1 We then
asked whether 17B1.3 prevents signaling by binding to a
2 We exploit this comprehensive resource to
ask whether a common Rab compartment architecture underl
3 We
ask whether a similar set of L chains exists in the huma
4 We also
asked whether a CB1 positive allosteric modulator would
5 abel adult-generated granule cells, we first
asked whether a comparable process occurs in the adult h
6 s into the cellular phase of coagulation, we
asked whether a functional vwf gene is present in the At
7 Here, we
asked whether a generalized Bayesian magnitude estimatio
8 To address this question, we
asked whether a missense mutation associated with human
9 We
asked whether a single transient episode of HSV-1 epithe
10 Here, we
asked whether a south-north expansion wave of L. granate
11 We
asked whether a viral bZIP transcription factor, Zta (BZ
12 ons with different biological activities, we
asked whether Abeta can assume structurally and function
13 T kills macrophages rapidly and potently, we
asked whether ACT would also kill neutrophils.
14 xperimentally address this knowledge gap, we
asked whether activation of PZ(Vgat) neurons could atten
15 Here, we
ask whether adaptation to historical conditions can incr
16 Finally, we
asked whether adaptor function conferred by Zap70 tyrosi
17 enhance the function of a compound, we then
asked whether adding a glucose makes aesculin activate N
18 As proof of concept we
asked whether administering low-level TLR2 ligands in ad
19 ert this apparent neuroprotective effect are
asking whether agonists, antagonists, partial agonists,
20 Finally, we must also
ask whether an elixir for aging would be applicable to e
21 In this study, we
asked whether an augmented ability to sense Ag complemen
22 Here, we
asked whether an increase in expression level of the cel
23 different approaches to their treatment, we
ask whether and how an unexpected event occurring at a p
24 Here we
ask whether and how motor control and redirected somatos
25 Here we
ask whether and how the dynamics of finding mates intera
26 ons across the continental United States and
asked whether and how fire-climate relationships vary ge
27 k of the measles virus hemagglutinin (H), we
asked whether and how it may facilitate membrane fusion
28 Here, we
ask whether,
and to what extent, landscape simplificatio
29 Here, we
asked whether any other co-factors associate with TAPBPR
30 o the evolutionary and heterosis mechanisms,
asking whether any effects of genetic mixture occurred i
31 We addressed this issue by
asking whether articulatory configurations can influence
32 A (DORmO) mouse model of type 1 diabetes, we
asked whether autoimmune insulitis was associated with c
33 Physiological data are limited, so we
asked whether basal transmission and long-term potentiat
34 Next, we
asked whether basic image properties could also explain
35 Here we
asked whether BCL6 controlled helper function through do
36 Here, we
asked whether BD might be characterized by enhanced risk
37 iment with humans with frontal lobe lesions,
asking whether behavioral impairments can be linked to l
38 In this study, we
asked whether beta-cell dedifferentiation can be prevent
39 We
asked whether blockade of Netrin-1 or its receptors [Unc
40 We
asked whether bone marrow neuropathy and the associated
41 We
asked whether brain connectomics can predict response to
42 We
asked whether bumblebees specialize over timescales shor
43 Thus, we
asked whether C1q binds to CD33 and if C1q mediates CD33
44 Here we
asked whether C5a generated locally in response to CNV r
45 Using this in vivo model, we
asked whether CB-BF populations that express characteris
46 In the current study, we
asked whether CD30 is expressed on the surface of neopla
47 First, we
asked whether CD4-induced Env conformational changes aff
48 We
asked whether cells have the ability to limit the degree
49 We therefore
asked whether cells in the tumour stroma might explain t
50 sonance energy transfer tension biosensor to
ask whether changes in forces across cell-cell junctions
51 We
asked whether chronic early life stress disrupts maturat
52 Here, we
asked whether CK2 interacts with other proteins involved
53 Therefore, the current study
asked whether CodY also regulates sporulation and CPE pr
54 In this study, we
asked whether combining innate immune activation in the
55 omplement components may deposit on RBCs, we
asked whether complement deposits on the surface of RBC
56 ularly effective opsonin for Francisella, we
asked whether complement-dependent uptake of F. tularens
57 Here we
ask whether computerized CL auditory training can enhanc
58 We
asked whether continuous host variation in susceptibilit
59 We
ask whether cooperative behavior is further promoted in
60 We
asked whether CP55,940 [(-)-3-[2-hydroxy-4-(1,1-dimethyl
61 ed to be in close proximity to pericytes, we
asked whether cross-talk between pericytes and NSCs was
62 the chondrocyte differentiation pathway, we
asked whether Ctgf (Ccn2) is the direct target gene of S
63 Here we
ask whether currently available ELISAs have similar prog
64 The Euro-EWING99-R1 trial
asked whether cyclophosphamide may replace ifosfamide in
65 given to human subjects, it may be useful to
ask whether cytokine polarization is the appropriate fra
66 Here, we
ask whether DA release is modulated by delivery of rewar
67 We
asked whether decreased Cyfip1 levels altered the functi
68 We
asked whether differences between sun-exposed and shaded
69 Here, we
ask whether different ploidy levels > 2x express differe
70 We first
asked whether different combinations of facultative symb
71 Here we
asked whether directly activating single alphaIIbbeta3 m
72 ration, and metabolic regulation in vivo, we
asked whether DJ-1 contributes to MG detoxification in v
73 Here, we directly
ask whether DNA methylation is altered following skin ag
74 all been shown to have roles in mitosis, we
asked whether DNA-PKcs phosphorylates SAF-A in mitosis.
