ライフサイエンスコーパス: asparagine

1 CBG steroid-binding sites is replaced by an asparagine.

2 ach if D21 is mutated into the corresponding asparagine.

3 outing even in the presence of glutamine and asparagine.

4 d residue in the -2 position relative to the asparagine.

5 e reversed by exogenous supplementation with asparagine.

6 provide non-essential amino acids, including asparagine.

7 on the extracellular pool of the amino acid asparagine.

8 design of variants that attain a low KM for asparagine.

9 ansporter highly selective for glutamine and asparagine.

10 w/w) for beta-ODAP and 0.006-0.47% (w/w) for asparagine.

11 rast, human enzymes have a millimolar KM for asparagine.

12 f the nonessential amino acids aspartate and asparagine.

13 human L-asparaginases have millimolar KM for asparagine.

14 rolysis and compensating for the lack of the asparagine.

15 nonical RhoGAP domains lacking the auxiliary asparagine.

16 the T19A, T116A, and K188M mutants soaked in asparagine.

17 correct positioning of l-glutamine but not l-asparagine.

18 Mutagenesis analysis in Kir1.1 revealed that asparagine 171 (N171) is the only pore-lining residue re

19 -P-X(140) motif, leucine-66, proline-67, and asparagine-176 may account for the broad substrate speci

20 e and asparagine 1788 to lysine) and Nav1.6 (asparagine 1768 to aspartic acid and leucine 1331 to val

21 both Nav1.1 (arginine 1648 to histidine and asparagine 1788 to lysine) and Nav1.6 (asparagine 1768 t

22 tinal rod photoreceptors, is glycosylated at asparagines-2 and 15 on its N-terminus.

23 ns are presented, including one with deleted asparagine 254, suggesting a role for this amino acid, w

24 e of a single N-linked glycosylation site at asparagine 297 of the Fc, with deglycosylation resulting

25 e bond between N-myristoylated glycine-2 and asparagine-3 of human ARF1, thereby providing a new mech

26 a resistant M2 variant encoding a serine to asparagine 31 mutation (S31N) with improved efficacy ove

27 hreonine (Thr)-244, Thr-312, serine-379, and asparagine-381 are related to substrate binding or catal

28 the constant domain 3 (Cepsilon3) of IgE, at asparagine-394 (N394) in human IgE and N384 in mouse.

29 Asparagine 46 is considered to be important for the stru

30 ghest amount of acrylamide was produced from asparagine (5987.5microg/kg) and the lowest from phenyla

31 glycosylation of the variable heavy chain on asparagine 85 in Framework Region 3.

32 trast, substitution of p5M to a conventional asparagine abolished recognition by the H-2D(b)/Trh4-spe

33 acing the glutamines in these sequences with asparagines abolished suppression and converted them to

34 which the aspartic acid is substituted by an asparagine (Ac-LPFFN-NH2).

35 spartic acid residue at position 398 with an asparagine (alpha5DN), has recently been associated with

36 the Na(+) affinity of site III aspartate to asparagine and alanine mutants is rescued by second-site

37 66Y, Y176F, and K288S/Y176F rapidly depleted asparagine and also down-regulated the transcription of

38 vide S Typhimurium the ability to catabolize asparagine and assimilate nitrogen.

39 The fructose, asparagine and chitosan mixture had more MRPs compared t

40 n a food model system consisting of glucose, asparagine and chitosans.

41 es and their combination including fructose, asparagine and different molecular weight chitosans.

42 e (ASNS) converts aspartate and glutamine to asparagine and glutamate in an ATP-dependent reaction.

43 A constant amount of asparagine and glutamine in palm olein and soy bean oils

44 es, aspartate and glutamate, were mutated to asparagine and glutamine, respectively.

45 an enzyme that catalyzes the hydrolysis of L-asparagine and isoaspartyl-peptides.

46 ibactin C (6) was converted to N-myristoyl-d-asparagine and its corresponding colibactin by colibacti

47 l plants, but contained diminished levels of asparagine and much higher levels of lysine.

48 Acrylamide is produced from free asparagine and reducing sugars during high-temperature c

49 rylamide is formed from the reaction between asparagine and reducing sugars.

50 amide and HMF in baked biscuits, nor between asparagine and the sum of glucose and fructose concentra

51 d of three stacked concentric phenylalanine, asparagine and tyrosine rings that may guide the extende

52 plants exhibited extremely reduced levels of asparagine and were greatly affected in their phenylalan

53 insically disordered, enriched in glutamines/asparagines and depleted in hydrophobic residues.

