ライフサイエンスコーパス: articular surface

1 have revealed chondrocyte progenitors at the articular surface.

2 fully covered with hyaline cartilage in the articular surface.

3 ulture and the depth of the explant from the articular surface.

4 tilage, with T2 values increasing toward the articular surface.

5 was increased only in chondrocytes near the articular surface.

6 subchondral bone segment from the remaining articular surface.

7 s increase in T2 from the radial zone to the articular surface.

8 ne and fluid within the line extending to an articular surface.

9 at was highest near the subchondral bone and articular surfaces.

10 yroid hormone-related peptide (PTHrP) at the articular surfaces.

11 Gd(DTPA)2- penetrated cartilage from the articular surface after intraarticular injection and fro

12 on, peak cartilage shear is highest near the articular surface and decreases markedly with depth.

13 lubricant, Exz was highest (0.056) near the articular surface and diminished monotonically with dept

14 rt by proteoglycan 4 (PRG4) molecules at the articular surface and in synovial fluid.

15 the transport of active TGF-beta across the articular surface and into the cartilage layer.

16 sential for maintaining the growth plate and articular surface and is required for sustaining trabecu

17 on of Ihh signaling upregulates PTHrP at the articular surface and prevents chondrocyte hypertrophy i

18 changes of cartilage collagen begin near the articular surface and progress to the deeper cartilage w

19 hat collagen matrix disruption starts at the articular surface and progresses into the deeper layers

20 involved the layer of collagen covering the articular surface and protecting cartilage from degradat

21 r intraarticular injection and from both the articular surface and the subchondral bone after intrave

22 IDEAL GRASS during the second to grade each articular surface and to determine the presence of menis

23 dy was sensitive to mild degeneration at the articular surface and was insensitive to changes associa

24 and localized by immunohistochemistry at the articular surface and within chondrocytes in explants an

25 nts, better delineation of the plantar plate articular surface, and better evaluation of the integrit

26 ts were subdivided into level 1, with intact articular surface, and level 2, containing middle and de

27 ween juxtaposed skeletal elements, irregular articular surfaces, and hypoplasia of ligaments.

28 ], and old [>/=60 years]), and a macroscopic articular surface appearance that was either normal or m

29 mature peptide at the location of the future articular surface but not in the future joint space.

30 omous expression of diphtheria toxin to kill articular surface chondrocytes in mice and determined th

31 ence or absence of cartilage lesions on each articular surface, first by using the routine MR protoco

32 Near the articular surface, G was lower with degeneration (0.06 M

33 hat clusterin expression was confined to the articular surface in both immature and mature samples.

34 Furthermore, destruction of the articular surface in long bones and premature fusion of

35 s for high signal intensity extending to the articular surface in the posterior third of the labrum,

36 ical intervention because of collapse of the articular surface in the treatment area.

37 Samples with articular surfaces in apposition exhibited little integr

38 surgery, osteoarthritis, joint instability, articular surface incongruity or dysplasia, disturbances

39 We propose that GPI-anti-GPI complexes on articular surfaces initiate an inflammatory cascade via

40 ular cartilage were harvested, some with the articular surface intact and others without.

41 markedly increased cartilage shear near the articular surface may contribute to progressive cartilag

42 obtained from the superficial (including the articular surface), middle, and deep layers were culture

43 We recently showed that the entire articular surface of a synovial joint can regenerate by

44 est that modulation of FN synthesis near the articular surface of cartilage may be one of the factors

45 Histologic staining revealed changes in the articular surface of level 1 explants following injury,

46 solute adsorption and distribution near the articular surface of mechanically injured cartilage.

47 dings are located at the lateral edge of the articular surface of the distal phalanx.

48 evaluated at separate sittings to grade each articular surface of the hip joint.

49 We tested the hypothesis that the articular surface of the synovial joint can regenerate w

50 Our findings suggest that the entire articular surface of the synovial joint can regenerate w

51 Each articular surface of the tibiofemoral joint was graded a

52 shed protocol, solute distributions near the articular surface of three commonly used fluorophores (f

53 The entire articular surface of unilateral proximal humeral condyle

54 moral head containment and congruency of the articular surfaces of the hip.

55 of abnormal signal intensity extending to an articular surface on intermediate-weighted images, (b) a

56 and 3), predict the effect of depth from the articular surface on pi(PG) in human aging.

57 e bursal surface partial-thickness tears, 10 articular surface partial-thickness tears, and 14 patien

58 murine growth plates, chondrocytes near the articular surface (periarticular chondrocytes) prolifera

59 xpression was observed in all samples at the articular surface, regardless of OA severity.

60 ath was more pronounced, particularly in the articular surface region.

61 Biological resurfacing of entire articular surfaces represents an important but challengi

62 t, orthokeratotic scales that fractured over articular surfaces, secondary to bending.

63 retrospectively and independently graded all articular surfaces seen at MR imaging by using a similar

64 Chondrocytes from the articular surface showed approximately twice the number

65 on MR images, and the presence or absence of articular surface tear was recorded.

66 ema was also seen beneath four (1.4%) of 238 articular surfaces that appeared normal at arthroscopy.

67 The chondrocytes of the presumptive articular surface therefore appear to arise directly fro

68 sGAG loss, these changes may predispose the articular surface to further softening and tissue damage

69 ndicate a physiologic response of the entire articular surface to repair the damaged matrix, which is

70 es was determined for the femoral and tibial articular surfaces (total maximum score of 60).

71 The articular surface was replaced by bone or hypertrophic c

72 Each articular surface was then evaluated at arthroscopy.

73 With PBS as lubricant, Exz values near the articular surface were approximately 50% higher than tho

74 All articular surfaces were graded at arthroscopy by using t

75 Cartilage disks with intact articular surfaces were harvested from immature bovines.

76 Lesions to the articular surface, which are thought to progress to OA,