75 We
ask whether domain interactions and calcium binding affe
76 Here we
asked whether dopaminergic signaling supports a TD learn
77 other aspects of female differentiation, we
asked whether dsx-expressing neurons mediate virgin fema
78 We
asked whether eltrombopag, a thrombopoietic agent, would
79 Here, we
asked whether engagement in an auditory detection task c
80 ty of homeoproteins, including Engrailed, we
asked whether Engrailed intercellular transfer was also
81 We
asked whether environmental factors impact SNP309G funct
82 gene expression via epigenetic phenomena, we
asked whether epigenetic factors contribute to down regu
83 Here we
asked whether epileptic activity induces plastic changes
84 We
asked whether ERalpha expression contributes to the deve
85 In this study, we
asked whether ex-T reg Th2 cells develop and contribute
86 In this study, we
asked whether exosomes from uninfected cells could activ
87 To test this hypothesis, we
asked whether experimentally elevating levels of the glu
88 ith the bacterium Caulobacter crescentus and
asked whether exposure to a moderate concentration of so
89 Here we
asked whether fatigue induced by time-on-duty (~24 hours
90 Here, we
ask whether feedbacks can increase the information avail
91 I
ask whether fluctuation in Chinook salmon (Oncorhynchus
92 stic, multidimensional learning environment,
asking whether focal lesions affected trial-by-trial att
93 Here, we
asked whether folding of a complex RNA can be understood
94 Here, we
asked whether generalization occurs in a nonhuman vocal
95 Here, we
asked whether genetic heterozygosity of PGC-1alpha modul
96 In this study, we
asked whether genetically distant NS3 proteins can alter
97 Therefore, we
asked whether Gi-GPCR signaling regulates beta-cell mass
98 We have
asked whether glucose metabolism is enhanced by either a
99 Here, we
asked whether gneY and gneZ are required for the synthes
100 Here we
asked whether H3K9me3, through mediating gene repression
101 To
ask whether handshakes are used to sample conspecific so
102 certained in genome sequencing data sets and
ask whether human populations differ in their predicted
103 Here we
ask whether humans are also sensitive to how the other's
104 Here, we
asked whether hyperthermia alters the response to chemot
105 In this study we
asked whether IL-27 is produced by human secondary lymph
106 n to interfere with T cell functionality, we
asked whether IL-7 affects the functionality of human DN
107 mation transfer as a sign of decoupling, and
asked whether impaired local information processing lead
108 Here we address a further aspect,
asking whether impression formation is mandatory.
109 In this study, we
ask whether in addition to promoting local translation,
110 We
asked whether in blindness, occipital cortices: (1) deve
111 egulate the activity of axonemal dyneins, we
asked whether in vitro acetylation or deacetylation of l
112 Here, we
asked whether inactivation of the UDP-galactose:beta-gal
113 Here, we
ask whether increased expression of RNF168 or 53BP1 impa
114 We
asked whether increased serotonin levels in newly identi
115 Finally, we
asked whether individual variation in wheel use within a
116 We
ask whether individuals with more general education and
117 Exp. 1
asks whether intended harm motivates blame or merely dem
118 Here we
asked whether interaction of gD with a cognate entry rec
119 We
asked whether it is possible to study these mechanisms i
120 ed genes is higher in wake than in sleep, we
asked whether it is specifically wake associated with sy
121 nculin (VCL) is an actin-binding protein, we
asked whether it participates in skeletal degradation.
