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1 dulatory and motion signals conveyed by this ascending pathway.
2 rons contribute to functions mediated by the ascending pathway.
3 hA4+ spinal neurons and the formation of the ascending pathway.
4 ayer 4 (the granular layer) is the target of ascending pathways.
5 y respiratory networks via direct or relayed ascending pathways.
6  with contributions from both descending and ascending pathways.
7 rent nociceptor engage the same or different ascending pathways.
8 ve the potential to contribute axons to both ascending pathways.
9 communication in addition to their action on ascending pathways.
10 e feeds information through several parallel ascending pathways.
11 pinal cord and synapse on cells of origin of ascending pathways.
12 e suggest functional independence of the two ascending pathways.
13 tical slow waves, suggesting their origin in ascending pathways.
14 ally placed to influence both descending and ascending pathways arising in the inferior colliculus.
15  supramammillary nucleus (SUM) is part of an ascending pathway conveying behavior-dependent drive to
16 lamocortical pathways relay information from ascending pathways (first order thalamic relays) and oth
17 hat glutamate may be a main component of the ascending pathway from the rNST to the PBN.
18 e primary somatosensory cortex is part of an ascending pathway from the sensory thalamus that mediate
19 In addition, we examined the neuroanatomy of ascending pathways from the antenna II and lateral anten
20                These studies showed that the ascending pathways from these neuropils innervate the sa
21 his article begins with an overview of these ascending pathways identified in adult rats using a stan
22  rationale for the apparent recession of the ascending pathway in motor cortex.
23 how that a lack of normal sensory inputs via ascending pathways in the dorsal spinal cord during earl
24               HSD2 neurons may be part of an ascending pathway involved in the salt-seeking behavior
25 ive contribution of the Sol and Pa5 to these ascending pathways is not known.
26 ion is known to be processed along a central ascending pathway leading to auditory cortex (AC).
27 excite vagal afferent neurons to activate an ascending pathway leading to inhibition of food intake.
28 s can modulate information processing in the ascending pathway, leading to improvements in signal det
29 e activation of Vi/Vc neurons and associated ascending pathways may facilitate somatoautonomic and so
30                      The data suggest (a) an ascending pathway medial to BIC that carries the fear-in
31                                              Ascending pathways mediated by monoamine neurotransmitte
32 stem represent an essential component of the ascending pathway mediating this effect of epinephrine.
33 uggest that noxious stimulation activates an ascending pathway of the spino-bulbo-spinal excitatory c
34 e report on the distribution of PICK1 in the ascending pathways of the central auditory system of the
35 visceral illness responses, depending on the ascending pathways of the GLP-1 system that are recruite
36 tency tuning is simply relayed along sensory ascending pathways or generated by local circuits remain
37  of pain information may take place in these ascending pathways or in descending pathways.
38 itry in the region of the lesion site and of ascending pathways originating near the injury.
39 medial nucleus outputs follow three distinct ascending pathways: periventricular, coursing through th
40 rect, postsynaptic dorsal column, and direct ascending pathways, respectively, that terminate as clim
41                         Comparison along the ascending pathway suggests that the increased jitter fou
42              Their axons form an ipsilateral ascending pathway that develops during embryogenesis and
43 n a millisecond basis, and could result from ascending pathways that utilize slow, second-messenger s
44  and device-reared owls in two nuclei in the ascending pathway: the external nucleus of the inferior
45                                    Are there ascending pathways through which the trigeminovascular s
46 ns to the AA-deficient or devoid diet via an ascending pathway to the APC.
47 nergic cells from the PT not only project an ascending pathway to the striatum, but send a descending
48 e relative contribution of brainstem-derived ascending pathways to forebrain immune-responsive nuclei
49  (LTP) and long-term depression (LTD) of the ascending pathways to layers IV and II/III in the mouse
50 e show fundamental differences between these ascending pathways using an in vitro slice preparation.
51 othesized that such pain is mediated through ascending pathways via the nucleus gracilis (NG) and is

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