54 This domain contained a conserved asparagine, and its mutation resulted in an increase in

55 isoleucine metaclusters, along with serine, asparagine, and the previously studied phenylalanine, ar

56 f select amino acids and peptides, including asparagine, arginine and proline.

57 Here we describe a role for asparagine as an amino acid exchange factor: intracellul

58 Gold nanoparticles synthesized using L-Asparagine as reducing and stabilization agent were empl

59 y the Maillard reaction of glucose (GL) with asparagine (AS).

60 L-ASP from Escherichia coli deamidates asparagine (Asn) and glutamine, with an ~10(4) higher sp

61 was able to simultaneously separate Gln and asparagine (Asn) deamidation products even for those pep

62 tion, aspartic acid (Asp) isomerization, and asparagine (Asn) deamidation.

63 silk, pure cystine (dimer of cysteine), and asparagine (Asn) did not show signs of racemization at t

64 This, in turn, increases asparagine (ASN) synthetase expression and the productio

65 ne-rich region contains two highly conserved asparagines (Asn-638 and Asn-652).

66 amino acids accumulated, and an imbalance of asparagine/aspartate, glutamate/glutamine, and nutrient

67 MS analysis of alanine, alpha-ketoglutarate, asparagine, aspartic acid, cystathionine, total cysteine

68 hain dicarboxylacylcarnitine metabolites and asparagine/aspartic acid could serve as biomarkers assoc

69 roxyisovalerylcarnitine/malonylcarnitine and asparagine/aspartic acid were associated with worse clin

70 contrast, only those animals homozygous for asparagine at codon 146 (NN animals) succumbed to oral c

71 s concluded that only animals homozygous for asparagine at codon 146 succumb to scrapie under natural

72 l distribution of the animals homozygous for asparagine at codon 146 was significantly shorter than t

73 with an additional mutation in M (serine to asparagine at position 220), strongly implying that Thr2

74 ycosylated in their Fc domain at a conserved asparagine at position 297.

75 for permissive CD4 alleles, which encode an asparagine at position 39 of the receptor.

76 ged avian-origin H7N9 virus also contains an asparagine at position 52 and shows reduced Mx sensitivi

77 resulted in a single exchange of tyrosine to asparagine at position 52 in NP (in close proximity to t

78 We found that lysine at position 627 and asparagine at position 701 in PB2 are essential for mamm

79 lysine at position 627 and aspartic acid to asparagine at position 701) of A(H7N9) viruses for mamma

80 were mediated via a conserved tyrosine- and asparagine-based motif in the cytoplasmic domain of DNAM

81 mutation of these pUS9 arginine residues to asparagine blocked the binding of both recombinant and n

82 uld be engineered to retain activity against asparagine but not glutamine.

83 propionic acid (beta-ODAP), homoarginine and asparagine by a simple and fast capillary electrophoreti

84 property allowing for the depletion of blood asparagine by bacterial asparaginases is their low micro

85 Initially, N-CDs were prepared from L-asparagine by pyrolysis and characterized by different s

86 Collectively, these results indicate that asparagine catabolism contributes to S Typhimurium virul

87 the translational fidelity of glutamine and asparagine codons.

88 but substitutions of serine by aspartate or asparagine completely abolished substrate transport.

89 tipping point in the ratio below which free asparagine concentration could affect acrylamide formati

90 scuits with glucose, also having the highest asparagine concentration.

91 e synthetase knockdown and altering of media asparagine concentrations, we show that intracellular as

92 Rather, conserved asparagines contributed to suppression of the substrate

93 his terpene synthase revealed an active site asparagine critical for water capture and specificity du

94 show that substituting Asp(25) of medin with asparagine (D25N) impedes assembly into fibrils and stab

95 of methionine oxidation and higher levels of asparagine deamdiation were observed.

96 that modifies a eukaryotic target through an asparagine deamidase activity, which in turn elicits hos

97 Asparagine deamidation and aspartate isomerization were

98 In this study, methionine oxidation and asparagine deamidation are the only two modifications id

99 Asparagine deamidation occurs spontaneously in proteins

100 tions as well as faint modifications such as asparagine deamidation or aspartic acid isomerization.

101 vel beta-sandwich adhesion domain and unique asparagine-dependent super-helical stalk.

