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5 uniquely activated genes associated with the aryl hydrocarbon hydroxylase (Ah) receptor (Cyp1a1, Cyp1
7 hat HIF-2alpha and its dimerization partner, aryl hydrocarbon nuclear transferase localize to the ROR
8 o the known targets p27 and p57, we identify Aryl hydrocarbon nuclear translocator (Arnt) messenger R
10 murine models, deletion of the gene encoding aryl hydrocarbon nuclear translocator (ARNT, also known
11 stent with their role in detoxification, the aryl hydrocarbon receptor (Ahr) (r(2) = 0.18, p = 0.045
12 gistically elicit allergic inflammation, and aryl hydrocarbon receptor (AhR) activation and signaling
15 is paper deals with the characterization and aryl hydrocarbon receptor (AhR) agonist activities of a
16 il fractions were tested for the presence of aryl hydrocarbon receptor (AhR) agonist and androgen rec
17 induction of CYP450 activity in response to aryl hydrocarbon receptor (AhR) agonist omeprazole and a
18 Rodent cancer bioassays indicate that the aryl hydrocarbon receptor (AHR) agonist, 2,3,7,8-tetrach
19 an photoproduct and endogenous high-affinity aryl hydrocarbon receptor (AhR) agonist, acts as a nanom
21 coordinate activation or inactivation of the aryl hydrocarbon receptor (AhR) and cyclic adenosine mon
23 s and controlled IL-22 production through an aryl hydrocarbon receptor (AhR) and IL-23 receptor pathw
24 lso resulted in nuclear translocation of the aryl hydrocarbon receptor (AhR) and in phosphorylation o
25 ro research suggests dioxins may bind to the aryl hydrocarbon receptor (AhR) and induce telomerase ac
26 (-) immature NK (iNK) cells uniquely express aryl hydrocarbon receptor (AHR) and interleukin-22 (IL-2
28 in LKO mice correlated with the elevation of aryl hydrocarbon receptor (AHR) and mediator 1 (MED1), t
30 ted the ligand-operated transcription factor aryl hydrocarbon receptor (AhR) and the hepatic enzyme t
31 ia the ligand-activated transcription factor aryl hydrocarbon receptor (AHR) and the suppressor of cy
33 In silico pathway evaluation suggested the aryl hydrocarbon receptor (AhR) as one possible target o
34 monocytes underexpressed the IL-1 inhibitor aryl hydrocarbon receptor (AHR) at baseline and accumula
38 nes and the whole liver established that the aryl hydrocarbon receptor (AhR) can disrupt G1-phase cel
40 aternal exposure to pollutants that bind the aryl hydrocarbon receptor (AhR) correlates with poorer a
41 es link exposure to pollutants that bind the aryl hydrocarbon receptor (AHR) during development with
85 y, we show that an environmental sensor, the aryl hydrocarbon receptor (AhR) is highly induced upon B
93 l exposure to contaminants that activate the aryl hydrocarbon receptor (AHR) lead to suppression of i
94 human fibroblasts by a candidate endogenous aryl hydrocarbon receptor (AhR) ligand 2-(1'H-indole-3'-
95 We have characterized previously a class of aryl hydrocarbon receptor (AHR) ligand termed selective
96 esidues involved in Hsp90 binding within the aryl hydrocarbon receptor (AhR) ligand-binding domain an
99 udies have shown that the toxicant-activated aryl hydrocarbon receptor (AHR) may disrupt fat metaboli
102 expression of IL-22, either by mutating the aryl hydrocarbon receptor (AhR) or inhibiting AhR signal
104 The ligand-activated transcription factor aryl hydrocarbon receptor (AHR) participates in the diff
105 diates its effects via the activation of the aryl hydrocarbon receptor (AhR) pathway and the subseque
