ライフサイエンスコーパス: arthropod vector

1 ersal of cells within the mammalian host and arthropod vector.

2 s impacts B. burgdorferi colonization of its arthropod vector.

3 context of the gut epithelial barrier of an arthropod vector.

4 during the transition between human host and arthropod vector.

5 by a rickettsial pathogen to survive in its arthropod vector.

6 AP) restricts bacterial colonization of this arthropod vector.

7 xic levels of heme present in the gut of the arthropod vector.

8 y are initiated to allow colonization of the arthropod vector.

9 tant to B. burgdorferi's survival within its arthropod vector.

10 nd the maintenance of spirochetes within the arthropod vector.

11 migrate to the salivary gland, and exit the arthropod vector.

12 mammalian hosts, but is downregulated in the arthropod vector.

13 er potential for acquisition of virus by the arthropod vector.

14 emerging flavivirus transmitted primarily by arthropod vectors.

15 ard vertebrate hosts and their blood-feeding arthropod vectors.

16 approach; IVM strategies aim only to control arthropod vectors.

17 ens in either their vertebrate reservoirs or arthropod vectors.

18 that could also apply to other blood-feeding arthropod vectors.

19 mals, and plants are transmitted by specific arthropod vectors.

20 rate viral pathogens that are transmitted by arthropod vectors.

21 fe, and highly unlikely to be transmitted by arthropod vectors.

22 that are transmitted between vertebrates by arthropod vectors.

23 imal reservoirs and transmitted to humans by arthropod vectors.

24 lichiosis, which is naturally transmitted by arthropod vectors.

25 ition to harboring beneficial microbes, many arthropods (vectors) also transmit pathogens to the anim

26 Filarial nematodes require both an arthropod vector and a mammalian host to complete their

27 isease, must adapt to two diverse niches, an arthropod vector and a mammalian host.

28 between a human pathogenic bacterium and its arthropod vector and delineate what we believe to be a n

29 st adapt to the distinct environments of its arthropod vector and mammalian host during its complex l

30 Thus survival of this spirochaete in an arthropod vector and mammalian host requires that it can

31 capabilities during its enzootic cycle in an arthropod vector and mammalian host.

32 ue as B. burgdorferi transitions between its arthropod vector and mammalian host.

33 disease, selectively expresses genes in the arthropod vector and mammalian host.

34 multiple adhesins to interact with both the arthropod vector and mammalian hosts it colonizes.

35 vironmental signals as it cycles between the arthropod vector and mammalian hosts, including temperat

36 ession in response to changes imposed by its arthropod vector and mammalian hosts.

37 gy employed by B. henselae to survive in the arthropod vector and the mammalian host.

38 throughout the spirochaete life cycle in the arthropod vector and the murine host.

39 burgdorferi shuttles back and forth between arthropod vector and vertebrate host, it encounters vast

40 c agent of Lyme disease, persists in both an arthropod vector and vertebrate hosts, usually wild rode

41 Establishing the source of alpha-gal in arthropod vectors and the immune response to vector bite

42 ow fever virus (YFV) are transmitted between arthropod vectors and vertebrate hosts.

43 al transfer between a vertebrate host and an arthropod vector, and acquisition of virus from an infec

44 of the Lyme disease agent within the feeding arthropod vector, and strategies for interfering with th

45 development within and transmission from the arthropod vector are known for many bacterial and protoz

46 ruses are transmitted via a diverse range of arthropod vectors, as well as rodents, and have establis

47 isolating viruses from humans, animals, and arthropod vectors at field stations in Latin America, Af

48 In the gut of the obligately hematophagous arthropod vector, bartonellae are exposed to concentrati

49 nce of working with viruses originating from arthropod vector cells in investigations of the cell bio

50 ently transit between its mammalian host and arthropod vector during tick feeding.

51 direct transmission, arboviruses utilize an arthropod vector (e.g., mosquitos, sandflies, and ticks)

52 adaptation of B. quintana to the hemin-rich arthropod vector environment.

53 Thus, transit in its arthropod vector exposes Y. pestis to favourable conditi

54 Saliva from arthropod vectors facilitates blood feeding by altering

55 ble component of Rickettsia biology involves arthropod vectors: for instance, typhus group rickettsia

56 Recently, arthropod vectors have been involved in emerging anaphyl

57 tablishment of a persistent infection in the arthropod vector; however, the nature of the virus-arthr

58 tracellular bacteria that cause a variety of arthropod vectored human diseases.

59 the evolution of the DTN in a diverse set of arthropod vectors, including ticks, and its role in prot

60 ued expansion of the range and number of its arthropod vectors increases the likelihood that OROV wil

61 nd C; as B. burgdorferi transitions from its arthropod vector into mammalian tissue, ospC is upregula

62 Efficient transmission of pathogens by an arthropod vector is influenced by the ability of the pat

63 of the mammalian host and colonization of an arthropod vector is required for the ongoing transmissio

64 ature in an enzootic cycle that involves the arthropod vector Ixodes scapularis and mammalian reservo

65 first encounters natural antibodies when its arthropod vector, Ixodes scapularis, begins feeding on a

66 Control of feline infestation with this arthropod vector may provide an important strategy for t

67 virus particles produced in and delivered by arthropod vectors may preferentially target vertebrate h

68 ng different stages of its life cycle in the arthropod vector or the mammalian host.

69 ression in response to signals unique to its arthropod vector or vertebrate hosts.

70 others circulate among multiple hosts, need arthropod vectors, or can survive in environmental reser

71 nimal cross-reactivity to mammalian host and arthropod vector organisms.

72 of infectivity of the mammalian host for the arthropod vector, plague epizootics require a high flea

73 stand their dynamics of infection in natural arthropod vector populations.

74 ty, induction of the PhoP-PhoQ system in the arthropod vector prior to transmission may preadapt Y. p

75 ion of sfRNA for flavivirus infection of the arthropod vector, providing an explanation for the stric

76 attenuated isolate of B. burgdorferi by the arthropod vector results in the generation of spirochete

77 Whether arthropod vectors retain competence for transmission of

78 ility, and specific loci that correlate with arthropod vector, serotype, and disease severity.

79 e demonstrate the existence of two plausible arthropod vectors, specifically reptile ticks.

80 viruses are transmitted by distantly related arthropod vectors such as mosquitoes (class Insecta) and

81 re transmitted to vertebrate hosts by biting arthropod vectors such as mosquitoes, ticks, and midges.

82 in both an immunocompetent mammal and in an arthropod vector suggests that they have evolved elegant

83 Historical exposure to animals and arthropod vectors that can harbor hemotropic Mycoplasma

84 acteria and a variety of vertebrate host and arthropod vector tissues.

85 in the transmission of spirochetes from the arthropod vector to the mammalian host.

86 to successfully make the transition from the arthropod vector to the vertebrate host.

87 Arthropod vectors transmit African and American trypanos

88 aviruses establish a persistent infection in arthropod vectors which is essential for the effective t

89 less clear, especially for parasites such as arthropod vectors, which generally spend only a short ti

90 context, that the core genome evolved in the arthropod vector with differential regulation, allowing

91 us Orthobunyavirus, which are transmitted by arthropod vectors with a broad cellular tropism in vitro