ライフサイエンスコーパス: arthropathy

1 ead to chronic disabilities with haemophilic arthropathy.

2 th effusion, meniscal tear, and degenerative arthropathy.

3 and knees, and may be associated with severe arthropathy.

4 or as seen in hemophilia in general leads to arthropathy.

5 and prevention of foot ulcers and neurogenic arthropathy.

6 or anular hyperintense zone (HIZ), and facet arthropathy.

7 ritical to the development of B27-associated arthropathy.

8 ubrication is an early event in inflammatory arthropathy.

9 as it had been proposed to do in hemophilic arthropathy.

10 thritis (RA) is the most common inflammatory arthropathy.

11 nfected individuals who presented with acute arthropathy.

12 d the relationship of chopsticks use to hand arthropathy.

13 al skin rash, and a characteristic deforming arthropathy.

14 and are associated with severe degenerative arthropathy.

15 y susceptible mouse can mimic B27-associated arthropathy.

16 J, and BALB/cJ beta2m(0) mice developed this arthropathy.

17 mino-transferase values, and hemochromatotic arthropathy.

18 l) and suffers from hemarthroses and chronic arthropathy.

19 tive arthritis, Reiter's syndrome, and other arthropathies.

20 nt enzymes and inhibitors in the destructive arthropathies.

21 ing of cartilage-subchondral interactions in arthropathies.

22 imulated renewed interest in crystal-induced arthropathies.

23 dvances in the management of crystal-induced arthropathies.

24 ial fluid samples from patients with various arthropathies.

25 argets for management of the crystal-induced arthropathies.

26 om patients with OA or other noninflammatory arthropathies.

27 ncluding 14 patients with spinal neuropathic arthropathy (12 radiographic, seven CT, and six MR studi

28 Of the 20, 5 had arthropathy, 5 had gastrointestinal symptoms, 4 had a ri

29 axis use and its impact on key indicators of arthropathy across the life-span among participants with

30 s can reliably differentiate between chronic arthropathies and inflammatory conditions with discrete

31 II activity may prove beneficial in limiting arthropathies and other degenerative bone diseases.

32 ails in patients with more severe peripheral arthropathy and axial involvement, and alternative treat

33 as a pathogenic factor in both degenerative arthropathy and chondrocalcinosis in aging.

34 ion might be linked to development of severe arthropathy and chronic infection.

35 ted with human aging-associated degenerative arthropathy and directly stimulate chondrocalcinosis, ma

36 lomerulonephritis, liver fibrosis, deforming arthropathy and increased anti-DNA antibodies.

37 s of TRPV4 function is associated with joint arthropathy and osteoarthritis.

38 rol bleeding and complications, particularly arthropathy and physical disability.

39 autosomal-dominant human motor neuropathies, arthropathies, and skeletal malformations of varying sev

40 and chronic red cell aplasia, fetal hydrops, arthropathy, and other disorders.

41 stosis, crystalline arthropathy, neuropathic arthropathy, and tendinopathy.

42 sis; multicentric osteolysis, nodulosis, and arthropathy; and Winchester syndromes, skeletal dysplasi

43 The crystal-induced arthropathies are characterized by self-limiting episode

44 is, including poor general health, diabetes, arthropathies, arrhythmias, impotence, and skin pigmenta

45 e participant, who had substantial, advanced arthropathy at baseline, administered factor for bleedin

46 Patients with arthropathy, B. cepacia infection, or younger age derive

47 members had multiple shoulder, hip, and knee arthropathies, beginning in the pre-teen years and conti

48 atients with already existing advanced joint arthropathy benefit from tertiary prophylaxis with signi

49 e acute arthritis and occasionally a chronic arthropathy, both in children and adults.

50 ay that has been associated with neuropathic arthropathy but has not been assessed in knee osteoarthr

51 ints is closely associated with OA and other arthropathies, but the precise role of HA in arthritis p

52 Rheumatoid arthritis is a destructive arthropathy characterized by chronic synovial inflammati

53 The crystal deposition arthropathies comprise a host of disorders that may occu

54 f-function mutations result in camptodactyly-arthropathy-coxa vara-pericarditis (CACP) syndrome, whic

55 n autosomal recessive disorder camptodactyly-arthropathy-coxa vara-pericarditis syndrome (CACP).

56 e autosomal recessive disorder camptodactyly-arthropathy-coxa vara-pericarditis syndrome (CACP; MIM 2

57 omal recessive, human disorder camptodactyly-arthropathy-coxa vara-pericarditis syndrome.

58 athy and vascular disease (DI), with Charcot arthropathy (DA), and without complications (D), and in

59 ebilitating, crippling arthritis, hemophilic arthropathy, develops.

60 Tendon diseases are among the most common arthropathy disorders; thus knowledge of tendon gene reg

61 lupus erythematosus, and a group of chronic arthropathies, expression of CD44-dependent primary adhe

62 Not surprisingly, osteoarthritis and crystal arthropathy frequently coexist.

63 ks may help differentiate spinal neuropathic arthropathy from infection.