106 e expression data revealed activation of the aryl hydrocarbon receptor (AhR) pathway in laquinimod-tr
107 acco smoke contains numerous agonists of the aryl hydrocarbon receptor (AhR) pathway, and activation
119 eceptor repressor (AhRR) is known to repress aryl hydrocarbon receptor (AhR) signaling, but very litt
121 tions, we show that MSI2 directly attenuates aryl hydrocarbon receptor (AHR) signalling through post-
122 xia inducible factor-1alpha (HIF1-alpha) and aryl hydrocarbon receptor (AHR) supports the differentia
123 we used lentivirus-mediated knockdown of the aryl hydrocarbon receptor (AHR) to demonstrate that 1023
124 orodibenzo-p-dioxin (TCDD) interact with the aryl hydrocarbon receptor (Ahr) to induce osteoclastic b
125 y, StemRegenin 1 (SR1), an antagonist of the aryl hydrocarbon receptor (AhR) transcription factor kno
126 rrepresentation of cognate sequences for the aryl hydrocarbon receptor (AhR) transcription factor, wh
127 promote NK cell IL-10, and activation of the aryl hydrocarbon receptor (AHR) was also required for ma
128 ealthspan in worms and flies depend upon the aryl hydrocarbon receptor (AHR), a conserved detector of
129 cities require binding and activation of the aryl hydrocarbon receptor (AhR), a ligand activated tran
134 ll TCDD toxicities require activation of the aryl hydrocarbon receptor (AHR), a ligand-activated tran
135 the modulation of the immune response by the aryl hydrocarbon receptor (AhR), a ligand-activated tran
137 lecular weight PAHs are known ligands of the aryl hydrocarbon receptor (AhR), a nuclear receptor that
138 ly polar structure, kynurenine activates the aryl hydrocarbon receptor (AHR), a PER, ARNT, SIM (PAS)
139 e show are modified by interactions with the aryl hydrocarbon receptor (AhR), a protein functioning b
143 e Il17 promoter through interaction with the aryl hydrocarbon receptor (Ahr), a transcription factor
145 y, bioassays indicative of activation of the aryl hydrocarbon receptor (AhR), activation of the pregn
147 DO2 in TNBC cells was sufficient to activate aryl hydrocarbon receptor (AhR), an endogenous kynurenin
150 nes represent known ligands of the mammalian aryl hydrocarbon receptor (AHR), and UPEC infection of A
152 achlorodibenzo-p-dioxin (TCDD), activate the aryl hydrocarbon receptor (AhR), causing it to transloca
154 cript levels of five gene targets, including aryl hydrocarbon receptor (Ahr), interleukin-1 beta (Il1
155 ICZ), an enigmatic endogenous ligand for the aryl hydrocarbon receptor (AHR), may explain adverse phy
156 to compare relative risks of activating the aryl hydrocarbon receptor (AhR), nuclear factor erythroi
157 ntrols, we found that coal tar activated the aryl hydrocarbon receptor (AHR), resulting in induction
158 and the anti-lipogenic transcription factor aryl hydrocarbon receptor (Ahr), the latter of which we
160 -induced Jag1 expression was mediated by the aryl hydrocarbon receptor (AhR), which bound to and acti
162 their toxic action through activation of the aryl hydrocarbon receptor (AhR), which is believed to re
163 at the ligand-activated transcription factor aryl hydrocarbon receptor (AhR), which was induced by IL
164 sed in vitro and in vivo studies to quantify aryl hydrocarbon receptor (AhR)-dependent effects of PCB
165 uction of anti-inflammatory cytokines via an aryl hydrocarbon receptor (AhR)-dependent mechanism.
166 r findings uncover an activin-A-induced IRF4-aryl hydrocarbon receptor (AhR)-dependent transcriptiona
170 this induction was abrogated by CH223191, an aryl hydrocarbon receptor (AhR)-specific antagonist.