102 ginase activity above defined thresholds and asparagine depletion compared with SS-PEG2500 and has a

103 Also, asparagine depletion via the ASNS inhibitor amino sulfox

104 property to allow the required therapeutic l-asparagine depletion.

105 In addition, we previously showed that asparagine deprivation such as that mediated by l-aspara

106 h an ~10(4) higher specificity (kcat/Km) for asparagine despite only one methylene difference in leng

107 of Na2 affinity by substituting Asp-420 with asparagine dramatically increased cation permeability in

108 delta-secretase, also known as asparagine endopeptidase (AEP) or legumain, is a lysosom

109 Here we show that asparagine endopeptidase (AEP), a lysosomal cysteine pro

110 Here we show that asparagine endopeptidase (AEP), a pH-controlled cysteine

111 Asparagine endopeptidase (AEP), also called legumain, is

112 Delta-secretase, a lysosomal asparagine endopeptidase (AEP), simultaneously cleaves b

113 are sensitive to proteases (cathepsin B and asparagine endopeptidase) that are over-expressed by res

114 that the substrate 2-ABA itself supplies the asparagine-equivalent amino function that assists in cat

115 ere we report beta5i-selective inhibition by asparagine-ethylenediamine (AsnEDA)-based compounds and

116 an amino acid exchange factor: intracellular asparagine exchanges with extracellular amino acids.

117 ant with His(273) and His(274) exchanged for asparagines exhibits a much less pronounced pH dependenc

118 terica fra locus, which encodes the fructose-asparagine (F-Asn) utilization pathway, are highly atten

119 Salmonella-specific nutrient source fructose-asparagine (F-Asn), to the probiotic bacterium Escherich

120 berculosis revealed the general relevance of asparagine fermentation and a variable contribution of t

121 10N) that resulted in the substitution of an asparagine for an aspartic acid at position 10 of ACT1 (

122 racteristic arginine finger and an auxiliary asparagine for catalysis.

123 expressing a mutant pRB protein carrying an asparagine for phenylalanine substitution at position 75

124 te lymphoblastic leukemia, acts by depleting asparagine from the blood.

125 Using a glucose-glycine and asparagine-fructose system as a Maillard reaction model,

126 Asparagine further proved crucial in glutamine-deprived

127 mination of six amino acids namely (alanine, asparagine, glutamine, proline, serine and valine) for S

128 PC6 (BCAAs and aromatic AAs) and PC10 (asparagine, glycine, and serine) made the largest contri

129 While both antibodies share a canonical asparagine-glycine (NG) motif in a structural loop, this

130 rge primarily from a single AA substitution (asparagine-->threonine) via a single nucleotide mutation

131 h the same pUS9 arginine residues mutated to asparagine (HSV-1pUS9KBDM) and then restored them being

132 for their ability to cleave and to catalyze asparagine hydrolysis, in addition to being examined str

133 The asparagine hydroxylase, factor inhibiting HIF (FIH), con

134 Studies investigating whether asparagine hydroxylation is a general regulatory oxygen-

135 glutamine-derived nitrogen and aspartate to asparagine) impaired EC sprouting even in the presence o

136 due 74 corresponding to lysine in mGluR4 and asparagine in mGluR7 might play a key role, and, indeed,

137 There was much greater free asparagine in potatoes grown at the Doncaster site compa

138 uantification of beta-ODAP, homoarginine and asparagine in seed extracts of 52 Lathyrus local landrac

139 Our structures reveal bound asparagine in the active site that has unambiguously not

140 ance of deamidation on glutamine rather than asparagine in the archeological samples was attributed t

141 thionine residue and deamidation of the same asparagine in the corresponding acidic fractions generat

142 vates Rho GTPases by deamidating a conserved asparagine in the GTPase switch-I region.

143 to accommodate the replacement of leucine by asparagine in the N-terminal cluster revealed the existe

144 A conserved asparagine in the oxyanion hole, Asn-169, is found to be

145 Glycosylation of this asparagine in zebra finch CBG does not influence its ste

146 Furthermore, we identified two conserved asparagines in MTN1 and MTN2 from Arabidopsis that confe

147 s, that native TRPP2 is glycosylated at five asparagines in the first extracellular loop.

148 gosaccharyltransferase from a lipid donor to asparagines in the sequon NX(S/T) of secreted polypeptid

149 red by replacing four of five glutamines for asparagines in various combinations via site-directed mu

150 or more conservative residues, glutamine or asparagine, in the GERAMT-binding site.

151 coma cells combined with depletion of plasma asparagine inhibited tumor growth in vivo.

