ライフサイエンスコーパス: asbestos exposure

1 f the pleura and is strongly associated with asbestos exposure.

2 on homeostasis, with a more marked effect in asbestos exposure.

3 s observed after adjustment for occupational asbestos exposure.

4 ant mesothelioma is strongly associated with asbestos exposure.

5 hic evidence of lung changes consistent with asbestos exposure.

6 n an urban setting, and with a potential for asbestos exposure.

7 ar of birth, sex, and potential occupational asbestos exposure.

8 lved and that mesothelioma predominates upon asbestos exposure.

9 ss mesothelioma - the cancer often linked to asbestos exposure.

10 d neoplasm that is typically associated with asbestos exposure.

11 an aggressive cancer largely associated with asbestos exposure.

12 ted to TP53 mutation status and occupational asbestos exposure.

13 , and are often associated with occupational asbestos exposure.

14 ancer that is commonly associated with prior asbestos exposure.

15 lls after mechanical wounding or crocidolite asbestos exposure.

16 information including cigarette smoking and asbestos exposure.

17 M) is an aggressive neoplasm associated with asbestos exposure.

18 vitamin A carotenoids, tobacco exposure, and asbestos exposure.

19 to develop fibroproliferative lesions after asbestos exposure.

20 likely to have been confounded by smoking or asbestos exposure.

21 osis and work-related histories of long term asbestos exposure.

22 ar duct regions by 24 hours after the single asbestos exposure.

23 ronchiolar epithelial cells after chrysotile asbestos exposures.

24 e induction of fibrosis in workers with high asbestos exposures.

25 Lung cancer mortality was increased by asbestos exposure alone among nonsmokers (rate ratio = 3

26 with 4.3 (95% CI, 1.9-9.8) for occupational asbestos exposure alone.

27 ent in individuals at high risk of MM due to asbestos exposure, although those with the 1125A polymor

28 om mesothelial cells and are associated with asbestos exposure and approximately 50% contain simian v

29 Mesothelioma arises in a context of asbestos exposure and chronic inflammation, which would

30 er adjustment for the confounding effects of asbestos exposure and cigarette smoking.

31 The relation between asbestos exposure and colorectal cancer remains controve

32 Malignant mesothelioma has been linked to asbestos exposure and generally has a poor prognosis bec

33 Analyses of associations between cumulative asbestos exposure and lung cancer mortality among textil

34 lignant mesothelioma (MM) is associated with asbestos exposure and the presence of SV40 viral sequenc

35 Occupational asbestos exposure and TP53 mutation were significantly a

36 The relationship between asbestos exposure and upper-lobe location of tumor was a

37 al membranes which are often associated with asbestos exposure and with Simian virus 40 (SV40) infect

38 ome, exposure to tobacco smoke, occupational asbestos exposure, and additional molecular alterations

39 in the United States are not associated with asbestos exposure, and only a minority of people exposed

40 r, not all mesotheliomas are associated with asbestos exposure, and only a small minority of people e

41 onmental history revealed no other source of asbestos exposure, and the initial clinical diagnosis wa

42 nterval, 1.5-15.4) compared to those without asbestos exposure, and this association remained after a

43 of lung cancer; smoking history; history of asbestos exposure; and intakes of yellow-orange vegetabl

44 nflammation occurred after 10 and 20 days of asbestos exposure as evidenced by increases in total pro

45 omas, clinicians should continue to consider asbestos exposure as the most likely and most thoroughly

46 For example, environmental asbestos exposures associated with a former mine in Libb

47 e a large number of pulmonary diseases, with asbestos exposure being the most risky.

48 elopment of mesothelioma is linked mainly to asbestos exposure, but the combined contributions of gen

49 Asbestos exposure causes activation of extracellular sig

50 e tumors, 14.6% had a history of significant asbestos exposure compared with 5.4% of those with lower

51 involved in lung injury and remodeling after asbestos exposure could aid in defining mechanisms of ai

52 r among those with a history of occupational asbestos exposure (crude odds ratio, 4.8; 95% confidence

53 The case-control study confirmed that asbestos exposure did not account for the observed assoc

54 f adenocarcinomas (p < 0.02), but history of asbestos exposure did not predict tumor histology.

