ライフサイエンスコーパス: asplenia

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1 ut PS-exposed autophagic vesicles because of asplenia.

2 Q) in a child with both severe 46,XY-DSD and asplenia.

3 7.1 years and 64% had functional or anatomic asplenia.

4 ciency causes organ growth defects including asplenia.

5 eptibility to infection caused by functional asplenia.

6 e molecular determinants of human congenital asplenia.

7 t underlie spleen development and congenital asplenia, a condition linked to increased risk of overwh

8 ents with severe sepsis or septic shock with asplenia and 52 without asplenia were included.

9 s, we searched for patients with and without asplenia and community-acquired severe sepsis/septic sho

10 ctive lateralization including dextrocardia, asplenia and intestinal malrotation, suggesting that BCO

11 A patient with asplenia and multiple red blood cell transfusions acquir

12 ng Diamond-Blackfan anemia (DBA), congenital asplenia and T cell leukemia.

13 or C3 deficiencies, 1 of isolated congenital asplenia, and 2 of Bruton disease (X-linked agammaglobul

14 Although functional asplenia from infarctions may be a major contributor to

15 Isolated congenital asplenia (ICA) is characterized by the absence of a sple

16 esis and the etiology of isolated congenital asplenia (ICA), a life-threatening human condition, are

17 spleen development, and Tlx1 deletion causes asplenia in mice.

18 n proposed in the pathogenesis of congenital asplenia in patients carrying mutations of the gene-enco

19 posplenism in older mice, recapitulating the asplenia of an HO-1-deficient patient.

20 Functional asplenia or hyposplenism may predispose patients to spon

21 % of OPSI patients vs 5% of patients without asplenia (P = .038).

22 quently in OPSI patients (42% vs 12% without asplenia; P < .001) and more frequently manifested as bl

23 ow overall (42% vs 8% among patients without asplenia; P < .001).

24 iary and organ laterality defects as well as asplenia, paralleling symptoms of the human condition ri

25 g chronic mucocutaneous candidiasis, AI, and asplenia, respectively, in 49 of 121 (40%) and 10 of 121

26 itulating the clinical symptoms of the human asplenia syndrome.

27 Asplenia was present in 76 patients (54%).

28 On multivariable Poisson regression, asplenia was the only predictive variable independently

29 or septic shock with asplenia and 52 without asplenia were included.

30 by a perinatal lethal skeletal dysplasia and asplenia, with severe malformation or absence of specifi

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