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1 ein expression was significantly enhanced in ascending colon.
2 of the most proximal inflection point in the ascending colon.
3 lly, overlying the position of the cecum and ascending colon.
4 the ischemically damaged small intestine and ascending colon.
5 ut first in the stomach, small intestine and ascending colon.
6 patic flexure in 4 patients (36%), cecum and ascending colon (4 pts, 36%), rectosigmoid (2 pts, 18%)
7 ion (positive criteria); (c) air or stool in ascending colon and (d) cecal air or stool (negative cri
8 erry extract by microbiota obtained from the ascending colon and descending colon compartments of a d
9 be divided into three major clusterings: (1) ascending colon and transverse colon, (2) descending col
10 lon, two in the transverse colon, one in the ascending colon, and one in the cecum.
11 c biopsy specimens from the duodenum, ileum, ascending colon, and rectum.
12                              The jejunum and ascending colon are the most common sites of involvement
13 mucosal biopsies from the terminal ileum and ascending colon during surgery and post-operative colono
14                  Overall colonic transit and ascending colon emptying (AC t((1/2))) were significantl
15 as a significant overall treatment effect on ascending colon emptying half-time (P = .015) and overal
16 ificantly faster overall colonic transit and ascending colon emptying with the 4-mg dose.
17            Areas of incidental uptake in the ascending colon had the greatest chance (42 %) of being
18 s defined as any tumor arising in the cecum, ascending colon, hepatic flexure, or transverse colon.
19 sigmoid colon, or rectum vs appendix, cecum, ascending colon, hepatic flexure, or transverse colon.
20 sigmoid colon, or rectum vs appendix, cecum, ascending colon, hepatic flexure, or transverse colon.
21 ormal colorectal anatomical locations (i.e., ascending colon (n = 182), transverse colon (n = 249), d
22 e were cecum (n = 65), sigmoid (n = 60), and ascending colon (n = 50).
23 could be detected in the small intestine and ascending colon of the normoxia group.
24 signal intensity (r = 0.88, P = .033 for the ascending colon; r = 0.82, P = .006 for the descending c
25 g sensitivities (SE) and specificities (SP) (ascending colon: SE: 1.10%, SP: 91.02; transverse colon:
26 ering all subjects, the percentage change in ascending colon volume rose significantly after CRF.
27 ructose malabsorption, as shown by increased ascending colon volumes.
28            Mean thickness of the wall of the ascending colon was significantly greater in patients wi
29                              The jejunum and ascending colon were the most common sites of gastrointe
30 ative lesions from the terminal ileum to the ascending colon with a non-specific histo-pathologic fin

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