutations cause capillary malformation-arteriovenous malformation (CM-AVM); whether it also fun
70 rhage, including age at initial diagnosis of arteriovenous malformation, co-existing extranidal aneur
74 or stroke in patients with unruptured brain arteriovenous malformations followed up for 33 months.
79 data regarding factors associated with brain arteriovenous malformation hemorrhage and different trea
80 al and genetic factors associated with brain arteriovenous malformation hemorrhage, as well as studie
82 ges but with concerns about the formation of arteriovenous malformation in the lung excluded from hep
84 emorrhagic telangiectasia, especially occult arteriovenous malformations in visceral organs that may
85 ing effective multidisciplinary treatment of arteriovenous malformations, including those previously
86 This resulted in defective angiogenesis and arteriovenous malformations, leading to embryonic lethal
87 moptysis caused by thoracic vascular injury, arteriovenous malformation, leaking thoracic aneurysm wi
88 tracranial stenosis, intracranial aneurysms, arteriovenous malformations, malignant gliomas, and meni
90 dy, 18 patients with tumors (n = 14), facial arteriovenous malformations (n = 3), and vertebral arter
93 Final histology showed that 2 patients had arteriovenous malformations: one had a benign hemangioma
99 pulmonary angiography in detecting pulmonary arteriovenous malformation (PAVM) in children with cavop
102 ss is a recognized complication of pulmonary arteriovenous malformations (PAVMs) that allow systemic
106 These mice die by mid-gestation with severe arteriovenous malformations resulting from fusion of maj
108 areful angiographic assessment of individual arteriovenous malformations should be performed before e
110 ability, progressive cyanosis from pulmonary arteriovenous malformations, systemic to hepatic venoven
111 angiodysplastic lesions (telangiectases and arteriovenous malformations) that affect many organs.
112 st the hypothesis that, for unruptured brain arteriovenous malformations, there is no difference betw
113 a lipomatous mass, an epidermoid naevus and arteriovenous malformation tissue, all of which were sam
114 levant for understanding the causes of human arteriovenous malformations, tumor angiogenesis, and dia
116 hese conditions include renal hemangiomas or arteriovenous malformations, ureteropelvic junction obst
117 ients (>/=18 years) with an unruptured brain arteriovenous malformation were enrolled into this trial
119 ), the hallmark vascular lesion is termed an arteriovenous malformation, which involves the direct co
120 stroke in patients with an unruptured brain arteriovenous malformation who are allocated to either m
121 n 21 subjects with epilepsy, brain tumor, or arteriovenous malformation who had undergone IAP and MEG
122 icentre randomized trial of unruptured brain arteriovenous malformations) will be of major importance
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