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1 phosphatidylinositol transfer protein (PITP) as being responsible for a powerful latent, nucleotide-i

2 e proteins, which were identified originally as being responsible for a step in the maturation of the

3 In our model, 74 fragments were identified as being responsible for all the chemical's HIV antivira

4 n of AR and the MH2 of Smad3 were identified as being responsible for binding.

5 dence points toward small oligomeric species as being responsible for cell dysfunction and death.

6 n and coordination in the chromophore pocket as being responsible for Dathail's photoswitching.

7 es of the haloacid dehalogenase superfamily, as being responsible for dephosphorylating the C-termina

8 as focused on local influences in the retina as being responsible for development of this diabetic co

9 gene approach, actin was identified recently as being responsible for dilated cardiomyopathy.

10 identified a single cysteine residue, C466, as being responsible for direct oxidation-mediated activ

11 Nevertheless, this protein was described as being responsible for driving the vitamin K-mediated

12 dynamics, rather than global rearrangement, as being responsible for dual-site phosphorylation.

13 ational modes in proteins have been proposed as being responsible for efficiently directing biochemic

14 file of the wild-type enzyme rules out His67 as being responsible for either of the pK(a) values.

15 ng from the mid/hindbrain boundary (isthmus) as being responsible for induction of different structur

16 ignaling by oncogenic Ras is generally cited as being responsible for induction of premature senescen

17 rminus of Rad52, a region previously defined as being responsible for its DNA binding ability.

18 and validate casein kinase 1alpha (CK1alpha) as being responsible for LRRK2 phosphorylation, includin

19 rophy (DM) locus has recently been suggested as being responsible for maintaining the frequency, in t

20 d amyloid assembly have long been recognized as being responsible for many devastating human diseases

21 ompounds, have been unequivocally identified as being responsible for most of the anthropogenic destr

22 namely, Glu71-Asp and Glu126-Lys, in DEN2 E as being responsible for mouse neurovirulence.

23 tified cytochrome P450 monooxygenase CYP89A9 as being responsible for NDCC accumulation in wild-type

24 axis, suggested previously in X-ray studies as being responsible for NTE behavior, in fact make negl

25 neighboring enolase dimer can be identified as being responsible for ordering and stabilization of t

26 to be exacerbated by undernutrition as well as being responsible for permanent growth deficits acqui

27 d HERC2, a HECT domain-containing E3 ligase, as being responsible for polyubiquitination of USP33.

28 that has been implicated in modeling studies as being responsible for poor inhibition by BPTI.

29 Fe(II) to iron oxide coatings was identified as being responsible for promotion of surface dissolutio

30 ther than identifying a single region on PSR as being responsible for PSR function, the results sugge

31 on may be part of the system often described as being responsible for reward learning: these neurons

32 its, TnI, TnC, and TnT, widely characterized as being responsible for separate aspects of the regulat

33 red over 40 years ago and are now recognized as being responsible for several genetic syndromes.

34 previous work, and we had identified His-497 as being responsible for the acidic block of CLC-K chann

35 tSBP1 complex identified Cys(21) and Cys(22) as being responsible for the binding of one selenium.

36 he first two YWTD-EGF repeat domains of LRP5 as being responsible for the binding.

37 some 40 kpc to the south--has been proposed as being responsible for the complexity of its H I struc

38 so the role of ferrocene moiety are included as being responsible for the cytotoxic response.

39 identified P2RX7 purinoceptor up-regulation as being responsible for the death of muscles in the mdx

40 transition, and protein kinase C inhibition as being responsible for the decrease in respiration rat

41 on of Yp genes to XO females implicated Zfy2 as being responsible for the deleterious Y chromosomal e

42 the lipid-binding pleckstrin homology domain as being responsible for the differential in vitro prope

43 nts identified the notochord and the somites as being responsible for the DV patterning of the neural

44 of the otherwise structured fibrin molecule as being responsible for the elastic recoil of the fiber

45 thin AtDBR1 was confirmed by complementation as being responsible for the embryo-lethal phenotype, an

46 We further identify AP1 as being responsible for the EV71-induced expression of

47 ed a change in AMPA-type glutamate receptors as being responsible for the expression of LTP but there

48 A sensitivity analysis identified one trial as being responsible for the heterogeneity; after exclus

49 This study identifies CaMKIV as being responsible for the increased expression of CRE

50 x) (1.26), virtually ruling out residue C221 as being responsible for the inverse effects reported fo

51 reonine-to-alanine difference at residue 128 as being responsible for the kinetic differences between

52 The characterization of this gene as being responsible for the loss of adhesion indicates

53 dence points to differences in contrast gain as being responsible for the majority of individual vari

54 The open reading frame postulated as being responsible for the metal-citrate transport obs

55 ecal specimens and was the strain identified as being responsible for the outbreak.

56 y at the binding steps of the reaction cycle as being responsible for the phenomenon, but the literat

57 f the T-box family of transcription factors, as being responsible for the physical malformations of t

58 more, knockdown experiments identified HDAC2 as being responsible for the regulation of RAD51.

59 metalloproteins first identified in mammals as being responsible for the synthesis of the wide-sprea

60 ntified two monooxygenases, AbeX1 and AbeX2, as being responsible for the transformation of an indolo

61 f6alpha arm of the unfolded protein response as being responsible for the up-regulation of Lmf1 in ER

62 structs implicate the lever arm/rod of MyoVc as being responsible for these properties.

63 0 by the dicarbonyl metabolite methylglyoxal as being responsible for this decreased association.

64 ifies the DYT1 gene on human chromosome 9q34 as being responsible for this dominant disease.

65 itochondrial oxidant stress is widely viewed as being responsible for this loss, but why these partic

66 he nonbonded nucleobase overhangs on the DNA as being responsible for this stability.

67 We identify the CsrA RNA binding protein as being responsible for this, and show that CsrA differ

68 on with parasitic worms (helminths), as well as being responsible for widespread suffering in allergi

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