152 -asparaginases to catalyze the hydrolysis of asparagine into aspartic acid and ammonia has been recen

153 eukemia, the data suggest that intracellular asparagine is a critical suppressor of apoptosis in many

154 Collectively, our results indicate that asparagine is an important regulator of cancer cell amin

155 Asparagine is formed by two structurally distinct aspara

156 sm and nucleotide synthesis, suggesting that asparagine is involved in coordinating protein and nucle

157 However, the auxiliary asparagine is missing in the RhoGAP domain of Myo9b (Myo

158 Asparagine is present in plums at relatively high concen

159 tes 96 and 107 are conservatively mutated to asparagine is strongly impaired in dimer formation but m

160 d-linked oligosaccharide (LLO) onto acceptor asparagines is catalyzed by the integral membrane protei

161 examine the metacluster formation of serine, asparagine, isoleucine, and tryptophan.

162 Several carbon sources (L-Asparagine, L-Aspartic Acid, L- Glutamic Acid, m- Erythr

163 ed by a group of amino acids that includes l-asparagine, l-glutamine, l-threonine, l-arginine, l-glyc

164 The continuous hydrogen-bond network (asparagine ladder) formed among the asparagine residues

165 a single-domain architecture with an intact asparagine ladder, a three-domain architecture with the

166 gs, buried polar residues, salt bridges, and asparagine ladders.

167 The serum asparagine level was not always completely depleted with

168 from increased branched chain amino acid and asparagine levels and altered expression of key enzymes

169 y, we show that maintenance of intracellular asparagine levels is critical for cancer cell growth.

170 e concentrations, we show that intracellular asparagine levels regulate uptake of amino acids, especi

171 idase) that is significantly associated with asparagine levels, with an effect that is independent of

172 Asparagine-linked (N-linked) glycosylation of ZIP14, par

173 Labeling of released asparagine-linked (N-linked) oligosaccharides from glyco

174 es the transfer of fucose from GDP-fucose to asparagine-linked GlcNAc of the N-glycan core in the med

175 ells upon knockdown of Hivep3 and identified asparagine-linked glycosylation 2 (Alg2), which encodes

176 Asparagine-linked glycosylation is a post-translational

177 e central enzyme in the Campylobacter jejuni asparagine-linked glycosylation pathway is the oligosacc

178 Asparagine-linked glycosylation plays important roles fo

179 IgG bears asparagine-linked oligosaccharide side chains in the Fc

180 Asparagine-linked protein N-glycosylation, the most comp

181 lyzed by strategies similar to proteases and asparagine lyases.

182 N61 on binding Loop D, suggesting these two asparagines may interact.

183 novel link between endothelial glutamine and asparagine metabolism in vessel sprouting.

184 serine, tryptophan, phenylalanine, tyrosine, asparagine, methionine, and lysine.

185 In 09HA, a serine-to-asparagine mutation coincided with a salt bridge destabi

186 Deamidation of glutamine (Q) and asparagine (N) has been recognized as a marker of degrad

187 he mutant containing an aspartic acid (D) to asparagine (N) substitution at position 405 (D405N) muta

188 A missense mutation, replacing asparagine (N) with lysine (K), at position 46 in the Gl

189 mammals reportedly lack proteins displaying asparagine (N)-linked paucimannosylation (mannose(1-3)fu

190 Previously, antibodies specific to a gluco-asparagine (N-Glc) glycopeptide, CSF114(N-Glc), were ide

191 The composition of asparagine(N)297-linked glycans can modulate the binding

192 We found that a highly conserved asparagine (N220) in the first transmembrane segment is

193 en bond between a glutamic acid (E90) and an asparagine (N258) residue suffices to keep the gate of C

194 fer of the oligosaccharide onto the acceptor asparagine of nascent proteins during the process of N-g

195 ation of hSERT at a single, highly conserved asparagine on TM1 (Asn-101) to provide several lines of

196 The substitution of Ser-254 with either an asparagine or a glutamine increases the l-asparagine spe

197 n conversion involved the gain or loss of an asparagine or glutamine residue.

