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1 -isoxazole-propionate (AMPA), and N-methyl-d-aspartic acid receptors.
2 n of CA2 synapses relies on NMDA (N-methyl-D-aspartic acid) receptor activation, calcium and calcium/
4 asing glutamatergic excitation at N-methyl-D-aspartic acid receptors, alters both the amplitude and f
5 vents were strongly influenced by N-methyl-D-aspartic acid receptor- and cyclic AMP-dependent signali
6 (CSF) levels of the glia-derived N-methyl-D-aspartic acid receptor antagonist kynurenic acid (KYNA)
10 ent, we found abnormally enhanced N-methyl-d-aspartic acid receptor-dependent long-term depression in
15 ed to glucocorticoids, exhibit an N-methyl-d-aspartic acid receptor-independent form of long-term pot
16 the other hand, experiments using N-methyl-d-aspartic acid receptor inhibitors suggested that these r
17 e injured independently via NMDA (N-methyl-D-aspartic acid) receptors located on peripheral oligodend
18 panoic acid receptor-mediated and N-methyl-D-aspartic acid receptor-mediated synaptic currents in lam
19 to LRP1, we demonstrate that the N-methyl-D-aspartic acid receptor (NMDA-R) is expressed by macropha
20 K-801, a specific pore blocker of N-Methyl-D-aspartic acid receptor (NMDAR) channels, and this occurr
24 ver, the majority of these larger N-methyl-d-aspartic acid receptor subunit immunoreactive spots was
25 atment also significantly reduced N-methyl-d-aspartic acid receptor subunit NR2B phosphotyrosine labe
26 late absorption and activation of N-methyl-d-aspartic acid receptors, the authors examined relationsh
27 te-binding GluN2A subunits of the N-methyl D-aspartic acid receptor upon binding agonists of varying
28 eptors were also decreased, while N-methyl-D-aspartic acid receptors were not different compared with
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