183 ls, IAA activated an inflammatory nongenomic aryl hydrocarbon receptor (AhR)/p38MAPK/NF-kappaB pathwa
184 ently been shown to be endogenous ligands of aryl hydrocarbon receptor (AhR; a unique cellular chemic
185 esticus) and common tern (Sterna hirundo) to aryl hydrocarbon receptor 1 (AHR1)-dependent changes in
189 y of Il1rl2(-/-) mice could be rescued by an aryl hydrocarbon receptor agonist, which was sufficient
190 ould parallel that in other clients like the aryl hydrocarbon receptor and HIF1alpha, which also inte
192 ghly expressed CD90 ( approximately 63%) and aryl hydrocarbon receptor and produced IL-17 ( approxima
193 ed in this process through activation of the aryl hydrocarbon receptor and subsequent mitochondrial r
194 ility and safety of StemRegenin-1 (SR-1), an aryl hydrocarbon receptor antagonist that expands CD34+
196 nhibits mouse 3'IghRR activation and induces aryl hydrocarbon receptor binding to dioxin response ele
199 Effects of developmental activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-
200 of the ligand-activated transcription factor aryl hydrocarbon receptor by 2-(1'H-indole-3'-carbonyl)-
202 that IL-23-mediated restoration of IL-22 is aryl hydrocarbon receptor dependent, whereas IL-17 requi
204 nital amaurosis (LCA) caused by mutations in Aryl hydrocarbon receptor interacting protein like-1 (Ai
206 Defects in the photoreceptor-specific gene aryl hydrocarbon receptor interacting protein-like 1 (Ai
207 lic mutations in the photoreceptor-expressed aryl hydrocarbon receptor interacting protein-like 1 (AI
209 of P351Delta12 hAIPL1 and the mouse isoform, aryl hydrocarbon receptor interacting protein-like 1 (mA
211 on of the aryl hydrocarbon receptor, and the aryl hydrocarbon receptor ligand restores FLG expression
212 inarof, a bacteria-derived polyphenol, is an aryl hydrocarbon receptor ligand that attenuated inflamm
213 ct 6-formylindolo[3,2-b]carbazole (FICZ), an aryl hydrocarbon receptor ligand, has been found to be a
216 hypoxia-inducible factor 1alpha (HIF1alpha)/aryl hydrocarbon receptor nuclear translocator (ARNT) an
217 rategy that led to the identification of the aryl hydrocarbon receptor nuclear translocator (ARNT) as
219 eted the HIF-alpha dimerization partner, the aryl hydrocarbon receptor nuclear translocator (ARNT) in
225 alpha subunit and a constitutively expressed aryl hydrocarbon receptor nuclear translocator (ARNT) su
226 hypoxia-inducible factor complex (HIF-alpha.aryl hydrocarbon receptor nuclear translocator (ARNT)) r
227 unoglobulin heavy chain enhancer 3', and the aryl hydrocarbon receptor nuclear translocator (ARNT), d
228 and NPAS3 must each heterodimerize with the aryl hydrocarbon receptor nuclear translocator (ARNT), t
229 ible factor (HIF) basic helix-loop-helix Per-aryl hydrocarbon receptor nuclear translocator (ARNT)-Si
230 and a constitutively expressed beta subunit, aryl hydrocarbon receptor nuclear translocator (ARNT).
231 With hypoxia, the stabilized HIF-alpha and aryl hydrocarbon receptor nuclear translocator (ARNT, al
232 ) and a constitutively present beta subunit, aryl hydrocarbon receptor nuclear translocator (HIFbeta/
233 cates to the nucleus where it dimerizes with aryl hydrocarbon receptor nuclear translocator and modul
234 ng the core clock component brain and muscle aryl hydrocarbon receptor nuclear translocator like 1 (B
235 ne hepatoma Hepa1c1c7 cells and its AhR- and aryl hydrocarbon receptor nuclear translocator-deficient
236 breaks in AhR-deficient cells compared with aryl hydrocarbon receptor nuclear translocator-deficient
237 he circadian clock transcriptional activator aryl hydrocarbon receptor nuclear translocator-like (Bma
238 f circadian clock transcriptional activators aryl hydrocarbon receptor nuclear translocator-like (Bma
241 ptional factor BMAL1 (brain and muscle ARNT [aryl hydrocarbon receptor nuclear translocator]-like pro
242 etabolic enzyme activity, likely through the aryl hydrocarbon receptor pathway, and generation of rea
243 hanced by IL-21 expression through the c-Maf/aryl hydrocarbon receptor pathway, independent of APCs.