122 sented with severe upper limb impairment and
asked whether it was possible to differentiate those wit
123 In the present study, we
asked whether it was possible to immediately change comp
124 esenting the ending portion of one voice and
asking whether it came from the cued voice.
125 is automatic has previously been studied by
asking whether it can occur implicitly (outside awarenes
126 glucopyranoside, suppresses inflammation, we
asked whether its anti-inflammatory activity is associat
127 Here we
ask whether lactate is responsible for T cells remaining
128 Here we
asked whether learning in young and older people may ref
129 We
asked whether LECs play a constitutive role in T cell de
130 of Inhibitor of DNA binding (ID) proteins to
ask whether lineage priming affects self-renewal of huma
131 More generally, we
ask whether linguistic knowledge is fully embodied or wh
132 We therefore
asked whether LPL functions downstream of Mst1.
133 We
asked whether lung function trajectories from birth to 1
134 We
asked whether MarA expression also has a stochastic comp
135 biophysical properties of the tissue, and we
asked whether matrix rigidity modulated the sensitive ve
136 he cellular level or the community level, we
asked whether matrix-defective mutant strains could be c
137 mplitude of afterhyperpolarization led us to
ask whether METH alters the activity of Ca(2+)-activated
138 We
asked whether minimal residual disease (MRD) determined
139 We then
asked whether miR-3189 was expressed in clinical samples
140 ers have been linked to oxidative stress, we
asked whether mitochondrial reactive oxygen species (ROS
141 e we used multimodal neuroimaging methods to
ask whether modulating reward-related signaling in the A
142 We
asked whether N deposition experiments adequately repres
143 We therefore
asked whether neonate-specific mechanisms prevent lympho
144 ng to become living kidney donors frequently
ask whether nephrectomy will affect their future pregnan
145 We
asked whether NEU1 might regulate EC capillary-like tube
146 ficiency in Col2a1-positive chondrocytes, we
asked whether neurofibromin is required to control FGFR1
147 These puzzling results prompted us to
ask whether neuroligin-3 mutant phenotypes may be reshap
148 otosynthetic proteins has led researchers to
ask whether nontrivial quantum phenomena are biologicall
149 Here, we
asked whether OFC lesions in humans disrupted the abilit
150 Then, we
asked whether or not these two neurotrophins exert their
151 We therefore
asked whether OSNs in different locations evolve unique
152 Here we
ask whether our recognition of sound sources and spaces
153 Using range expansion simulations, we
asked whether our ability to detect genomic regions invo
154 Here we
ask whether PACAP is also involved in producing reinstat
155 sms regulate CXCR4 expression; therefore, we
asked whether PAF activates epigenetic mechanisms in mas
156 We first
asked whether pairs of young children (M = 74 months) wo
157 We
asked whether patients with FHL with immunodeficiency bu
158 Specifically, we
ask whether patterns of social contact (group membership
159 d with progressive capillary rarefaction, we
asked whether PDGF-CC neutralization in fibrosis might h
160 Using a genetic ablation model, we
asked whether pericyte loss alone is sufficient for capi
161 Here we
ask whether perturbations to two subtypes--parvalbumin-p
162 We here
ask whether piwi/piRNAs are also expressed and have func
163 Even so,
asking whether population genetics explains endosymbiosi
164 ss the specificity of inhibitory mechanisms,
asking whether preparatory inhibition affects task-irrel
165 We
asked whether previously reported anomalous white matter
166 We
ask whether primates are sensitive to global changes tha
167 We
asked whether prolonged AICAR treatment is beneficial in
168 We then
asked whether prolonged PSAP/IntA experience would never
169 We next
asked whether proprioceptive nerve endings are similarly
170 We first
asked whether reassembled communities differ from undist
171 Therefore, we
asked whether replacement of brain-resident myeloid cell
172 A separate item
asked whether respondents used a condom at last sexual i
173 lored factors that govern such patterns, and
asked whether reward specialists made more foraging bout
174 We
asked whether risk alleles disrupt transcription factor
175 We
asked whether ROM1 could modify the phenotype of a PRPH2
176 Here, we
asked whether salience-driven distraction is prevented b
177 In this study, we
asked whether short- and/or long-term behavioral consequ
178 We
asked whether similar individual variation exists for co
179 We
asked whether single nucleotide polymorphisms (SNPs) in
180 isite for sleep-like states, prompting us to
ask whether sleep is present in Cnidaria.
