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1 aline phosphatase and Pseudomonas aeruginosa arylsulfatase.
2 ystallographic data for three highly related arylsulfatases.
3 hows no sequence similarity with other known arylsulfatases.
4 ctrophotometric method, the determination of arylsulfatase A (ARSA) activity toward the natural subst
7 fy small molecules that enhance the residual arylsulfatase A (ASA) activity found in patients with me
11 nization of a recombinant glycoprotein human arylsulfatase A even when their rapid interconversion pr
12 Sap B) is an essential activator protein for arylsulfatase A in the hydrolysis of sulfatide, a lipid
15 lipid rafts isolated from wild-type mice and arylsulfatase A-deficient (ASA knockout) mice that accum
16 of canine HSS, based on homology with human arylsulfatases A and B, suggested the pathogenic effect
18 th nutrient-replete growth and P limitation; arylsulfatase activity and S deficiency-responsive genes
20 r1sac1snrk2.2 triple mutants exhibit reduced arylsulfatase activity compared to snrk2.2 or psr1snrk2.
23 demonstrate that the mammalian Sulfs exhibit arylsulfatase activity with a pH optimum in the neutral
27 specific enzymes (1, 4-beta-cellobiosidase, arylsulfatase, alkaline phosphatase, and phenol oxidase)
28 and consequently these cells synthesize some arylsulfatase and exhibit elevated levels of transcripts
29 xemplified by the synthesis of extracellular arylsulfatase and the accumulation of transcripts encodi
32 iggered accumulation of transcripts encoding arylsulfatases (ARS), an extracellular polypeptide that
33 nes that encode nitrate reductase (NITI) and arylsulfatase (ARS2) transcriptionally fused to sequence
34 tein (Rasa), dihydrofolate reductase (Dhfr), arylsulfatase (As-1), thrombin receptor (Cf2r), hexosami
35 sents evidence that deficiency of the enzyme arylsulfatase B (ARSB; N-acetylgalactosamine 4-sulfatase
36 n, and decline in the activity of the enzyme arylsulfatase B (ARSB; N-acetylgalactosamine-4-sulfatase
37 enzymes, N-acetylgalactosamine-4-sulfatase (arylsulfatase B (ASB)) and galactose-6-sulfatase (GALNS)
39 disorder caused by the deficient activity of arylsulfatase B (ASB; N-acetylgalactosamine 4-sulfatase)
41 intracellular activity of recombinant human arylsulfatase B (rhASB) on its natural glycosaminoglycan
42 colonic epithelial cells through decline in arylsulfatase B activity, with subsequent impact on C4S,
43 by loss of function of the steroid sulfatase arylsulfatase C (STS), to develop a model of corrective
45 s to sulfur limitation; it cannot synthesize arylsulfatase, does not take up sulfate as rapidly as wi
46 ibrary for HYDA1 promoter activity using the arylsulfatase-encoding ARYLSULFATASE2 gene as a selectio
48 oding cluster and a region encoding putative arylsulfatase enzymes, which were conserved across geogr
49 oduct shows homology to a well-characterized arylsulfatase family of enzymes found in eukaryotes, as
54 notypes, the cheY gene was inserted into the arylsulfatase gene of 81-176 to generate a strain with t
55 ere we report the functional analysis of the arylsulfatase insulator (ArsI) derived from the sea urch
57 for microorganism identification or exhibit arylsulfatase, lactonase, or phosphotriesterase activiti
59 fication; of note, N. nova were positive for arylsulfatase, N. farcinica were positive for opacificat
60 mains of PbgA resemble the structures of the arylsulfatase protein family and contains a novel core h
62 Tween 80 hydrolysis, tellurite reduction, or arylsulfatase reduction; grew best at low salt concentra
64 3)CR1-expressing cell membranes treated with arylsulfatase to desulfate tyrosine residues also showed
65 activity upon phosphorus deprivation and the arylsulfatase upon sulfur deprivation, suggesting that t
66 e as a cofactor in a class of enzymes termed arylsulfatases, which catalyze the hydrolysis of various
67 ystal structure shows structural homology to arylsulfatases with conservation of the core alpha/beta-
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