248 okers, including an intragenic region of the aryl hydrocarbon receptor repressor gene (AHRR; cg055759
251 of immunology, presenting information on the aryl hydrocarbon receptor signaling pathway, the immunom
255 nhibition of transcriptional activity of the aryl hydrocarbon receptor, a key regulator of ILC3 maint
256 cated within a putative binding site for the aryl hydrocarbon receptor, a master regulator of IL-22 p
257 Accordingly, we show that expression of the aryl hydrocarbon receptor, a transcription factor import
259 on the ligand-activated transcription factor aryl hydrocarbon receptor, and the third on how docosano
261 nduces the expression of CYP2E1, CYP1A2, and aryl hydrocarbon receptor, but not of CYP3A4, hepatocyte
262 ound mutations further demonstrated that the aryl hydrocarbon receptor, but not RORgammat, was requir
263 ndogenous ligand of the transcription factor aryl hydrocarbon receptor, could change the expression o
264 , which, along with the environmental sensor aryl hydrocarbon receptor, forms a multipartite transcri
265 ith other PDE4 inhibitors, an agonist of the aryl hydrocarbon receptor, Janus kinase inhibitors, and
266 ABCG2 coincided with increased occupancy of aryl hydrocarbon receptor, Sp1, and Nrf2 within the ABCG
267 transcription 3 and the cell cycle regulator aryl hydrocarbon receptor, the data suggest a disturbed
268 totype, is independent of suppression of the aryl hydrocarbon receptor, which targets cells with more
269 l for Th9, via suppressing the expression of aryl hydrocarbon receptor, without an increase in IL-10.
270 associated with increased transcription from aryl hydrocarbon receptor- and oxidative stress-regulate
271 ve and tolerant populations, we identify the aryl hydrocarbon receptor-based signaling pathway as a s
272 by TCDD, and antagonist studies supported an aryl hydrocarbon receptor-dependent activation, which wa
273 s close homolog Cyp1a1 was upregulated in an aryl hydrocarbon receptor-dependent manner, hence indica
275 Here we show that homozygous deletion of the aryl hydrocarbon receptor-interacting protein (AIP), a s
276 ar chaperones, including Hsp90, p23, and the aryl hydrocarbon receptor-interacting protein (AIP), als
279 ations in either the enzyme itself or AIPL1 (aryl hydrocarbon receptor-interacting protein-like 1), l
287 adhesion kinase (FAK)/RhoA alteration by the aryl-hydrocarbon receptor (AhR) agonist 3-methylcholanth
288 ed single nucleotide polymorphisms (SNPs) in aryl-hydrocarbon receptor (AHR) and cytochrome P450 1A1
289 d in cultured cells following treatment with Aryl-hydrocarbon receptor (Ahr) ligands including the ub
290 forms together with the transcription factor aryl-hydrocarbon receptor (AhR), compared to unprimed co
291 derivatives of tryptophan that activated the aryl-hydrocarbon receptor in CD4(+) T cells, allowing Th
292 questionnaires as well as DNA methylation in aryl-hydrocarbon receptor repressor (AHRR), a sentinel e
293 (LIMR), enhanced the interaction of LIMR and aryl-hydrocarbon receptor repressor (AHRR), and promoted
294 que for TAA at 1 week (Folh1, Cubn), whereas aryl-hydrocarbon receptor signaling might be important f
295 prominent example is hypomethylation of the aryl hydrocarbon-receptor repressor (AHRR) locus, which
296 ese regulatory factors, we identify AHR, the aryl hydrocarbon-receptor which controls a healthy immun
297 pothesis that coplanar PCBs act at adipocyte aryl hydrocarbon receptors (AhRs) to promote adipose inf
298 netic and functional similarities in species aryl hydrocarbon receptors (AHRs), which mediate DLC sen
299 ted to xenobiotic metabolism (pregnane X and aryl hydrocarbon receptors), hormone-mediated modes of a
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