181 natomical foundations of this hypothesis, we
asked whether space-time interactions can be observed in
182 oups of putative target genes (regulons) and
asked whether specific regulons are enriched for genes a
183 Here, we
asked whether SPL and other RGS18 binding proteins such
184 To address this question, we
asked whether splenic immune populations were altered in
185 We have characterized the SSc epidermis and
asked whether SSc-injured epidermal cells release factor
186 Finally, we
ask whether stem cell aging establishes an epigenetic 'm
187 community in Boston, Massachusetts, who were
asked whether strabismus was present.
188 Here we
asked whether Stx1a and Stx2a act differently when deliv
189 We
ask whether such 1D responses are consistent with the sy
190 Here, we
ask whether such directional post-mating (that is, crypt
191 ollowing direct injury of sensory nerves, we
asked whether such a dysregulation also plays a role in
192 Here, we
asked whether targeting of ribosome biogenesis can be us
193 e of autologous antigen-presenting cells, we
asked whether Th17 cells are present in young infants th
194 onstrated topographic maps of numerosity, we
ask whether the brain also contains maps of object size.
195 nce than clinical research, it is natural to
ask whether the dynamic patterns of neural circuit activ
196 Here we
ask whether the excitability of the motor pathways to th
197 splicing modifies different channels, and to
ask whether the functional consequences of this splicing
198 es developed from small-scale experiments to
ask whether the functional diversity within a range of w
199 We show that an important first step is to
ask whether the functional unit of pathogenesis is a bac
200 llowed up on the previous proposals [1,2] to
ask whether the image features that increase seizure lik
201 We
ask whether the new resolution conferred by molecular in
202 Here we
ask whether the precision of stereopsis varies with prof
203 We
ask whether the prevalence and medical history of angina
204 Here, we
ask whether the quantitative invariants of V1 architectu
205 We
asked whether the >80% sequence differences throughout t
206 We then
asked whether the aberrant tangential migration of GABAe
207 Here we
asked whether the anatomical features of individual huma
208 Here, we
asked whether the anterior temporal lobe (ATL) serves as
209 We
asked whether the CD161 ligand LLT1 could be expressed o
210 Hence, we
asked whether the CD8(+) Treg cells could directly targe
211 Here, we
asked whether the central electrosensory system actually
212 minals of the MF is target specific, we here
asked whether the corelease of glutamate and GABA was al
213 Finally, we
asked whether the defining molecular signature of J-RGCs
214 ther who had severe itch for several months,
asked whether the dermatologist accepted the relevant pl
215 We therefore
asked whether the DNA-binding signature of ERalpha diffe
216 This study
asked whether the EBV major membrane glycoprotein could
217 This study
asked whether the functional expression of voltage-gated
218 We therefore
asked whether the functionality of Nodal can be augmente
219 s genomes enter the nucleus as naked DNA, we
asked whether the HIRA chaperone complex affects herpesv
220 In this study, we
asked whether the hyperpolarization-activated cyclic nuc
221 action (go or no-go) were orthogonalised, we
asked whether the impact of stress on learning was actio
222 ith PLA gene expression information and were
asked whether the lesions should be biopsied.
223 Here, we
asked whether the membrane composition of neurons, at th
224 linked with the underlying actin network, we
asked whether the myelin instability associated with PMP
225 We
asked whether the presence of kin might act on energy al
226 We
asked whether the proteome of secreted cytokines and sig
227 We
asked whether the rates of these underlying processes ca
228 We then
asked whether the replication and the mitotic phenotypes
229 In this issue of the JCI, Lu and colleagues
asked whether the results of neural stem cell transplant
230 Yet we have never
asked whether the same is true for animals.