198 The presence of asparagine or threonine in over 99% of all human seasona

199 sual maturation process involving the N-acyl-asparagine pro-drug intermediates preamicoumacins, which

200 midotransferase that converts aspartate into asparagine, produced the strongest inhibitory effect on

201 any prion-forming proteins contain glutamine/asparagine (Q/N) rich domains, and there are conflicting

202 Most yeast prion proteins contain glutamine/asparagine (Q/N)-rich prion domains that drive prion act

203 Asparagine reacted with fructose to form a Schiff base b

204 Through its exchange factor role, asparagine regulates mTORC1 activity and protein synthes

205 In addition, we show that asparagine regulation of serine uptake influences serine

206 to develop a mechanistic model, based on the asparagine-related pathway, for acrylamide and HMF forma

207 Asparagine reliance of sarcoma cells may represent a met

208 ion at the mitotic cortex of leucine-glycine-asparagine repeat protein (LGN) and nuclear mitotic appa

209 the mechanistic aspects of the synthesis of asparagine repeats and about their implications in the g

210 an extreme AT-rich genome and a profusion of asparagine repeats associated with low complexity region

211 An asparagine residue (Asn-106) in transmembrane segment 2

212 n 1 reveals that a glycan emanating from the asparagine residue at position 25 (Asn-25) is located wi

213 t because the decisive role of the conserved asparagine residue for determining sugar specificity has

214 dent acyltransferase and identified a unique asparagine residue in the acyltransferase domain of KATm

215 inked oligosaccharide and its transfer to an asparagine residue in the sequon NX(S/T) of a secreted p

216 ariants had a mutation at either a conserved asparagine residue in transmembrane helix 8 or a threoni

217 vated EcPlt toxin modifies a proximal lysine/asparagine residue instead.

218 A conserved CTE asparagine residue is required for ubiquitylation and de

219 r study on one peptide (PepB2) pinpointed an asparagine residue necessary for CPP activity.

220 id-linked oligosaccharide (LLO) donor to the asparagine residue of a nascent polypeptide chain is cat

221 elop a bidentate interaction with a critical asparagine residue resulted in the incorporation of a py

222 asaccharide with a beta-glucose linked to an asparagine residue which is not located in the typical s

223 y introducing multiple negatively charged or asparagine residues at the edges of CDR3, whereas other

224 Peptides with deamidation sites at asparagine residues but lacking a typical asparagine-X-s

225 h transfers preassembled glycans to specific asparagine residues in target proteins.

226 Mass spectrometry analysis identified two asparagine residues in the helicase 2i domain of RIG-I t

227 nown that N-linked glycans usually attach to asparagine residues in the N-X-S/T motifs of proteins.

228 attachment of an oligosaccharide to selected asparagine residues in the sequence N-X-S/T (X not equal

229 been found to hydroxylate aspartic acid and asparagine residues on epidermal growth factor (EGF)-dom

230 network (asparagine ladder) formed among the asparagine residues on the concave surfaces of neighbori

231 ecule coordinated by conserved histidine and asparagine residues seems to serve as the catalytic base

232 typically involved mutating the glycosylated asparagine residues to structurally similar glutamines o

233 e uses nucleotide-activated sugars to modify asparagine residues with single monosaccharides.

234 mutagenesis to replace six deamidation-prone asparagine residues, at positions 408, 466, 537, 601, 71

235 ide-activated monosaccharides to glycosylate asparagine residues.

236 TRPP2 because mutations of the glycosylated asparagines result in strongly decreased protein express

237 8-oxo-dGMP, the substitution of Ser(266) to asparagine resulted in a dramatic increase in 8-oxo-dGMP

238 e clustered lysine residues with alanines or asparagines results in recombinant PrP amyloid fibrils w

239 que in size and composition: glutamine free, asparagine rich, and the smallest defined to date.

240 An N-terminal glutamine/asparagine-rich nucleation domain is required for nuclea

241 en were strongly enriched for glutamine- and asparagine-rich prion-like proteins.

242 Although prion activity of glutamine/asparagine-rich proteins is predominantly determined by

243 position occupied by the analogous bacterial asparagine, screened for ALAS function, and characterize

244 the leaves for aspartate or in the roots for asparagine, serine and glycine.

245 to interference by the amide function of the asparagine side chain with Na(+)-coordinating residues i

246 s coupled to the nitrogen atom (N-linked) of asparagine side chains or to the oxygen atom (O-linked)

247 Significantly, two highly conserved asparagine side chains, each one located between two try

248 While ECs can take up asparagine, silencing asparagine synthetase (ASNS, which

249 berrant glycan modification on this specific asparagine site of E-cadherin was demonstrated to affect

250 ctin isoform is N-glycosylated at a specific asparagine site that is required for interactions with s

251 preamicoumacins, which are hydrolyzed by the asparagine-specific peptidase into the active component

252 an asparagine or a glutamine increases the l-asparagine specificity but only when combined with the E

253 into the molecular basis for the increased l-asparagine specificity.