55 e favors adenocarcinomas, but the history of asbestos exposure does not appear to influence the tumor

56 er-lobe location while those associated with asbestos exposure favor a lower-lobe location.

57 We estimated cumulative asbestos exposure (fibers per cubic centimeters x months

58 In addition to asbestos exposure, genetic predisposition to asbestos ca

59 Asbestos exposure has been proposed as a risk factor for

60 with lung cancer, both cigarette smoking and asbestos exposure histories favor an upper-lobe location

61 In comparison with observed asbestos exposure, if the facility had operated under th

62 100 fiber-years per milliliter of cumulative asbestos exposure in a cohort study of textile workers i

63 Lack of information on asbestos exposure in the LLPC limited the ability to val

64 Asbestos exposure increased mitochondrial Ca(2+) influx

65 These data suggest that asbestos exposure increases the likelihood of mutation a

66 Additional studies revealed that asbestos exposure induced a highly significant increase

67 y all lung cancer, and it is well known that asbestos exposure interacts synergistically with tobacco

68 Asbestos exposure is one important environmental cause o

69 Asbestos exposure is strongly associated with the develo

70 Asbestos exposure is the major known risk factor for mes

71 examine the effects of cigarette smoking and asbestos exposure on location and histology of lung canc

72 s with rounded atelectasis have a history of asbestos exposure or pleural effusion due to various cau

73 between tobacco smoke and both occupational asbestos exposure (OR, 12.04 [CI, 4.32 to 38.28]) and ex

74 4 [CI, 4.32 to 38.28]) and extraoccupational asbestos exposure (OR, 8.42 [CI, 2.77 to 30.58]).

75 not vary according to age, sex, duration of asbestos exposure, or degree of radiographic changes and

76 istory of lung cancer (p = 0.03), history of asbestos exposure (p = 0.02), less intake of yellow-oran

77 [95% CI, 4.0-13.7]), and by smoking without asbestos exposure (rate ratio = 10.3 [95% CI, 8.8-12.2])

78 bestos, many issues related to environmental asbestos exposures remain unresolved.

79 ccupational Safety and Health Administration asbestos exposure standard of <0.1 fibers/mL, we estimat

80 PDGF-AA isoform expression immediately after asbestos-exposure, suggest a scenario in which a potent

81 c studies have established tobacco smoke and asbestos exposures synergistically interact to enhance l

82 th k-ras mutations had significantly greater asbestos exposures than those without mutations (P < 0.0

83 shed between malignant mesothelioma (MM) and asbestos exposure, the exact mechanism by which asbestos

84 ms increases the percent of HM that survives asbestos exposure, thus increasing the pool of asbestos-

85 A further reduction in asbestos exposure to a standard of <0.05 fibers/mL was e

86 a comprehensive characterization of possible asbestos exposure to investigate asbestos as the potenti

87 ers) when interpreting asbestos toxicity; d) asbestos exposure to susceptible populations; and e) usi

88 sociations between pleural abnormalities and asbestos exposure using multiple logistic regression to

89 mortality by age 90 years under the observed asbestos exposure was 9.44%.

90 In contrast to some prior reports, asbestos exposure was also associated with an upper-lobe

91 A history of asbestos exposure was associated with idiopathic RPF (od

92 We aimed to test whether occupational asbestos exposure was associated with k-ras codon 12 mut

93 case of a 65-year-old accountant whose only asbestos exposure was during a summer job 50 years earli

94 No history of asbestos exposure was elicited by careful questioning or

95 A dose-response trend based on years of asbestos exposure was less evident.

96 Chrysotile asbestos exposure was measured in fiber-years per millil

97 Asbestos exposure was most specifically associated with

98 e association of k-ras mutation and reported asbestos exposure was not dependent on the presence of r

99 justed for age, birth date, and occupational asbestos exposure were used to estimate odds ratios (OR)

100 ls selected from 1,150 men with occupational asbestos exposure who had undergone pulmonary function t

101 sible associations of tobacco carcinogen and asbestos exposure with DAP-kinase promoter methylation,

102 e was an association of smoking duration and asbestos exposure with FHIT exon loss, indicating that c