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1 aline phosphatase and Pseudomonas aeruginosa arylsulfatase.
2 ystallographic data for three highly related arylsulfatases.
3 hows no sequence similarity with other known arylsulfatases.
4 ctrophotometric method, the determination of arylsulfatase A (ARSA) activity toward the natural subst
5 e disorder caused mainly by mutations in the arylsulfatase A (ARSA) gene.
6 22S1743 and D22S1744, were developed for the arylsulfatase A (ARSA) region of chromosome 22q.
7 fy small molecules that enhance the residual arylsulfatase A (ASA) activity found in patients with me
8 eural precursors (NPs) that are deficient in arylsulfatase A (ASA) activity.
9 omote brain delivery of the lysosomal enzyme arylsulfatase A (ASA).
10 ase II and by assaying enzyme activities for arylsulfatase A and carnitine palmitoyltransferase.
11 nization of a recombinant glycoprotein human arylsulfatase A even when their rapid interconversion pr
12 Sap B) is an essential activator protein for arylsulfatase A in the hydrolysis of sulfatide, a lipid
13                                              Arylsulfatase A is an endogenous enzyme that is responsi
14 epsin A, cathepsin D, alpha-galactosidase A, arylsulfatase A, and alpha-iduronidase.
15 lipid rafts isolated from wild-type mice and arylsulfatase A-deficient (ASA knockout) mice that accum
16  of canine HSS, based on homology with human arylsulfatases A and B, suggested the pathogenic effect
17 accumulate sulfatides due to a deficiency in arylsulfatase-A, directly activate iNKT cells.
18 th nutrient-replete growth and P limitation; arylsulfatase activity and S deficiency-responsive genes
19          Clinical isolates were positive for arylsulfatase activity at 3 days, nitrate, iron uptake,
20 r1sac1snrk2.2 triple mutants exhibit reduced arylsulfatase activity compared to snrk2.2 or psr1snrk2.
21                    The sac3 mutant expresses arylsulfatase activity even when grown in nutrient-reple
22                          In wild-type cells, arylsulfatase activity is detected only during sulfur li
23 demonstrate that the mammalian Sulfs exhibit arylsulfatase activity with a pH optimum in the neutral
24                 In contrast to its effect on arylsulfatase activity, Sac3 positively regulates the hi
25 ntial for endosulfatase activity but not for arylsulfatase activity.
26 in has a negative effect on the induction of arylsulfatase activity.
27  specific enzymes (1, 4-beta-cellobiosidase, arylsulfatase, alkaline phosphatase, and phenol oxidase)
28 and consequently these cells synthesize some arylsulfatase and exhibit elevated levels of transcripts
29 xemplified by the synthesis of extracellular arylsulfatase and the accumulation of transcripts encodi
30 lagellation responsiveness to a promoterless arylsulfatase (ARS) gene.
31 ferred partial acid shock inducibility to an arylsulfatase (ARS) reporter gene.
32 iggered accumulation of transcripts encoding arylsulfatases (ARS), an extracellular polypeptide that
33 nes that encode nitrate reductase (NITI) and arylsulfatase (ARS2) transcriptionally fused to sequence
34 tein (Rasa), dihydrofolate reductase (Dhfr), arylsulfatase (As-1), thrombin receptor (Cf2r), hexosami
35 sents evidence that deficiency of the enzyme arylsulfatase B (ARSB; N-acetylgalactosamine 4-sulfatase
36 n, and decline in the activity of the enzyme arylsulfatase B (ARSB; N-acetylgalactosamine-4-sulfatase
37  enzymes, N-acetylgalactosamine-4-sulfatase (arylsulfatase B (ASB)) and galactose-6-sulfatase (GALNS)
38 disorder caused by the deficient activity of arylsulfatase B (ASB).
39 disorder caused by the deficient activity of arylsulfatase B (ASB; N-acetylgalactosamine 4-sulfatase)
40                                              Arylsulfatase B (N-acetylgalactosamine-4-sulfatase; ARSB
41  intracellular activity of recombinant human arylsulfatase B (rhASB) on its natural glycosaminoglycan
42  colonic epithelial cells through decline in arylsulfatase B activity, with subsequent impact on C4S,
43 by loss of function of the steroid sulfatase arylsulfatase C (STS), to develop a model of corrective
44                   For Pseudomonas aeruginosa arylsulfatase-catalyzed aryl sulfate hydrolysis, Bronste
45 s to sulfur limitation; it cannot synthesize arylsulfatase, does not take up sulfate as rapidly as wi
46 ibrary for HYDA1 promoter activity using the arylsulfatase-encoding ARYLSULFATASE2 gene as a selectio
47            We also evaluated the efficacy of arylsulfatase enzymatic activity as a reporter and found
48 oding cluster and a region encoding putative arylsulfatase enzymes, which were conserved across geogr
49 oduct shows homology to a well-characterized arylsulfatase family of enzymes found in eukaryotes, as
50                                              Arylsulfatase G (ARSG) is a recently identified lysosoma
51              Here we show that the lysosomal arylsulfatase G (ARSG) is the long-sought glucosamine-3-
52                                Deficiency of arylsulfatase G (ARSG) leads to a lysosomal storage dise
53                                          The arylsulfatase gene from Campylobacter jejuni 81-176 enco
54 notypes, the cheY gene was inserted into the arylsulfatase gene of 81-176 to generate a strain with t
55 ere we report the functional analysis of the arylsulfatase insulator (ArsI) derived from the sea urch
56               A novel member of this family, arylsulfatase K (ARSK), was identified bioinformatically
57  for microorganism identification or exhibit arylsulfatase, lactonase, or phosphotriesterase activiti
58 mined to be homologous to E. coli K-12 aslA (arylsulfatase-like gene).
59 fication; of note, N. nova were positive for arylsulfatase, N. farcinica were positive for opacificat
60 mains of PbgA resemble the structures of the arylsulfatase protein family and contains a novel core h
61           The ars76 mutant cannot accumulate arylsulfatase protein or mRNA and shows marked alteratio
62 Tween 80 hydrolysis, tellurite reduction, or arylsulfatase reduction; grew best at low salt concentra
63                                              Arylsulfatases require a maturating enzyme to perform a
64 3)CR1-expressing cell membranes treated with arylsulfatase to desulfate tyrosine residues also showed
65 activity upon phosphorus deprivation and the arylsulfatase upon sulfur deprivation, suggesting that t
66 e as a cofactor in a class of enzymes termed arylsulfatases, which catalyze the hydrolysis of various
67 ystal structure shows structural homology to arylsulfatases with conservation of the core alpha/beta-

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