254 aluation of immunosensors fabricated using L-Asparagine stabilized gold nanoparticles and citrate sta

255 d gold nanoparticles and (3) directly onto L-Asparagine stabilized gold nanoparticles modified electr

256 Here we show that a single serine-to-asparagine substitution [Ser(139)-->Asn(139) (S139N)] in

257 Here we demonstrate that the asparagine substitution of the aspartate associated with

258 combination of TCA cycle replenishment plus asparagine supplementation restored the metabolic aberra

259 anistically, glutamine provided nitrogen for asparagine synthesis to sustain cellular homeostasis.

260 ent rapid apoptosis when glutamine-dependent asparagine synthesis was suppressed, and expression of a

261 Asparagine synthetase (ASNS) converts aspartate and glut

262 Silencing of asparagine synthetase (ASNS), an amidotransferase that c

263 against cancer cells that express measurable asparagine synthetase (ASNS).

264 While ECs can take up asparagine, silencing asparagine synthetase (ASNS, which converts glutamine-de

265 One is the ammonia-utilizing asparagine synthetase A (AsnA), and the other is asparag

266 . possess asparagine-tRNA synthetase paralog asparagine synthetase A (LdASNA) that is ammonia-depende

267 and also down-regulated the transcription of asparagine synthetase as compared with WT-EcA.

268 ragine synthetase A (AsnA), and the other is asparagine synthetase B (AsnB) that uses glutamine or am

269 NS gene have been clinically associated with asparagine synthetase deficiency (ASD).

270 Through asparagine synthetase knockdown and altering of media as

271 synthesis was suppressed, and expression of asparagine synthetase was statistically correlated with

272 agine is formed by two structurally distinct asparagine synthetases in prokaryotes.

273 In WMS glucose/asparagine systems, examination of three different conce

274 the binding groove while the first residue, asparagine, tethers the peptide via an interaction with

275 ted mutagenesis of Cfp4, we identified three asparagines that function as the principal sites of N-li

276 the importance of Asp-139; upon mutation to asparagine the Q reductase activity is inhibited by 75%.

277 Mutation of this residue (Ser(266)) to asparagine, the residue present in most PolXs, had a str

278 cacy of L-asparaginases is micromolar KM for asparagine to allow for complete depletion of this amino

279 es catalyze the hydrolysis of the amino acid asparagine to aspartate and ammonia.

280 ic properties and analyzed the conversion of asparagine to aspartate using NMR.

281 As drugs, these enzymes act to hydrolyze asparagine to aspartate, thereby starving the cancer cel

282 The use of asparaginase to convert asparagine to aspartic acid may provide a means to reduc

283 most potent peptide from this library was an asparagine to diaminopropionic acid substitution that po

284 acid route, i.e., reducing sugars react with asparagine to form the Schiff base before decarboxylatio

285 f the relationship between the ratio of free asparagine to reducing sugars and the levels of acrylami

286 Catalysis is based on a cysteine-histidine-asparagine triad, which is shared with human PAD1-PAD4 a

287 s, we had shown that Leishmania spp. possess asparagine-tRNA synthetase paralog asparagine synthetase

288 synthesis of aminoacylated glutamine and/or asparagine tRNAs, involving the glutamine amidotransfera

289 strated a crucial, energy-generating role of asparagine utilization and non-generic usage of the glyo

290 modulation of key metabolites like proline, asparagine, valine and several flavonoids.

291 r residues revealed that the amide moiety of asparagine was crucial for GlyR activation.

292 ls and animal fat using a constant amount of asparagine was measured.

293 We found that asparagine was necessary and sufficient to suppress glut

294 ated asparaginase on induction day 4, plasma asparagine was undetectable for 11 days for SS-PEG2500 a

295 0.95 and 0.94 respectively (p<0.05), whereas asparagine, was poorly correlated (p>0.05).

296 In dry glyoxal/asparagine waxy maize starch (WMS) systems, 9 out of 10

297 olar ratios of total fructose and glucose to asparagine were investigated.

298 Heating asparagine with various cooking oils and animal fat at 1

299 Replacing asparagines with glutamines created stronger suppressors

300 at asparagine residues but lacking a typical asparagine-X-serine/threonine sequons (N-X-S/T, X is any