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1 cterial infections, and mortality at lower CD4 cell counts, as expected.
2 eting six chemokine 3'-UTRs increased chemokine mRNA levels as expected.
3 reased body weight, adiposity, and serum metabolic hormones as expected and also elicited an increase in mammary ERalpha
5 ditioned fear extinction and response-outcome conditioning, as expected based on prior studies using other inactivation t
6 HCC cells and ATRA-PLLA did not inhibit normal liver cells, as expected because ATRA selectively inhibits active Pin1 in
8 the phase transfer step and in the stereoinduction process, as expected, but also in the unproductive reaction that provi
13 tg deletion resulted in increased adherence to M cells and, as expected, decreased adherence to Caco-2 cells.
15 ith wider peak widths in the mini-CIT than on the Synapt G2 as expected due to high-pressure operation.
17 istances near the sum of the van der Waals and ionic radii, as expected for predominantly electrostatic interactions.
18 oud feedback) and with a less negative lapse-rate feedback, as expected from a decrease in stability.
22 Grade 4 non-haematological toxicity was uncommon; as expected, haematological toxicity was more common in patie
25 e of cleaning incitation signals, clients did not retaliate as expected (i.e., by chasing) in response to increased cheat
26 on and secretion remained intact in LRKO mice and increased as expected in the setting of diet-induced insulin resistance
27 c acid cause substantial dissolution of carbonate minerals, as expected, increasing effective fracture volume (fracture v
36 n of vessel normalization reduced T lymphocyte infiltration as expected, reciprocal depletion or inactivation of CD4(+) T
50 a single mutation in SLC30A9 within this locus, segregating as expected within the kindred and not found in a homozygous
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