231 Here, we
asked whether the size of IPS is affected by manipulatio
232 Here, we
asked whether the synaptic organizer agrin might regulat
233 ow the intestinal microbiome develops, so we
asked whether the transfer of an antibiotic-perturbed mi
234 In this study, we
asked whether the tyrosine kinase Zap70 is required to t
235 re on angina (symptomatology) and a question
asking whether the respondent had ever been told by a me
236 orestation regulations in some countries, we
ask whether their movements are determined partly by dif
237 in alloantigen-induced T cell activation and
asked whether their absence might impact in vivo allores
238 At baseline, patients were
asked whether their oncologist had disclosed an estimate
239 We
asked whether there is a common neural code that underpi
240 The patient
asked whether there is any role for definitive local the
241 Here, we
ask whether these documented latitudinal patterns can be
242 Here we
ask whether these elevated cortisol levels shift risk pr
243 To
ask whether these findings might be due to abnormalities
244 nections between alpha-Syn and tau led us to
ask whether these proteins might be regulated through a
245 Herein, we
asked whether these assays were able to help determine t
246 In this study, we
asked whether these factors also predict diversity and t
247 However, we
asked whether these findings were an artifact of conside
248 homologs of FGF3, FGF8 and FGF receptors and
asked whether these functions are ancestral features of
249 Here we
asked whether these proneural genes also regulate lamina
250 Here, we
asked whether these SOC components and Homer1 are presen
251 We
asked whether these TALENs could create targeted somatic
252 The present study
asks whether these contradictory conclusions result from
253 emporally distinct modes of inactivation and
ask whether they can be distinguished pharmacologically.
254 this process in the BLA and, if present, to
ask whether they would still be dependent on OFC input.
255 en the apparent power of search rankings, we
asked whether they could be manipulated to alter the pre
256 Sophia, Athens, Reykjavik and Vilnius) were
asked whether they had allergic symptoms associated with
257 ns were alerted to the initial diagnosis and
asked whether they had previously treated patients with
258 , early-stage breast cancer, physicians were
asked whether they intended to administer adjuvant CT be
259 are associated with nonlymphoid tissues, we
asked whether they populated secondary lymphoid organs (
260 Institutions were
asked whether they were familiar with "Image Gently," th
261 Participants were
asked whether they would opt for diagnostic testing on t
262 cts using allozymes and microsatellites, and
ask whether this is consistent with its colonization his
263 sequentially, in a cascade-like manner, and
ask whether this is likely to be an inherent property.
264 Here we
ask whether this is maintained when the body is made mor
265 We
ask whether this pair of muscles acts as a muscle synerg
266 rotein kinase II (alpha-CaMKII), and here we
ask whether this role is conserved in the adult brain.
267 we extend the biological market framework to
ask whether this theory is of use to evolutionary biolog
268 Here, we
ask whether this VIP-SST circuit enhances plasticity dir
269 Now we
asked whether this cascade also controls axon bifurcatio
270 In this work we
asked whether this hypothetical weakness is a problem in
271 ent receptor potential ankyrin 1 (TRPA1), we
asked whether this increase in lipid content occurs in T
272 Here we
asked whether this is also true of native and endemic Me
273 We
asked whether this is due to neuronal loss or selective
274 Here, we
asked whether this mechanism generalizes to OFF-selectiv
275 sa first touches an object, and we therefore
asked whether this parameter could provide the rat with
276 We
asked whether this peculiar expression pattern (earlyOff
277 We
asked whether this polarized effect of ATP is, in fact,
278 In this study, we
asked whether this protective allele was associated with
279 s the developing skin along the A-P axis, we
asked whether this strain alone drives global planar pat
280 Recently, a number of authors have
asked whether this type of motor learning problem might
281 infections during the first year of life and
asked whether this was modified by prenatal and early-li
282 Here, we
ask whether TIN2 needs to interact with both TRF1 and TR
283 We
asked whether top-scoring PPARgamma activators identifie
284 Our second experiment
asked whether trajectory control could be generated if t
285 We
ask whether trees differ from WBE symmetry and if it mat
286 Here we
asked whether trichromats express chromatic-specific cir
287 Moreover, we
ask whether TRPV4-mediated Ca(++)-influx evokes mast cel
288 In this study, we
asked whether tumor clone-initiating cells (TcICs) were
289 Experts and non-experts were
asked whether two concurrent bumblebee images depicted t
290 Here, we
asked whether valACV suppresses VL in HIV-1 infected HSV
291 We then
ask whether variation in movement based on (i) age, (ii)
292 Here, we
ask whether variations in ocean phytoplankton biodiversi
293 sequencing in mouse collecting duct cells to
ask whether vasopressin signaling selectively increases
294 Here we
ask whether video game play also alters temporal process
295 We
asked whether visits to physician offices and hospital o
296 Here, we
asked whether visual training can induce recovery outsid
297 Now is a fair time to
ask whether we are far down a trail leading to a deep un
298 anges in species across multiple regions, we
ask whether we can discriminate an urban signature of ph
299 ressed mainly in pancreatic acinar cells, we
asked whether we could find structural pancreatic change
300 Here, we
asked whether WNT signaling directs facial morphogenesis