ライフサイエンスコーパス: ascospore

1 ting bodies (perithecia) or meiotic progeny (ascospores).

2 ication of fungicides, which target airborne ascospores.

3 carp-forming fungi that physically discharge ascospores.

4 be induced leading to production of meiotic ascospores.

5 a and chlamydospores and in wild habitats by ascospores.

6 and development of barren perithecia without ascospores.

7 ating type and the formation of self-sterile ascospores.

8 y but which were complete devoid of asci and ascospores.

9 le in more mature asci containing developing ascospores.

10 the haploid nuclei are not partitioned into ascospores.

11 killer haplotypes suppress MSUD even though ascospores are not killed.

12 Ascospores are the primary inoculum in the wheat scab fu

13 )-anchored protein thought to be involved in ascospore assembly.

14 In addition, ascospores carrying null mutations of either gene are in

15 f Spore killer (Sk) x WT (Sk-sensitive), the ascospores containing the Spore killer allele survive, w

16 Ascospore delineation does not occur, however, and no se

17 s of temperature, relative humidity (RH) and ascospore density in multiple controlled environment exp

18 Tetrad analyses showed that all small ascospore-derived strains lacked the missing DNA between

19 o direct repeats that was found in all large ascospore-derived strains.

20 rip mutant corrected the defect in ascus and ascospore development in crosses with wild-type.

21 killer targets not only late but also early ascospore development.

22 lement and prevents it from interfering with ascospore development.

23 g that these genes play specialized roles in ascospore development.

24 y shown to control light driven conidial and ascospore development.

25 al pathogen Sclerotinia trifoliorum exhibits ascospore dimorphism and unidirectional mating type swit

26 l in growth and conidiation but defective in ascospore discharge due to the premature breakdown of it

27 editing of its transcripts is important for ascospore discharge in F. graminearum.

28 itable allele rescued the defects of amd1 in ascospore discharge.

29 of N. tetrasperma contain four self-fertile ascospores each with nuclei of both mating types (matA a

30 Deletion of FgSRP1 also reduced ascospore ejection and deoxynivalenol (DON) production.

31 al treatment, but also affected conidiation, ascospore formation and pathogenicity.

32 Ascospore formation in yeast is accomplished through a c

33 tant was reduced in growth, conidiation, and ascospore formation.

34 rate, macroconidiation, microconidiation, or ascospore formation.

35 tead resulted in severe defects in ascus and ascospore genesis.

36 inimum, maximum and optimum temperatures for ascospore germination were 0.0, 29.9 and 21.7 degrees C

37 lar between-slope differences were found for ascospore germination-resistance to acriflavine, with mu

38 spores, it formed eight small, single-celled ascospores in each ascus.

39 evelop on lettuce plants inoculated with dry ascospores in the absence of apparent leaf wetness (requ

40 tical in light of the fact that formation of ascospores in these species requires a long period of ti

41 Instead of producing four-celled ascospores, it formed eight small, single-celled ascospo

42 d a novel screen based on the sensitivity of ascospores lacking dityrosine to treatment with lytic en

43 The Neurospora crassa Asm-1+ (ascospore maturation 1) gene encodes an abundant nucleus

44 , cloning, and molecular analysis of Asm-1+ (Ascospore maturation 1), the Neurospora crassa homologue

45 Using the Ascospore maturation-1 (Asm-1) gene, we defined what nee

46 e defective in female sexual development and ascospore maturation.

47 d for formation of female structures and for ascospore maturation.

48 Progeny ascospores not carrying the killer element fail to matur

49 The self-fertile ascospores of N. tetrasperma result from first-division

50 Ascospores of Neurospora tetrasperma normally contain nu

51 involves microdissecting the four products (ascospores) of a single meiosis and analyzing the config

52 etes produce eight homokaryotic self-sterile ascospores per ascus.

53 otal mutation frequencies over many loci for ascospore pigmentation were 2.3, 3.5 and 4.4% for three

54 ng - self-fertile strains derived from large ascospores produce both self-fertile (large-spores) and

55 The WT RSK protein is dispensable for ascospore production and is not a target of the spore-ki

56 itions, the Delta odeA strain was delayed in ascospore production but produced more ascospores than w

57 d sufficient for perithecial development and ascospore production.

58 cotoxin fumonisin B1 (fum1) among 121 random ascospore progeny from a single cross.

59 Disruption of pairing causes failure of ascospore progeny to mature.

60 A total of 111 ascospore progeny were analyzed for segregation at 235 l

61 ficantly reduced in virulence and delayed in ascospore release.

62 ns, the ratio of conidia (asexual spores) to ascospores (sexual spores) is affected by linoleic acid

63 ed in ascospore production but produced more ascospores than wild type over time.

64 After germination from ascospores, the camk-1 null strains grew slowly, indicat

65 rm or ascus containing intracystic bodies or ascospores, the products of sexual replication.

66 ploids undergo meiosis, but are defective in ascospore wall maturation for they fail to give the fluo

67 ue to the dityrosine-containing layer in the ascospore wall.

68 Double-deleted ascospores were able to germinate but had a limited capa

69 l cleistothecia carrying a reduced number of ascospores, whereas DeltagprADeltagprB eliminated fruiti

70 ed on its formation of oblate, smooth-walled ascospores within yellow or yellow-green tufts of aerial