64 te inflammation and tissue damage in crystal arthropathies have been further clarified.

65 vival of less than 50% without B. cepacia or arthropathy have improved survival.

66 ticarial rash, aseptic meningitis, deforming arthropathy, hearing loss, and mental retardation.

67 oad suggests the negative role of hemophilic arthropathy in bone density loss.

68 Progression of preexisting chronic arthropathy in one participant was the only serious adve

69 Joint arthropathy in primary prophylaxis develops over many ye

70 Effective ways to prevent arthropathy in severe hemophilia are unknown.

71 se acute arthritis, and occasionally chronic arthropathy, in infected adults.

72 any of the first descriptions of crystalline arthropathies, including gout, calcium pyrophosphate dep

73 olderia cepacia, and cystic fibrosis-related arthropathy increased the post-transplantation hazard of

74 s; unspecified/mixed CTD; other inflammatory arthropathy), increased ASCVD rates were found in nearly

75 or effusion, meniscal tear, and degenerative arthropathy, independent of one another.

76 esting as cutaneous vasculitis, inflammatory arthropathy, intermittent polyclonal lymphoproliferation

77 is and are often associated with destructive arthropathies involving cartilage degeneration.

78 Distal interphalangeal (DIP) joint arthropathy is characteristic of both psoriatic arthriti

79 f events from joint bleeding to synovitis to arthropathy is well documented, the component or compone

80 = 0.55), Modic changes (kappa = 0.59), facet arthropathy (kappa = 0.54), and posterior HIZ (kappa = 0

81 = 0.74), Modic changes (kappa = 0.64), facet arthropathy (kappa = 0.69), and posterior HIZ (kappa = 0

82 te and chronic parvoviral B19 infections and arthropathy may provide insights into virus-host interac

83 rding rubella as a possible cause of chronic arthropathy, more negative evidence accumulated with two

84 (2- to 2.3-fold), but was not affected by an arthropathy mutation (1.1-fold).

85 c entrapment (n = 19), neoplasm (n = 9), and arthropathy (n = 11).

86 diopathic skeletal hyperostosis, crystalline arthropathy, neuropathic arthropathy, and tendinopathy.

87 Hemophilic arthropathy occurs in all patients with severe and moder

88 An unusual destructive and hypertrophic arthropathy of the atlantoaxial joint in calcium pyropho

89 images in patients with diabetic neuropathic arthropathy of the foot were examined by two reviewers i

90 e of any increased risk of new onset chronic arthropathies or neurologic conditions in women receivin

91 f Philadelphia were examined for evidence of arthropathy or arthritis.

92 Transplanted adults with B. cepacia, arthropathy, or a 5-year predicted survival of greater t

93 elated systemic pathology (e.g., early onset arthropathy, premature aging, ovulation, late onset of p

94 Degenerative arthropathy resulting from recurrent hemarthrosis remain

95 Patients with the systemic inflammatory arthropathy, rheumatoid arthritis (RA), were evaluated a

96 Joint arthropathy secondary to recurrent hemarthroses remains

97 nti-IL-17 antibody prevented the destructive arthropathy seen in vaccinated and challenged IFN-gamma(

98 CHIKV disease, usually a self-limiting viral arthropathy, share multiple inflammatory processes.

99 The arthropathy showed many features characteristic of human

100 These include viral arthropathies such as O'nyong-nyong, chikungunya, and de

101 ssociated with inflammatory and degenerative arthropathies such as rheumatoid arthritis.

102 were less likely to be seen for nontraumatic arthropathies than nonpostpartum women (4.5% vs 7.2%, P=

103 rthritis is a heterogeneous group of chronic arthropathies that are influenced by complex genetic and

104 described as an inflammatory crystal-induced arthropathy that afflicts peripheral joints.

105 to end-stage joint degeneration (hemophilic arthropathy), the major morbidity of hemophilia.

106 Among the 124 subjects with chronic arthropathy, the onset occurred between 1 week and 6 wee

107 joint has been referred to as an "analgesic arthropathy." This article discusses common treatments o

108 ) were classified as having undifferentiated arthropathy (UA).

109 collateral ligament injury, and degenerative arthropathy was recorded.

110 eumatoid arthritis [RA], 20 OA, and 27 other arthropathies) was evaluated.

111 osteoarthritis (OA) or other noninflammatory arthropathies were used as controls.

112 findings for diagnosis of spinal neuropathic arthropathy were vacuum disk on radiographs and CT image

113 me assessment as an indicator of early joint arthropathy when followed by ultrasound or magnetic reso

114 Gout is a form of crystal arthropathy where monosodium urate (MSU) crystals deposi

115 y represent a familial type of pyrophosphate arthropathy with a phenotype that includes peripheral an

116 d rubella vaccine in the US and some chronic arthropathy with an onset between 1 week and 6 weeks aft