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1 ein expression was significantly enhanced in ascending colon.
2 of the most proximal inflection point in the ascending colon.
3 lly, overlying the position of the cecum and ascending colon.
4 the ischemically damaged small intestine and ascending colon.
5 ut first in the stomach, small intestine and ascending colon.
6 patic flexure in 4 patients (36%), cecum and ascending colon (4 pts, 36%), rectosigmoid (2 pts, 18%)
7 ion (positive criteria); (c) air or stool in ascending colon and (d) cecal air or stool (negative cri
8 erry extract by microbiota obtained from the ascending colon and descending colon compartments of a d
9 be divided into three major clusterings: (1) ascending colon and transverse colon, (2) descending col
13 mucosal biopsies from the terminal ileum and ascending colon during surgery and post-operative colono
15 as a significant overall treatment effect on ascending colon emptying half-time (P = .015) and overal
18 s defined as any tumor arising in the cecum, ascending colon, hepatic flexure, or transverse colon.
19 sigmoid colon, or rectum vs appendix, cecum, ascending colon, hepatic flexure, or transverse colon.
20 sigmoid colon, or rectum vs appendix, cecum, ascending colon, hepatic flexure, or transverse colon.
21 ormal colorectal anatomical locations (i.e., ascending colon (n = 182), transverse colon (n = 249), d
24 signal intensity (r = 0.88, P = .033 for the ascending colon; r = 0.82, P = .006 for the descending c
25 g sensitivities (SE) and specificities (SP) (ascending colon: SE: 1.10%, SP: 91.02; transverse colon:
26 ering all subjects, the percentage change in ascending colon volume rose significantly after CRF.
30 ative lesions from the terminal ileum to the ascending colon with a non-specific histo-pathologic fin
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